Module 3: Trauma Flashcards
What makes an event traumatic (Giller)?
- it overwhelms the individual’s ability to cope
- it is perceived as threatening
- it exceeds the ability to integrate the emotional experience into schemas
What are the typical reactions to trauma?
- physical,
- behavioral
- cognitive
- emotional
Types of traumatic events
- Acts of mass violence
- Natural disasters
- Acts of interpersonal violence
- Accidents
- Psychological abuse
- Deprivation of human rights
- Illness/Injury and its treatment
What is vicarious trauma?
- the individual does not directly experience the traumatic event, but feels traumatized in response to hearing about or seeing images (e.g., photos or videos) of the event.
- can also be from witnessing family/friends go through an event
What factors increase risk of developing vicarious trauma?
o organisational environment
o the therapist’s work experience
o interactions with clients
What is the strongest risk factor of vicarious trauma?
sense of isolsation both at work and home
How to protect psychologists from vicarious trauma?
CARE:
- Connection (managers, friends)
- Awareness (of symptoms)
- Resourcing (work-life balance)
- Effectiveness (training and supervision)
Assumptive World Model
- Janoff Bulman
- We live with 3 underpinning sets of assumptions:
1. benevolent world
2. meaningful world
3. worthiness of self. - these can become shattered and trauma leads to new world views
- Dual Representation Model
- Brewin
- information processing occurs at two levels from the outset of the traumatic event
- Verbally accessible memories (VAM): superficial level, can be accessed as desired, explicit memories.
- Situationally accessible memories (SAM): unconscious level, cannot be deliberately accessed by the victim, automatically triggered
Psychodynamic model - Freud
Traumatic neuroses
Failure of the pleasure principle
Wounding of the mind
Psychodynamic model - Horowitz
Drive towards completion
Active memory storage
Processing overload
Humanistic/existential models
- Considers how the client’s perceived world is changed by the trauma and sees that trauma divides a person’s life into: 3 Phenomenological ‘movements’: Before, during and after trauma
2 major concepts:
1. Emotional processing conflict model
(unfinished business to resolve)
Trauma-related emotion schemes model
(changes in the client’s view of the world, others and self)
Biological impact of trauma:
- attempting to keep homeostasis through allostasis
- neurotransmitters: catecholamines (norepinephrine, epinephrine and dopamine) and glucocorticoids (cortisol).
- emotional loop between the hypothalamus and the amygdala (allostatic load)
What percentage of Australians will experience PTSD in 12 months?
4.4% / 1 million
Life time prevalence of PTSD in Aus?
7.2%
What gender is more likely to experience PTSD?
Women 2 x more likely
What is the military rate of PTSD?
8.3% per 12 months
What are the neurological factors in PTSD?
- endocrine factors
- nuerochemistry
- brain circuitry
What are endocrine factors of PTSD?
- abnormal cortisol and thyroid regulation
- dysregulation of HPA axis
- hypocortisolism (low cortisol)
- HPT axis: elevated levels of tri-odo-thyronine (T3 - anxiety) and thyroxine (T4) in trauma victims
What are neurochemistry factors of PTSD?
Abnormal regulation of:
- catecholamine (dopamine, norepinephrine and epinephrine)
- serotonin
- amino acid (GABA and glutamate),
- peptide,
- opiod neurotransmitters.
What does norepinephrine do PTSD:
- central in the brain’s response to stress,
- norepinephrine and corticotropin-releasing hormone interact in a feed-forward circuit of the amygdala, hypothalamus and locus ceruleus to increase fear conditioning
What does Elevated catecholamines contribute to?
sustained hyperactivity within the autonomic sympathetic nervous system, thereby increasing physiological markers of stress
What does serotonin contribute to? PTSD:
impulsivity, hostility, aggression, depression and suicidality
What does Impaired GABA-benzodiazepine receptors contribute to? PTSD:
may lessen GABA’s ability to reduce the physiological response to stress.