memory, reward, motivation Flashcards
declarative memory
explicit memory, can clearly state them, conscious memories, 2 types
non-declarative memory
implicit memory, motor memory
2 types of declarative memory
episodic- events, semantic- facts
brain areas important for declarative memory
MTL, Hippocampus, and mammilary bodies
brain areas important for non-declarative memory
basil ganglia
working memory
temporary storage of info
spatial memory
memory for spacial information, using the hippocampus
place cells
fire at different rates based on location and place
amnesia
memory loss
retrograde amnesia
memory loss of times before injury
anterograde amnesia
impaired ability to form new memories
what can cause memory loss
injury, neurodegenerative disease, infectious disease, nutritional deficiency, anoxia (loss of oxygen)
dementia
substantial loss of memory and other cognitive functions
intelligence
set of knowledge and skills
PFC/vmPFC
responsible for executive functions and abilities
osmotic thirst
when fluid in cells decreases
hypovolemic thirst
when there is a decrease in blood volume
arcuate nucleus (ARC)
master control for hunger
orexigenic neurons
start eating
POMC neurons
stop eating
lateral hypothalamus
initiates eating
ghrelin
starts in the stomach, triggers eating through ARC
CCK
Starts in intestines, terminates eating through PVN
Leptin
Starts in fat cells, maintains long term through ARC
Insulin
Starts in pancreas, maintains long term through ARC
Batille’s model for working memory
phonological loop, visual sketchpad, and central executive
Case of H.M
Injury to bilateral MTL via resuction, resulted in impairments to declarative memory–anterograde amnesia and temporally graded retrograde amnesia
Case of S.M
Bilateral amygdala damage due to neurodevelopmental disorder which resulted in a lack of fear response
Case of Boswell
bilateral MTL damage due to HSE resulting in impaired declarative memory
psychoactive drugs
effects brain and nervous system
opiates
from the plant poppy, relieves pain, induces sleep, positive feels–morphine, heroin
depressants
decreases central nervous system activity, calms, reduces anxiety, sleep inducing–alcohol, benzodiazpines
stimulants
activate the CNS, increased arousal, increased alertness, mood–caffeine, nicotine, meth
caffeine
blocks binding of adenosine, increases dopamine and ocetylocine
cocaine
blocks reuptake of dopamine and serotonin and dopamine
meth
increases dopamine and norephinephrine, blocks dopamine reuptake
weed
binds to endogenous signal receptors (canaboid receptors)–releases dopamine