Mod D Tech 13 Shock and Fainting Flashcards
1
Q
Definition of shock
A
Insufficient perfusion of the tissues with blood carrying oxygen and nutrients causing anaerobic metabolism and thus metabolic acidosis
2
Q
Shock
Name Factors effecting
A
- Blood volume
- Cardiac output
- Functioning blood vessels
3
Q
Name Risk factors for shock
A
- Trauma
- Pregnant patients
- Elderly patients
- Cardiac patients
4
Q
Name Types of Shock
A
Cardiogenic shock
Neurogenic shock
Toxic/Septic shock
Anaphylactic shock
Anaphylaxis
5
Q
Cardiogenic shock
A
•Impaired cardiac function due to a damaged myocardium from either an infarct, arrhythmia or tamponade.
6
Q
Describe Hypovolemic shock
A
- Reduced circulating volume
- Fluid loss
•
Major causes
- Haemorrhage
- Burns & scalds
- Diarrhoea & vomiting
7
Q
Describe Neurogenic shock
A
- Head injury
- Transection of the spinal cord
Spinal shock
- Loss of nervous control
- Relative Hypovolaemic
8
Q
Describe Toxic/Septic shock
A
- Infection such as meningitis causes release of toxins causing arteriolar and capillary dilatation
- Loss of venous tone
- Mismatch between the circulating blood volume and increased capacity of the blood vessels
- Relative hypovolaemia
9
Q
Describe Anaphylactic shock what does it affect
A
- Exposure to Antigen
- Previous exposure to Antigen
- Excessive Antigen/ Antibody reaction
- Histamine released with systemic effect
•
Affecting:
- Tissues
- Respiratory function
- Circulatory function
- Mismatch between the circulating blood volume and increased capacity of the blood vessels
•
•Loss of fluid into tissues, causing sudden oedema
•
•Causes difficulties with airway if around the face and neck
•
•Histamine may also cause brochospasm preventing adequate pulmonary ventilation
10
Q
Anaphylaxis
A
- Anaphylaxis - hypersensitivity reaction
- Anaphylactoid - similar reaction but not dependent on hypersensitivity
- Manifestations and management similar
11
Q
Causes for Anaphlaxic
A
- Drugs (e.g. penicillin, aspirin, vaccines)
- Food substances (e.g. peanuts, shellfish, eggs)
- Venom (e.g. wasp stings, bee stings)
- Almost any foreign substance can become an allergen
12
Q
what is Anaphylaxis
A
A severe allergic reaction, often affecting the whole body
It often occurs when patients are exposed to an allergen to which they have become over sensitive.
When an allergen is reintroduced into the body, mast cells may vigorously react and release an overdose of histamine
13
Q
what does Histamine do
A
- Released by mast cells and basophils
- Causes vasodilation and increased blood vessel permeability
14
Q
Leukotrines
A
- Produced by mast cells and basophils
- Cause bronchoconstriction, an increase in blood vessel permeability and coronary vasoconstriction
15
Q
Physiological effects of anaphalaxiis
A
- Decreased coronary blood flow
- •Systemic vasodilation
- •Reduced cardiac output
- Increased permeability of the blood vessels
- Spasm and oedema of the bronchi
- •Pulmonary vasoconstriction
- •Laryngeal oedema
16
Q
Clinical Signs & Symptoms of Anaphlaxis
A
- Extreme dyspnoea (coughing, wheezing, prolonged sneezing)
- •Tachycardia (often with arrhythmias)
- •Hypotension
- Warm, flushed skin
- •Oedema (particularly facial)
- •Nausea & Vomiting
- •Abdominal cramps
- Urticaria (hives)
- Pruritis (itching)
17
Q
Anaphylaxis facts and figures
A
•Hypovolaemia occurs quickly – 50% of blood plasma volume is lost in 10 – 15 minutes
•
•>50% of fatalities occur within the first hour
•
•75% of fatalities are due to asphyxia from airway obstruction or bronchoconstriction
•
•25% of fatalities occur as a result of circulatory failure and hypotension
18
Q
things to consider /mangerment
A
- Consider when compatible history of severe allergic-type reaction with respiratory difficulty and/or hypotension especially if skin changes present – firstly remove from source
- Oxygen - high flow then maintain 94-98%
- Stridor, wheeze, respiratory distress or clinical signs of shock
- Epinephrine (adrenaline) 1:1000 solution i.m. Repeat every 5 minutes as clinically indicated
- Consider Salbutamol for Bronchospasm resistant to IM Adrenaline.
- If clinical manifestations of shock do not respond rapidly to drug treatment:
- Consider paramedic assistance for administration of antihistamine (Chlorpheniramine), fluids and hydrocortisone if the call is more than 30 mins from the hospital
19
Q
What is Adrenaline 1:1000 (Epinephrine)
what type
what effects
A
- A sympathomimetic drug
- •Stimulates alpha and beta receptors
- •Alpha effect – reverses vasodilation and reduces oedema
- •Beta effect – causes bronchodilation, increase heart rate and strength of contraction. Suppresses histamine and leukotrine release
20
Q
Adrenaline 1:1000
side effects:
A
- Tachycardia
- •Angina
- •Hypertension
- •Ventricular arrhythmias
- Anxiety
- •Headache
- •Cerebral bleeding
- •Nausea & Vomiting
21
Q
Adrenaline dosage
A
22
Q
Salbutamol Dosage
A
23
Q
How does the body respond to shock?
Compensatory mechanisms
A
Abnormal fluid loss:
- Stimulates the Sympathetic Nervous System
- Increased heart rate
- •Selective artery constriction to non vital organs
- •Increased venous tone mobilises reserve blood volume & reduces circulatory capacity
Hormonal Regulation
- RAA= Renin-angiotensin-aldosterone system
- •Activated by baroreptors sensing lowered BP and reduced renal blood flow
- Renin released by kidneys works as an enzyme and converts angiotensinogen (made in the liver) to angiotensin I.
- •Angiotensin I in lungs with ACE (Angiotesin converting enzyme) produces Angiotensin II, a potent vasoconstrictor
- Angiotensin II stimulates release of Aldosterone by adrenal cortex, which increases sodium and water reabsorption
•
•Epinephrine and Noradrenaline
–Increase heart rate
–Increase vascular tone
–•ADH = Antidiuretic Hormone, produced by Hypothalamus and released by pituitary gland in response to blood loss to cause vasoconstriction
•
24
Q
Hypovolaemic shock
how many stages
split in to what
A
Four Stages
Compensated and Decompensated
25
Stage 1
(up to 15% blood volume loss
Signs and symptoms
* Pale skin
* •Capillary refill \< 2 seconds
* •Pulse up to 100 bpm
* •Blood pressure - no change
26
Stage 2
signs and symptoms
| (15% - 30% blood volume loss)
* Pale, cool, clammy skin
* •Capillary refill \> 2 seconds
* •Pulse \> 100 bpm
* •Increased respiratory rate
* •Blood pressure - normal systolic pressure but raised diastolic pressure
* The body’s compensatory mechanisms are at their limit
27
Stage 3
(30% - 40% blood volume loss)
signs symptoms
* Anxiety, restlessness & agitation
* Pulse \> 120 bpm
* •Blood pressure - systolic pressure 100mmHg or less
* ***_The patient is in decompensated shock_***
28
Stage 4
(\> 40% blood volume loss)
signs and symptoms
* Moribund appearance
* •Central cyanosis
* •Altered level of consciousness
* •Pulse - weak with marked tachycardia
* •Respiratory distress
* •Blood pressure - systolic pressure 70mmHg or less
* The patient is in critical condition
29
Septic Shock
Definitions:
•***_Sepsis_***:- Presence of bacteria in the blood stream
***_•Septic shock_***:- Begins with the development of septicaemia usually from bacterial infections, but can be viral in origin.
This is the most common type of Distributive shock
30
what is SIRS
Systematic Inflammatory Response Syndrome
## Footnote
The ‘normal’ response to injury doesn’t stop
The body develops SIRS
* HR \>90
* RR \>20 or ETCO2 \<32mmhg (4.3kPa)
* Temp \<36c \>38c
* White cell count \<4000 \>12000
(normal range 4300 - 11,000 with both low and high counts can be attributed to bacterial infection)
•\>10% immature neutrophils
31
Stages of Sepsis how many
32
Septic Shock
signs symptoms
* Described as distributive shock reflecting the effects of the disease process on the movement of fluid around the different compartments of the body (Morton & Fontaine 2009)
* Early symptoms of systemic vasodilation
i. red & flushed appearance
ii. full bounding pulse and rapid capillary refill
iii. increased peripheral circulation
iv. increased micro-vasculature permeability creating a major loss of volume from the core circulation (Jenkins et al. 2007)
v. Blood sugar levels increase to cope with the metabolic demands
33
Recognition of Shock
General Signs & Symptoms
* Increased pulse rate, becoming weaker
* Hypotension
* Pale, cool, clammy skin
* Increased respirations becoming shallow
* Nausea & vomiting
* Thirst
* Pupil reaction slow
Faintness & dizziness
34
Signs & Symptoms
Spinal Shock
* Loss of feeling
* Pins & Needles
* Paralysis
* Displaced vertebrae / swelling
* Fixation of spine
* Priapism (men)
* Hypotension
* Normal Pulse
* Normal colour above level controlled by point of transection
* Warm & flushed below level controlled by point of transection
35
Signs & Symptoms
Anaphylactic shock
* Severe dyspnoea
* Tachycardia
* Severe hypotension
* Warm, dry, flushed skin becoming pale as the condition worsens
* Oedema
* Itching
* Nausea & vomiting
Abdominal cramps
36
General management of shock
* Safety
* Do I need help
* Ensure open airway
* Oxygen to 94-98%
* ? Assisted ventilation
* Treat other injuries
* Keep Pt. Temp. normal
* Pt. Positioning (autotransfusion)
* Reassurance
* Monitor vital signs
* Rapid / smooth transfer to hospital
* Alert hospital using ATMIST
37
Management of spinal shock
* Safety
* Do I need help
* Ensure open airway
* High conc. Oxygen
* ? Assisted ventilation
* Immobilise fractures
* Control bleeding
* Avoid unnecessary movement
* Keep Pt. temp. normal
* Reassurance
* Monitor vital signs
* Rapid / smooth transfer to hospital
* Alert hospital using ATMIST
38
Fainting or Syncope
Causes and Types
* Postural fainting
* •Emotional stress
* •Cardiac origin
39
Postural Fainting
•Standing quickly from a recumbent position
•
•Prolonged periods of standing particularly in hot weather
•
•Athletes may be vulnerable should they suddenly stand following vigorous exercise
40
Fainting or Syncope
Emotional Stress
Stress or fright
## Footnote
•
•Reflex dilation of blood vessels
•
•Pooling of blood in the extremities
41
Fainting or Syncope
Cardiac origin
•Transient decrease in cardiac output as a result of severe bradycardia or tachycardia
•
•Diminishes cardiac output
•
•Usually sudden onset which resolves when arrhythmia settles
42
Fainting or Syncope
Clinical signs and symptoms
* Slow pulse initially (increases as recovery occurs)
* •Cold and clammy skin
* Pale colour
* Pupils equally dilated
* •Shallow to normal breathing
* •Reduced level of consciousness
43
Fainting or Syncope
Management
•Maintain in recumbent position (facilitates cerebral perfusion)
•
•Ensure open airway
•
•Loosen tight or restrictive clothing
•
* Elevate lower limbs (facilitates venous return)
* Initiate secondary survey and treat any injuries caused by the fall
•
•Encourage patients to remain in recumbent position until fully recovered
•
•Monitor for any change in condition
