Module 3 : Vascular Diseases Flashcards

(124 cards)

1
Q

what are 5 conditions that can alter blood flow in and out of organs

A
  • increase in vessel pressures
  • thrombosis/tumor invasion
  • atherosclerosis
  • congenital abnormalities
  • aneurysms
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2
Q

what is the normal aortic waveform pattern prox and distal

A
  • prox = moderate

- distal = high resistance

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3
Q

renal artery normal wave form pattern

A
  • low resistance
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4
Q

sma normal wave form patter prandial and postprandial

A
  • pre prandial = high resistance

- post prandial = low resistance

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5
Q

normal portal venous flow

A
  • hepatopedal

- monophasic flow with slight undulations

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6
Q

hepatic veins and ivc normal flow

A
  • phasic flow
  • influence by the cardiac and respiratory cycles
  • IVC often described as a saw tooth pattern
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7
Q

normal ao prox measuremnt

A

2-2.5cm

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8
Q

normal ao dist measurement

A

1.5cm

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9
Q

at what measurement is the ao considered aneurysmal

A

3cm

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10
Q

ivc size

A

5-29mm (usually less than 25mm)

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11
Q

when is the IVC considered dilated

A

> 3.7cm

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12
Q

what is the IVC size on initial inspiration

A

decreases in size

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13
Q

what is the IVC size on suspended respiration

A

increases in size

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14
Q

4 indications for arterial assessment

A
  • pulsatile abdominal mass
  • abdominal pain
  • abdominal buit
  • hemodynamic compromise of the lower limbs
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15
Q

definiton of arteriosclerosis

A

hardening of arteries

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16
Q

definition of atheroma

A

lipid deposits in arterial intima

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17
Q

atherosclerosis definiton

A
  • form of arteriosclerosis occurring in larger and medium arteries
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18
Q

plaque definiton

A

platelets forming cap over fatty deposits

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19
Q

characteristics of atheromatous disease (atherosclerosis)

A
  • lipid deposits on intimal lining of any artery

- alteration of lining provokes fibrosis and calcification

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20
Q

what 3 things is atherosclerosis associated with

A
  • hypertension
  • smoking
  • diabetes
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21
Q

does incidence of atherosclerosis incense or decrease with age

A
  • increase
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22
Q

does atherosclerosis effect females or males more

A

males

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23
Q

sonographic appearance of atherosclerosis

A
  • wall irregularities
  • tortuous vesels
  • calcifications
  • narrowed vessel lumen
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24
Q

do aneuysms effect only one artery

A
  • no can affect any artery
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25
6 types of aortic aneurysms
- abdominal aortic aneurysms - splanchnic artery aneurysms (mesenteric) - renal artery aneurysms - iliac artery aneurysms - mycotic aneurysms - inflammatory aneurysms
26
what is an abdominal aortic aneurysm
- a weakening of the aortic wall that leads to a focal dilatation
27
what is a true aneurysms
- involves all 3 layers of the artery wall
28
what is ectasia
- slight widening of the aorta up to 3cm
29
where does AAA usually occur
- occur mainly below the level of the renal vessels (infrarenal)
30
5 causes of AAA
- atherosclerosis MOST COMMON - syphilis - systemic infection - cystic medical necrosis - other diseases (marfans)
31
4 increased risk factors for AAA
- men > 60 - hypertenison - family history - hypercholesteremia
32
6 signs and symptoms of AAA
- usually asymptomatic - palpable mass - incidental finding on X-ray - lower back pain - abdominal pain - leg pain
33
two types of AAA
- fusiform | - saccular
34
fusiform AAA
- uniform tubular dilation
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saccular AAA
- sac like protrusion towards one side connected to the aortal - most often due to trauma or infection
36
sonographic appearance of a AAA
- dilation of the aorta 3cm or greater - aorta projects anterior and left (ivc and spine) - wall irregularities - thrombus on anterior and lateral walls -
37
what is thrombus
- clot attached to the vessel wall | - poorly attached thrombus can result in the release of emboli
38
what three things cause thrombus
- slowing of the blood stream - injury to vessel - alterations to the blood constituents
39
two associated findings with AAA
- iliac artery aneurysms | - popliteal aneurysms
40
iliac artery aneurysms
- bilateral - 2cm or greater - usually asymptomatic - older men - can rupture - causes hydronephrosis = compress ureters
41
popliteal aneurysms
- 25% of cases | - >1cm
42
protocol for measuring AAA
- place callipers outer to outer wall | - measure perpendicular to the vessel
43
6 things you want to document with AAA
- length, width and AP dimension - shape - location in aorta - does it involve renal or iliac arteries - describe the wall thickening - flow pattern
44
follow up treatment for 3-5cm aneurysm
- increasing in size 2-5mm/year + serial ultrasound exams - increasing in size 10mm/year + surgery aortic graft
45
follow up and treatment for 5-6cm aneurysm
- surgery with good prognosis | - at 6cm surgery is considered imperative
46
follow up and treatment for >7cm aneurysm
- 1 year survival rate 25% - 75% risk of fatal rupture - surgery = aortic graft
47
4 complications with AAA
- stenosis/occlusion MOST COMMON - rupture - dissection - thrombosis + with distal emboli
48
characteristics of AAA rupture
- surgical emergency - mortality rate >50% - operative mortality rate >40-60%
49
signs and symptoms of AAA rupture
- pain - shock - expanding abdominal mass
50
sonographic appearance of AAA rupture
- free fluid in abdomen - complex fluid collection - compression/displacement of surrounding structures
51
two types of AAA repair
- traditional graft | - endovascular aortic stent
52
traditional graft
- surgical bypass graft - open repair - made by flexible synthetic material (teflon or Dacron)
53
endovascular stent
- less invasive - stent inserted and ballooned out - more popular and less invasive quicker recovery
54
sonographic appearance of end-vascular stent
- ribbed appearance - straight distinct borders - increased echoes - post op = small layer of fluid normal - graft older than 8 years may bee difficult to see
55
6 complications of surgical repair
- fluid collection - hematomas seromas - abscesses - pseudoaneurysms - stenosis - endoleaks
56
splanchnic artery aneurysms
- celiac, SMA, IMA | - rare but life threatening
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order of arteries effected by splanchnic artery aneurysms
- splenic artery - hepatic artery - SMA
58
underlying causes of splanchnic artery aneurysms
- congenital - atherosclerosis - mycotic (fungal/bacterial) - inflammatory
59
sonographic appearance of splanchnic artery aneurysms
- anechoic or complex area continuous with the vessel - doppler signal - can be mistaken for abscess/cyst/lymph node
60
renal artery aneurysms
- extra renal location
61
common underlying cause of renal artery aneurysms
- athersclerosis | - polyarteritis
62
signs and symptoms of renal artery aneurysms
- palpable mass - hypertension (kidneys controls blood pressure) - hematuria
63
sonographic appearance of renal artery aneurysms
- anechoic mass connected to artery with doppler signal
64
mycotic aneurysms
- rare - due to bacterial or fungal infection - most are saccular with asymmetric wall thickening - difficult to diagnose with ultrasound alone
65
inflammatory aneurysms
- uncommon - dense fibrotic reaction around the aneurysm - involves retroperitoneum - high morbidity and mortality rate - adherent to bowel, ureter, iliac veins and IVC
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sonographic appearance of inflammatory aneurysms
- dilated aorta - wall thick - hypo echoic mantle around aorta
67
what is an aortic dissection
- tear of the intimal lignin got the aorta (and sometimes media) - false lumen created as blood pulsates through the vascular wall layers - begins at the thorax - presents typically with chest pain
68
what are ao dissections related to
- hypertension
69
3 types of aortic dissections
- involving aortic arch and movie down the aorta - marfans + ascending ao only - dissection of descending ao after origin of left subclavian + MOSR COMMON
70
sonographic appearance of ao dissection
- thin, linear, echogenicity within the arterial lumen + flap moves with cardiac cycle - doppler + demonstrates blood flow on both sides of the flap
71
what is a psuedoaneurysms
- blood escapes through a tear in the vessel wall and Is contained by surrounding tissues
72
causes of pseudo aneurysms
- failed graft - post angiogram - trauma
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sonographic appearance of pseudoaneurysms
- round/oval appearnce - blood circulates through in systole - pulsatile jet entry - variable waveform - always identify neck of mass
74
treatment of pseudoanerysm
- ultrasound guided compression - ultrasound guided thrombin injection + cause blood to clot off - monitored with color doppler imaging
75
what is an arteriovenous AV fistula
- abnormal communication between and artery and vein - blood moves from high to low pressure - most secondary to trauma, can be complication to aortic aneurysm
76
clinical presentation of AV fistula
- lower back and abdominal pain - hemodynamics altered - swelling of lower trunk and extremities - dilated veins
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sonographic appearance of AV fistula
- dilated IVC - pulsatile IVC - irregular waveform high velocites - reduced distal arterial flow
78
vascular stenosis
- can occur in varying degree | - caused by atherosclerotic plaque
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common 2D and doppler findings of vascular stenosis
- narrowed lumen - post stenotic dilation - increased velocities at stenosis - downstream changes
80
renal artery stenosis
- associated with incontrollable hypertension - decreased glomerular filtration rate - causes ischemic renal damage - commonly at the origin from the aorta within the first 2cm
81
4 things to look for when assessing the IVC
- changes with respiration - compressibility - echo free lumen - patency
82
what is a dilated IVC
- caliber increases below the point of obstruction
83
what is a dilated IVC associated with
- right ventricular failure
84
4 signs and symptoms of dilated IVC
- abdominal pain - ascites - hepatomegaly - lower extremity edema
85
characteristics of ivc tumors
- primary metastatic or extension | - present with leg edema, ascites and abdominal pain
86
primary IVC tumors
- uncommon - leiomyoma/leiomyosarcoma + tumor of smooth muscle
87
metastatic ivc tumors
- most commonly RCC
88
sonographic appearance of ivc tumors
- intraluminal ehcogenic foci - can be isoechoic or heterogeneous - caliber of ivc increased - loss of respiratory changes
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characteristics of IVC thrombus
- life threatening - MOST COMMON ABNORMALITY OF IVC - spreads from other veins
90
sonographic appearance of IVC thrombosis
- intraluminal filling defect expanding the vessel - echogenicity of the thrombus depends on its age - respiratory changes decreased or absent - color doppler is useful will show no flow in the region of the thrombus - IVC filters used for treatment
91
signs and symptoms of IVC thrombus
- leg edema - lower back pain - GI complaints - enlarged liver - ascites
92
renal vein thrombosis characteristics
- serious complication in a dehydrated state or septic infant - in adults occur as result of shock, renal tumor, renal transplants or trauma - associated with diabetes and high blood pressure
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3 signs an symptoms of renal vein thrombosis
- flank pain/mass - hematuria - proteinuria
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sonographic appearance of renal vein thrombosis
- enlarged kidney hypo echoic - dilated renal vein - filing defects of variable echogenicty - decreased or absent venous signal
95
congenital abnormalities of the IVC
- most occur at or below level of the renals
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what are the most common congenital abnormalities of IVC
- duplication and transposition
97
when does azygous/hemiazygous abnormality occur
- when hepatic segment of the IVC fails to develop
98
three types of congenital IVC abnormality
- transposition - duplication - azygous continuation
99
what is hepatic congestion
- passive edema of liver secondary to vascular congestion - related to heart failure - blood flow to the right side of heart is impaired
100
sonographic appearance of acute hepatic congestion
- enlarged liver - IVC dilated/ no respiratory changes - hepatic veins = highly pulsatile, flow reversal - portal vein flow = pulsatile
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sonographic appearance of chronic hepatic congestion
- shrunken liver - hepatic veins distended - LFT may be altered
102
what is portal hypertension
- due to increase portal venous pressure | - 2 types based on whether hepatic vein pressure are normal or elevated
103
2 types of portal hypertension
- presinusoidal + normal hepatic vein pressure - intraheptaic + abnormal hepatic vein pressure
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2 types of presinusoidal hypertension
- extrahepatic | - intrahepatic
105
extra hepatic presinusoidal hypertension
- PV and SV thrombosis - results in ascites, splenomegaly, varices - possible causes + malignancy, infection, inflammation, trauma, splenectomy, hypercoaguable states
106
intrahepatic presinusoidal hypertension
- caused by diseases affecting the portal zones + schistosomiasis (panc infection) + primary biliary cirrhosis
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intrahepatic portal hypertension
- most common in western world = cirrhosis - normal liver parenchyma is replaced which leads to increased resistance to PV flow and obstruction of HV outflow - results in ascites, splenomegaly, collaterals
108
sonographic appearance of both types of portal hypertension
``` - dilated PV in the early stages + upper limit of normal 13mm - collateral flow - patent umbilical vein/coronary vein - ascites - splenomegaly - monophasic >> biphasic>> hepatofugal ```
109
5 most common collateral routes
``` - gastroesophageal + fatal hemorrhage - paraumbilical + leads to caput medusa - splenorenal/gastrorenal - intestinal - hemorrhoidal ```
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what is portal vein thrombosis
- clot in portal venous system | - can result in cavernous transformation
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causes of portal vein thrombosis
- malignancy - infection/inflammation - trauma - splenectomy
112
why is color doppler important for portal vein thrombosis
- useful in distinguishing benign and malignant thrombi | - acute thrombus may be relatively anechoic
113
what is cavernous transformation
- formation of venous channels around a thrombodes vein portal vein
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what is Budd chairi syndrome
- rare | - obstruction of hepatic veins with possible IVC occlusion
115
causes of Budd chiari syndrome
- coagulation/congenital abnormalites - trauma - ORAL CONTRACEPTIVES - tumor invasion
116
sonographic appearance of Budd chiari
- stage dependant - enlarged liver (acute) - poor visualization of hepatic veins - thrombus in IVC - caudate lobe enlarged/hypoechoic - HV flow absent or reversed
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signs and symptoms of Budd Chiari
- RUQ - hepatomegaly - occasionally splenomegaly
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treatment of portal hypertension
- portosystemic shunts
119
portisystemic shunts characteristic
- shunts venous blood flow from congested venous system to a systemic vein - decompress PV system - can be placed surgically or percutaneously - TIPS
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TIPS shunt
- trans jugular intrahepatic portosystemic shunt - stent inserted percutaneously via jugular vein - inserted HV to PV - blood flows from PV system into hepatic venous system
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what is an infarction
- sudden interruption of blood supply that may lead to encores
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splenic infarct
- common cause of focal splenic lesions - caused by occlusion of the splenic artery - sudden onset of LUQ pain - underlying causes = sickle cell anemia
123
sonographic appearance of splenic infarct
- hypo echoic wedge shaped round area | - echogenicity changes over time (becomes hyperechoic)
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renal infarct
- can be segmental or diffuse - appearance is time dependant + early = hypo + late = hyper - renal function often remains normal