Miscellaneous Flashcards
Etiologies of hammer toes
- Flexor stabilization
- Flexor substitution
- Extensor substitution
Flexor stabilization
MOST COMMON
o Pronation of STJ leads to unlocked/hypermobile foot and therefore excessive “gripping”
o Flexors (FDL, FDB) fire earlier and stay contracted longer to stabilize the foot, but end up overpowering the interosseous muscles
o STJ pronation also leads to forefoot abduction making the QP muscle weak, allowing adductovarus rotation of 5th digit
Flexor substitution
LEAST COMMON
o Supinated foot type with weak gastro-soleal complex (Achilles rupture, Achilles surgery)
o Flexors gain advantage over interosseous due to weak gastro-soleal complex, they try to “substitute” for the weak muscle group
o Deep posterior and lateral muscles attempt to make up for weak gastro-soleal complex
Extensor substitution
o Pes cavus, neuromuscular contracture, equinus (these need to be corrected as well)
o Extensor digitorum longus gains advantage over the lumbricals, bowstringing of EDL
o Initially only swing phase hammering which will partially resolve during weight bearing
o Orthotics will not work due to the deformity predominating during swing phase
Virchow’s triad
Three factors that are commonly associated with the formation of thrombi
- Stasis
- Blood vessel injury
- Hypercoagulability
Stasis
Reasons for stasis
- Arrhythmias
- MI
- CHF
- Heart failure
- Immobilization
- Obesity
- Varicose veins
- Dehydration
Blood vessel injury
Reasons for blood vessel injury
- Trauma
- Fracture
- IV
Hypercoagulability
Reasons for hypercoagulability
- Neoplasm
- Oral contraceptives
- Pregnancy
- Surgery
- Polycythemia
Calculate ABI
- Determine brachial systolic pressure
- Determine ankle systolic pressure
- Ankle pressure divided by brachial pressure
Ranges of ABIs
1 = normal 0.5-0.8 = intermittent claudication <0.5 = rest pain, ulcers
Toe pressures in diabetics
> 55 mmHg = healing
45-55 mmHg = uncertain healing
<45 = no healing
At least 30 mmHg required for healing a wound on the digits
Seddon classification for nerve injury
- Neurapraxia
- Axonotmesis
- Neurotmesis
Neurapraxia
Bruised nerve - results in numbness that is reversible
Axonotmesis
Injury to axon that results in Wallerian degeneration, will regenerate over several months as long as gap is not too big
Neurotmesis
Complete severance of the nerve resulting in irreversible numbness
Sunderland’s classification for nerve injury
- First degree
- Second degree
- Third degree
- Fourth degree
- Fifth degree
First degree
A conduction deficit without axonal destruction
Second degree
Axon is severed without reaching the neural tube. Wallerian degeneration with regeneration. Regeneration is likely (axonotmesis)
Third degree
Degeneration of axon with destruction of fascicle with irregular regeneration
Fourth degree
Destruction of axon and fascicle and no destruction of nerve trunk, but a neuroma-in-continuity exists