NCD Prevention Flashcards

1
Q

Primary prevention

A

Reduce incidence

Pre-pathogenesis

Health promotion and specific protection

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2
Q

Health promotion (general level)

A

Env modifications

Nutritional interventions

Lifestyle and Behavioural changes

Health education

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3
Q

Specific protection

A

Targets type or group of diseases

Immunisation

Specific nutrients or supplementations

Protection against occupational hazards

Control of env hazards

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4
Q

High risk approach

A

Focuses on Indivs at greatest risk of disease

But they may contribute little to overall burden of disease in popn

Adv: Subject and Physician motivation , interventions appropriate to indiv , cost effective use of resources , favourable benefit to risk ratio

Disadv: temporary (does not alter underlying causes), Behaviorally inappropriate (inconvenient, against social norms) , power to predict future disease weak —> indiv with risk factor can remain well while unexpected illness can happen to someone with clear screening

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5
Q

Population approach

A

Reduce average risk in popn

Adv: radical (attempt to remove underlying causes), small reduction can lead to significant reduction , behaviourally appropriate (social norm changes)

Disadv: bring much benefit to community but offer little to each indiv , poor subject and physician motivation

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6
Q

Secondary prevention

A

Reduce prevalence by shortening duration

Detect disease sooner and make treatment more effective

Target indivs who are free or asymptomatic

Screening —> important health problem, high prevalence , natural history understood, long latency period , early detection improves prognosis

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7
Q

Tertiary prevention

A

Reduce complications/progression of disease

Target: patients

Treatment and rehabilitation

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8
Q

Life course approach

A

Understanding processes that operate across lifespan or generations

NCDs can be prevented and controlled at multiple stages of life

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9
Q

Critical period

A

Limited time window in which exposure can have adverse or protective effects on outcome

Outside this window, no excess disease risk

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10
Q

Sensitive period

A

Not temporarily fixed

Exposure can have greater impact than at another time

Increases risk but does not necessarily result in irreversible damage

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11
Q

Foetal origins hypothesis

A

Barker hypothesis

Adaptation for survival made by foetus raise risk of chronic disease risk in later life

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12
Q

Preconception, foetal development

A

Micronutrient deficiencies

Exposure to potentially harmful substances

Gestational diabetes, congenital anomalies

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13
Q

Infant development

A

Breastfeeding

Reduction in adolescent and adult obesity rates

Reduction in risk for asthma, leukemia and other NCDs

Reduction in incidence of diabetes

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14
Q

Screen time

A

Interactive — video games, communicating via Skype

Not interactive — sitting and watching movies

Educational — doing math hw online

Recreational — games or watching videos

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15
Q

Childhood and adolescence

A

Development of habits that will carry over into adulthood

Healthy behaviours initiated in childhood shd be maintained during adolescence

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16
Q

Adults, Elderly

A

Adult: maintaining highest possible level of function

Older age: maintaining independence and preventing disability

How well you started determines how well you end

Acceleration in decline can be reversible at any age and influenced thru env, indiv, policy level measures

Early life preventive measures require long term investment but can lead to large reductions in disease risk

17
Q

Diet quality

A

Prevent heart disease, diabetes and other NCDs

Prevention weight gain

Appropriate intake of micronutrients and macronutrients

18
Q

Micronutrient

A

Needed only in minuscule amounts

Enable body to produce enzymes, hormones

19
Q

Folic acid

A

Green leafy veg, legumes (peas)

Folate deficient: cells unable to divide —> macrocyte , megaloblastic ; neural tube defects

20
Q

Vitamin A

A

Liver, milk, cheese, eggs

Deficiency: Xerophthalmia (night blindness) , infections , anaemia (low iron status)

21
Q

Macronutrients

A

Carbohydrate: glycaemic index —> how slowly/fast food causes increase in sugar level

Fruits and veg: vitamins, minerals, dietary fiber and antioxidants

Proteins: Lean/white meats , plant oils

No saturated and trans fat: increase risk of heart disease and stroke

No high sodium/salt: increase BP, CVD, stroke

No high sugars: increase risk of overweight/obesity, tooth decay, NCDs

22
Q

Physical inactivity

A

6 to 10% of major NCDs worldwide attributable

23
Q

Alcohol use

A

Heart disease, stroke, pancreatic, liver damage, poor judgement, slower reaction time (depressant), impaired speech and motor skills, loss of balance, depression, anxiety

Drink in moderation: per day not more than 1 drink (women) , 2 drinks (men)

24
Q

Tobacco smoking

A

Kills up to half of users — direct use , exposed to secondhand smoke

Sensation short lived —> smoke a no of cigarettes thru the day to maintain stimulation (addiction)

25
Metabolic risk factors
Overweight and obesity Raised blood pressure Raised total cholesterol Elevated blood glucose
26
Body mass Index (BMI)
BMI = weight (kg) / [(height(m)]^2 Shd not be used to calculate risk in pregnant women, ppl with muscular build and elderly, youths below 18 —> increase cld be due to muscle mass increase Measure of excess weight rather than excess body fat — does not distinguish btw excess fat, muscle or bone mass nor does it provide indication of distribution of fat among indivs