Module Four: Atrial Fibrillation + DVT Flashcards

1
Q

What is an arrhythmia?

A

irregular heartbeat is an arrhythmia (also called dysrhythmia). May or may not include a change in heart rate

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2
Q

What might cause an arrhythmia?

A

Dec K+, CHF, stroke, pulmonaray disease.

Pretty much anything that may cause CAD, HTN, ETC

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3
Q

Normal Sinus Rhythm (NSR) vs Afib heart Rate

A

NSR- 75 and AFIB 150-300

AFIB often Irregular irregular, no P wave distinction

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4
Q

Risk factors for Afib?

A
Risk factors:
Age 
inflammation
enlarged atria
Hormonal Abnormalities
Alcohol abuse
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5
Q

Signs ans Symptoms of Afib?

A
!  HR 100bpm(Dec CO)
!  Dizzy
!  Weakness
!  SOB
!  Fainting
!  Palpitations (skipped beats)
!  Irregular pulse
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6
Q

Interventions for Afib?

A

Dependent on severity and symptoms but…

Non-Pharmacological
! Cardioversion and defibrillation
! Electrical shock stops all electrical impulses. ( requires Ventricuar tachycardia)
! SA node to restart at normal sinus rhythm
! Pharmacology
! Rate Control
! Decrease risk of complications

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7
Q

Nursing considerations for Afib?

A

! Heath History (Heart failure/HTN/CAD/ Allergies?)
! Is cardiac alteration producing symptoms???
-> Can they walk with the traid of symptoms Fatigue, dizziness, palpatations
! Vitals (BP/HR/SpO2)
-> Bp not key factor, HR more important
! Electrolytes (k+ levels, other ions, dec urine?)
! Baseline ECG

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8
Q

What is a palpitation?

A

a noticeably rapid, strong, or irregular heartbeat

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9
Q

Common pharmacological tx

Class 1-5

A

1) Sodium channel blockers (class1)
2) Beta-Blockers
3) Potassium channel blockers
4) Calcium channel blockers
5) Positive inotrope- digoxin

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10
Q

Other key Medications in Afib?

A

Antiplatelet- prevents thrombus/embolus

Anticoagulant

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11
Q

What is the most common medication for Afib?

What does it do?

What’s bad about it?

A

Digoxin

! Increases force of myocardial contraction
! Prolongs conduction through SA and AV nodes ! Slows and strengthens myocardial contraction
! Therapeuticeffect -$CO,useful for CHF

Can be toxic, watch for bradycardia

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12
Q

Do anticoagulants break up clots?

A

Not really
They clotting time (seconds) to prevent thrombi from
forming, or growing larger by Inhibit specific clotting factors in the coagulation cascade

clot busters are called thrombolytics

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13
Q

When do you avoid thrombolytics?

A

Post Sx or with any new clot formation preventing excessive bleeding

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14
Q

Reasons someone might be on anticoagulants

A
!  Immobility (post surgical)
!  Hx. DVT/pulmonary embolus 
!  Dysrhythmias ( A-Fib)
!  Mechanical heart valve
!  Post MI or stroke
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15
Q

Why might the Doc order LMWH and not heparin?

A

! Duration is 2-4 times longer then heparin (heparin half life 90mins)
! Produce more stable response than heparin
! Reduced risk thrombocytopenia
! Dosages based on patient weight
! Dec follow up lab tests
! Patient/family member can be taught to self administer

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16
Q

Pro and Cons of Warfarin as anticoagulant?

A

! Can be taken only oral
! Can take 3-5 days to reach therapeutic level
! Long half life ! 1-3 days
! Must Monitor PT-INR

NOTE
! Antidote is Vitamin K

17
Q

Two most common Antiplatelet Meds?

A

ASA
! Anticoagulant properties can last up to a week after one single dose

Clopidogrel (Plavix)

! Anticoagulant properties can last up to 5 days after
last dose
! Usually reserved for patients who cannot tolerate ASA
! Given to patients with recent MI or stroke

18
Q

Nursing Considerations for Anticoagulants?

A
•  Monitoring for S & S of bleeding
–  Hematuria, epitaxis, bloody stools, bruising
•  Monitor vital signs
•  Monitor lab values
–  CBC, PTT, PT-INR
•  Hepatic/renal failure (can they metabolize and excrete the med?)
•  Drug to drug interactions
•  Patient teaching
19
Q

What is the D-Dimer Test for?

A

Detecting fibrin degradation product- small protein fragment detectable in blood after clot breakup. Allows you to know if a thrombus has recently broke possibly causing embolus to be circulating in blood

20
Q

Why is Thrombus formation a concern in Afib?

A

Anything that causes pooling can lead to platelets aggregation

21
Q

Diagnostics Associated with Anticoagulant therapy?

A

! Platelets
! D-Dimer
! PTT, PT, INR
! Ultrasound (Monitoring for clots in vasculature DVT)
! CT Scan (monitor for pulmonary embolism, or stroke)

22
Q

Risk Factors for DVT?

AKA Virchow’s triad

A

Blood stasis
Vessel wall injury
Altered blood coagulation

23
Q

Conditions associated with Blood Clotting?

A
Immobility
AFIB
Heart failure
Obesity
Vericose veins
24
Q

Conditions associated with Vessel Wall injury?

A
Sx
Atherosclerosis
Central venous catheters CVC
Dialysis
TRAUMA
Diabetes
25
Q

Conditions associated with Altered blood coagulation?

A

Cancer
pregnancy
Oral contraceptives
Polycythemia

26
Q

Major complications of DVT

A

CVA or MI

Embolus travelling through body causing major blockage

27
Q

Signs ans Symptoms

A
Swelling (calf, foot)
Pain
Cool or warm to touch 
Redness
Can be non-specific..... hard to diagnose
28
Q

Common tx for DVT Prevention

A

! Increasing mobility (post surgical++)
! Compression stockings
! Intermittent Pneumatic Compression Devices
! Prophylactic Anticoagulation Therapy