Module 7 : Fetal Chest Pathology Flashcards
1
Q
contents of the chest
A
- lung
- heart
- muscle and diaphragm
- trachea
- esophagus
- ribs
2
Q
what is the diaphragm
A
- hypo echoic liner structure between lungs and abdomen
- easier to see in later gestations
- note shape and continuity
- usually visualized on parasagittal scans
3
Q
why is the diaphragm hypo echoic
A
- lung tissue is filled with fluid and collapsed so diaphragm appears hypo echoic
4
Q
fetal lungs
A
- homogenous
- echogenicity compared to liver can be
+ isoechoic
+ hyperechoic
5
Q
fetal heart position and chamber
A
- 4 chambers
- occupies 1/3 of the fetal chest
- apex points to the left with axis about 45º
6
Q
9 lung pathology
A
- pulmonary hypoplasia
- congenital cystic adenomatoid malformation (CCAM)
- congenital pulmonary airway malformation (CPAM)
- pulmonary sequestration
- bronchogenic cyst
- diaphragmatic hernia
- pleural effusions
- tracheal atresia
- esophageal atresia
7
Q
pulmonary hypoplasia
A
- when one or both lungs are underdeveloped
- can be lethal
- fetuses < 24 weeks are not considered viable due to pulmonary immaturity
8
Q
causes of pulmonary hypoplasia at term
A
- restricted chest cage \+ some skeletal dysplasia - decreased amniotic fluid - chest masses - pleural effusion
9
Q
what is the most frequent cause of pulmonary hypoplasia
A
- due to lack of fluid
+ PROM = premature rupture of membranes
+ GU anomalies = genitourinary
10
Q
amniotic fluid things
A
- produced by membranes until fetal kidney begin to produce urine
- fetus swallows amniotic fluid
- fluid goes through the GI track absorbed by the GI track excreted by the kidneys as urine fetus urinates back into the amniotic cavity
11
Q
cause of polyhydramnious and oligohydramnios
A
- poly= any high GI obstruction/swallowing deficits
- oligo = any bilateral GU obstruction/renal agenisis
12
Q
thoracic/ chest circumference
A
- method to predict pulmonary hypoplasia
- transverse chest circumference at level of 4 chamber heat
- measure outer to outer
+ except in cases of skin edema/hydrops
13
Q
congenital cystic adenomatoid malformation CCAM
A
- now called CPAM \+ congenital pulmonary airway malformation - three classifications \+ type I, type II, type III - type of hamartoma (1 lobe effected) - communicates with tracheobronchial tee - large may see mediastinal shift -
14
Q
what is CPAM associated with
A
- hydrops
- pulmonary hypoplasia
- polyhydramnious
15
Q
type I CPAM
A
- macrocystic
- > 2cm
16
Q
type II CPAM
A
- macro and microcystic
+ < 1.5cm
17
Q
type III CPAM
A
- microcystic
- monographically hyperechoic
18
Q
prognosis and treatment of CPAM
A
- depends on mediastinal shift and hydrops
- may regress or disappear on its own
- no intervention is performed unless hydrops is noted
- may drain cyst or surgically resect the mass