MSK US Flashcards

1
Q

Skin & subcutaneous tissue

A
  • high freq* linear probe
  • only need a shallow depth of penetration & high detail
  • use B-mode & Colour Doppler
  • watch yr probe pressure
  • use a good layer of gel or a water bath (glove filled w water)
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2
Q

Abscess formation

A
  • often starts w separation of fatty areas, like cellulitis
  • maybe be hypoechoic but may not be
  • will have increased vascularisation with time
  • May swirl or appear to move in increased fluid, but not always if more solid contents
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3
Q

Benign masses US

A
  • anechoic - true cyst is benign
  • thin walled
  • well defined
  • wider than tall
  • a vascular/minimally vascular
  • hyperechoic non-shadowing
  • does not cross tissue layers
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4
Q

Benign soft tissue masses

A
  • well defined
  • avascular
  • variable echogenicity from hypo/hyper or mixed
  • rare to be in muscle
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5
Q

Epithelial inclusion/sebaceous cyst

A
  • sebaceous cells push into deeper layers
  • typically not anechoic
  • may have edge shadowing
  • may be at varied depths
  • Check: sinus, leaking and adjacent structures
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6
Q

Pilonidal cyst

A
  • abscess
  • sinus
  • hair
  • often hypoechoic compared to surrounding
  • white evidence of hair within cyst
  • can be quite large so ensure U define its entire extension under the skin
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7
Q

Malignant masses US

A
  • cannot diagnose precisely from ultrasound alone

Suggestive if:
- large
- not from synovial space (bursa or tendon sheath)
- hyperechoic & hypervascular
- Ill-define & solid
- posterior shadowing
- invading tissue planes/destructive (very suspicious!!)
- thick walled

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8
Q

Vascular subcut* masses

A

Haemangiomas

Arteriovenous malformations

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9
Q

Bone shaft & Joints

A
  • highest freq* probe possible
  • excellent for joints, not so great for bone shafts (except for stress #s)
  • scan in two planes: if u can’t see it in both planes, it’s not real
  • use colour to ensure it’s not a nutrient channel
  • lots of gel/stand off pad/bath
  • move & stress joints: to get movement to move fluid/items to show things better
  • be aware of heating: esp in smaller places (esp hands)
  • talk to the Pt, esp when looking for stress #s, they’ll be quite tender & a Pt’s response shows if neurological Vs MSK
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10
Q

Normal adult bone Appearance

A
  • echogenic
  • linear
  • reverberation artifacts: mirror images
  • nutrient channels present: vessels going thru periosteum
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11
Q

Normal child bone Appearance

A
  • echogenic
  • linear
  • reverb* artifacts
  • periosteum
  • Sharpeys fibres - thicker cortical bone layer (creates hypo echoic line on cortical surface.
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12
Q

Normal Adult joint appear*

A
  • echogenic
  • reverb* artifact
  • articulate/hyaline cartilage: hypoechoic layer between joint capsule & bone
  • joint capsule
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13
Q

Normal child joint Appear*

A
  • echogenic
  • growth plate/physis
  • reverb* artifact
  • articular/hyaline cartilage: thicker than Adults, still quite dark (not joint effusions, compare to collateral side!!)
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14
Q

Abnormal bone appearance

A

Fractures:
- stress #s in kids - bone bruising (bony bruising, wider sharpeys fibres, along the cortex)
- trauma
- pathological

Adult #s:
- step deformity & adjacent soft tissue inflammation/haematoma

Aged #s:
- loss of normal tissue planes & raised cortical surface with healing

Shoulders:
- # humeral head may be found, not just a torn tendon!

Pubic symphysis:
- erosive changes possible
- bowing out of the joint capsule indicates underlying changes

Pathological #s:
- normal cortical margin that gets lost
- adjacent soft tissue mass, normal tissue planes have been disrupted
- cloud like appearances around bone
- loss of smooth cortical surfaces

Exostosis:
- benign, bony outgrowth if cartilaginous tissue
- bursitis around bony outgrowth

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15
Q

Abnormal joint appearance

A

Traumatic:
- simple effusion: twisted knee
- complex effusion: rheumatoid/septic arthritis
- can be hypoechoic or echogenic region

Infective: septic Arthritis

Degenerative:
- erosions: deterioration in cortical surface, fluid & swelling around joint region
- osteophytes: loss of smoothness of bone surface, loss of volume of joint space,

Vascularity:
- bowing out of joint capsules
- colour: vasc*
- no colour: synovial hypertrophy

Cartilage:
- articular: hard cartilage, graded changes, 0-3 (often involves osteophyte formation: grades 0-3)
- fibrocartilage: spongy cart*, meniscus can protrude w trauma/degenerative changes (MRI better at detecting)

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16
Q

Normal tendon & sheath Appearance

A
  • type 1 tendon: straight from muscle to bone
  • type 2 tendon: crosses over a synovial joint

Type 1:
- peritenon
- paratenon
- epitenon- fills spaces between fascicles & contains collagen & elastic fibres
- endotenon
- fascicles

Type 2:
- synovial lining/bursa (internal & external linings)
- epitenon (fills space between fascicles & contains collagen & elastic fibres)
- endotenon
- fascicles

17
Q

Elastography

A
  • assess the elasticity of structures
  • tendon should have a high elastic modulus or be ‘stiff’
  • abnormal tendons become less stiff & less able to store & release energy.
18
Q

Subluxation

A

Causes:
- acute
- chronic
- congenital laxity if fibrous hood
- congenitally shallow grove

Intermittent/reducible?

Permanent subluxation

Permanent dislocation

Assoc* abnormalities:
- associated inflammation & disruption of tissues
- cystic changes
- changes to cortical surface

19
Q

Abnormal Tendon Sheath appearance

A

Type 1:
- hypoechoic, inflammation shown by increased vascularity

Type 2:
- thickening of the sheath around the tendon
- increased fluid & vascularity

20
Q

Synovial Proliferation (Panus)

A
  • rheumatological Dx; rheumatoid & psoriatic arthritis
  • colour very useful esp if thickened synovium quite hypoechoic
  • probe pressure & dynamic assessment also essential to increase sensitivity
21
Q

Infective synovial fluid

A
  • US not 100% sensitivity to detecting this
  • tends to be more echogenic with synovial thickening & increased vascularity BUT cannot be sure based solely on US
22
Q

Abnormal tendon appearance: grades

A

Grade 1:
- no fibre disruption
- often hypoechoic
- excessive activity may appear as diffuse hyper echogenicity
- let the Pt’s symptoms guide U
- use colour/power Doppler

Grade 2:
- partial tear, moderate disruption
- often at myotendinous junction but direct trauma may result in muscle body tear
- can be difficult to define edges, so use movement
- can be massive

Grade 3:
- complete disruption
- define size
- distance from attachment
- use movement

23
Q

Enthesis - normal appearance

A
  • change in orientation in fibres & results in thin black line where tendons joint the bone
  • manual labour/exercise => double the thickness

Can be mistaken for a tear, but it’s the enthesis:
- change position or change Pt’s position to test

24
Q

Enthesis - abnormal appearances

A
  • irregularity
  • subchondral cysts
  • calcific stripes
  • osteophytes
  • failure
  • bursa/fat pad
  • seronegative arthropathy (rheumatoid issues)
25
Q

Muscle injuries: extrinsic Vs intrinsic

A

Extrinsic:
- external trauma; contusion or laceration (thigh & the vastus intermedius is against the femur)

Intrinsic:
- often exercise induced; disruption of the muscle fibres
- usually near a myotendonus junction

26
Q

Pathobiology if muscle injury

A

1- distructive phase: 0-3 days

2- early remodelling: 3-12 days

3- late remodelling: >12 days

27
Q

Grades muscle strain injuries

A

Grade 0 - normal

Grade 1 - tear affecting small No of muscle fibres w an intact fascia (elongation injury; flame shaped hypoechoic defects one cm or longer in muscle belly)

Grade 2 - mod tear w fascia remaining intact (partial rupture; more than 5% muscle but less than full diameter of muscle)

Grade 3 - tear of many fibres w partial tearing of the fascia

Grade 4 - complete tear of the muscle & fascia (complete rupture; muscle totally torn, the muscle stump floats in fluid ‘clapper in the bell sign’)

28
Q

Complications of muscle tears

A
  • cysts
  • myositis ossificans
  • calcific myonecrosis (rare)
29
Q

Myositis ossificans (MO)

A
  • ongoing symptoms/failure to improve 2wks post injury
  • intense inflammatory reaction around tear site; hypervascularity and oedema
  • usually anterior muscles of thigh or arm
  • comorbid conditions can increase risks
  • CAN look like aggressive tumour like growths!
30
Q

Scanning techniques of muscle imaging

A

Look for;
- haematoma
- inflammatory reaction
- regeneration & scar
- denervation change & MO!!

31
Q

Inflamm* & ischaemia cond*

A
  • idiopathic inflammatory myopathies
  • pyomyositis & hydatid Dx
  • diabetic muscle infarction & rhabdomyolysis
32
Q

Acute compartment syndrome US

A
  • increased pressure within the muscle compartment
  • single insult; trauma or exercise overload
  • increasing pain w movement
  • pain can be delayed up to 12 hrs post exercise
  • most often anterior, posterior & lateral lower leg compartments
  • increased compartment size
  • sometimes bowing or fascia
33
Q

Rhabdomyolysis

A
  • skeletal muscle necrosis
  • accompanied by an acute rise in enzymes
  • often involves gluteal muscles
  • IVDU & statins users
  • hard to diagnose
34
Q

Diabetic myonecrosis

A
  • usually lower limbs or pelvis
  • usually poorly controlled diabetic pts