Muscle Tone Assessment Flashcards

1
Q
  • resistance of m. to passive elongation or stretch

- resistance increases with increasing amplitude and velocity of passive stretch

A

increased m. tone/spasticity (velocity dependent)

  • caused by hyper-excitability of the stretch reflex
  • sign of UMN damage
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2
Q

what diagnoses do you most often see spasticity?

A

UMN damage: CP, stroke, MS, SCI

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3
Q

simple way to differentiate spasticity and rigidity

A

increase speed when testing:

  • spasticity will change with increasing speed
  • rigidity will not feel different with increasing speed
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4
Q

if spasticity occurs which tracts/pathways are likely damaged?

A

lateral corticospinal tracts

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5
Q

what is clasp knife spasticity?

A
  • type of spasticity
  • initially high resistance but then lets go at end of joint range (more than a catch)
  • seen most when flexing a joint/stretching an extensor (triceps, quads)
  • no Ashworth number
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6
Q

clonus

A

muscular spasm involving repeated (often rhythmical) contractions

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7
Q

what part of the body do you observe clonus?

A

most commonly tested at the ankle (forced DF while relaxed) but can be seen elsewhere including jaw, wrist, patella, etc.

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8
Q

resistance to passive movements involving both agonist and antagonist muscles (both directions whether or not the m. is on stretch)

A

rigidity (not velocity dependent)

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9
Q

cardinal sign observed in Parkinson’s disease

A

rigidity

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10
Q

ratchet-like jerks

A

cogwheel rigidity (seen more in UE with PD)

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11
Q

rigidity posturing

A
  • decorticate (flex UE)

- decerebrate (ext UE, worse prognosis)

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12
Q

decreased m. tone

A

hypotonia or flaccidity

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13
Q

does hypotonia/flaccidity occur b/c of LMN or spinal shock?

A

spinal shock; 24-48 hrs of hypotonia and absent DTRs followed by hypertonia and exaggerated DTRs

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14
Q

hyperkinetic movement disorder characterized by tone and involuntary repetitive twisting (writhing motions) movement in large portions of the body

A

dystonia (impaired tone)

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15
Q

tone varies and is affected by:

A
  • volitional movement
  • stress, anxiety
  • position
  • medication
  • health
  • environmental temp.
  • state of the CNS (arousal, alertness)
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16
Q

procedure for assessing m. tone

A
  • palpate m. belly
  • PROM, speed up as you go
  • all limbs/joints
  • compare R and L
  • compare UE and LE
  • cue pt to stay relaxed
17
Q

Modified Ashworth Scale

A
0 = no inc. in m. tone
1 = slight inc. in m. tone, manifested by catch and release or by minimal resistance at end ROM when moved in flex/ext
1+ = slight inc. in m. tone manifested by a catch followed my minimal resistance throughout less than half of the ROM
2 = marked inc. in m. tone through most of ROM, but affected parts easily moved
3 = considerable inc. in m. tone, passive movement difficult
4 = affected parts rigid in flex/ext
18
Q

DTR scoring/scale

A
0 = absent
1+ = present, but depressed
2+ = normal
3+ = brisk
4+ = very brisk, hyperactive with clonus
19
Q

major pathological reflexes

A

babinski and hoffman’s sign

20
Q

reflexes present in infancy that become integrated by CNS; obligatory responses that dominate motor behavior are always considered pathological

A

primitive reflexes

21
Q

synergistic patterns

A
  • abnormal obligatory movements that emerge with spasticity
  • inability to perform isolated movements at each joint
  • PT records when patterns occur, under what circumstances, and variations observed
  • flexion/extension synergies of UE and LE
22
Q

MMT validity with neuro conditions

A
  • based on movement capabilities
  • isolated movement may not be possible
  • in that case, MMT is not reliable or valid
23
Q

inhibits isolated movements making MMT unreliable/not valid

A

presence of synergies, spasticity, abnormal posturing, contractures

24
Q

how to estimate strength if MMT is unreliable/not valid

A
  • observation of active movement (i.e. squats, sit to stands, heel raises/toe raises)
  • modified MMT position documented in notes