Mechanisms of Activating forces Flashcards
what is somatic dysfunction?
Impaired or altered function of related components of the somatic (body framework) system: skeletal, arthrodial, and myofascial structures, and related vascular, lymphatic and neural elements.
what type of texture does a viscerosomatic SD typically have?
rubbery
what is tissue texture of arthrodial SD?
bony end feel at the restrictive barrier
what is the feel of Muscular SD
tense end feel
ropey
tight
what does SD associated wtih strain/counterstrain have?
tender points have more tenderness
what are factors that predispose people to SD?
posture
gravity
anatomical anomalies
transitional areas
muscle hyperirritability (emotional stress, infection)
trauma
compensation for other structural deficits (short leg, muscle imbalance)
what do DO’s call arthrodial somatic dysfunction?
won’t complete full motion
NOT “subluxed” “out of place” “out of joint”
2 main theories of why we have SD?
Proprioceptive
Nociceptive
usually combo of the two
what did Korr have to say about proprioceptive theory
“A muscle, by changes in the degree of activation and deactivation of its contractile mechanisms, becomes the major and highly variable impediment to mobility of the lesioned joint.”
alteration in both intrinsic and extrinsic reflexes
what is gamma gain
inappropriate activity which creates inappropriate muscle length and tone
resulting in a functionally imbalanced joint
can be helped with OMT
it is one of the determinants of the physiological motion barrier and the motion barrier of SD
what are the three types of neural feedback that provide proprioceptive input to the spinal cord about muscle length and tension in the musculoskeletal system?
Primary annulosprial endings of afferent fibers (muscle spindle)
secondary flower spray endings (muscle spindle)
golgi tendon organs
what do primary annulospiral endings transmit ?
info on the length and rate of change in length of muscles
what is the muscle spindle?
connective tissue sheath that encloses intrafusal muscle fibers with each end of the spindle attached to extrafusal muscle fibers
innervated by a single group Ia afferent fiber and a single group II afferent fiber
DOES NOT transmit info about absolute length of the muscle, just the length of the spindle relative to the length of the muscle
connected in PARALLEL
intrinsic (in the muscle itself)
what do secondary flower spray endings transmit info about?
relative muscle length
what are golgi tendon organs
specialized stretch receptors located in tendons that transmit info on muscle tension
connected in series with extrafusal fibers
contraction induces firing of the golgi tendon organs and this sends info into the spinal cord
what is the extrinsic reflex system?
anterior horn cells of the alpha and gamma efferents to the muscle receive synaptic impulses from sensory nerves originating in other muscles or organs
what supervises both intrinsic and extrinsic reflex systems?
suprasegmental reflex systems = cerebral cortex
this system is probably what is responsible for us not getting more SD than we have as well as activities of daily living
in SD the muscles and reflex activity have isolated themselves from the suprasegmental control.
example?
reciprocal inhibition of antagonist muscles (biceps/triceps)
what is spinal facilitation
areas of the spinal cord will stay hyperexcitable and hyperirritable playing a role in SD
muscles in this area of hypertonic
these areas sometimes have increased muscle activity as well as pain and tenderness when palpated
somatosomatic reflexes
defensive reflex
info coming through soma and causes reflex and get response back through motor neurons to other area of body (example→ defensive reflex such as stepping on a nail) (effecting body part and going back out and affecting another body part)
viscerovisceral reflexes
organ to another organ
digestion→ when we eat, distention of the gut causes increased contraction of the gut muscle
somatovisceral reflexes
info in from soma and affects visceral organ
irritate abdominal skin it slows down digestion→ in rats
viscerosomatic reflexes
problem in the viscera, input that goes from that organ into the spinal cord and causes reflex muscular activity of the paraspinal levels at that level of the spinal cord
example upper back pain with an MI (will probably feel tight muscles, ropey)
nociceptive theory
noxious stimulus comes in and stimulates the nociceptor
the nociceptor can then stimulate sypathetic activation, which leads to visceral effects or immune effects
the nociceptor can also stimulate spinal cord nocifensive reflexes –> leading to shortened skeletal muscle –> maintained shortened muscle–> connective tissue reorganization in shortened form (scar tissue)
why is pain generated by parts of the body that are adapting and not by parts that have impaired function?
Nociceptors are more likely to be stimulated by joint movements especially abnormal movements from adaptation or hypermobility, than by joint restriction.