Module 4 - Abdomen Flashcards

1
Q

imaging features of appendicitis

A

Inflamed: >6mm diameter, surrounded by inflamed hyperechoic fat, may have faecolith, may be hypervascular

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2
Q

Features of an amoebic abscess on CT

A

Amoebic abscess: hyperdense capsule, hypodense oedema beyond capsule

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3
Q

What is the characteristic finding of achalasia on a barium swallow?

A

bird-beak deformity

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4
Q

Outline the most common imaging techniques for assessing the oesophagus and stomach

A

XR

  • barium swallow
  • gastrograffin swallow

EUS: local staging of oesophageal cancer

CT: with oral contrast, useful for staging

FDG PET: for staging

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5
Q

Based on location, what are the likely diagnoses for oesophageal masses that are

  • submucosal/intramural
  • extrinsic
  • mucosal
A
  • Submucosal intramural: smooth filling defects
    ○ Smooth muscle tumor of oesophageal wall
  • Extrinsic lesions are often longer, obtuse angles not fixed to esophageal wall, epicentre outside oesophagus
    ○ Lymph node or cyst
  • Mucosal lesions show mucosal irregularity
    Adenocarcinoma or SCC
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6
Q

Which is more sensitive for intra-abdominal bleeding, CTA or Angiography?

A

CTA

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7
Q

Where is it best to embolise the splenic artery to preserve collateral perfusion?

A

proximal to the short gastric

helps avoid infarction

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8
Q

Describe 6 different interventional radiological procedures that address liver cancers, in particular HCC

A
  • TAE: transarterial embolisation, selective arterial embolisation to supply of tumour
  • Ethanol ablation: injection of ethanol to induce chemical necrosis
  • TACE: intra-arterial gel/substance or microspheres to deliver sustained release of drug while decreasing/obstructing the blood flow to the tumor(s)
  • TARE: Selective intra-arterial infusion of radioactive microspheres into the arterial supply of a tumor
  • Microwave ablation
  • Cryotherapy
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9
Q

What is the expected appearance of post-treatment HCC?

What about recurrent or residual HCC?

A

CT/MR/CEUS no arterial contrast enhancement
Recurrent or residual tumor is commonly located along the periphery of an ablation zone and manifests as focal nodular arterial phase hyperenhancement (APHE)

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10
Q

Diagnostic features on U/S of cholecystitis vs choledocholithiasis

A

CHOLECYSTITIS
Sonographic Murphy’s sign
Pericholecystic fulid
Gallbladder thickening >4mm

CHOLEDOCHOLITHIASIS
Dilated CBD >6mm infers stones

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11
Q

Features of interstitial vs necrotising pancreatitis on CT

A
  • Interstitial oedematous pancreatitis (80-90%)
    ○ Diffuse enlargement, stranding and lack of necrosis
  • Necrotising pancreatitis
    ○ Presence of tissue necrosis, commonly involving pancreatic parenchyma and peripancreatic tissue
    ○ Low signal intensity on CT
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12
Q

Imaging features of portal HTN/cirrhosis

A
- Steatosis
	○ low hounsfield units on CT
	○ MRI: out of phase image significant drop in signal indicating intra-voxular fat
- Small, nodular liver
- Varices
- Splenomegaly
- Ascites
- Dilated portal vein >13mm
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13
Q

Who is in the population for HCCC screening and what does screening encompass?

A
  • hep C
  • asian
  • cirrhosis

6 monthly U/S + AFP looking for nw hypoechoic lesions
If enlarging or >10mm for MRI, multiphase CT or contrast enhanced ultrasound
If definitive diagnosis not achieved with the above, for biopsy

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14
Q

CT and MRI appearance of FNH

A

CT

  • central hypodense scar
  • homogenous arterial hyperenhancement
  • isoenhancing on PV

MRI

  • T2 hyperintense scar
  • retains primovist
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15
Q

Imaging appearance of hepatic adenoma

A

arterial enhancement
early washout
fat containing

NO retention of primovist

classic population: young woman on COCP, no risk factors

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16
Q

CT and MRI appearance of liver haemangioma

A

CT

  • slow enhancement
  • peripheral nodular and gradual “centripetal” filling in of contrast

MRI
- lightbulb bright on T2

17
Q

CT and MRI appearance of HCC

A

CT

  • arterial enhancement
  • washout
  • capsular retraction

MRI

  • T2 fat-saturated hyperintense
  • diffusion restriction
18
Q

What are 2 common complications post liver transplant, time frame and their imaging characteristics ?

A

Hepatic arterial thrombosis
○ Post-op or years after
○ Increased likelihood if sepsis, graft ischaemia, rejection, hypercoagulable
○ Doppler: no detectable flow in hepatic artery with colour or spectral doppler

Hepatic Artery Stenosis
○ At anastomosis usually >3months post-transplant
○ Elevated peak systolic velocity >200cm/sec
○Parvus tardus waveform in intra-hepatic arteries

19
Q

What is the appearance of RCC on CT?

What are some mimics? (4)

A
  • Solid and Solid/cystic
  • Enhancement: on MRI and CT
    ○ Often hypervascular compared to background parenchyma, hypovascular on late phase
  1. Renal cysts with internal complexity
  2. Angiomyolipoma: contains fat, echogenic on ultrasound, low attenuation on CT, fat suppression on MRI
  3. TCC: involvement of renal pelvis
  4. Renal oncocytoma : benign tumour, indistinguishable from RCC
20
Q

What are some findings consistent with chronic renal impairment on imaging?

A

loss of corticomedullary differentiation, thinning cortex, increased echogenicity of renal parenchyma

21
Q

Differentials for collections post-renal transplant and characteristic ultrasound appearance

A
  • Haematoma: central black areas, fluid filled, thickened margins
  • Lymphocele: thin wall, thin septations
  • Urinoma: no septations, pure fluid
22
Q

Imaging (U/S) features (6) and management of acute renal graft rejection

A
  • diffusely enlarged graft with thickened cortex
  • loss of corticomedullary differentiation
  • prominent pyramids
  • urothelial thickening
  • Doppler: resistive index >0.8 but this is non-specific, if diastolic flow absent or reversed, more suggestive of acute rejection
  • Renal artery thrombosis in rare severe cases

Treatment: high dose steroids and antibiotics

23
Q

Imaging (U/S and nuc med) features of chronic graft rejection

A

U/S: enlargement of sinus fat and cortical thinning, dystrophic calcification, mild hydronephrosis
Doppler: Increased intrarenal RI
Nuclear med: reduced uptake of radiopharmaceuticlas but normal parenchymal transit or increased parenchymal retention

24
Q

Complications of solid organ embolisation

A
  • GB necrosis
  • liver ischemia
  • renal ischemia
  • splenic infarct
  • abscesses