Medical Dx, Acute Mgmt, and Stabilization Flashcards
Pathophysiology
SCI level determined by cause of injury and age
Non-traumatic- often in thoracic area, result in incomplete injury
Vertebral dysplasias- upper cervical
Birth trauma- CT junction
8-10 y/o- cervical spine has greater mobility d/t lig laxity, shallow facets, incomplete ossification, and underdevelopment of neck mm for size of head (i.e upper cervical injury more common in children vs adults
SCIWORA- SCI without radiographic abnormality– no signs of bone damage, 6-38% of SCI
55% tetraplegia, 45% paraplegia
Emergent Stabilization (in the field)
stabilize spine to prevent further damage
immobilization during transport and throughout all assessments and procedures
use spine board appropriate to pediatric pt
D/t increased head to torso ration in children- must modify board with occipital cutout, elevation of torso to allow for neutral spinal alignment—normal board may result in increased cervical flexion = increased injury
Diagnostic Studies
Neuro exam- determines motor and sensory level of SCI and completeness of injury
Spinal shock usually present
X-rays performed to identify any fractures/dislocations/subluxations, completed on entire spine– obtain AP and lateral views
CT and MRI- dx root impingement, cord compression, bone fragments in spinal canal, and hemorrhage
D/t high incidence of SCIWORA in children– MRI is indicated in all children (SCIWORA specifically common in children less than 10 y/o)
Surgical Stabilization
Main goal = prevent later deformity, pain , or loss of neuro function
Surgery may not be needed if can achieve alignment with traction and orthosis
Halo traction preferred and safer than tonged cervical traction– if Halo cannot be applied may need to use CTLSO or external halo orthoses
Surgery indicated if there is a penetrating injury, if traction has failed to reduce dislocation, if nerve root impingement exists, if the spine is highly unstable, or if bone fragments are compressing cauda equina
Use of orthosis following surgery
Central Cord Syndrome
Hemorrhage in central part of cervical SC = flaccid weakness in arms and strong but spastic legs, preserved BB control
Ambulation is potential goal for this pop, but hand function may be impaired depending on level of injury
Anterior Cord Syndrome
Incomplete SCI d/t damage of anterior spinal artery causing infarction to SC
Variable motor paralysis, reduced sensation of pn and temp with preserved dorsal column function
Poor prognosis for return to function
Posterior Cord Lesion
Rare
Produce selective loss of proprioception with preserved motor function
Ambulation unlikely due to loss of proprioception
Brown-Sequard Lesion
ipsilateral paralysis and proprioceptive loss
contralateral loss of pn and temp
Cause: penetrating trauma to one side of SC, stab wound
Prognosis Good for ambulation and BB control
Cauda Equina
injury at lumbosacral roots
le weakness and areflexia of legs and bladder
lesion of peripheral nerve or LMN–> may show recovery over several years d/t resolution of neurapraxia
Spinal Shock
mm are flaccid below SCI and all cutaneous and DTR are absent
State persists for hours to weeks
Over when sacral reflexes (bulbocavernous and anal reflexes) are present
Underlying Injuries and Comorbidities
TBI commonly associated with SCI ~38% of cases
Injury to brachial plexus- should be considered if MOI included any type of traction to shoulder or impingement/fracture of clavicle in presence of asymmetrical weakness in ues