Neuro Flashcards

1
Q

2 Nervous System

A

Central and Peripheral Nervous System

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

CNS is about

A

Brain and Spinal Cord

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

2 Types of PNS

A

Somatic and Autonomic Nervous System

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Voluntary Movements

A

Somatic Nervous System

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

2 Types of Autonomic Nervous System

A

Sympathetic
Parasympathetic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Basic functional unit of The Nervous System

A

NEURONS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Extension that carry the impulses toward the cell body.

A

Dendrite

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Transmits impulses away from the cell body

A

Axon

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Allows impulse to transmit

A

Myelin Sheath

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Types of Neurons

A

Sensory Neurons (Affarent)
Motor Neurons (Efferent)
Interneurons

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Transports impulse from receptors to the CNS

A

Sensory Neurons

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Transports impulse from CNS to the receptors

A

Motor Neurons

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Found entirely within the central nervous system
Specialized to transmit sensory / motor impulse

A

Interneurons

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Communicate message from one neuron to another or from a neuron to a specific target tissue

A

Neurotransmitters

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Excitatory
Control movements, motivation, and cognition
Regulates emotion response

A

DOPAMINE

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Excitatory
Causes sympathetic stimulation
Causes changes in attention and learning

A

NOREPINEPHRINE

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Excitatory
Control fight or flight response

A

EPINEPHRINE

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Inhibitory
Controls emotion (happy and sad)

A

SEROTONIN

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Inhibitory / Excitatory
Signals muscles to be alert
Controls wakefulness and sleep cycle

A

ACETYLCHOLINE

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Inhibitory
Relaxation hormone

A

GABA

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Excitatory
Responsible for relaying messages between MOTOR neurons

A

GLUTAMATE

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

CENTRAL NERVOUS SYSTEM

A

CEREBRUM
CEREBELLUM
BRAIN STEM
DIENCEPHALON

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Cerebrum Lobes

A

Frontal
Parietal
Temporal
Occipital

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

Personality
Attention / Focus
Speech: Brocha’s Area (Expressive)
Thinking / Judgment

A

Frontal Lobe

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Touch Taste Temperature
Parietal Lobe
26
Memory of sound Hearing and Smelling Speech: Wernicke’s Area (Receptive)
Temporal Lobe
27
Vision Memory
Occipital Lobe
28
Balance and Coordination
Cerebellum
29
Brain Stem
Mid Brain Pons Medulla Oblongata
30
Auditory and Visual REFLEX
MId Brain
31
Pattern of Breathing
Pons
32
Controls heart and respiratory rate, coughing and vomiting
Medulla Oblongata
33
Diencephalon
Hypothalamus Thalamus
34
Controls BP and Temperature Hormone release
Hypothalamus
35
Pain threshold Capable of suppressing minor sensation
Thalamus
36
Peripheral Nervous System
Cranial Nerves
37
CN Sense of Smell Sensory Assessment: With eyes closed, patient is asked to identify familiar odors (Coffee, Cinnamon) Abnormal: Anosmia – Loss of sense of smell
I - OLFACTORY
38
CN Sense of Sight / Vision Sensory Assessment: Snellen’s Chart (Normal: 20/20) Abnormal: Hemianopia, Blindness
II - OPTIC
39
CN Pupillary constriction & dilation Motor Assessment: PERRLA Abnormal: (-) PERRLA
III – OCULOMOTOR
40
CN 6 Cardinal Gaze movement Motor Assessment: Cardinal Field of Gaze Assessment Abnormal: Nystagmus
IV - TROCHLEAR
41
CN TriCHEWminal – For chewing Facial SENSATION Both Assessment: Wisp of cotton Abnormal: Absent of sensation and jaw weakness
V - TRIGEMINAL
42
CN Eye movement side-to-side (AbduSIDE-TO-SIDE) Motor Assessment: Test for bilateral eye movement Abnormal: Double Vision
VI – ABDUCENS
43
CN Facial MOVEMENT Anterior 2/3 of tongue sensation Both Assessment: Ask patient to smile and wrinkle forehead Abnormal: Facial weakness
VII - FACIAL
44
Sense of hearing, balance, and coordination Sensory Assessment: Weber’s / Rinne Test and Romberg Test Abnormal: Deafness and Impaired balance
VIII - VESTIBULOCOCHLEAR
45
CN Swallowing Posterior 1/3 of the tongue sensation Both Assessment: Food tasting Problem: Augeusia / Dysphagia
IX – GLOSSOPHARYNGEAL
46
CN Movement of Uvula / Gag Reflex Parasympathetic sensation Both Assessment: Tongue Depressor Abnormal: Absent Gag Reflex
X - VAGUS
47
CN Neck movement Motor Assessment: Ask the patient to turn head and shrug shoulders Problem: Weak / Absent Shoulder Shrug
XI - ACCESSORY
48
CN Tongue movement Motor Assessment: Ask patient to move tongue side to side Problem: Dysphagia / Slurred Speecj
XII - HYPOGLOSSAL
49
31 PAIRS SPINAL NERVES
CERVICAL – C1 – C8 THORACIC – T1 – T12 LUMBAR – L1 – L5 SACRAL – S1 – S5 COCYX - 1
50
DACALGG
C1 – C4 – Diaphragm C5 – T1 – Arms T2 – T6 – Chest T7 – T12 – Abdomen L1 – L5 – Legs S1 – S3 – GI & GU S4 – S5 – Genitals
51
Fight or Flight Response Increase Everything; Decrease GI & GU
SYMPATHETIC NERVOUS SYSTEM
52
Dominates during relaxed situations Decrease Everything; Increase GI & GU
PARASYMPATHETIC NERVOUS SYSTEM
53
Neuro Assessment
CEREBRAL FUNCTION Assess degree of wakefulness / alertness Note the intensity of stimulus to cause a response Apply a painful stimulus over the nailbed with a blunt instrument Ask questions to assess orientation to person, place, and time GLASGOW COMA SCALE 3 Areas: Eye Opening Verbal Response Motor Response Scores Interpretation: 15 = highest score; patient is fully oriented and alert <7 = comatose patient 3 = Deep coma
54
DIAGNOSTIC TEST
SKULL AND SPINAL X-RAY Identifies fracture, dislocation, compression, and spinal cord problem Nursing Care: 1. Provide support for the confuse of combative client 2. Remove metal items 3. Maintain immobilization CT - SCAN Used for diagnosing neurological disorder of the brain or the spine Can detect: 1. Hemorrhage 2. Tumors 3. Abscess Nursing Care: 1. Assess for iodine allergy 2. Instruct to lie still on a movable table 3. Inform the patient of possible discomforts 4. Remove any metallic object MAGNETIC RESONANCE IMAGING Used for diagnosis of degenerative diseases, intracranial and spinal abnormalities Not useful when looking bony abnormalities ELECTROENCEPHALOGRAPHY (EEG) Graphic recording of electrical activity of the brain by placing electrodes to the scalp
55
Unilateral inflammation of CN 7 (Facial) Cause: Infection Autoimmune Viral Infection (Herpes simplex) Signs and Symptoms: Drooping of the eyelid (Ptosis) Inability to close eye completely Unilateral facial weakness (temporary)
BELL’S PALSY
56
Management: 1. Facial Massage (Moist Heat Massage) 2. Artificial tears 3. Antibiotics / Antiviral Drugs 4. Corticosteroids 5. Eye patch on affected side / sun glasses 6. Soft diet - Chew on the unaffected side 7. Avoid hot fluids / food 8. Facial Exercise (grimacing, wrinkling, puffing of cheeks, blowing of air
Bell's Palsy
57
Other name: Tic Douloureux Problem in CN 5 (Trigeminal) Cause: Chronic compression and irritation of CN 5 Degenerative changes in the Gasserian Ganglion Pressure from surrounding tissue Risk factors: Patient with multiple sclerosis (Damage to myelin sheath around trigeminal nerve Men with MS > Women with MS Signs and Symptoms: Sudden severe unilateral facial pain Sensitive to extreme changes in temperature Difficulty chewing
TRIGEMINAL NEURALGIA
58
other name of TRIGEMINAL NEURALGIA
Tic Douloureux
59
Medical Management: 1. Pharmacologic Therapy Anti-seizure (Carbamazepine) – To manage pain by decreasing electrical impulse on trigeminal nerve Alcohol / Phenol injection of the Gasserian Ganglion – To make the nerve numb 2. Surgical Treatment Microvascular Decompression of the Trigeminal Nerve Percutaneous Balloon Micro-Compression 3. Radiofrequency Thermal Coagulation Nursing Management: 1. void extreme changes in temperature 2. No facial massage / teaching 3. Pat dry to cleanse face 4. Chew on unaffected side 5. Soft diet 6. Post op care: Instruct not to rub the eye Assess the eyes for redness Artificial tears
TRIGEMINAL NEURALGIA
60
Autoimmune diseases that damages the myelin sheath and would lead into delayed impulse transmission
Demyelination Disorders
61
Demyelination of CNS nerves Has remission and exacerbation Common cause: Autoimmune Post Viral Infection Common in: 20-40 years old (female)
MULTIPLE SCLEROSIS
62
Signs and Symptoms: -Visual Disturbances: Scotoma -white patches in vision (EARLIEST SIGN) Diplopia Blurred Vision -Respiratory Depression -Cerebellum/Basal Ganglia Involvement: Tremors Muscle Weakness (descending) MS (Mataas Simula) Ataxia -Sensory Nerve Disturbances: Paresthesia Pain -Cognitive Disturbances: Memory Loss Decrease Concentration Dementia -Bowel and Bladder Dysfunction -Lhermitte's Sign
Multiple Sclerosis
63
Is an electric shock sensation that radiated from the back of your neck down your spine.
Lhermitte's Sign
64
MS Due to impaired cerebellar function Intentional Tremors Scanning Speech Nystagmus
CHARCOAT'S TRIAD
65
Medical Management: 1. Corticosteroid (Methylprednisolone) 2. Baclofen – Muscle relaxant 3. Oxygen and Mech Vent - If (+) respi involvement Nursing Management: 1. Warm pack application – To minimize spasm 2. Avoid hot baths – Due to head intolerance 3. Moderate exercise (swimming, stationary biking) 4. Unilateral eye patch 5. Promote regular elimination: - Increase fluid and fiber intake - Stool softener 6. Priority: SAFETY due to impaired muscle control 7. WOF: UHTHOFF’S SIGN - Transient worsening of symptoms after exposure to hot environment
Multiple Sclerosis
66
Demyelination of PNS nerves Cause: Autoimmune History of GI or Respi Infxn (Campylobacter Jejuni) Signs and Symptoms: Dyskinesia (Leg weakness / Clumsiness ) – Early Sign Paralysis of Diaphragm Dec. GIT and GUT function Numbness & Spasticity Ataxia
GUILLAIN – BARRE SYNDROME
67
Complications: Respiratory failure Cardiac dysrhythmias Orthostatic hypertension Medical Management: 1. Plasmapheresis 2. Corticosteroids 3. Dantrolene Sodium – Muscle relaxant Nursing Management: 1. Maintain adequate ventilation 2. Perform range-of-motion exercises 3. Assess gag reflex prior to feeding 4. Assistive devices 5. No to hot baths 6. Priority: SAFETY 7. Promote regular elimination: - Increase fluids and fiber intake
GUILLAIN – BARRE SYNDROME
68
Nurse Marty is monitoring a client for adverse reactions to Dantrolene Sodium. Which adverse reaction is most common? Excessive tearing Urine retention Muscle weakness Slurred speech
Muscle weakness
69
Nurse Kim is assessing a 37 year old client diagnosed with multiple sclerosis. Which of the following symptoms would the nurse expect to find? Vision changes Absent deep tendon reflex Tremors at rest Flaccid muscles
Vision changes
70
The nurse is teaching a female client with multiple sclerosis. When teaching the client how to reduce fatigue, the nurse should tell the client to: Take a hot bath Rest in an air-conditioned room Increase the dose of muscle relaxants Avoid naps during the day
Rest in an air-conditioned room
71
A male client with Bell’s Palsy asks the nurse what caused this problem. The nurse’s response is based on an understanding that the cause is: Unknown, but possibly includes ischemia, viral infection, or an autoimmune problem Unknown, but possibly includes long term tissue malnutrition and cellular hypoxia Primary genetic in origin, triggered by exposure to meningitis Primary genetic in origin, triggered by exposure to neurotoxins
Unknown, but possibly includes ischemia, viral infection, or an autoimmune problem
72
Although an adverse reaction to Tensilon is considered rare, which medication should be readily available to give as an antidote in case a patient should experience complications? Protamine Sulfate Narcotic Analgesic Atropine Sulfate Regitine
Atropine Sulfate
73
Cranial Nerve Disorders
Bell's Palsy Trigeminal Neuralgia
74
Demyelination Disorders
Multiple Sclerosis Guillain-Barre Syndrome
75
Neurotransmitter Disorders
Myasthenia Gravis Parkinson's Disease Amyotropic Lateral Sclerosis Huntington's Disease
76
Defect in transmission of nerve impulse at myoneural/neuromuscular junction Cause: Autoimmune that will attack acetylcholine receptors Antibody will bind to acetylcholine receptors Acetylcholine can’t bind anymore Acetylcholine accumulates in the synapse Cholinesterase will kill acetylcholine in the synapse POOR MUSCLE MOVEMENT
Myasthenia Gravis
77
Diagnostics: TENSILON TEST (Confirmatory) - Cholinesterase inhibitor (Endrophonium) is injected - (+) MG = Temporary (5 mins) improve in muscle movement - Prepare Atropine Sulfate (Symphatomemitics) for possible side effects. Signs and Symptoms: Descending Paralysis (Mataas Galing) Diplopia & ptosis – Early Sign ! Muscle weakness in the afternoon Dysphonia (voice impairment)
Myasthenia Gravis
78
Management: 1. Priority: Airway 2. NGT feeding 3. Plasmapheresis to remove excess antibody 4. Mechanical Ventilator Stand-by 5. Tracheostomy Set at Bed Side 6. Medications: (anticholinesterase/Cholinergics) Neostigmine (Prestigmine) Pyrastigmine (Mestonin) Steroids
Myasthenia Gravis
79
MYASTHENIA GRAVIS Complications (CHOLINERGIC CRISIS VS. MYASTHENIC CRISIS)
CHOLINERGIC CRISIS Overmedication Tensilon Test: Muscle weakness Parasympathetic effects Prepare: Atropine Sulfate MYASTHENIC CRISIS Undermedication Tensilon Test: Temp. Relief Sympathetic effects Prepare: Cholinergics
80
Slowly progressing neurologic movement disorder caused by Decrease Dopamine Level Causes: Idiopathic Degenerative Viral infection Head trauma Use of antipsychotic medications Pathophysiology: Damage to Substantia Negra Decrease dopamine production Abnormal muscle movement
Parkinson's Disease
81
Signs and Symptoms: Cog-wheel rigidity Pill-rolling tremors Resting tremors – Early sign! Stooped posture – Osteoporosis like Bradykinesia – Dec. Muscle Movement Shuffling gait – Dragging 1 foot when moving Mask – Like appearance Dysphagia Decrease GIT and GUT function Respiratory depression
Parkinson's Disease
82
Medical Management: 1. Pharmacologic treatment: Antiparkinsonian Drug - Levodopa - Carbidopa Antiviral drugs - Amantadine Dopamine Agonist - Bromocriptine - Pergolide Antihistamines - Benadryl Anticholinergic Agents - Cogentin - Artane - Akineton
Parkinson's Disease
83
NursingManagement: 1. Activity: Anything that involves fingers 2. Priority: Safety 3. Mobility: Marching walk ; if they freeze, move to opposite side 4. Diet: Puree diet because of dysphagia 5. Aspiration precaution
Parkinson's Disease
84
Other name of Amyotropic Lateral Sclerosis
Lou Gehrig's Disease
85
AKA: Lou Gehrig’s Disease Progressive, degenerative condition that affects MOTOR NEURONS Causes: Unknown 5-10% Genetically transmitted Increase GLUTAMATE in the brain Pathophysiology: Increase Glutamate in the brain Voluntary muscle problem FINE MOTOR Fasciculation - Twitching of muscle - Earliest Sign Tremors Dysphagia GROSS MOTOR Respiratory Depression - Common cause of death (2 years)
AMYOTROPIC LATERAL SCLEROSIS
86
AMYOTROPIC LATERAL SCLEROSISMedical Management: 1. Riluzole (Rilutek) – To decrease glutamate in the brain - Can delay symptoms for as long as 3 months 2. Mechanical ventilator stand-by
AMYOTROPIC LATERAL SCLEROSIS
87
Nursing Management: 1. Maximize functional abilities: - Prevent complications of immobility - Promote self care - Maximize effective communication - Promote use of assistive devices 2. Ensure adequate nutrition 3. Prevent respiratory complications: - Promote measures to maintain adequate airway
AMYOTROPIC LATERAL SCLEROSIS
88
Progressive atrophy of basal ganglia and some parts of cerebral cortex Basal Ganglia – Helps to control muscle movements - Produces GABA Pathophysiology: Decrease GABA Impaired muscle relaxation Overactivity of muscle Cause: Autosomal Dominant - Only takes 1 parent to pass the gene - 50% transmission to child
HUNTINGTON’S DISEASE
89
Neurotransmitter abnormalities: Decrease GABA - Increased voluntary movements - Chorea – Sudden muscle jerking - Impaired chewing and swallowing Increase Dopamine - Psychosis - Personality changes Abnormal Serotonin - Depression / Manic
HUNTINGTON’S DISEASE
90
Nursing Management: 1. Foster independence in ADL 2. Reinforce use of assistive devices 3. Aspiration precaution 4. Provide safe environment 5. Genetic counseling
Huntington's Disease
91
NEURO - INFECTIOUS DISORDERS
Meningitis
92
Inflammation of meninges in the brain and spinal cord Causes: Bacterial, Viral, or Protozoan Infection Signs and Symptoms: Increase ICP Hearing loss Photophobia Projectile vomiting Nuchal Rigidity - Neck Stiffness Kernig's Sign - Knee Brudzinki's Sign - Batok
Meningitis
93
Management: 1. Droplet precaution 2. Antibiotics within 24 hours para dili na contagious 3. Isolate the patient 4. Place in private, non-stimulating room to prevent seizure 5. WOF: Aspiration
Meningitits
94
SEIZURE – RELATED DISORDERS
Seizure
95
Abnormal transmission of impulse at motor complex of the brain. Cause: Information overload Infection Trauma (Brain) Autoimmune Heredity Vegan diet – Vitamin B12 deficiency
Seizure
96
Types of Seizure
Epilepsy Status Epilepticus Petit Mal Grand Mal Myoclonic Akinetic Symptomatic Simple Partial / Focal Awareness Complex Partial / Focal Impaired Awareness
97
Chronic, recurrent episodes of seizure that is unprovoked
Epilepsy
98
- A continuous seizure that last 5-30 minutes - Risk for hypoxia: 3-5 mins – Normal tolerance 6-8 mins – Cerebral damage >8 mins – Brain stem damage
Status Epilepticus
99
- Absence seizure - Blank facial expression - “Blank Stare” - Common in pedia
Petit Mal
100
- Tonic – Clonic Seizure (Preceeded with Aura) - Tonic – Stiffening - Clonic – Involuntary muscle jerking - Generalized seizure
Grand Mal
101
- Brief, involuntary muscle jerking of the body - 1– 2 seconds
Myoclonic
102
- Drop attack seizure - Sudden loss of postural tone & consciousness
Akinetic
103
- Benign seizure - Temporary until the cause will be addressed - ex: Febrile (convulsion)
Symptomatic
104
- Conscious seizure - Purposeless behavior
Simple Partial / Focal awareness
105
- Preceded with Aura - Impaired consciousness - Cannot remember what happened
Complex Partial / Focal Impaired awareness
106
Pre - Ictal Phase - Aura: Flashing lights Smells burning wire Metallic taste Dizziness - Loss of consciousness: 1. Protect the head and neck then lay down 2. Turn to sides 3. Loosen constrictive clothing 4. Clear the area - Priority: SAFETY ! ! !
Seizure
107
What phase: - Aura: Flashing lights Smells burning wire Metallic taste Dizziness - Loss of consciousness: 1. Protect the head and neck then lay down 2. Turn to sides 3. Loosen constrictive clothing 4. Clear the area - Priority: SAFETY ! ! !
Pre-Ictal Phase
108
Management: 1. Turn patient to side (Left lateral side lying) 2. Remove objects that may harm the patient 3. Loosen restrictive clothing 4. Raise 2-3 side rails 5. Do not put anything inside the patient’s mouth 6. Remove pillow if seizure on bed; Put pillow if happens on the floor 7. Suction machine at bedside 8. Medications: Anticonvulsants
Seizure
109
Seizure: Anticonvulsants PhenyCarDiLaToDe
PHENYtoin CARbamazepine DIazepam LAmotrigene TOpamax DEpakene
110
SEIZURE anticonvulsant: First line in the hospital; given via IV Fast acting Sedative / Relaxant: Can induce sleep HIGHLY ADDICTIVE Antidote: Flumazenil
Diazepam (valium)
111
SEIZURE anticonvulsant: For maintenance due to its least toxic effect Side effect: GINGIVAL HYPERPLASIA - Have a good oral hygiene - Massage gums - Use soft bristle toothbrush dip in warm water Adverse effct: AGRANULOCYSTOSIS – Dec. WBC Don’t discontinue abruptly because it may lead to Status Epilepticus Therapeutic level: 10 – 20 mg/dL (DilanTEN – PhenyTOINte)
PHENYTOIN (DILANTIN)
112
Monro – Kellie Hypothesis: “Skull is incompressible” Brain – 80% CSF - 10% Blood - 10% Normal: 0-15 mmHg Causes: Brain abscess Brain Hemorrhage Brain edema Hydrocephalus Initial Sign: Altered Level of Consciousness (Cerebrum will be the first affected) - Restlessness - Confusion - Disorientation - GCS alteration Late Sign: - Decrease Level of Consciousness (Lethargy) - Seizure - Projectile Vomiting - Indicates medulla oblongata damage - Cheyne – Stoke Respiration – Indicates pons damage - Decerebrate Posture - High - pitched cry (newborn) - Bulging fontanels (newborn) - Cushing’s Triad – Indicates brain stem damage
Increased Intracranial Pressure
113
9. Medications: IV mannitol - For cerebral edema - Check BP before administration - Check urine output during therapy Dexamethasone - Steroids for cerebral inflammation 10. DON’TS: Valsalva Maneuver Routine suctions Lumbar puncture Coughing / Sneezing
Increased Intracranial Pressure
114
Disruption of blood supply to the brain Causes: Thrombus Formation Hypertension Atherosclerosis Diabetes Mellitus Aneurysm Risk Factors: Age: 45 years old and above Obesity Estrogen Therapy – Increases clotting ability Hereditary Sedentary lifestyle Smoking Alcoholism
CEREBROVASCULAR ACCIDENT
115
2 types of CEREBROVASCULAR ACCIDENT
Ischemic Hemorrhagic
116
2 Areas affected: Left Hemispheric Stroke - Paralysis to the right side of the body - Right visual field deficit - Aphasia - Altered intellectual ability - Slow, cautious behavior Right Hemispheric Stroke - Paralysis to the left side of the body - Left visual field deficit - Increase distractibility - Lack of awareness of deficits What to assess: Facial drooping Arm defect Slurred speech Time – To measure the severity of brain damage and for the drug administration
CEREBROVASCULAR ACCIDENT
117
Signs and Symptoms: Aphasia: - BROCHA’S APHASIA – Unable to speak fluently - WERNICKE’S APHASIA – Unable to comprehend - GLOBAL APHASIA – Combined - Management: 1. Short, one at a time task 2. Independence promotion 3. Make simple direction 4. Alternative communication style 5. Provide time to verbalize concerns -Paralysis (Hemiparesis) Weakness of 1 side of the body Management: 1. Quad cane on the AFFECTED SIDE 2. Slipping Tub Bath 3. Electric wheel chair 4. Avoid - Roller Walker Foot drop (Plantar Flexion) - Management: 1. High topped sneakers 2. Foot board Neglect Syndrome (Unilateral Neglect) - Inability to identify for weak side - Management: 1. Instruct the patient to touch the weak side 2. Offer a mirror Homonymous Hemianopia (Half Vision) -Management: 1. Scan the environment 2. Move side-to-side 3. Initially; Approach from unaffected side 4. Latter; Approach from affected side Drug Management: -Striptokinase -Thrombolytics (for ischemic stroke) -Ideally given within 4-8 hours -NOt given in Hemorrhagic stroke -Antihypertensives
Cerebrovascular Accident
118
Intracranial Hemorrhage Types:
Epidural Subdural Subarachnoid Intracerebral
119
Type of Intracranial Hemorrhage: Location: Between skull and dura mater Blood Vessel Affected: Middle Meningieal Artery Hallmark: Lucid Interval
Epidural
120
Type of Intracranial Hemorrhage: Location: Between Dura and Arachnoid Blood vessel affected: Cortical vein Hallmark: Gradual Deterioration
Subdural
121
Type of Intracranial Hemorrhage: Location: Between Pia Mater and Arachnoid Blood Vessel Affected: Berry Aneurysm Hallmark: Thunder drop headache (worst headache of his/her life)
Subarachnoid
122
Type of Intracranial Hemorrhage: Location: Brain parenchyma Blood vessel affected: Hemorrhagic stroke Hallmark: Sudden onset
Intracerebral
123
Management: 1. Craniotomy -Supratentorial - superior to tentorium cerebelli - Post op position: Semi-Fowlers -Infratentorial - Inferior to tentorium cerebelli - Post op position: Flat
INTRACRANIAL HEMORRHAGE
124
2 types of Traumatic Brain Injury
Closed Open (Skull Fracture)
125
Types of CLOSED traumatic brain injury
Concussion Contusion
126
Jarring of the brain
Concussion
127
Bruising of the brain
Contusion
128
Types of Contusion
Coup Counter-Coup Coup-Counter-Coup
129
Same side of the site of injury
Coup
130
Opposite side to the site of injury
Counter-coup
131
Bouncing Back traumatic brain injury
Coup-Counter-Coup
132
Types of OPEN (Skull fracture) traumatic brain injury
Linear Depressed Comminuted Basal-Skull
133
Fine line on the skull most common
Linear
134
Skull is driven inward
Depressed
135
skull is fragmented
comminuted
136
Signs and symptoms: Raccoon’s eye - Periorbital Edema Battle sign - Ecchymosis of Mastoid Bone CSF leakage
Open skull fracture
137
Injury to the spinal cord which characterized by a decrease or loss of sensory and motor functions below the level of injury , Causes: . Motor vehicle accidents • Gunshot injuries • Falls • Sports injuries • Whiplash injury - Neck • Transection - Due to sharp objects • Hyper rotation Risk factors: Young age Alcohol and drug base Male
Spinal Cord Injury
138
types of spinal cord injury
Cervical SCI Thoracolumbar SCI
139
Paralysis: Quadriphlagia Priority: Respiratiory
Cervical SCI
140
Paralysis: Paraphlagia (Lower body) Priority: Elimination
Thoracolumbar SCI
141
Management 1. Stabilize the airway (Jaw Thrust Maneuver) 2. Immobilization (Flat, firm surface) 3. Cervical collar 4. Transport client as a unit 5. Do not attempt to realign body parts 6. Suctioning may be indicated, but with caution 7. Position change q 2 hours 8. Intermittent catheterization for bladder distention 9. Anticoagulants 10. Anti-embolic stockings
Spinal Cord Injury
142
Complication of Spinal Cord Injury
Autonomic Dysreflexia
143
Complication: AUTONOMIC DYSREFLEXIA - Life threatening condition that occurs in patients with SCI above T6 level. - Impairs the normal equilibrium between sympathetic and parasympathetic divisions • Causes: Bladder distention (Most common) • Bowel impaction . UTI . Pressure ulcers
Spinal Cord Injury
144
Manifestations: • Hypertension • Throbbing / Pounding headache • Diaphoresis • Piloerection • Bradycardia • Blurring of vision • Warm and flushed - Above the level of injury • Cold and Pale - Below the level of injury
Spinal Cord Injury
145
Management: 1. Position the patient in sitting position to decrease BP 2. Catheterization 3. Check for fecal impaction 4. Monitor blood pressure 5. Antihypertensive medication: HYDRALAZINE
Spinal Cord Injury
146
Antihypertensive medication in spinal cord injury
HYDRAZALINE