Mike MI Flashcards

1
Q

What is a MI

A

Myocardial infarction
Occurs when there is sustained myocardial ischemia.
This usually occurs when an atherosclerosis plaque ruptures and a thrombosis blood clot form at the site causing coronary occlusion.

A blockage of blood flow to.the heart causes necrosis due to lack of oxygen…within 20 mins

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2
Q

What is a MI

A

Myocardial infarction
Occurs when there is sustained myocardial ischemia.
This usually occurs when an atherosclerosis plaque ruptures and a thrombosis blood clot form at the site causing coronary occlusion.

A blockage of blood flow to.the heart causes necrosis due to lack of oxygen…within 20 mins

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3
Q

Necrotic myocytes

A

When they become necrotic the plasma membrane of the muscle cells (sarcoma) ruptures and there is a leakage of cardiac enzymes (biomarkers).
These become detectable in the blood.
Myocardial ischemia
Hypoxia
Myocardial necrosis
Sarcolemmq breakdown
Leakage of contractile proteins

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4
Q

Risk factors for mike

A

Smoking
Male
Genetics..family hx

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5
Q

Risk factors for mike

A

Smoking
Male
Genetics..family hx

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6
Q

Modifiable risk factors

A

Obesity
Hypertension
Hypercholesterolemia
Physical inactivity
Hyperglycemia
Hyperlipidemia
Smoking

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7
Q

Signs and symptoms of MI

A

Diaphoresis
Chest pain/discomfort
SoB
Anxiety
Nausea/vomiting
Pain/discomfort in shoulder, neck, arm jaw
Pallor and pale peripherals

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8
Q

Mike’s symptoms

A

Indigestion type pain
Angina pain
Pain in chest
Pain in left arm
Sweating profusely
Tingling hand
Pallor
Felt cold
Pain not relieved by morphine or aspirin
Elevated S-T

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9
Q

Mike’s symptoms

A

Indigestion type pain
Angina pain
Pain in chest
Pain in left arm
Sweating profusely
Tingling hand
Pallor
Felt cold
Pain not relieved by morphine or aspirin
Elevated S-T

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10
Q

Myocardial stunning

A

Temporary loss of contractibility lasting hours to days after repercussion (tissue damage caused when blood returns)

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11
Q

Myocardial hibernation

A

Prolonged loss of contractihility when there is sustained ischemia

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12
Q

Myocardial remodeling

A

Permanent loss of contractibility with cellular hypertrophy…increased in muscle cells

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13
Q

Complications of MI

A

Angina
Reincarnation
Arrhythmia
Valvular incompetence.. leaky valves
Cardiogenic shock…can’t pump enough blood to tissues
Left ventricular aneurysm
Pericarditis
Rupture of ventricilular wall
Heart failure
Death

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14
Q

Complications of MI

A

Angina
Reincarnation
Arrhythmia
Valvular incompetence.. leaky valves
Cardiogenic shock…can’t pump enough blood to tissues
Left ventricular aneurysm
Pericarditis
Rupture of ventricilular wall
Heart failure
Death

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15
Q

Assessments and testing

A

BP
Hr
Presentation
Full blood count, cardiac enzymes, BSL, urea and electrolytes, TFT
Ecg
Angiography
Chest xray

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16
Q

Medication

A

MONA
Morphine, o2, gtn, aspirin
Clexane
Clopidigral..prevents thrombosis
Gtn dulates bvs to decrease workload
Metoprolol…to reduce HR..betablocker, reduces cardiac output and BP
Simvastin..lower cholesterol

Anti coag
Beta blockers
Nitroglycerin
Cholesterol lowering

17
Q

Medication

A

MONA
Morphine, o2, gtn, aspirin
Clexane
Clopidigral..prevents thrombosis
Gtn dulates bvs to decrease workload
Metoprolol…to reduce HR..betablocker, reduces cardiac output and BP
Simvastin..lower cholesterol

Anti coag
Beta blockers
Nitroglycerin
Cholesterol lowering

18
Q

Dietary and lifestyle changes

A

Low fat
Low salt
Increase fruit and veg
Increase soluble fibre
Stop smoking
Increase exercise

19
Q

Dietary and lifestyle changes

A

Low fat
Low salt
Increase fruit and veg
Increase soluble fibre
Stop smoking
Increase exercise

20
Q

Goal of care

A

Decrease heart workload
Increase O2 delivery to body tissues
Reperfusr rhe heart wall