module 5 Flashcards

1
Q

mood states

A
  • euthymia
  • feeling sad/upset/grief
  • major depressive episode
  • dysthymia
  • hypomania/hypomanic personality
  • mania
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2
Q

euthymia

A
  • the goal of treatment aka living without mood disturbances and achieving baseline again
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3
Q

hypomanic personality

A

individuals are extroverted, energetic, intensely emotional, hyper confident, ambitious, impulsive, need less sleep and tend to be rude, irritable and irresponsible

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4
Q

polarity of mood states

A
  • average length of tome for the first untreated major depressive episode is 9 months
  • treated MDE = 4-5 months
  • untreated manic episode lasts between 2-6 months but will often be noted well before
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5
Q

major depressive episode

A
  • a component of various mood disorders
  • must experience 5 or more symptoms out of 9, during the same two-week period
  • at least 1/5 symptoms experienced must be either depressed mood or anhedonia
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6
Q

major depressive episode symptoms

A
  • depressed mood
  • significantly diminished interest or pleasure in all activities
  • significant weight loss or gain
  • insomnia or hypersomnia
  • psychomotor agitation or retardation
  • fatigue/loss of energy
  • feeling worthlessness or guilt
  • diminished ability to think or concentrate
  • recurrent thoughts of death, suicidal ideation or suicide attempt
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7
Q

major depressive disorder criteria

A
  • one or more MDE (must be at least 2 months symptom free between episodes)
  • cannot be a normal response to a significant loss
  • no history of mania/hypomania
  • women are more likely to be diagnosed then men
  • age of onset is high in early puberty to late twenties
  • 12mo% = 5-8%
  • life% = 10-20%
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8
Q

persistent depressive disorder (dysthymia)

A
  • individual experiences depressed mood for most of the day, nearly every day, for 2 years
  • incredibly chronic
  • no history of mania/hypomania
  • can not be symptoms free for more than 2 months to receive a diagnosis
  • can have MDE and still have PDD but if both are experienced simultaneously it is called double depression
  • women are twice as likely as men to be diagnosed
  • onset can start in childhood but is typically in early adulthood
  • 12mo% = 1%
  • life% = 2-5%
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9
Q

PDD symptoms

A
  • 2/6 symptoms needed to receive a diagnosis on top of a depressed mood:
    1. poor appetite or overeating
    2. insomnia or hypersomnia
    3. low energy or fatigue
    4. low self-esteem
    5. poor concentration or difficulty making decisions
    6. feelings of hopelessness
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10
Q

manic episode

A
  • a 1 week period of significantly elevated, expansive, or irritable mood, and significantly increased goal-directed activity or energy
  • one week period doesn’t apply if they have been hospitalized
  • commonly leads to hospitalization
  • involve hallucinations or delusions
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11
Q

mania criteria

A
  • additional symptoms; need 3/7 or 4/7 if the primary presentation is the irritable component:
    1. inflated self-esteem or grandiosity
    2. decreased need for sleep
    3. more talkative than usual or pressure to keep talking
    4. flight of ideas
    5. distractibility
    6. increased goal-directed activity or psychomotor agitation
    7. excessive involvement in risky behaviour
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12
Q

hypomania episode

A
  • identical to a manic episode, except for the following differences:
    1. typically shorter in duration, but must last at least 4 days
    2. not severe enough to cause hospitalization
    3. no significant impairment in daily functioning
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13
Q

bipolar 1 disorder

A
  • has had a manic episode
  • highly recurrent
  • people will bipolar 1 have more hypomanic episodes then bipolar 2
  • often begins with a major depressive episode
  • rates are equal between men and women
  • average age of onset for the first manic episode is 18
  • 12mo% = 0.6%
  • life% - 1%
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14
Q

bipolar 2 disorder

A
  • has has a hypomanic episode
  • has had a major depressive episode
  • no history of mania
  • people with bipolar 2 have more total mood episodes then people with bipolar one
  • equal in men and women
  • average age of onset is mid 20s
    12mo% = 0.6-0.8%
  • life% = 1%
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15
Q

cyclothymic disorder

A
  • numerous periods of hypomanic symptoms and numerous periods of depressive symptoms for 2 years
  • can not been without symptoms for more than 2 months
  • no history of mania, hypomania, or major depressive episode
  • half of people with cyclothymia will go on to develop a bipolar disorder
  • rates are equal between men and women
  • age of onset is typically adolescents or early adulthood
  • 12mo% = 0.3-0.5%
  • life% = 0.4-1%
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16
Q

mood disorder specifiers

A
  1. psychotic features (hallucination/delusions)
  2. melancholic features anhedonia, guilt)
  3. atypical features (overeating, oversleeping)
  4. peripartum (before/after giving birth)
  5. seasonal pattern
  6. rapid cycling ( 4 episodes in 12 mo)
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17
Q

etiology - biological approaches

A
  • genetics
  • behavioural activation system (BAS)
  • neurotransmitters
  • sleep
18
Q

genetics - etiology

A
  • tends to run in families, especially in twins
  • more recurrent in families
  • BPD tends to be stronger in families (passed on)
19
Q

BAS - etiology

A
  • sensitivity to rewards and motivation
  • positive emotions and hope
  • linked to dopamine
  • individuals can have a global reaction to activity happening in the environment (i.e. finding things rewarding or less sensitized); this occurs by getting a signals from the environment that something may be rewarding
  • designed to increase our contact with positively rewarding activities
20
Q

neurotransmitters - etiology

A
  • low levels of serotonin; allowed norepinephrine and dopamine to range wildly
  • serotonin helps achieve homeostasis
21
Q

sleep - etiology

A

sleep disruptions may lead to depression or mania

22
Q

mood disorders etiology - biological approaches

A
  1. tricyclic antidepressants
  2. monoamine oxidase inhibitors (MAOIs)
  3. selective serotonin reuptake inhibitors (SSRIs)
  4. mood stabilizers
23
Q

tricyclic antidepressants - mood disorder etiology

A
  • increase the amount of norepinephrine and serotonin in synaptic cleft so they get more reuptake for overtime effects
  • can be lethal if overdosed
24
Q

MAOIs - mood disorder etiology

A
  • increase the amount of norepinephrine, serotonin and dopamine
  • lots of dietary precautions and risks of severe reactions if mixed with other drugs
  • used less frequently than SSRIs
25
Q

SSRIs - mood disorder etiology

A

increase amount of serotonin

26
Q

mood stabilizers - mood disorder etiology

A
  • lithium is used to helped decrease the frequency and intensity of manic episodes
  • anticonvulsants (e.g. valproate)
  • prevents and treats manic episodes
27
Q

electroconvulsive therapy - mood disorders - biological approaches

A
  • unilateral vs. bilateral
  • shock is administered for less than 1 second, once every second day 6-10 times
  • last line of defense for people with treatment resistant depression or BPD
  • relapse tends to be common and people have to go on medication after ECT
  • couple circumstances where individuals may not try medication and referred directly to ECT
  • 50% respond
28
Q

psychological approaches etiology - interpersonal psychotherapy (IPT)

A
  • perceived stressful life event triggers onset of mood disorders
  • stressful relationships worsen the disorder or relationships can also be a result of these mood disorders
  • relationship problems significant factor for women
  • there is also an assumed genetic vulnerability that can bring the onset of these disorders too
  • for men depression is what damages a relationship
29
Q

psychological approaches treatment - interpersonal psychotherapy (IPT)

A
  • resolve problems in current relationships and form new important relationships
  • address role disputes and address the loss of relationships
  • correct social skills
  • tend to be more effective than support groups, brief psychodynamic therapy, placebo
  • takes about 15-20 weeks
  • 60-70% effective
30
Q

psychological approaches etiology - beck’s CBT

A
  • perceived stressful life event or automatic thoughts trigger onset of a mood disorder
  • recognizes the interplay of thoughts, feelings and behaviours
  • some people may be more prone to having more negative thoughts, leading to the onset of a mood disorder
  • lack a sense of mastery/achievement
  • numerous cognitive distortions
  • depression can exacerbate them
31
Q

numerous cognitive distortions - becks cbt etiology

A
  • overgeneralization; such as taking one instance of something and applying it to many instances
  • arbitrary inference; such as jumping to negative conclusions without evidence
  • catastrophizing; such as thinking the worst possible outcome will happen
32
Q

psychological approaches treatment - beck’s CBT

A
  • decrease the plausibility and strength of negative thoughts
  • behavioural activation
  • identify dysfunctional thinking styles
  • correct negative thoughts and substitute more realistic thoughts
  • challenge deeper held schemas
33
Q

behavioural activation as a psychological approaches treatment

A
  • for people who lack mastery, behavioural activation is a good place to start
    - helps people identify the smallest thing they can do, do that small thing and then you get a sense of mastery of having done something which can build momentum for doing greater things
    - focused on getting people active and developing a sense of mastery
  • this can be a standalone treatment
34
Q

identifying dysfunctional thinking styles as a psychological approaches treatment

A
  • may use tools such as a thought record
  • a thought record involves identifying what was happening before the thought, what the thought was, and what impact the thought had
35
Q

treatments for bipolar disorder - psychological approaches

A
  1. CBT
  2. family therapy
36
Q

CBT as a treatment for bipolar disorder

A
  • standard CBT for depression symptoms
  • decreasing desire for mania because of medication by showing the pros to it
  • increasing medication compliance
  • interpersonal and social rhythm therapy
37
Q

family therapy as a treatment for bipolar disorder

A
  • decreasing family tension because mania affects the family of the individuals with mood disorders
  • help family members understand the disorder
38
Q

interpersonal and social rhythm therapy for BPD

A
  • individuals all have social rhythms/schedules, and so people that experience mood disorders can be triggered with sudden changes in their social rhythms/schedules
    - interpersonal and social rhythm theory helps individuals stick to their schedule
39
Q

combined approaches

A
  • there is no definite advantage to combining both medication and psychotherapy
  • possible advantages include:
    1. medication may work faster (debate)
    2. IPT improves interpersonal relationships
    3. CBT changes negative thinking styles and prevents relapse
40
Q

relapse/recurrence

A
  • most individuals have multiple episodes
  • 50% of clients who stop their antidepressants relapse within 4 months
  • CBT generally leads to less relapse so combining it with meds may bring relapse rates lower
41
Q

suicide

A
  • women attempt suicide more often
  • men die due to suicide more often because they choose more lethal means
  • other high risks groups include minorities, elderly and teenagers
  • when working with clients, the best predictor of if they’re going to harm themselves is if they tell you
42
Q

risk factors of suicide

A
  • degree of current ideation and planning
  • previous attempts
  • hopelessness
  • depression or comorbid disorders
  • emerging from severe depression
  • low levels of serotonin
  • impulsivity & recent significant stressors
  • family history