NCLEX Flashcards
Diseases with too much aldosterone
Cushings (too much of all steroids), hyperaldosteronism (Conn’s)
too little aldosterone
Addisons- Need to ADD steroids- will go into FVD
ANP action
in response to atrial stretch (from vol excess) released to cause excretion of sodium and water
goes up with concentration and down in dilution
urine specific gravity, sodium and hematocrit
Anti diuretic hormone action (ADH)
causes retention of only water- think ADH-H20. ADH from pituitary in head- head injury or sx? increased ICP?
too much ADH
SIADH- too many letters, too much water! Na does not follow the water. retain water- FVE. urine concentrated (being retained) and blood dilute
too little ADH
DI= Diurese. Lose water, FVD, urine dilute and blood concentrated.
normal CVP
2-6 mmHg, or 5-10 cmH20
Fluid retention think
heart problems first. Watch the weight on all heart patients.
assessment or evaluation think
signs and symptoms
Bed rest induces
diureses- blood comes back to core- atrial stretch- ANP and reduction of ADH. Everything thinker- pulmonary secretions (pneumonia), blood (DVT), kidney stones- push fluids unless contraindicated. watch for crackles, edema, weight, BP, CVP
IVF slowly to
old, young, hx of heart or kidney problems
polyuria think
shock. loss of fluids from anywhere: thoracentesis, paracentesis, vomiting, diarrhea, hemorrhage, third spacing.
sxs FVD
decreased weight, BP, UO, skin turgor, dry membranes, increased respiratory rate, concentrated urine (increased specific gravity), cool and clay. SAFETY- risk for ORTHOSTATIC HYPOTENSION, risk for falls and overload
isotonic fluids action
increases blood pressure, NS goes with blood products. DO NOT give to HTN, cardiac or renal d/s can cause FVE, HTN, or hypernatremia (ones with sodium)
types isotonic fluids (4)
NS, LR, D5W, D5 1/4 NS
hypotonic fluids action and types (3)
(hypO= Out of vessel) shifts into cells- rehydrates without causing HTN. For those with hx of HTN, cardiac or renal d/s, to dilute hypernatremia. D2.5W, 1/2 NS, 0.33%NS ALERT- watch for cellular edema/ too much fluid to cells can cause FVD in vascular space and decrease BP
hypertonic fluids action and types
(hypEr= Enter the vessel) Volume expanders
for hyponatremia or severe third spacing. ALERT- monitor for FVE- BP, pulse, CVP. D10Wm, 3 or 5%NS, D5LR, D5 1/2NS, D5 NS, TPN
2 types of cancer
solid tumors and hematologic malignancies
sarcoma
solid tumor in connective tissue- bones, cartilage, tendons
carcinoma
solid tumor in epithelial tissues- line organs- and skin! most common type of cancer
hematologic malignancy
originate from blood or lymphatic system- leukemia, lymphomas..
metastasis
ability to travel- can travel by direct invasion, through blood stream or through lymphatic system
cancer primary prevention
no smoking, exercise good nutrition, maintain normal weight, limit or eliminate alcohol, vaccine for viruses, avoid exposure to carcinogens
secondary prevention for cancer (female)
screenings to detect early. Breast SE day 7-12, clinical breast exam q3yrs 20-39 and q1yr >40. paps q3yr >21 y/o. mammogram starting at 40 y/o
colonoscopy and fecal occult blood > 50 y/o
secondary prevention for cancer (male)
breast self awareness, yearly testicular exam, monthly self exam, (majority ages 15-36!!) teach testical self exam early. Digital rectal example, PSA, and fecal occult blood annually after 50. colonoscopy at 50 and q10yr.
Cachexia
extreme wasting and malnutrition (used in cancer)
Cancer symptoms think
Caution Chang in bowel/bladder habits A sore that does not heal Unusual bleeding or discharge Thickening or lump Indigestion or difficulty swallowing Obvious change in wart or mole Nagging cough or hoarseness Also- weight loss, FATIGUE, pain Fever- first in leukemia and lymphoma (infection! )
Cancer can invade bone marrow causing (3)
Anemia -hypoxia risk
leukopenia- infection risk
thrombocytopenia- bleeding risk
rupture of innominate artery
bleeding massively from the trach- call Dr, OR ASAP
sputum specimen
get first thing in the morning, sterile technique! sterile specimens. have client rinse out mouth first to decrease mouth bacteria, don’t let mouth touch the sterile cup. if they have a trach- get the sputum specimens from the trach.
total laryngectomy
removed vocal cords and epiglottis- have a permanent trach. can use an electrolarynx or Blom- Singer to talk. they cannot whistle, drink through a straw or swim, can smoke through the trach.
suctioning a trach
hyper oxygenate before and after, sterile procedure, stop advancing at resistance/ cough, intermittent suction on the way out for no longer then 10 seconds, monitor HR/arrythmias, can stimulate the vegas nerve and cause HR to drop
adjuvant
using two complimentary treatments together- like chemo and radiation
neoadjuvant
time specific therapies- one is done before the next- like surgery to remove tumor followed by chemo.
Grade of cancer
1-4 based off of how different cells are from the original cells- more different- more aggressive.
TNM system
cancer staging- TNM
Tumor size, lymph Node involvement, Metastasis?
mastectomy with lymph node removal
protect that side for the rest of life- no constriction, no: watch, purse on that side, IV, BP, sunburn, nail biting, elastic blouses, watch small cuts
brachytherapy
internal radiation close to cancer. both pts will emit radiation
unsealed- IV or PO, radioactive for 24-48 hrs and radioactive body fluids
sealed or solid- body fluids not radioactive, can be temp or permanent implants
*safe to assume all emit radiation!!!
nursing internal radiation precautions
time, distance, iron shield. rotate assignment daily, nurse should only get one radiation implant pt a shift, private room, film badge, wear gloves with risk of exposure to body fluids, mark room with instructions for specific isotope, prevent dislodgment- bed rest, fiber restriction, cath to prevent bladder distention
internal radiation pt visitors
limit, visitors must stay 6 ft away, cannot visit for more than 30 minutes. no visitors who are < 16 years old or pregnant.
after internal radiation precautions
client is immunosuppressed. stay 6 ft away from people for up to 10 days, close lid and flush toilet 2-3 times
radiation implant dislodgement
gloves, forceps, tongs- put in lead lined container and call radiation dept- leave it in the room
external radiation
AKA tele therapy or external beam radio therapy with a cobalt or gamma machine- pt is not radioactive- do not wash off markings or put lotion on them, protect the site from UV for 1 year
sxs from external radiation
1 c/o fatigue, location and dose related. erythema, shedding, altered taste, and pancytopenia (all blood components decreased)
calcitonin
hormone made by the thyroid gland. it takes ca from blood and puts it back into bones- so it decreases serum calcium. the drug calcitonin is given to pts with osteoporosis. need iodine to make hormones
don’t give beta blockers to
asthmatics or diabetics. hides the symptoms of hypoglycemia and can cause an asthma attack
parathyroid think
calcium. parathyroid secretes PTH (parathyroid hormone) which pulls calcium from bone and deposited it in the blood- increases serum calcium
hyperparathyroidism=
hypercalcemia= hypophosphatemia
too much PTH, serum ca is high and phosphate is low- client is sedated (Ca acts as a sedative) fix with partial parathyroidectomy
pheochromocytoma
benign tumors in adrenal medulla (on top of kidneys) that secrete boluses of epi and norepinephrine causing increase BP, pulse, flushing, HA, palpitations- check the catecholamine levels (vanilla test or metanephrine test, 24 urine collection) DO NOT palpate ABN, can cause bolus and severe HTN
4 major actions glucocorticoids
adrenal cortex steroid. Think MI-FI Mood, Immunosuppressed, Fat, Insulin
1- change mood
2- alter defense mech- suppress immune system
3- Breakdown fats and proteins -to glucose
4- inhibit insulin- hyperglycemia- monitor glucose
and don’t let them share room with infected b/c of suppressed immune system
Graves
hyperthyroid
addisons
insufficient steroids/ adrenocorticoids- glucocorticoids, mineralocorticoids (aldosterone), and sex hormones
aldosterone drug name
fludrocortisone
glucocorticoid administration
prednisone- give twice a day, 2/3 of dose in morning and 1/3 of dose in evening (for addisons disease)
addisons symptoms
Think no aldosterone or glucocorticoids. no aldosterone to help retain water and Na, so lose water and Na, retain K, hypotension; no glucocorticoids to make glucose or inhibit insulin, so hypoglycemic, confused, GI probs, fatigue, and skin discolorations- hyper pigment or depigment (vitiligo)
cushings
too many steroids- glucocorticoids, mineralocorticoids, sex hormones
magnesium and calcium think
sedatives/ muscles, loose, flaccid muscles, arrhythmia, decreased LOC, DTRs, pulse and respirations
normal magnesium
1.3-2.1 mEq/L
normal calcium
9.0- 10.5 mg/dL
low magnesium or calcium
tight rigid muscle tone, risk for seizure, stridor/ laryngospasm, chvosteks and or trousseaus sign, arrhythmia, DTRs increased, swallowing probs
calcium gluconate
given IVP very slowly (max 1.5-2mL/min) For hypermagnesium (antidote), or hypocalcemia
hypermagnesium causes, tx
renal failure or antacids (tums have magnesium) tx: vent if resp depression, dialysis if kidney prob, calcium gluconate antidote
hypercalcemia cause and tx
hyperparathyroid (too much PTH), Thiazides (retain Ca), immobilization- bear weight to keep Ca in bones, fluids to prevent kidney stones, DVT, sodium phosphate- inverse relationship, steroids, phosphorous foods, calcitonin, vitamin D
hypomagnesium cause and tx
diarrhea, alcoholism- give Mg but monitor kidney fin!, seizure precautions, eat magnesium
foods high in magnesium
spinach, mustard greens, summer squash, broccoli, halibut, turnip greens, pumpkin seeds, peppermint, cucumber, green beans, celery, kale, sunflower seeds, sesame seeds, flax seeds
hypocalcemia cause and tx
hypoparathyroidism, radical neck dissection, thyroidectomy (all not enough PTH)
Vit D, phosphate binders (sevelamer, calcium acitate), IV Ca- give slowly with pt on heart monitor
Na think
Neuro! neuro changes when sodium is high or low
hypernatremia= dehydration cause
feeding tube, hyperventilation, heat stroke, DI. give fluids, restrict Na, Daily weights, I&O, lab work
hyponatremia= Dilution cause and tx
Replacing fluid with only water, psychogenic polydipsia, D5W, SIADH
client needs salt, not water, give hypertonic saline if having neuro probs- 3 or 5% NS- very dangerous, messes with brain and can throw into FVE
hypernatremia symptoms
neuro and dry mouth, thirsty, swollen tongue
hyponatremia symptoms
neuro and HA, Seizure, Coma
hyperkalemia cause and tx
kidney probs, aldactone (spironolactone-retain K),
dialysis if kidney prob, calcium gluconate for arrhythmia, glucose and insulin, Kayexalate (sodium polystyrene sulfonate)
hyperkalemia ECG changes
bradycardia, tall peaked T waves, prolonged PR intervals, flat or absent P wave, widened QRS, conduction blocks, v fib
hypokalemia ECG changes
U waves, PVCs, v tach
hyperkalemia symptoms other than ECG
first muscle twitching, then weakness, then flaccid paralysis- then life threatening arrythmia
hypokalemia symptoms (not ECG)
muscle cramps and weakness then life threatening arrythmias
hypokalemia cause and tx
vomiting, NG suction, diuretics, not eating
potassium supps
GI upset with PO- give with food. IV always on a pump, mix well- settles, never IVP, burns during infusion, Assess kidneys/ urinary output before giving
potassium and metabolic A/B imbalance
metabolic acidosis= hyperkalema
metabolic alkalosis= hypokalemia
rule of 9s
head and neck-9
each arm-9
each leg-18
front-18, back-18
consensus formula
aka parkland formula
4mL LR X Kg X % burned= total for 24 hours
1st 8 hours- 1/2
next 16 hours- 1/2
titrate to 0.5mL/kg/hr urinary output (1mL for kids)
prealbumin
most sensitive indicator for nutritional status/ nitrogen balance
diet for burns
protein and vitamin C- always for max nutrition
meds to prevent GI stress ulcer
curlings ulcer
antacids= HYDROXIDE (a base!)-aluminum hydroxide, mag hydroxide (milk of magnesia). liquid on empty stomach at bedtime, when stomach empty more risk of acid on the ulcer, protects overnight.
H2 antagonist= DINEs, ranitidine, famotidine..-take at night to inhibit acid secretion
proton pump inhibitors= ZOLE- pantoprazole, esomeprazole..take before first meal
Sucralfate- barrier over ulcer. 1 hr b4 meals and at bedtime, empty stomach, with glass of water- no other meds at same time.
tetanus toxoid
booster- takes 2-4 weeks for body to make antibodies- active immunity. give with burns unless overdue for booster or don’t know- then give immune globulin (antibodies)-passive immunity
circumferential burn
circulatory checks- pulse, color, temp, cap refill
if bad- escharotomy or fascitomy to relieve pressure
brown or red urine post burn
myoglobin from damaged tissues- can clog kidney- call dr. may give Mannitol to flush (diuretic) also given for increased ICP. EXCEPTION- normally want to give fluids not diuretics to burn pt- trying to save kidneys
burns and potassium
tissue damaged- k leaks out of cells, hyperkalemia
enzymatic debridement
sutilains or collagenase- eat dead tissue
not for face, if PG, over large nerves or if area is opened to a body cavity.
mycin drugs
cause nephrotoxicity and ototoxicity- monitor BUN, creatinine and hearing
autograft
(burns) graft from own skin, dressing at donor site until stops bleeding, then open to air, can re harvest from same donor site every 12-14 days in healthy pt
electrical burn
heart monitor for 24 hours, at risk for v fib, c collar until cervical fracture is r/o
Cardiac output assessment
focused assessment- do anytime a hear rhythm change!
LOC, Chest pain?, wet lungs, skin cold and clammy or warm and dry?, Urinary output, peripheral pulses
what is coronary artery disease
broad term that includes chronic stable angina and acute coronary syndrome (MI and unstable angina)
drugs for chronic stable angina
nitro- dilates all vessels, decreased BP, ASA
and BLOCK from happening again (prevention) with BB and CCB!
beta blockers (prevention)- decrease BP, pulse and contractility
ca channel blockers (prevention)- dilates arteries, including coronary arteries and decreases BP
don’t leave a hypotensive client- unstable!
given to protect kidneys from iodine dye
acetylcysteine preprocedure; iodine based dye makes them feel warm and flushed and palpitations are normal- cardiac cath uses dye!
Triad MI symptoms
in women with MI- indigestions/ abdominal fullness, chronic fatigue and SOB. women also c/o pain btwn shoulders, aching jaw or choking sensation
1 sign MI in elderly
SOB. they may faint or have a behavior change
anytime ELDERLY + BEHAVIOR CHANGE = PROBLEM could be UTI, but r/o life threatening causes first
MI labs
troponin most sensitive and specific- elevates 3-4 hours and remains elevated for 3 weeks
myoglobin increases within one hour and peaks in 12 hours- not specific for diagnosis, but can be used to R/O
troponin levels
troponin T < 0.10 ng/ml
troponin I < 0.03 ng/ml
digoxin
normal level- 0.5-2 ng.ml
increases contractility and decreases heart rate (check apical pulse) =increase CO
toxicity GI probs then arrhythmia and vision change
apical pulse location
5th intercostal , left midclavicular
fluid retention, think
heart problems (heart failure) report weight gain of 2-3 lbs (1-2 kg)
loss of capture
when pacemaker fires but does not result in a heart contraction
failure to sense
when pacemaker fires at inappropriate times when it is not needed
cardiac tamponade
blood fluid or exudates have leaked into pericardial sac resulting in compression of heart. hallmark signs: INCREASING CVP with DECREASING BP. NARROWED Pulse Pressure=CARDIAC TAMPONADE
WIDENED PP= INCREASED ICP
decreased CO= worry shock
TX- pericardiocentesis