Module 3 - Cardiovascular and Medical Emergencies Flashcards
Hypoxic Hypoxia
aka Altitude Hypoxia Is a deficiency in the alveolar oxygen exchange, which can be caused by low barometric pressure.
Hypemic hypoxia
Is a reduction in the oxygen-carrying capacity of the blood.
Stagnant hypoxia
Occurs when conditions exist that result in reduced total cardiac output, pooling of the blood within certain regions of the body, a decreased blood flow to the tissues, or restriction of blood flow.
Histotoxic hypoxia
aka tissue poisoning Occurs when metabolic disorders or poisoning of the cytochromic oxidase enzyme results in a cell’s inability to use molecular oxygen.
Which patient is not affected with altitude temperature changes?
A. Cardiac patient
B. Burn patient
C. Head injured patient
D. Spinal cord injured patient
A: Cardiac patients are usually not affected with altitude temperature changes.
Your patient presents with epigastric pain, nausea, and vomiting for the last hour. He describes his chest pain as “heavy in nature.” What does the following 12-lead ECG show? [inferior mi ecg]
C: Inferior wall MI presents with ST elevation in leads II, III, and aVF. Reciprocal changes are present in leads I, aVL, and V1-V4.
Inferior MI - II, III, aVF would show reciprocal changes in
reciprocal changes seen in I, aVL, V1-V4
Anterior-septal wall MI identified in V1-V4, would show reciprocal changes in
reciprocal changes seen in II, III, aVF, aVL
Lateral wall MI identified in I, aVL, V5, V6 would show reciprocal changes in
reciprocal changes seen in II, III, aVF
Posterior wall MI identified in V6 would show reciprocal changes in
reciprocal changes seen in V1-V4
CPK as a marker
onset 4-6 hours, peaks 24 hours
CPK-MB as a marker
onset 4-6 hours, peaks 12-20 hours
LDH as a marker
onset 8-12 hours, peaks 2-4 days
Troponin I normal range (most sensitive test)
0-0.1 ng/mL; onset 4-6 hours, peaks 12-24 hours, returns to normal in 4-7 days
Troponin T normal range (most sensitive test)
0-0.2 ng/mL; onset 3-4 hours, peaks 10-24 hours, returns to normal in 10-14 days
Your patient is experiencing left ventricular diastolic failure. Therapy should be focused on
A. Augmentation of left ventricular clearing
B. Decreasing afterload
C. Decreasing preload
D. Diuretics and relief of anxiety
D. Diuretics and relief of anxiety. Relieving ischemia, treating atherosclerosis, and correcting renal artery stenosis are most helpful. In addition, efforts to keep patients dry, maintain a slow sinus rhythm, and control blood pressure provide a basic approach to diastolic dysfunction. When
Your patient is exhibiting ST elevation in leads II, III, and AVF. ST depression is noted in V1-V3. Which of the following may prove hazardous?
A. Isotonic fluid bolus
B. Heparin
C. GII/BIIIa inhibitors
D. Nitroglycerin
D. Nitroglycerin
Explain consideration of Nitro administration with a RVI
Hypotension, with distended neck veins and clear lungs occurs in patients with (RVI). This is because of occlusion of the proximal right coronary artery, the blood supply to the right ventricle. RVI patients are volume dependent because of inadequate preload to the left ventricle. The basis of therapy for a RVI is large volumes of intravenous fluids prior to administering medications that may decrease preload and inotropic support. RVI accompanies inferior-posterior wall MIs in 30-50% of patients.
Clot busting and prevention
ASA G2BIIIA inhibitors—Reopro Integrelin Aggrastat Heparin Thrombolytics—Retavase tPA Streptokinase
Treatments for Heart rate and myocardial O2 demand reduction
Beta-blockers Calcium-channel blockers
Thrombolytics - Relative & Absolute CI’s, Complications
Relative contraindications: HTN, recent trauma, pregnancy Absolute contraindications: Active internal bleeding, Suspected aortic dissection, Known intracranial neoplasm, Previous hemorrhagic stroke, Stroke within the last year. Complications: bleeding, intracranial hemorrhage, dysrhythmias, cardiac tamponade, pulmonary edema
Preload reduction management for AMI/Unstable angina
Nitrates (5-200 μg/min) Morphine (2-4 mg every 5-15 minutes)
Electrical alternans may be caused by
A. Pulmonary embolus
B. Pericardial tamponade/effusion
C. Tension pneumothorax
D. Diaphragmatic rupture
B. Pericardial tamponade/effusion
Antidote for Coumadin overdose is
A. Protamine sulfate
B. Glucagon
C. Vitamin K, FFP
D. Physostigmine
C. Vitamin K, FFP
Anterior infarct – Coronary artery involved?
LAD
Inferior infarct – Coronary artery involved?
RCA
Lateral infarct – Coronary artery involved?
LCX
Septal infarct – Coronary artery involved?
LAD
Posterior infarct – Coronary artery involved?
LCX or RCA
Wellen’s syndrome is ____ and it presents on ECG by __.
Associated with critical stenosis of the proximal LAD and impending infarct ECG: V2-V3 segment turns down into a negative T at a 60-90-degree angle
A Transmural MI vs Subendocardial (nontransmural)
Extends through the full thickness of the myocardium and includes the endocardium and epicardium Vs. Necrosis is limited to the subendocardial surface
Dressler’s syndrome
Is a secondary form of pericarditis that occurs in the setting of injury to the heart or the pericardium, the outer lining of the heart. It is also known as post MI syndrome and postcardiotomy pericarditis. Largely a self-limiting disease that very rarely leads to pericardial tamponade. Consisting of a persistent low-grade fever, chest pain (usually pleuritic in nature), a pericardial friction rub, and /or a pericardial effusion. The symptoms tend to occur within 2-5 days post MI but can be delayed for a few weeks or even months after infarction. It tends to subside in a few days.
Pericardial effusion
Is the development of pericardial fluid as response to injury, acute pericarditis. Electrical alternans can be present on the ECG.
Cardiac tamponade
Consists of cardiac output being compromised by the fluid around the heart. Beck’s triad can be indicative for the presence of tamponade, which includes muffled heart tones, hypotension, and jugular vein distension (JVD).
Beck’s Triad is
can be indicative for the presence of tamponade which includes: muffled heart tones hypotension jugular vein distension (JVD).
Management of cardiac tamponade
ABCs intravenous fluids pericardiocentesis
S1 heart sound: Description - Location to hear - May indicate -
S1 heart sound: Description - A normal S1 sound is low pitched and of longer duration than S2. Location to hear - Best heard over the mitral area at the apex which is approximately at the fifth intercostal space, midclavicular left side of the chest. May indicate - Corresponds to the closure of the mitral and tricuspid valves (atrioventricular—AV valves) at the beginning of ventricular contraction or systole.
S2 heart sound: Description - Location to hear - May indicate -
S2 heart sound: Description - A normal S2 sound is higher pitched and of shorter duration than S1 Location to hear - The second heart tone, or S2, “Dub” is best heard over the aortic area which is located at the second intercostal space at the base of the heart, right sternal border. A normal S2 sound is higher pitched and of shorter duration than S1. May indicate - Corresponds to closure of the aortic and pulmonic valves (semilunar valves) at the end of ventricular systole.
S3 heart sound: Description - Location to hear - May indicate -
S3 heart sound: Description - Abnormal heart sound that may sound like the word “Ken-tu-cky” in three fairly evenly spaced sounds. Location to hear - The S3 sound is heard immediately after S2 and is normal in children and young adolescents but usually disappears after the age of thirty. May indicate - When heard in adults, an S3 is called a “gallop” and indicates left ventricular failure.
S4 heart sound: Description - Location to hear - May indicate -
S4 heart sound: Description - Abnormal heart sound that may sound like the word “Tennessee” where the three syllables come quick followed by a pause. Location to hear - The S4 sound is heard immediately before the S1 and may be present in infants and children. May indicate - The S4 is produced with decreased compliance of the ventricle and may indicate MI or shock.
Protamine sulfate is used for
heparin overdose
Your patient has a chief complaint of dyspnea and weakness with the following vitals: BP 72/64, HR 112, RR 28, SpO2 88%, temp. 99.1°F. He is on 6 L/minute of oxygen via NC. The ECG shows ST with frequent PVCs. Physical exam reveals profound vesicular rales and bronchial wheezing. Your most likely diagnosis is
A. CHF
B. ARDS
C. Asthma
D. Cardiogenic shock
D. Cardiogenic shock
Treatment for compensated bradydysrhythmias with a pulse and heart rate <60 beats per minute.
Compensated: SBP > 90-100 mmHg without S/S of shock Good LOC Treat the underlying cause, place combo pads as an anticipatory measure.
Treatment for uncompensated bradydysrhythmias with a pulse and heart rate <60 beats per minute.
Decompensated: SBP < 90-100 mmHg, ALOC may be present and other S/S of shock present F—Fluids A—Atropine (not rec. in 2nd ˚ AVB Type II or CHB) E—External pacing D—Dopamine drip E—Epinephrine drip
Treatment for compensated tachydysrhythmias with a pulse and heart rate >160 beats per minute.
Compensated: SBP > 90-100 mmHg without S/S of shock and good LOC Supportive care Vagal maneuvers—narrow complex tachycardia (SVT) Medications Application of combo pad as an anticipatory measure
Treatment for uncompensated tachydysrhythmias with a pulse and heart rate >160 beats per minute.
Decompensated: SBP < 90-100 mmHg with S/S of shock; ALOC may be present. Consider sedation Synchronized cardioversion beginning at 100 joules Medications
Tx for Cardiogenic shock (hypotension present with S/S of pulmonary edema—left ventricular failure)
Inefficient pumping of the heart with hypotension Dobutrex, dopamine, and Inocor to improve cardiac output Vasodilators such as Nipride, NTG, and Levophed IABP and LVAD therapy
Right-side ventricular heart failure:
Back up of fluid to the right side of the heart and body Increased jugular venous distention (JVD) Sacral edema Pedal edema
Left-side ventricular heart failure:
Back of fluid to the left side of the heart and lungs Rales
Treatment of cardiac tamponade includes all of the following, except
A. Force fluids
B. Pericardiocentesis
C. Rapid transport
D. Needle thoracostomy
D. Needle thoracostomy Intravenous fluids, pericardiocentesis, and rapid transport are all indicated for management of a patient presenting with cardiac tamponade. Needle thoracostomy is indicated for a patient presenting with a tension pneumothorax.
You are transporting a forty-five-year-old man with acute respiratory distress syndrome (ARDS) and MODS secondary to probable organ rejection after a heart transplant. During transport the patient becomes bradycardic with heart rate in the 30s with hypotension. Which of the following therapies will likely prove fruitless?
A. 250-500 mL saline bolus
B. Dopamine 5-20 μg/kg/min
C. Transcutaneous pacing
D. Atropine 0.5-1 mg IV push
D: The administration of Atropine will not work with patients who have had a heart transplant because of denervation of the vagus nerve. Atropine works by blocking the vagus nerves, thereby increasing heart rate. Symptomatic bradycardia, second degree Type II block, high-grade AVB and CHB require placement of a pacemaker. Complications of pacing can include oversensing and failure to sense; failure to capture, myocardial penetration/perforation, and cardiac tamponade.
Your patient presents with following parameters: CVP 20, CI 1.1, PA S/D 8/4, wedge 3, and SVR 1,800. What is your diagnosis?
A. Hypovolemic shock
B. RVMI
C. CHF/LVF
D. Sepsis
B: RVI because of the low PAWP. Review
You are transporting a fifty-year-old man from ICU to another facility for further evaluation. The patient has been diagnosed with AMI. He has been complaining of increasing CP, SOB, and dramatic weight loss. He appears very nervous, and you note tremors. His ECG shows AF at 148. The patient may be experiencing
A. Addison’s disease
B. Thyrotoxicosis (grave’s dieases)
C. Myxedema coma
D. Cushing’s syndrome
B: Thyrotoxicosis, also known as Grave’s disease, thyroid storm and hyperthyroidism. Avoid Aspirin because it increases T3, T4 levels and can worsen condition.
Grave’s disease
Hyperthyroidism Exophthalmos— “Marty Feldman” or “Betty Davis” protruding eyeballs Atrial fibrillation is common Anxiety, tremors Weight loss Treatment: correct electrolytes and glucosteroids
Myxedema coma
Hypothyroidism Infection common cause; coarse hair, deep voice, thinning or loss of the outer third of the eyebrows (Queen Ann’s sign) Women > 60 years; occurs in the winter months Fatigue Weight gain Treatment: levothyroxine
Addison’s disease
Acute renal insufficiency Hypotension is common: caution with etomidate administration Fatigue Weight loss Treatment: supportive care
Cushing’s disease
Hyperaldosteronism Hypertension Women have facial hair, moon-face, buffalo hump Upper body obesity, thin arms and legs Treatment: decrease or initiate steroids
The formula to calculate MAP is:
A. 2/3 DBP × SBP
B. 2 × DBP + SBP divided by 3
C. 2 × SBP + DBP
D. 2 + DBP × SBP divided by 3
B: (2 × DBP) + SBP divided by 3 = MAP. Normal MAP is 80-100 mmHg.
Normal coronary perfusion pressure (CPP) is
A. 50-60 mmHg
B. 70-90 mmHg
C. 80-100 mmHg
D. <50 mmHg
A: Normal coronary perfusion pressure is 50-60 mmHg. It can be calculated by using the following formula: DBP − PWCP. Remember that your HEAD is higher than your HEART (50-60 mmHg).
Cerebral perfusion pressure can be calculated by using the following formula: MAP − ICP. Normal range for cerebral perfusion pressure is 70-90 mmHg. Remember that your HEAD is higher than your HEART.
MAP − ICP. Normal range for cerebral perfusion pressure is 70-90 mmHg. Remember that your HEAD is higher than your HEART (50-60 mmHg).
When performing a pericardiocentesis, the insertion site is
A. Below the subxyphoid process
B. Just right of the subxyphoid process
C. Just left of the subxyphoid process
D. Above the subxyphoid process
C: The emergent treatment of choice is pericardiocentesis. A large bore needle is placed just to the left of the patient’s sub-xyphoid process and with negative pressure applied to the syringe, it is directed toward the left scapula (shoulder) while monitoring the ECG for the presence of ventricular ectopy. As little as 15-20 mL of blood to improve the patient’s condition.
The initial treatment of a patient with a suspected cardiac tamponade
The initial treatment of a patient with a suspected cardiac tamponade is a rapid intravenous fluid bolus. This measure improves filling pressures and temporarily improves cardiac output until pericardiocentesis can be performed.
You are transporting a seventy-five-year-old man with a diagnosis of inferior wall MI. During the flight you note the following rhythm. Vital signs are: 70/palp, HR 150, RR 24, SpO2 94% on high flow oxygen with NRM at 15 L/min. He is awake and complains of chest pain and SOB. How will you manage this patient? [image vtach]
A. Administer lidocaine and nitroglycerin
B. Administer normal saline bolus
C. Consider sedation and synchronize cardiovert at 100 joules
D. Have the patient cough forcefully
C. Consider sedation and synchronize cardiovert at 100 joules
sixty-year-old man complains of chest pain for three days with a low-grade fever. Patient complains of increased pain when lying in supine position and states that the chest pain decreases when sitting forward. What is the most likely diagnosis?
A. Pulmonary embolism
B. Pleurisy
C. Pericarditis
D. Pericardial tamponade
C: Pericarditis is an inflammation of the pericardium which can present with chest pain radiating to the back and relieved by sitting up forward and worsened by lying down, is the classical presentation. Other symptoms of pericarditis may include dry cough, fever, fatigue, and anxiety.
How is the coronary perfusion pressurecalculated?
A. DBP − PCWP
B. DBP + PCWP
C. SBP − DBP
D. SBP − PCWP
A. DBP − PCWP
Inferior wall MI is caused by an occlusion of which coronary artery?
A. LAD
B. RCA
C. Circumflex
D. Inferior vena cava
B. RCA
What medications would you expect to administer to a patient presenting with severe chest/abdominal pain, diaphoresis, and is restless? SBP is 170/palp and heart rate in 116. You note a difference in blood pressures when taken on each arm.
A. Nitroglycerin and atenolol
B. Nipride and b-blockers
C. Lasix and nitroglycerin
D. Bumex and Dobutrex
B: Nipride and Beta-blockers.
Three types of Aortic dissection
Debakey classification system describes three types: I = occurs in the ascending aorta and extends distally beyond the aortic arch II = process is limited to the ascending aorta (Marfan’s) III = dissection distal to the origin of the left subclavian artery and extends distally to abdominal aorta.