Memory and Amnesia Flashcards

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1
Q

are metaphors of memory good

A

no
account for memory but arent a good way of doing so
based on technology of the time went from wax tablet to computer

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2
Q

what is the broad memory process

A

encoding - processing information into the memory system
storage - retention of encoded material over time
retrieval - getting the information out of the memory storage

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3
Q

are memories fixed

A

no they are representations of what we remember and change all the time
they get manipulated they are not fixed in an archive

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4
Q

why can memory fail us

A
  • We may not notice something
  • We may notice it but not encode it
  • Encode but not consolidate
  • Consolidate but not retrieve
  • Retrieve it wrongly
  • Retrieve correctly but forget it quickly.
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5
Q

what is the testing effect

A

memory is increased when some of the learning period is spent retrieving the to be remembered info
counters the effects of forgetting
actively rehearsing increases learning

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6
Q

according to roediger HL should you test immediately after learning

A

no, testing immediately is bad and rereading produces a better result immediately after. 2 days between learning and revising testing method proves better results

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7
Q

does the testing effect relate to the elderly and people with neurological issues

A

yes
single testing forget more than young or healthy people
repeated testing = people attain much more and age affect disappears if they keep testing

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8
Q

what is the difference in question asked between clinical and cognitive neuropsychology

A

clinical - what has happened to cause symptoms

cognitive - what does pattern of impaired and intact capabilities show about how the normal mind and brain work

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9
Q

what are the two traditions of human neuropsychology

A

1) What functions disrupted by damage to the region x? (classical neuropsych approach) Addresses the questions of functional specialisation converging evidence and functional imaging. Tends to use study group methods.
2) Can a particular function be spared/impaired relative to other cognitive functions. Addresses questions of what the building blocks of cognition are (irrespective of where they are). Tends to use a single case methodology.

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10
Q

what is plasticity

A

the brains ability to change as a result of experience and this persists throughout life
the changes in neural connectivity are greatest in childhood.

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11
Q

what is phrenology

A
  • Differences in personality traits manifest in differences in cortical sizes and bumps on the skull
  • Crude division of psychological traits e.g love of animals and this is not grounded in science.
  • Now discredited but used in the 19th century.
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12
Q

how did phrenology contribute to modern day psychology

A

it was the start of studying parts of brains doing diff things and different parts of the cortex serving diff functions

this has stood the test of time

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13
Q

what is functional specialisation

A

diff areas doing diff things
modern cog neuroscience uses empirical methods to ascertain different functions
doesnt assume each region has one function or each function has a discrete location
does assume some degree of specialisation of neurons in particular regions

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14
Q

what is the problem with using single cases for establishing patterns

A

they show a pattern but some cases dont verify what we want
Black swan analogy always have to look for black swans (karl popper)

is still value in single cases can change the way we think about memory, hard to recreate same injuries to increase study size anyway

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15
Q

what is the function of the hippocampus

A

most encoding and storage occurs here

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16
Q

what is a double dissociation

A

used to determine if an anatomical structure is responsible for a process. need two patients with lesions, one performs well on task a and pathologically on task b. have second ppt that is pathological on a but performs well on b. from this we can infer that task b is related to function b and task a is related to function a and that they are run by different neuronal and cognitive networks

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17
Q

what is the short conclusion for when a double dissociation is observed

A

When double disassociations are observed we are allowed to postulate that the two relevant memory systems involve different cognitive systems possibly mapped onto different neuronal networks

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18
Q

what is anterograde amnesia

A

memory deficit of learning new things.

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19
Q

what is the back story for the HM 1950 case study

A

young man suffered from difficult to treat epilepsy
experimental surgery removed medial temporal lobes (bilaterally) which contained the hippocampi
temporal lobectomy

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20
Q

what were the results on HM memory after the surgery

A

profound amnesia
cant remember new memories (anterograde amnesia)
can still remember how to speak and learn new motor skills
bilateral removal of the hippocampus and surrounding area also lead to retrograde amnesia deificts back 10 years before surgery

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21
Q

double dissociation to show that long term and short term memory are different things

A

HM global amnesiac - normal digit span remembered but impaired ltm
KF - normal ltm for word lists but impaired digit span
dissociation between ltm and stm allows us to postulate that they are controlled by different memory systems and the damage is in different neural systems

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22
Q

what are the characteristics of stm

A

limited capacity
limited duration
holding available recent and relevant information in a temporary store

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23
Q

what are the characteristics of ltm

A

unlimited storage
relatively permanent
store for episodic and semantic memory

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24
Q

what are the primacy and recency effects

A

in free recall more items are recalled from the start of the list (primacy) and from the end of the list (recency)

25
Q

what is the serial position curve

A

demonstrates the recall of items as you progress through a list and demonstrates the recency and primacy effects on a u shape

26
Q

what are the two case studies that show pathological recency and primacy effect

A

amnesiacs show no primacy effect and it is pathological, no encoding benefit - HM remember last few words when not interrupted but primacy effect gone.

Patient KF intact primacy effect but an impaired recency effect.

27
Q

how can we use the primacy and recency effects to pin down the kind of amnesia new patients are showing

A

Two effects predicted the amnesic patients’ impairments and can predict performance on classical psychological tasks.
Can use this to pin down kind of amnesia new patients show.

28
Q

what are the hippocampal and parahippocampal regions responsible for and where are they

A

hippocampal formation is responsible for episodic memory
parahippocampal region for semantic memory
they are very close in the brain
lesions can impair them both because they’re close

29
Q

what does the case study of KC teach us

A

intact semantic memory but impaired episodic. cant learn anything but can remember facts from before had motorcycle accident with very severe brain lesion

therefore semantic and episodic memory are different.

MRI displays extensive damage to patient KCs MTL/hippocampus in axial and coronal sagittal views

30
Q

what is the difficulty with defining normality in memory

A

it’s unreliable hence need to define it within imperfect systems in order to diagnose a pathological memory deficit
example of unreliable memory = eyewitness testimony.

brain reconstructs events and is an organ or re presentation

31
Q

how has evolution selected for an inaccurate brain

A

ancestors see grass move assume tiger and flee, even though they were wrong 99% of the time. brain incorrectly tells people to run, a computer however would always stay as the probability of it being a tiger is so low its not worth running so the computer would die from the tiger one day.

fearful and false assumptions kept us alive and false memories were selected for evolution

32
Q

what was sir Frederick Bartletts central insight in memory

A

memory isnt like a tape recorder doesnt faithfully play back our experiences.
Instead reconstructs them imaginatively

33
Q

what was Bartletts study that highlighted the reconstructive nature of memory

A

presented story of ghosts to ppt of indian origin the people reconstructed based on what they believe. Investigated how memory of a story is affected by previous knowledge, does cultural background and unfamiliarity with a text lead to distortion of memory, reconstructed on cultural schemas

34
Q

what is change blindness

A

when we encode what we expect to encode dont notice a change

35
Q

what is the study that demonstrates change blindness

A

ppt asked q by experimenter on the street, during qs a piece of wood goes between and the sight is blocked off, experimenter changes as it passes. ppt doesnt realise because they are wearing similar clothes
information is mediated by our beliefs didnt expect the person to change so they didnt notice

36
Q

what is the study highlighting the inaccuracy of eye witness testimony

A

loftus and palmer
ppt watch video of car accident, asked to remember if glass broken using different words “hit” vs “smashed” each other was there broken glass. people who heard hit reported broken glass 14% of the time but smashed incorrectly reported smashed glass double the amount of times

memory is unreliable so eye witness testimony is too, questions change the answer

37
Q

what is the disney experiment

A

waited for tourists to leave and asked whether they remember seeing buggs bunny. lots of people said they did but hes not a disney character so its impossible
expectation that it could have happened caused them to remember something that didnt happen

38
Q

what do we need to sacrifice to plan the future

A

In order to plan the future, which is what memory is about, we need a system of representations rather than mere recordings of events. The price we pay is a memory system which is fallacious and prone to errors.

39
Q

what real life story proves that memory is reconstructive

A

karl popper and wittgenstein discussing if there is a science in morality. Wittgenstein picked up a poker from the fire to play with as the conversation got heated. then asked popper to think of an example of a moral stance and he said not to threaten lecturers with a poker as it had offended

The people who were in the room many years later asked to recall the event all 9 said different things

40
Q

what is the information deficit model

A

dispel a myth e.g vaccines are bad
people agree immediately after
return a month later no notion of what is false ortrue they have mixed into the same thing
educating people can backfire and lead to people remembering misinformation about things

41
Q

what do we need to define for deciding what is pathological

A

errors and false memories

42
Q

what is forgetting

A

Forgetting is defined as the lack of availability, partial or total, temporary or permanent, of memories of lived events or of information previously encountered, processed or learned.

43
Q

when does forgetting occur

A

when something is disregarded, erased or transformed during encoding or retrieval. After the learning phase, information may be inaccessible because of inhibition, removal or interference.

44
Q

what does forgetting identify

A

the difference between what has been encoded consciously or unconsciously and what is remembered + available for retrieval

45
Q

how does forgetting manifest

A

omission errors (with or without awareness of not remembering) or commission errors (false memories, intrusions, and substitutions). Different error types reflect different neurobiological and cognitive mechanisms.

46
Q

is forgetting necessary

A

yes
it reflects normal functioning
essential for categorisation, learning and abstract reasoning
for maintaining social and affective relationships

47
Q

what does pathological forgetting look like

A

disproportionate or abnormally fast forgetting are pathological
lack of forgetting is also abnormal

48
Q

is forgetting ever voluntary

A

it can be induced voluntarily to preserve feelings and pursue personal goals in everyday life or following specific instructions in experimental settings

49
Q

how do we forget

A

trace decay theory - memory trace fades and the memory is no longer availabe

Interference theory - the memory trace is no longer accessible as another memory trace interferes with its retrieval

50
Q

how did Ebbinghaus’s create the forgetting curve

A

he started the idea of studying forgetting and determined the curve. Memories fade away as function of time. made curve by studying himself. on learning non sensical lists of syllables

51
Q

describe ebbinghaus’s forgetting curve

A

fast forgetting at the start then slowed down

plateaus later on, important to know if forgetting is abnormal in a patient

52
Q

experiment to show what determines forgetting

A

ppt repeat material different amounts of times, 6,4 or 2 times. more repeated the more is remembered in immediate memory. independent of initial learning stage the curves are parallel. more repetition = more remembered but decay rate is the same

53
Q

what is the experiment to prove consolidation occurs

A

psychologists wait at football game until someone had an injury found that these people if asked remembered exactly what happened, followed ambulance and asked again at diff points where they didn’t remember anything. It encoded but faded after 10 mins so failed to consolidate. People with no concussion show same remembering immediately to 20 mins later, people with concussion remember immediately but fades to 0 by 20 mins.

54
Q

how do we separate the passage of time causing forgetting and interference

A

ask ppt to encode material then either perform interference tasks or rest in a dark room (minimal interference) with ppt with normal and with amnesia. test both 30 mins to week later people in interference remember less than people with less interference, if time alone responsible there would not be a difference due to the condition of what happens during this time. Therefore interference plays a role in forgetting. People with dense amnesia with minimal interference remember much better as compared to the usual condition where they remember 5% as opposed to 60%. This interference changes according to how close the interference happens to the remembering event.

55
Q

how does closeness of interference to presentation of material relate to forgetting

A

longer interference is postponed the better the performance

the earlier a lack of consolidation occurs the worse remembering is

56
Q

what is the etymology of forgetting

A

Etymologically, the word “to forget” derives from the old English word forgytan, which is composed by for- and gietan (to grasp) and literally means “to lose (one’s) grip on” (Hoad, 1996). In Italian: dimenticare, from the Latin demens which means “without mind”. In most languages, forgetting denotes a defective memory.

57
Q

what are the adaptive reasons for forgetting

A

the loss of access to information through disuse is seen not as a failure of the system, but an adaptive feature that facilitates updating.
not retrieved = not reinforced = forgotten
not useful to remember every parking space has in your life

58
Q

describe transient epileptic amnesia

A

– TEA have often malfunctioning hippocampus as demonstrated by MRI scans. They learn something they are normal, then after 30 mins still normal, encoding, storage and first retrieval normal but showed diff forgetting curve compared to Ebbinghaus. Instead they drop dramatically (fast pathological forgetting)

59
Q

how should forgetting be viewed

A

Forgetting should not be conceived as the opposite of remembering, rather as functional to remembering, unless it becomes pathological.