MISC Flashcards

1
Q

Most common cause of conjunctival haemorrhage?

A

Diabetes and age

  • self limited condition if not associated with systemic lllness
  • associated conditions: HTN, diabetes, bleeding disorder, high cholesterol, coronary heart disease, medications, valsalva
  • usually takes 1-2 weeks to resolve
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2
Q

What is an orf?

A
  • zoonotic viral skin infections
  • contracted from sheeps and goats
  • generally solitary lesion
  • most common on hands
  • clinical diagnosis in people handling goats and sheep
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3
Q

ECG features of hyperkalaemia

A
  • peaked T waves
  • flat p waveas, PR prolongation, widened QRS
  • bradycardia
  • sine wave if severe
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4
Q

Management of hyperkalaemia

A
  • ABCD
  • calcium 10ml of 10% gluconate
  • insulin/dextrose: 10U in 50ml of 50% dextrose
  • salbutamol neb/IV
  • bicarb infusion: 1mmol/kg IV

If able calcium resonium 15-30g

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5
Q

Q fever

A
  • the most common zoonotic disease
  • fever, rigors, chills, headache, fatigue, weight loss
  • abnormal LFTs
  • coxiella burnetti
  • serology or PCR
  • main source: cattle, sheep, goats
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6
Q

Clinical signs and symptoms of Q fever

A
  • fever
  • headache
  • myalgia
  • cough
  • influenza like illness
  • weight loss
  • pneumonia
  • nausea
  • jaundice (rare)
  • meningeal signs (rare)
  • rash (rare)
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7
Q

Chronic Q fever

A
  • chronic fatigue
  • alcohol intolerance common
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8
Q

Complications of Q fever

A
  • Endocarditis
  • Pnuemonia
  • hepatitis
  • meningitis
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9
Q

Common blood finding of Q fever

A
  • LFT derangement
  • lymphopenia
  • thrombocytopenia
  • CRP elevated
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10
Q

Abx for Q fever

A

100mg doxycycline BD for 14 days

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11
Q

Vaccination for Q fever

A
  • contraindicated if previous Q fever due to risk of reactivity
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12
Q

What are the notifiable zoonotic disease in Australia?

A
  • brucellosis (pigs)
  • leptospirosis (infected urine/water)
  • Q fever (cattle, goats, sheep)
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13
Q

Brucellosis

A
  • fever, headache, weakness, sweats, chills, myalgia
  • inflammation or liver or pleen
  • may cause inflammation of testes and epipdymis
  • symptoms last 2-4 weeks followed by spontaneous recovery
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14
Q

Leptospirosis

A
  • fever, chills, headache, myalgias, conjunctival suffusion
  • Weil’s disease: jaundice, renal failure, haemorrhage, myocarditis
  • Meningitis
  • pulmonary haemorrhage and ARDS
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15
Q

Common presentation of acute and chronic Q fever

A
  • chronic: endocarditis or hepatitis
  • acute: pneumonia, hepatitis, osteomyelitis and meningitis or encephalitis.
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16
Q

Management of brucellosis

A
  • 6 weeks doxycycline + IV gentamicine for 7 days

or

  • doxycycline and PO rifampicin (not subsidised on PBS)
17
Q

What is effort thrombosis?

A
  • Axillosubclavian vein thrombosis
  • form of venous thoracic outlet syndrome
  • caused by venous injury due to mechanical compression
  • chronic repetative venous compression at costoclavicular junction causes inflammatory response hence leading t o thrombosis of subclavian vein
  • diagnosis: young, right handed with occupational or athletic history of overhead activity
  • acute pain, heavy arm
18
Q

Main problems in snake bites (4)

A
  1. Paralysis: ptosis, diplopia, opthalmoplegia, weakness, resp problems
  2. coagulopathy: bleeding from bite site, gums and at IVC
  3. myolysis: muscle pain and weakness, myoglobinuria, AKI, hyperk
  4. renal damage: coaguloapathy, AKI,
19
Q

General and local snake bite symptoms

A
  • headache, nausea, vomiting, abdominal pain, collapse, seziures
  • local symptoms: local pain, swelling, bruising
20
Q

Snake bite first aid

A
  • ABCD
  • broad compression bandage to the bite site, extended bandage to cover whole of bitten limb including fingers and toes
  • splint the limb
21
Q

Hospital management of snake bite

A
  • ABCD
  • IVC + IVF
  • admit or transfer for clinical observation and serial labs
  • maintain PBI until at a centre with lab
  • antivenom indicated if any evidence of envenomation
22
Q

Clinical evidence of evenomation

A
  • any paralysis (ptosis, opthalmoplegia, resp distress)
  • excessive bleeding
  • period of LOC, fitting, collapse, arrest
  • oliguria, anuria, myoglobinuria
23
Q

Lab tests of envenomation

A
  • INR, apTT, fibrinogen, d-dimer
  • FBC, UEC, CK

If initial tests are normal (pre PBI removal), repeat testing 1 hr post-removal of PBI and then at about 6 & 12 hours post-bite, or more urgently if the patient develops clinical evidence suggestive of envenoming

24
Q

Antivenom therapy

A
  • give IV
  • have adrenaline ready and drawn up in case of anaphylaxis
  • most cases give 1 vial of polyvalent
25
Q

Brown snake

A
  • coagulopathy, renal failure and paralysis
  • dominant feature is coagulopathy
26
Q

Red belly black snack

A
  • haemorrhagic and myolytic
27
Q

Death adder

A

Neurotoxic/paralysis

28
Q

Taipan

A

Haemorrhagic and neurotoxic

29
Q

Tiger snake

A

-haemorrhagic and neurotoxic

30
Q

Red back spider bites

A
  • common but unlikely lethal
  • bite felt, severe localised pain and sometime localised sweating
  • pain spreads proximally and causes lymphandopathy
  • generalised pain with sweating, hypertension and malaise
31
Q

Funnel web spider bites

A
  • potentially rapidly lethal, medical emergency
  • painful with fang marks present
  • systemic envenomatyion: perioral tingling, tongue fasciculation, increased salivation, lacrymation, piloerection, sweating, nausea, vomiting, headache, dyspnoea, pulmonary oedema
32
Q

Sting rays

A
  • hot water immersion for pain
  • ADT
  • analgesia + regiona nerve block
  • prophylaxtic Abx
33
Q

Blue ringed octopus

A

Most bites are minor, but potentially lethal. Bite may be painless. In significant cases, rapid development of systemic envenoming which may include: Perioral tingling. Progressive generalised weakness/paralysis. Respiratory paralysis. Hypotension. Maintain airway, respiration (intubate, ventilate), give IV fluids ± pressors to control hypotension. No antivenom is available.

34
Q
A