Memory Flashcards

1
Q

What are the four types of memory?

A

Episodic, semantic, autobiographical and emotional memory

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2
Q

What is episodic memory?

A

Memory of personal life experiences, contextualised.

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3
Q

What is semantic memory?

A

Recollection of facts, ideas and concepts.

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4
Q

What is autobiographical memory?

A

A combination of semantic and episodic memory, and a memory for your personal history.

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5
Q

What is emotional memory?

A

It is often important in episodic memory, emotion-memory interactions.

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6
Q

How is memory processed?

A

Encoding, storage, consolidation and retrieval.

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7
Q

What is encoding in memory?

A

Conversion of information into a form capable of being stored in memory.

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8
Q

What is storage in memory?

A

Creation of a trace of information within the nervous system.

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9
Q

What is consolidation in memory?

A

Strengthening of this trace over time, to convert it from a labile form to a more damage-resistant form.

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10
Q

What is retrieval in memory?

A

Attempt to recover a memory trace.

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11
Q

What is the unitary model of memory? Is it an accurate representation of memory? Why / why not?

A

The idea that STM and LTM are one mechanism, with retention for decades and unlimited capacity. It is inaccurate as our memory decays (15 - 20s)and has limited storage.

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12
Q

What is Atkinson & Shiffrin’s multi-store model of memory?

A

Comprised of sensory, ST and LT memories, information is first registered from the environment by sensory organs. If attended to, this information is stored ST, which is transformed into LT if it is rehearsed by being given meaning.

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13
Q

What is the serial position effect?

A

Tendency to remember the first or last thing learned, with poorer memory recall for middle words learned.

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14
Q

What is Baddeley & Hitch’s working memory model?

A

Emphasises that STM is a mental workbench, not a storage platform. Active manipulation of information through the central executive that engages the episodic buffer, phonological loop and visuospatial sketchpad

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15
Q

What is Craik & Watkins’ maintenance rehearsal?

A

It is the idea that information is stored better when you repeat it for a short time. However, it failed to improve memory performance.

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16
Q

What is Stein & Bransford’s elaborative rehearsal?

A

Organisation and relation of new information to material already held in LTM.

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17
Q

What is Craik & Tulving’s depth of processing?

A

Indicates that information processing has different levels, depending on the type of information (rhyme, semantics, case)

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18
Q

What did Bransford & Johnson realise about giving a sentence to summarise a paragraph before reading the entire paragraph?

A

Elaborative encoding improves comprehension and memory, as participants provided prior knowledge about the paragraph remembered it better.

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19
Q

What is a schema?

A

An organised mental framework with an idea of certain concepts.

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20
Q

What is anterograde amnesia?

A

Amnesia that prevents your ability to remember events post-trauma.

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21
Q

What is retrograde amnesia?

A

Inability to remember events pre-trauma.

22
Q

What is Dresler et al.’s method of loci?

A

Visualising items you want to remember in specific, familiar places, in order to improve memory.

23
Q

What is Godden & Baddeley’s encoding-retrieval context experiment about?

A

Divers recalled information presented better underwater, compared to on land, meaning that emulating similar environments for both learning and recollection improves memory.

24
Q

What does temporal lobe amnesia affect, and what types of memory are spared?

A

Episodic and declarative memories are impaired, whilst semantic and procedural memories (skills, habits, priming, conditioning) are spared.

25
Q

What are the four sins of memory / forgetting?

A

Transience (reduced memory over time), blocking (inability to remember needed information), absentmindedness (encoding failure, didn’t pay attention), persistence (resurgence of unwanted memories).

26
Q

What is retroactive interference?

A

New learning interferes with the old, such as learning a new language.

27
Q

What is proactive interference?

A

Old learning interferes with the new, such as ditching old habits.

28
Q

What are the four lobes of the cerebral cortex?

A

Parietal, occipital, temporal and frontal.

29
Q

What does the parietal lobe do?

A

Helps in registering sensory and spatial information.

30
Q

What does the occipital lobe do?

A

Registers visual information.

31
Q

What does the temporal lobe do?

A

Manages emotions and stores memories.

32
Q

What does the frontal lobe do?

A

Registers speech and language, personality, motor skills.

33
Q

What are neurons and what are the main components of one?

A

Neurons are the brain’s processing units, and are comprised of dendrites, nucleus, axon and the presynaptic terminal.

34
Q

What is the resting membrane potential?

A

The difference in electric charge between electrically inactive neurones, which is -70mhV.

35
Q

Explain how information is integrated in the dendrite using spatial summations.

A

Spatial / temporal summations of graded potentials occur within the dendrites, leading to a “pulse” known as an action potential (within the axon) if the threshold level of intensity at the initial segment is reached.

36
Q

How is information transmitted within a neuron?

A

Information flows from the dendrites to the axon and the presynaptic terminal. This occurs when there is a change in the neuron’s membrane potential, which is done through the contraction and expansion of ion channels.

37
Q

How does tetrodotoxin impact neurons?

A

It blocks the ion flow through channels, impairing the generation of action potentials.

38
Q

How do neurons communicate with each other?

A

Neurotransmitters are synthesised within the axon terminal where they are released into the synaptic cleft and bind to the dendrites of another neuron, creating graded potentials before reuptaking.

39
Q

In what ways can drugs affect neurotransmission?

A

They can influence the synthesis, storage, release, binding and reuptake of neurotransmitters.

40
Q

What are agonists?

A

Agonists are drugs that often mimic the action of a naturally occurring substance, binding to a receptor cell and triggering a response. Indirect agonists act as catalysts, enhancing synthesis / release of neurotransmitters.

41
Q

What are antagonists?

A

Antagonists are drugs that block or suppress agonist-mediated responses.

42
Q

What are the symptoms of Parkinson’s disease?

A

Tremors, muscular rigidity, shuffling wide-based gait leading to festination, bradykinesia (slowness of movement) and involuntary posture shifts

43
Q

What is often used to treat Parkinson’s?

A

Deep brain stimulation, where electrode probes are inserted in the brain and connected to pulse generators. The electrical stimulation mitigates cognitive and motor decay.

44
Q

What are the symptoms of schizophrenia?

A

Positive symptoms: hallucinations, delusions and disorganised thinking.

Negative symptoms: loss of empathy, poverty of speech & thought.

Cognitive symptoms: poor memory, impaired executive function

45
Q

How are schizophrenia and Parkinson’s disease related?

A

They both depend on dopamine levels. Dopamine antagonists are used to treat schizophrenia, but may result in Parkinsonian symptoms. Dopamine agonists (amphetamine, L-Dopa, cocaine) meant to treat Parkinson’s can produce schizophrenic episodes.

46
Q

What is J Olds & P Milner’s experiment about, and how does it relate to dopamine?

A

Involves starved rats who were given the option to push the lever for electrical stimulation (increased dopamine) or food, yet still chose the lever. This is known as intracranial self-stimulation, which activates dopaminergic pathways in the brain, including the nucleus accumbens (NA) and the ventral tegmental area (VTA).

47
Q

What is the human reward system? How is it activated?

A

It consists of the nucleus accumbens and ventral tegmental area (VTA). 50% of neurons in the VTA are dopaminergic, and projects to the NA and prefrontal cortex. It is activated by reinforcers such as sex, food, money, beauty, humour & drugs.

48
Q

How is complex behaviour produced?

A

The nervous system relies on the operation of networks of neurons to produce complex behaviour.

49
Q

What is the simplest behaviour-producing network?

A

The reflex, which includes simple automatic responses to stimuli, such as blinking, swallowing, pupil dilation.

50
Q

What is the difference between monosynaptic and polysynaptic reflex?

A

Monosynaptic reflex (knee-jerk) involves a singular synapse connected by the sensory and motor neurons. On the other hand, the polysynaptic reflex (withdrawal) involves multiple synapses, where the afferent neuron engages with the interneuron in the spinal cord, the inhibitory neuron and also neurons in the brain before the motor neuron.

51
Q

What is the vestibulo-ocular reflex? (VOR)

A

This reflex stabilises the gaze during head movement by focusing images on the retinas of the eyes.