Module 3 @Risk PP Family Flashcards

1
Q

What percentage of maternal deaths are preventable?

A

1/2

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2
Q

What race is the most susceptible to maternal death

A

African American women

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3
Q

What are the top 4 causes of maternal mortality? What’s the top?

A

1) sepsis
2) cardiac disease
3) hemorrhage
4) venous thromboembolism

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4
Q

Many women in this US almost die due to pregnancy related complications. What’s the top pregnancy complication?

A

Postpartum hemmorage

In the past 10 years there’s been an 183% increase in the # of women who had a blood transfusion around the time they gave birth.

The US is one of the only countries where maternal deaths and injuries have increased

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5
Q

Define postpartum hemmorage

A

1) blood loss of > 500ml after a vaginal birth
2) 1000ml after a C/s
3) any amount of bleeding that places the mother in hemodynamic jeopardy

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6
Q

Early postpartum hemmorage

A

First 24 hours after delivery

  • blood loss often underestimated
  • usual cause is uterine atony (uterus fails to contract)
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7
Q

Late or delayed postpartum hemorrhage

A

24 hours to 6 weeks postpartum

Usual case is retained placental tissue

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8
Q

Doctors usually underestimate the amount of blood loss. Why don’t we use hemoglobin and hematocrit levels

A

Change is dependent on the timing of the test and amount of fluid resuscitation given.

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9
Q

Do different women have different blood loss capacities?

A

Yes. A healthy women has a 30-50% blood volume increase during pregnancy and is more tolerant than a woman who has preexisting anemia, an underlying cardiac condition, or a volume contracted condition secondary to dehydration or preeclampsia

Hence the reason postpartum hemorrhage should be diagnosed with any amount of threatening blood loss.

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10
Q

Potential causes of postpartum hemorrhage:

A

4 Ts:

1) Tone
2) Tissue
3) Trauma
4) cloTTing

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11
Q

Describe Tone

A

Uterine atony
Lack of tone is the most common etiology of postpartum hemorrhage
Impaired uterine contractions after birth
Atony: abscence of tone, occurs as a result of that failure of contraction of the myometrial muscle fibers, which can lead to rapid or severe hemorrhage & hypovolemic shock

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12
Q

What causes uterine atony?

A

Overdistension of the uterus
Prolonged rapid labor
Placental abnormalities
bladder distention

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13
Q

What causes overdistension of the uterus?

A
  • multifetal gestation
  • fetal macrosomia
  • polyhydramnios
  • fetal abnormality (hydrocephalus)
  • retained blood
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14
Q

Prolonged rapid labor as it relates to Tone

A

Poor myometrial contraction can result from fatigue
-especially if artificially stimulates with Pitocin

Can be cause by medications that inhibit contractions: anesthetics, nitrates, non-steroidal, anti-inflammatory drugs (magnesium sulfate, beta-agonists, & nifedipine)

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15
Q

Why would the placenta be retained?

A

Placental Previa- Placental implantation at the lower uterine segment (doesn’t contract as well as upper uterus)

Gestation earlier than 24 weeks

Placenta accreta- when part of the placenta remains attached (placental adherence)

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16
Q

When with placental previa

A

Should be informed of the risk of severe postpartum hemorrhage, including the possible need for transfusion/ hysterectomy

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17
Q

What do you do if the woman keeps bleeding but uterine atony is not the cause?

A

Etiology may be from trauma (baby itself or use of forceps) or cloTTing issues

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18
Q

What is cervical laceration most commonly associated with?

A

Forcep delivery
The use of forceps or vacuum should never be attempted without the cervix being fully dilated.

Also happens when woman cannot resist “bearing down” before she’s at full dilation

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19
Q

Do C/S deliveries result in twice as much blood?

A

Yes

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20
Q

Are episiotomies common in L&D?

A

No
They > blood loss and the risk of anal sphincter tears

Should be avoided urgent delivery is necessary

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21
Q

How do hematomas present?

A

As a change in vital signs disproportionate to blood loss.

Patients with persistent volume loss or enlargening hemotoma may end up back in the OR and require incision and vacuation of the clot

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22
Q

Uterine rupture is most common

A

In women who have a history of significant uterine scarring

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23
Q

Women who have problems cloTTing probably

A

Have preexisting diseases such as hemophilia or Von Willebrand’s Disease

Could be cloTTing abnormalities caused by pregnancy though such as
HELLP, placenta abruption, DIC, or sepsis

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24
Q

CloTTing abnormalities brought on by pregnancy:

A

-HELLP: a serious complication of High blood pressure during pregnancy
Hemolysis, Elevated Liver Enzymes, Low
Platelet Count
-Placenta abruption- A serious pregnancy complication in which the placenta detaches from the womb (uterus).

  • DIC
  • Sepsis
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25
Q

DIC

A

Disseminated intravascular coagulation in pregnancy. The DIC syndrome is the most common cause of an abnormal hemorrhage tendency during pregnancy and the puerperium and reflects systemic activation of the coagulation cascade by circulating thromboplastic material, with secondary activation of the fibrinolytic system.

26
Q

Risk factors for PPH

A

1) overdistension 2) previous PPH
3) anesthesia (MgSO4)
4) uterine relaxants (nifidipine, terbutaline). 15) retained placenta
5) trauma 6)Grand multiparty (5+ pregnancies )
7) history of anemia or hemorrhage
8) infection (esp. endometriosis)
9) uterine inversion or rupture
10) placenta previa or accreta
11) abnormal labor pattern (hyper/hypotonic)
12) prolonged or fast labor. 13) obesity
14) oxytocin admin. during labor

27
Q

Placental Previa vs placental accreta

A

Placenta Previa- cervix entirely of partially blocked by placenta

Placenta accureta- placenta so low it can be palpated by the examiners finger

28
Q

Signs of impending hemmorage

A

Bleeding (>2pads/30-60min)

Light headedness, nausea, visual disturbances

Anxiety, pale/ashen color, clammy skin

Hematoma-painful. Contain 3-500 mL blood

> BP + R, BP same or lower
MAP/Pulse ~ 60 to perfuse adequately
AVG 80-100

29
Q

Sequelae

A

a condition which is the consequence of a previous disease or injury.
“the long-term sequelae of infection”

Sequelae of hypovolemia would be PPH probably. It’s the secondary result

30
Q

What might you see in a woman who is hemorrhaging

A

Pad soaked in 15 min of <

Blood pressure will elevated just before it drops

Vital sign changes are a late sign of hemorrhage. they would have lost a sig. amount of blood already

Notice pain!! Pain+ urge to poo= PPH

If blood is > prob need to go back to OR

31
Q

What’s he best intervention for all postpartum women

A

Intervention
Assess risk (assess placenta)
Have IV site ready, administer Pit @3rd stage of labor

Ensure bladder is empty. Massage fundus if PPH but don’t overmassage before (tire out)

Avoid balloon tamponade & hysterectomy
Frequent VS

Know medications to administer

32
Q

Cytotec/ Misoprostol/Sidotech

A

Used to induce labor in miscarriage

It causes uteral contractions and for the cervix to soften

33
Q

Methergine

A

Uterotonic and analgesic

It can treat severe bleeding from the uterus after childbirth

34
Q

Hemabate

A

For abortion

Stops postpartum bleeding

35
Q

The volume of a blood clot represents how much blood?

A

The actual blood amount is 2x the volume of the clot

Symptoms of hypovolemia may not develop until a large volume of blood has already been lost

36
Q

Puerperal sepsis

A

Any infection of the genital canal within 28 days after birth or abortion

37
Q

Common pathogens after birth

A

Group B strep

E.Coli

38
Q

Temperature that characterizes postpartum infection

A

Temp >100.4 on at least 2 of the first 10 days after birth, exclusive of first 24 hours

OR

Temp >101 within first 24 hours

39
Q

Common infections after birth

A
Endometriosis 
Wound infections
UTI
Mastitis
Respiratory Infection 

Prevention is the best intervention:
Hand hygiene, maternal perineal hygiene,
Antibiotic administration, wound management
Breast care

More common after c/s

40
Q

Preconception risk factors

A
History of previous DVT, UTI, mastitis, pneumonia
Diabetes
Alcoholism
Drug abuse
Immunosuppression
Anemia
Malnutrition
41
Q

Intrapartum risk factor for infection

A
Prolonged labor
Poor aseptic technique
Birth trauma.       C/s
Prolonged rupture of membranes (water breaks)
Chorioamnionitis.      Catheterization
Internal fetal/uteral ministering
Multiple vaginal exams after rupture of membranes
Epidural anesthesia        PPH
Manual removal of placental fragments
Episiotomy or lacerations 
Hematomas
42
Q

Endometriosis

A
Most cause of PP infection
Usually starts at placental site
Can involve entire endometrium &amp; spread to Fallopian tubes + ovaries
Lower abdominal tenderness, chills
Foul smelling Lochia 
Tachycardia 
Subinvolution 
Reason why we give c/s mom antibiotics prophactically prior to surgery
43
Q

Treatment for endometriosis

A

Administer broad spectrum antibiotic
Provide analgesia
Provide emotional support

44
Q

Interventions for postpartum endometriosis

A

Monitoring
Administering antibiotics
If risk of hemorrhage is noted, order medications to treat subinvolution

Encourage semi-Fowler’s position (90)

45
Q

Risk factors for developing mastitis

A

Engorgment
Cracked nipples
Blocked ducts
Pegged ducts

Mild engorgement means that milk is not coming due to edema and milk stasis

46
Q

What’s he best preventative measure for engorgment

A

A good latch and baby drinking

Education

47
Q

Interventions for engorgment

A

Warmth/ warm showers
Use of ice or cabbage leaves after breastfeeding
Anti-inflammatory meds and breast massage

Cracked nipples give bacteria an “in”

48
Q

Lanolin

A

Treats sore nipples

49
Q

What bacteria causes mastasis?

A

S. aureus

Engorgment and stasis of milk preceded mastasis

Typically happens in the lactating breast at the 2nd or 2rd week after pregnancy

Treatment: antibiotics, analgesics, & antipyretic

50
Q

What factors put the mom at risk for postpartum mastasis

A

Underwire bra
Missed feedings
Improper breast hygiene
Poor infant suck

NOT breast pumping

51
Q

Best prevention for UTI

A
Wipe front to back
Use peri bottles
Sitz bath
> fluid intake
Wash their hands
52
Q

Symptoms of UTI

A
Burning and pain while urinating
Lower abdominal pain
Low grade fever
Flank pain
Protuenuria, hematuria, bacteriuria, nitrates, and WBC
53
Q

Treatment for a UTI

A
assess vitals Q4
Encourage fluid intake monied I&amp;O
Administer antibiotics as ordered
Administer antipyretics, antipasmodics, and antiemetic
Encourage rest
54
Q

3 thromboembolic conditions concerned with the postpartum period

A

1) Superficial venous thrombosis
- involves superficial venous thrombosis

2) Deep vein thrombosis
Involves veins in the calves, thigh, or pelvis

3) pulmonary embolism
Complication of DVT
Occurs when part of a blood clot dislodges and is carried to pulmonary artery where it occludes the vessel & obstructs blood flow to lungs

55
Q

Why has incidence of thrombosis declined in the past 20 years?

A

Early ambulation after childbirth

Major causes of thrombosis are venostasis & hypercoagulation (to prevent hemorrhage PP women have high fibrinogen)

56
Q

Risk factors for thrombosis

A
> fibrinogen levels during pregnancy (normal)
History of thromboembolic disease or varicosities
> parity
Obesity.      Smoking
Advanced maternal age
Immobility
C/s
Tissue trauma
Blood type other than O
Dehydration
57
Q

What are the nurses psychological responsibilities for her patient family?

A

Assess and screen for women who are at risk for either devoting or are currently suffering from a mood disorder

We’re focusing less on baby blues and more on PP depression and PP psychosis

58
Q

Postpartum depression

A

10-20% of all postpartum patients

Symptoms generally noted within the 1st 3 months but can occur up to a year after delivery

59
Q

Treatment for PP depression

A
Primary prevention 
Psychotherapy
Medication
Electroconvulsive therapy 
Combination therapy
60
Q

Postpartum Psychosis

A

Days to weeks after delivery
EMERGENCY
Hallucinations, delusions, agitations, inability to sleep, bizarre or irrational behavior
History of bipolar disorder or history of postpartum psychosis

Risk of suicude or homicide is high.
Antipsychotics & mood stabilizers like Li+