Neural and Hormonal Mechanisms in Aggression Flashcards

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1
Q

What do biological explanations assume?

A

The potential for aggression is located within an individuals biological makeup

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2
Q

What are neurotransmitters?

A

Chemical substances that enable impulses to travel from one area to another

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3
Q

How does serotonin influence aggression?

A
  • Can inhibit responses to emotional stimuli so reduce aggression
  • Low levels are associated with increased susceptibility to impulsive, aggressive, violent and suicidal behaviours
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4
Q

What did Mann do in his study into serotonin?

A
  • 35 p’s given dexfemfluramine to reduce serotonin

- Questionnaire to assess hostility and aggression given after

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5
Q

What did Mann find in his serotonin study?

A

Drug was associated with an increase in hostility and aggression scores, providing support for a link between low serotonin levels and increased aggression

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6
Q

What is a limitation of Mann’s study into serotonin?

A
  • Self report methods may lower reliability due to SDB

- People want to paint themselves in the best light

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7
Q

What is a second methodological limitation of Mann’s study?

A
  • Only conducted on men - shows beta bias
  • May not be valid to women
  • May encourage stereotypical beliefs about female aggression and an andocentric view of human behaviour
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8
Q

What can we conclude from Mann’s study after taking methodological flaws into account?

A
  • While this study provides some support for the S/A link

- Lack of generalisability may limit it’s use in supporting the link as results may not be valid to wider population

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9
Q

Where does further support for the serotonin and aggression link?

A

Non-human studies - Raleigh

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10
Q

What did Raleigh’s study involve?

A
  • Half a group of monkeys fed diet high in tryptophan to increase serotonin
  • Half fed diets low in tryptophan to decrease serotonin
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11
Q

What did Raleigh’s study show?

A

Low serotonin - increase aggression, providing considerable research support for the role of neurotransmitters

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12
Q

However, what are 2 criticisms of Raleighs study?

A
  • May lack generalisability as we cannot apply to humans. Animals have different anatomy and their behaviour is a poor predictor of human behaviour, limiting the support we can gain.
  • However may offer us an expanded insight into physiological and psychological processes
  • Ethical concerns - could cause pain and suffering, lacking credibility outside the study
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13
Q

What did Bond find?

A

Evidence to support the idea that neurotransmitters can influence aggression from antidepressants

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14
Q

What were Bond’s findings in clinical studies of antidepressants?

A

Antidepressants that increased serotonin were found to reduce irritability and impulsive aggressiveness

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15
Q

How can dopamine lead to aggressive behaviour?

A

Increases in dopamine via amphetamines have been associated with increases in agg, whereas antipsychotics to reduce dopamine reduce aggressive behaviour

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16
Q

Who does evidence for dopamine link come from?

A

Couppis and Kennedy

17
Q

What did Couppis and Kennedy find?

A

Mice - reward pathway in brain becomes engaged in response to aggressive event - Dopamine acts as a positive reinforcer following an aggressive act as it causes feelings of euphoria

18
Q

Why might dopamine acting as a reinforcer cause aggressive behaviour?

A

Suggests individuals may intentionally seek out an aggressive encounter for the rewarding sensation

19
Q

Why do Couppis and Kennedys findings provide conflicting evidence?

A
  • Provides support for idea that dopamine is linked
  • As a cause rather than a consequence as initially suggested
  • Need for further research?
20
Q

What did a meta analysis of 29 studies involving antisocial children and adults find?

A
  • Low serotonin found in aggressive individuals but no difference in dopamine
  • Reduce serotonin found in all antisocial groups but particularly suicide attempters
  • Suggests that serotonin depletion leads to impulsive behaviour which can result in aggression in various forms
21
Q

What does the meta analysis of 29 studies show?

A
  • Biological, deterministic approach in explaining aggression
  • Serotonin levels are a predictor of future behaviour
22
Q

What hormonal mechanism is thought to explain aggressive behaviour?

A

Male sex hormone Testosterone

23
Q

When and why does testosterone influence aggression?

A

Influence aggression from young adulthood onwards due to its action on brain areas involved in controlling aggression

24
Q

Who tested the testosterone/aggression hypotheses?

A

Dabbs in a study measuring salivary testosterone levels in violent and non violent criminals

25
Q

What did Dabbs find in his testosterone study?

A

Highest testosterone levels found in those with a history of primarily violent crimes

26
Q

However, what could be argued as a result of Dabbs findings?

A

Lack of generalisability as research was conducted only on criminals. However, this was responded to by studies of non-prison population, finding similar results and so giving credit to original findings

27
Q

What is the challenge hypothesis?

A

Monogamus species: testosterone levels should rise only in response to social challenges like threat to status, partner or male-male aggression. Humans are M so we would expect the same findings

28
Q

What approach does the challenge hypothesis link to?

A

Evolutionary approach, as it suggests we are biologically adapted so our T levels rise only to conditions of threat in order to provide defence

29
Q

Which further studies provide further evidence for the challenge hypothesis and so add credibility to hormonal explanations as they can be applied to the real world?

A
  • Gun related crimes can be explained by testosterone increase by the presence of stimulus, as this is seen as a threat
  • Klinesmith: Child saliva measurements for testosterone post play with gun or childs toy. Gun - significant testosterone increase
30
Q

However, Klinesmith’s study may be limited because..

A

Conducted on male students so many not be valid to the general population e.g females and different ages

31
Q

There has been inconsistent evidence from research into testosterone…

A

Archer - meta analysis of 250 males in 5 studies. Weak positive correlation was found.

  • May be accredited to different measurements used over the studies (small samples, only men, etc) so show beta bias
  • Concluded that due to methodological issues, a correlation of 0.8 was more appropriate providing modest support
32
Q

What is Cortisol?

A

Another hormonal mechanism that may influence aggressive behaviour

33
Q

What does cortisol do?

A

Increases anxiety and likelihood of social withdrawal, thus has a mediating effect on other aggression related hormones such as testosterone

34
Q

What do low/high levels of cortisol do?

A

High levels inhibit testosterone which leads to aggression. Low levels are found in habitual violent offenders and school children.

35
Q

What do levels of cortisol suggest?

A
  • Although T is primary hormonal influence

- Low cortisol also plays a role by increasing the likelihood

36
Q

Evidence for the role of Cortisol comes from McBurnett on boys with behavioural problems..

A

Consistently low levels of cortisol boys began antisocial acts at a younger age and show 3x the number of aggressive symptoms than those with high or fluctuating levels

37
Q

The neural and hormonal mechanisms explanation follow a biological approach. What is a strength of this?

A

Such approaches allow for empirical data, easy to gather strong scientific evidence such as saliva samples, validating the theory as an explanation

38
Q

The neural and hormonal mechanisms explanation follow a biological approach. What is a limitation of this?

A

Reductionist - does not take factors such as social influencing factors into account