Module 3 @Risk PP Family Flashcards

1
Q

What percentage of maternal deaths are preventable?

A

1/2

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2
Q

What race is the most susceptible to maternal death

A

African American women

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3
Q

What are the top 4 causes of maternal mortality? What’s the top?

A

1) sepsis
2) cardiac disease
3) hemorrhage
4) venous thromboembolism

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4
Q

Many women in this US almost die due to pregnancy related complications. What’s the top pregnancy complication?

A

Postpartum hemmorage

In the past 10 years there’s been an 183% increase in the # of women who had a blood transfusion around the time they gave birth.

The US is one of the only countries where maternal deaths and injuries have increased

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5
Q

Define postpartum hemmorage

A

1) blood loss of > 500ml after a vaginal birth
2) 1000ml after a C/s
3) any amount of bleeding that places the mother in hemodynamic jeopardy

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6
Q

Early postpartum hemmorage

A

First 24 hours after delivery

  • blood loss often underestimated
  • usual cause is uterine atony (uterus fails to contract)
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7
Q

Late or delayed postpartum hemorrhage

A

24 hours to 6 weeks postpartum

Usual case is retained placental tissue

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8
Q

Doctors usually underestimate the amount of blood loss. Why don’t we use hemoglobin and hematocrit levels

A

Change is dependent on the timing of the test and amount of fluid resuscitation given.

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9
Q

Do different women have different blood loss capacities?

A

Yes. A healthy women has a 30-50% blood volume increase during pregnancy and is more tolerant than a woman who has preexisting anemia, an underlying cardiac condition, or a volume contracted condition secondary to dehydration or preeclampsia

Hence the reason postpartum hemorrhage should be diagnosed with any amount of threatening blood loss.

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10
Q

Potential causes of postpartum hemorrhage:

A

4 Ts:

1) Tone
2) Tissue
3) Trauma
4) cloTTing

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11
Q

Describe Tone

A

Uterine atony
Lack of tone is the most common etiology of postpartum hemorrhage
Impaired uterine contractions after birth
Atony: abscence of tone, occurs as a result of that failure of contraction of the myometrial muscle fibers, which can lead to rapid or severe hemorrhage & hypovolemic shock

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12
Q

What causes uterine atony?

A

Overdistension of the uterus
Prolonged rapid labor
Placental abnormalities
bladder distention

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13
Q

What causes overdistension of the uterus?

A
  • multifetal gestation
  • fetal macrosomia
  • polyhydramnios
  • fetal abnormality (hydrocephalus)
  • retained blood
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14
Q

Prolonged rapid labor as it relates to Tone

A

Poor myometrial contraction can result from fatigue
-especially if artificially stimulates with Pitocin

Can be cause by medications that inhibit contractions: anesthetics, nitrates, non-steroidal, anti-inflammatory drugs (magnesium sulfate, beta-agonists, & nifedipine)

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15
Q

Why would the placenta be retained?

A

Placental Previa- Placental implantation at the lower uterine segment (doesn’t contract as well as upper uterus)

Gestation earlier than 24 weeks

Placenta accreta- when part of the placenta remains attached (placental adherence)

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16
Q

When with placental previa

A

Should be informed of the risk of severe postpartum hemorrhage, including the possible need for transfusion/ hysterectomy

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17
Q

What do you do if the woman keeps bleeding but uterine atony is not the cause?

A

Etiology may be from trauma (baby itself or use of forceps) or cloTTing issues

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18
Q

What is cervical laceration most commonly associated with?

A

Forcep delivery
The use of forceps or vacuum should never be attempted without the cervix being fully dilated.

Also happens when woman cannot resist “bearing down” before she’s at full dilation

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19
Q

Do C/S deliveries result in twice as much blood?

A

Yes

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20
Q

Are episiotomies common in L&D?

A

No
They > blood loss and the risk of anal sphincter tears

Should be avoided urgent delivery is necessary

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21
Q

How do hematomas present?

A

As a change in vital signs disproportionate to blood loss.

Patients with persistent volume loss or enlargening hemotoma may end up back in the OR and require incision and vacuation of the clot

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22
Q

Uterine rupture is most common

A

In women who have a history of significant uterine scarring

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23
Q

Women who have problems cloTTing probably

A

Have preexisting diseases such as hemophilia or Von Willebrand’s Disease

Could be cloTTing abnormalities caused by pregnancy though such as
HELLP, placenta abruption, DIC, or sepsis

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24
Q

CloTTing abnormalities brought on by pregnancy:

A

-HELLP: a serious complication of High blood pressure during pregnancy
Hemolysis, Elevated Liver Enzymes, Low
Platelet Count
-Placenta abruption- A serious pregnancy complication in which the placenta detaches from the womb (uterus).

  • DIC
  • Sepsis
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25
DIC
Disseminated intravascular coagulation in pregnancy. The DIC syndrome is the most common cause of an abnormal hemorrhage tendency during pregnancy and the puerperium and reflects systemic activation of the coagulation cascade by circulating thromboplastic material, with secondary activation of the fibrinolytic system.
26
Risk factors for PPH
1) overdistension 2) previous PPH 3) anesthesia (MgSO4) 4) uterine relaxants (nifidipine, terbutaline). 15) retained placenta 5) trauma 6)Grand multiparty (5+ pregnancies ) 7) history of anemia or hemorrhage 8) infection (esp. endometriosis) 9) uterine inversion or rupture 10) placenta previa or accreta 11) abnormal labor pattern (hyper/hypotonic) 12) prolonged or fast labor. 13) obesity 14) oxytocin admin. during labor
27
Placental Previa vs placental accreta
Placenta Previa- cervix entirely of partially blocked by placenta Placenta accureta- placenta so low it can be palpated by the examiners finger
28
Signs of impending hemmorage
Bleeding (>2pads/30-60min) Light headedness, nausea, visual disturbances Anxiety, pale/ashen color, clammy skin Hematoma-painful. Contain 3-500 mL blood > BP + R, BP same or lower MAP/Pulse ~ 60 to perfuse adequately AVG 80-100
29
Sequelae
a condition which is the consequence of a previous disease or injury. "the long-term sequelae of infection" Sequelae of hypovolemia would be PPH probably. It’s the secondary result
30
What might you see in a woman who is hemorrhaging
Pad soaked in 15 min of < Blood pressure will elevated just before it drops Vital sign changes are a late sign of hemorrhage. they would have lost a sig. amount of blood already Notice pain!! Pain+ urge to poo= PPH If blood is > prob need to go back to OR
31
What’s he best intervention for all postpartum women
Intervention Assess risk (assess placenta) Have IV site ready, administer Pit @3rd stage of labor Ensure bladder is empty. Massage fundus if PPH but don’t overmassage before (tire out) Avoid balloon tamponade & hysterectomy Frequent VS Know medications to administer
32
Cytotec/ Misoprostol/Sidotech
Used to induce labor in miscarriage It causes uteral contractions and for the cervix to soften
33
Methergine
Uterotonic and analgesic | It can treat severe bleeding from the uterus after childbirth
34
Hemabate
For abortion | Stops postpartum bleeding
35
The volume of a blood clot represents how much blood?
The actual blood amount is 2x the volume of the clot Symptoms of hypovolemia may not develop until a large volume of blood has already been lost
36
Puerperal sepsis
Any infection of the genital canal within 28 days after birth or abortion
37
Common pathogens after birth
Group B strep | E.Coli
38
Temperature that characterizes postpartum infection
Temp >100.4 on at least 2 of the first 10 days after birth, exclusive of first 24 hours OR Temp >101 within first 24 hours
39
Common infections after birth
``` Endometriosis Wound infections UTI Mastitis Respiratory Infection ``` Prevention is the best intervention: Hand hygiene, maternal perineal hygiene, Antibiotic administration, wound management Breast care More common after c/s
40
Preconception risk factors
``` History of previous DVT, UTI, mastitis, pneumonia Diabetes Alcoholism Drug abuse Immunosuppression Anemia Malnutrition ```
41
Intrapartum risk factor for infection
``` Prolonged labor Poor aseptic technique Birth trauma. C/s Prolonged rupture of membranes (water breaks) Chorioamnionitis. Catheterization Internal fetal/uteral ministering Multiple vaginal exams after rupture of membranes Epidural anesthesia PPH Manual removal of placental fragments Episiotomy or lacerations Hematomas ```
42
Endometriosis
``` Most cause of PP infection Usually starts at placental site Can involve entire endometrium & spread to Fallopian tubes + ovaries Lower abdominal tenderness, chills Foul smelling Lochia Tachycardia Subinvolution Reason why we give c/s mom antibiotics prophactically prior to surgery ```
43
Treatment for endometriosis
Administer broad spectrum antibiotic Provide analgesia Provide emotional support
44
Interventions for postpartum endometriosis
Monitoring Administering antibiotics If risk of hemorrhage is noted, order medications to treat subinvolution Encourage semi-Fowler’s position (90)
45
Risk factors for developing mastitis
Engorgment Cracked nipples Blocked ducts Pegged ducts Mild engorgement means that milk is not coming due to edema and milk stasis
46
What’s he best preventative measure for engorgment
A good latch and baby drinking | Education
47
Interventions for engorgment
Warmth/ warm showers Use of ice or cabbage leaves after breastfeeding Anti-inflammatory meds and breast massage Cracked nipples give bacteria an “in”
48
Lanolin
Treats sore nipples
49
What bacteria causes mastasis?
S. aureus Engorgment and stasis of milk preceded mastasis Typically happens in the lactating breast at the 2nd or 2rd week after pregnancy Treatment: antibiotics, analgesics, & antipyretic
50
What factors put the mom at risk for postpartum mastasis
Underwire bra Missed feedings Improper breast hygiene Poor infant suck NOT breast pumping
51
Best prevention for UTI
``` Wipe front to back Use peri bottles Sitz bath > fluid intake Wash their hands ```
52
Symptoms of UTI
``` Burning and pain while urinating Lower abdominal pain Low grade fever Flank pain Protuenuria, hematuria, bacteriuria, nitrates, and WBC ```
53
Treatment for a UTI
``` assess vitals Q4 Encourage fluid intake monied I&O Administer antibiotics as ordered Administer antipyretics, antipasmodics, and antiemetic Encourage rest ```
54
3 thromboembolic conditions concerned with the postpartum period
1) Superficial venous thrombosis - involves superficial venous thrombosis 2) Deep vein thrombosis Involves veins in the calves, thigh, or pelvis 3) pulmonary embolism Complication of DVT Occurs when part of a blood clot dislodges and is carried to pulmonary artery where it occludes the vessel & obstructs blood flow to lungs
55
Why has incidence of thrombosis declined in the past 20 years?
Early ambulation after childbirth Major causes of thrombosis are venostasis & hypercoagulation (to prevent hemorrhage PP women have high fibrinogen)
56
Risk factors for thrombosis
``` > fibrinogen levels during pregnancy (normal) History of thromboembolic disease or varicosities > parity Obesity. Smoking Advanced maternal age Immobility C/s Tissue trauma Blood type other than O Dehydration ```
57
What are the nurses psychological responsibilities for her patient family?
Assess and screen for women who are at risk for either devoting or are currently suffering from a mood disorder We’re focusing less on baby blues and more on PP depression and PP psychosis
58
Postpartum depression
10-20% of all postpartum patients Symptoms generally noted within the 1st 3 months but can occur up to a year after delivery
59
Treatment for PP depression
``` Primary prevention Psychotherapy Medication Electroconvulsive therapy Combination therapy ```
60
Postpartum Psychosis
Days to weeks after delivery EMERGENCY Hallucinations, delusions, agitations, inability to sleep, bizarre or irrational behavior History of bipolar disorder or history of postpartum psychosis Risk of suicude or homicide is high. Antipsychotics & mood stabilizers like Li+