MAX FAX Flashcards

1
Q

What foramen does the ophthalmic branch of the trigeminal nerve pass through?

A

superior orbital fissure

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2
Q

What foramen does the maxillary branch of the trigeminal nerve pass through?

A

Foramen rotundum

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3
Q

What foramen does the mandibular branch of the trigeminal nerve pass through?

A

Foramen ovale

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4
Q

What is the origin, insertion, innervation and function of the muscles of mastication:

A

Masseter:
o Origin – zygomatic arch
o Insertion – lateral surface and angle of mandible
o Action – elevates and deep fibres retrude mandible
o Testing – clench teeth together
o Innervation – masseteric branch of mandibular division of trigeminal nerve

  • Temporalis:
    o Origin – floor of temporal fossa
    o Insertion – coronoid process and anterior border of Ramus
    o Action – elevates and retracts mandible
    o Testing – clench teeth and palpate all fibres (anterior, middle and posterior)
    o Innervation – anterior division of deep temporal nerve branches of mandibular division of trigeminal nerve
  • Medial pterygoid:
    o Origin – superficial head (maxillary tuberosity) Deep head (Medial side of lateral pterygoid plate)
    o Insertion – medial surface of angle of mandible
    o Action – elevates and assists in protrusion of mandible
    o Testing – intra oral can be painful
    o Innervation – nerve to medial pterygoid of the mandibular division of trigeminal
    nerve
  • Lateral pterygoid:
    o Origin – lateral surface of lateral pterygoid plate and base of skull
    o Insertion – pterygoid fovea and some fibres extend into the capsule of the TMJ
    independent heads – inferior to head of consult; superior to intra articulate disc
    Action- mandibular protrusion & depression
    o Testing – response to resisted movement by putting finger far back of maxilla
    and move jaw side to side
    o Innervation – anterior division nerve to lateral pterygoid branch of mandibular division of trigeminal nerve
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5
Q

What is the pathology of a squamous cell carcinoma?

A

2 main factors involved:
* Genetic
* Carcinogens (environmental factors)

Damage alters gene expression - altering cell function.
Initiation- carcinogen causing genetic change.
Promotion- cell multifplication
Transformation- production of malignant cell.
Progression- forming malignant tumours.

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6
Q

Patient comes in with a right body mandibular fracture
- Other than pain, bruising and swelling. List 6 other signs and symptoms
associated with mandibular fractures

2020 Paper 1 Q9

A
  • Limited function (opening and lateral movements)
  • occlusal derangement = Can’t bite as normal into ICP
  • Numbness lower lip
  • Mobility of teeth/loose teeth
  • Bleeding limited to area of fracture
  • AOB
  • –when ramus is shortened by trauma (posterior teeth meet prematurely)
    —-Subcondylar (bilateral) Causes shortening In vertical height of ramus = AOB
  • Facial asymmetry
  • deviation of mandible towards opposite side to fracture
  • Bleeding in FOM = sublingual haematoma
  • Steppy deformity of the teeth.
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7
Q

Two radiographic views required for mandibular fractures

2020 Paper 1 Q9

A

2 Plain views at 90 degree angles to each other
* OPT + posteroanterior mandible

OPT- Orthopantomogram
Other radiographs:
* Occlusal
* Lateral oblique
* Towns view
* SMV
* CT scan or CBCT

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8
Q

What factors cause displacement of mandible fractures? (6)

2020 Paper 1 Q9

A
  • Direction of fracture line (Muscle can encourage or prevent displacement)
  • Opposing occlusion: prevents fracture being displaced
  • Magnitude of force
  • Mechanism of injury
  • Intact soft tissue: intact tissue = displacement unlikely
  • Other associated fractures: > 1 fracture = higher chance of displacement
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9
Q

What are 6 signs and symptoms of TMD?

A
  • Intermittent pain of several months or years duration
  • Muscle / joint / ear pain, particularly on wakening
  • Trismus / locking/limited mouth opening
  • ‘Clicking/popping’ joint noises
  • Headaches
  • intra-oral signs: linea alba and tongue scalloping
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10
Q

What 2 muscles should be palpated when querying TMD?

A

masseter

temporalis

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11
Q

What are the common causes of TMD? (8)

A
  • Myofascial pain (common. Problem with muscles due to overworking)
  • Disc displacement (common)
    – Anterior with reduction: where the disc slips forward but can move back to its original place
    – Anterior without reduction
  • Degenerative disease (less common)
    – Localised e.g. osteoarthritis
    – Generalized (Systemic) e.g. rheumatoid arthritis
  • Chronic recurrent dislocation – condyle gets stuck in front of the eminence and mouth is locked open
  • Ankylosis – condyle fused to the base of the skull (most people have a psudoankylosis)
  • Hyperplasia – one condyle grows more than the other (can be bilateral but nota s common)
  • Jaw moves to the opposite side from where the hyperplasia is
  • Neoplasia (osteochondroma, osteoma, or sarcoma)
  • Infection – can result in ankylosis
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12
Q

What 3 nerves supplies the TMJ?

A

auriculotemporal, masseteric, posterior (deep) temporal nerve
- Patients with TMJ pain can also experience discomfort in the ear as the auriculotemporal nerve also provides sensation to the external auditory meatus

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13
Q

What conservative advice is given to manage A patient with TMD?

A

Patient education!!
Counselling/reassurance: why its happening, how it happens, what the causes are, how we manage etc.

Advice: (standard approach)
* Reassurance
* Soft diet
* Masticate bilaterally
* No wide opening
* No chewing gum
* Don’t incise foods
* Cut food into small pieces
* Stop parafunctional habits e.g. nail biting, grinding
* Support mouth on opening e.g. yawning

Medication
- NSAIDs
- Muscle relaxants
- Tricyclic antidepressants (have muscle relaxant properties)
- Botox of masseter = prevents clenching (last resort tx)
- Steroids

Physical therapy
- Physiotherapy
- Massage/heat
- Acupuncture
- Relaxation
- Ultrasound therapy (not used as much)
- TENS (Transcutaneous Electronic Nerve Stimulation)
- Hypnotherapy and CBT

Splints
- Bite raising appliances
- Anterior repositioning splint e.g. wenvac or Michigan splint

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14
Q

What are the mechanisms of a bite splint using in TMD?

A

They stabilize the occlusion and improve the function of the masticatory muscles, thereby decreasing abnormal activity.
They also protect the teeth in cases of tooth grinding

  • Eliminated occlusal interference
  • Habit breaker
  • Reduces loading on TMJ
  • Prevents the join head from rotating so far posteriorly in the glenoid fossa

(same answer as above but lara order) x
Elimates occlusal interference
acts as a habit breaker
Reduces load on TMJ.
Improves function of mastiagtory muscles by decreasing the abnormal activitu and protecting the teeth in case of tooth grinding.

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15
Q

What is arthrocentesis?

A

Arthrocentesis = wash of the joint = increase lubrication

  • Under LA or GA
  • Inject lactate, hyaluronic acid and steroid into the capsule
  • Can lead to reduction of the disc and increase function

Action: Breaks fibrous adhesion and flushes away the inflammatory exudate to increase lubrication.

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16
Q

Give 2 possible surgical options for TMD?

A
  1. Menisectomy = remove the disc completely
  2. Disc plication = move the disc to correct position
  3. Eminectomy = remove part of the boney eminence
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17
Q

What are 6 signs and symptoms of Zygomatico-orbital complex fractures involving orbit floor?

A

Diplopia and restricted eye movement

paralysis of eye

numbness under the eye

pain, bruising, swelling

flat face

facial asymmetry

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18
Q

What imaging would you take to confirm Zygomatico-orbital fracture diagnosis?

A

occipitomental view at 15 and 30 degrees

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19
Q

What are the management options for Zygomatico-orbital complex fractures? (7)
- initial and correction

A

Initial:
* Exclude ocular injury
* Prophylactic AB’s
* Avoid nose blowing

  • Review once swelling subsided
  • Further radiographs (+/- CT)
  • Informed consent

Correction:
* Closed reduction +/- fixation
- howard gillies approach
* Open reduction + internal fixation
- Most corrections have ORIF

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20
Q

oral cancer
- What does dentally fit mean?

A

Patient who is free of pain & infection or future sources of pain&infection

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21
Q

OC - What is a multi disciplinary team?

A

A team of individuals from a variety of disciplines who work together to provide holistic treatment/care for a patient

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22
Q

List 4 members of an MDT for someone being treated for oral cancer?

A

Oncologist, special care dentist, Maxillofacial surgeon, speech and language therapist, physiotheraptist, radiographer

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23
Q

What risks is the patient at following radiotherapy, apart from mucositis? (5)

A
  • traumatic Ulceration,
  • mucositis
  • Fibrosis of the muscles/soft tissues = Trismus
  • xerostomia = Dry mouth from damage to the salivary glands
  • Dental caries from the dry, acidic mouth
  • dental erosion
  • periodontal disease
  • increased candida infections
  • reactivation of herpes simplex virus
  • Endarteritis obliterans (damge to the blood vessels supplying the bone) = osteoradionecrosis
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24
Q

What are the oral side effects of chemotherapy? (5)

A

Cytotoxic
* mucositis
* Decreased salivary gland function (Xerostomia)

Bone marrow supression causing:
* Defective haemostasis (Decreased neutrophils/ platelets/RBCs) increasing bleeding risk.
* Greater infection risk (Oral candidiasis/ Herpes simplex)

Neurological
* Trismus
* Joint pain.

Nausea/vomiting = erosion

Any teeth mineralising during chemotherapy- Disturbed root formation/ microdontia/ crown hyperplasia/ Hypodontia.

General conditions:
reduced RBC = anaemia
reduced WBC = leukopenia
reduced platelets = thrombocytopenia
reduced neutrophils = neutropenia
hairloss
fatigue/tiredness

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25
Q

What are the grades of mucositis?

A

Grade 0 = no alterations

grade 1 = pain and erythema

grade 2 = erythema and ulcers

grade 3 = ulcers (liquid diet)

grade 4 = unable to feed

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26
Q

How is mucositis managed? (3)

A

Avoid:
- Smoking
- Spirits
- Spicy food
- Tea and coffee
- Non-prescription mouthwash (esp those with alcohol)

Topical:
- Lignocaine gel
- Saline mouthwash (good for use in radiotherapy)
- Sodium bicarbonate mouthwashes (good for use in radiotherapy)
- Caphosol (manmade mucous/saliva sub)
- Tea tree oil mouthwash
- Cooling using ice cubes

low level laser light therapy

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27
Q

How can mandibular fractures be classified? (7)

A
  1. Involvement of the surrounding soft tissue;
    * Simple: surrounding soft tissue intact
    * Compound: fracture exposed to the surrounding environment (soft tissue breached)
    Need AB’s
    * Comminuted: multiple small fragments e.g. from gunshot
  2. No of fractures
    * Single
    * Double
    * Multiple
  3. side of fracture
    * Unilateral
    * Bilateral
  4. Site of fracture
    * Angle
    * Below condyle (subcondyle)
    * Parasymphyseal (in the middle)
    * Body
    * Ramus
    * Coronoid
    * Condylar fracture (intra/extra caspular)
    * Alveolar process
  5. Direction of fracture line
    * Favourable: direction of fracture line limits the displacement of the fracture by the action of the surrounding muscles
    * Unfavourable: direction of fracture line encourages the displacement of the fracture
  6. Specific fractures
    * Green stick fracture: soft bone (children) very unlikely to completely fracture = still attachment in one of the cortices
    * Pathological: fracture caused by pathology
    e.g. osteoporosis, osteomyelitis, Padgets, expanding cystic lesion
  7. Displacement of the fracture
    * Displaced: requires surgical tx
    * Undisplaced: may not require surgical intervention
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28
Q

How is a mandibular fracture managed?

2020 Paper 1 Q9

A

Control pain and infection - analgesia and antibiotic (if a compound fracture)

  1. Undisplaced = usually no tx
  2. Displaced or mobile fracture = surgical intervention
    a) Closed reduction + fixation: reduce the fractured segments to their normal anatomical orientation without exposing the fracture line (we judge it using Intermaxillary fixation = if teeth go into ICP )

b) Open reduction + internal fixation
Reflect ST and expose the bone to reduce the segment
Open reduction and internal fixation (ORIF) = most common

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29
Q

What are the signs and symptoms of maxillary fractures? (7) *** have we done

A

Pain, asymmetry, diplopia, altered sensation, swelling, nose bleed, mobility.

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30
Q

Describe the Le Fort Classifications.

A

Type 1 – horizontal maxillary fracture spreading teeth from upper face (floating palate)
Type 2 – pyramidal fracture involving nasofrontal suture (floating maxilla)
Type 3 – transverse craniofacial disjunction where maxilla is detached from base
of skill (floating face)

Surgical Classifications:
Le Fort I osteotomy (most common)
- LF1 = Cut the maxilla from the base of the skull and move it in any direction
- LF2 = maxilla and nose moved together
- LF3 = Maxilla, nose and cheek bone moved together

31
Q

What radiographs would you use to image a maxillary fracture?

A

occipitomental view 15* & 30*

32
Q

how are maxillary fractures managed?

A
33
Q

What is a cyst?

A

A pathological cavity having fluid, semi-fluid or gaseous contents & which is not created by the accumulation of pus.
Allows it to gradually increase in size

34
Q

Give 2 inflammatory cysts (4)

A

(Odontogenic)
radicular cysts (&residual cysts)

Inflammatory collateral cysts
- paradental cyst
- buccal bifurctaion cyst

35
Q

Give 2 developmental cysts (4)

A

Odontogenic:
dentigerous cysts
odontogenic keratocysts
lateral periodontal cyst

non-dontogenic
nasopalatine duct cyst

36
Q

Give 2 non-odontologenic cysts (3)

A

nasopalatine duct cyst

solitary bone cyst

aneurysmal bone cysts

37
Q

Give 2 common treatment options for cysts with advantages and disadvantages for both?

A
  1. Enucleation

What is it?
All of the cystic lesion is removed (entire cyst lining removed with the associated tooth/root if present)

Treatment of choice for most cysts

What are the Advantages?
* Whole lining can be examined pathologically
* Primary closure
* Little aftercare needed

What are the Contraindications/disadvantages?
* Risk of mandibular fracture with very large cysts
* (Dentigerous cyst) wish to preserve tooth
* Old age/ill health can’t be put under GA
* Clot-filled cavity may become infected
* Incomplete removal of lining may lead to recurrence
* Damage to adjacent structures
* Daughter cysts in the keratocyst lining – to remove all of these would cause damage to adjacent structures/anatomy = have to use marsupialisation

  1. Marsupialisation (less invasive and can be done under LA)
    Requires cooperation
  • Creation of a surgical window in the wall of the cyst, removing the contents of the cyst & suturing the cyst wall to the surrounding epithelium
  • Encourages the cyst to decrease in size & may be followed by enucleation at a later date
  • Always a window present with something inside for up to 6 months e.g. an obturator to prevent tissues growing back over it

What are the Advantages?
* Simple to perform
* May spare vital structures

What are the Contraindications/disadvantages?
* Opening may close & cyst may reform
* Complete lining not available for histology
* Difficult to keep clean & lots of aftercare needed
* Long time to fill in

38
Q

How does a radicular cyst develop? (6)

A

Always associated with a non-vital tooth and always attached to a tooth

  • Initiated by chronic inflammation at apex of tooth due to pulp necrosis
  • Proliferating epithelium with central necrosis (bringing fluid in)
    OR epithelium surrounds fluid area
  • Continued growth
  • Osmotic effect with semi-permeable wall
  • Cytokine mediated growth
39
Q

How does a radicular cyst appear histologically and radiographically?

A

Radiographically:
* Well-defined, round/oval radiolucency
* Corticated margin continuous with lamina dura of non-vital tooth
* Larger lesions may displace adjacent structures
* Long-standing lesions may cause external root resorption &/or contain dystrophic calcification

Histologically:
- Epithelial lining (often incomplete) = non k stratified squamous epithelium
- Connective tissue capsule
- Inflammation in capsule
Dark blue dots nuclei of inflammatory cells

Cholesterol clefts
Mucous metaplsia
Hyaline/rushton bodies
(Niamh)

40
Q

Orthognathic surgery
- What are the indications for orthognathic surgery? (3)

A
  • Pt with aesthetic or functional concerns
  • Growth completed
  • Moderate/Severe skeletal discrepancy
  • A-P
  • Transverse
  • Vertical
41
Q
  • What are the risks of orthognathic surgery? (6)
A

Relapse
Nerve damage
Bleeding
Unobtainable results for patients with high expectations
Infections
TMJD

42
Q

What investigators are carried out before orthognathic surgery? (3)

A

Radiographs:
* OPT
* Cephalographs
* Periapicals
* Occlusal
* CT scanning
All patients have a CBCT nowadays.

  • Study Models (casts)
  • Photographic (2D, 3D)
    Stereophotogrammetry = 3D imaging
43
Q

Give 2 types of mandibular orthognathic surgery

A
  • Advancement = Sagittal split mandibular osteotomy
  • Ramus is split from the body to allow the anterior part of the (body and the dentition) mandible to move in any direction.
  • Equivalent of the LF1 osteotomy
  • vertical subsigmoidal osteotomy (VSSO)
44
Q

Give 2 types of maxillary orthognathic surgery

A

LF 1-3
* Le Fort I osteotomy (most common)
- Cut the maxilla from the base of the skull and move it in any direction
- LF2 = maxilla and nose moved together
- LF3 = Maxilla, nose and cheek bone moved together

Anterior maxillary osteotomy
- Can’t move the whole maxilla back because of the pterygoid plates, only the anterior part is moved back

45
Q

What might patient complain of it they have a sialolith? (5)

A

pain/pressure before mealtime

thick viscous saliva

salty saliva

dry mouth

swelling of duct which is fixed

46
Q

What gland/duct is most commonly affected by sialolith and why? (3)

A

Submandibular most commonly than parotid due to position of gland as;
the duct has a long uphill Path of saliva secretion = flow is slow and saliva can stagnate
Saliva is thicker and more mucous like
hilum present (90 degree bend) in some cases

47
Q

How do you manage siathoths

A

To remove the stone:
* It must be mobile
* Located
- within the lumen on the main duct distal to the posterior border of the
mylohyoid
-Distal to the hilum
-At the anterior border of the parotid gland.

If it is inflamed/ enlarged then refer patient to max fax.

48
Q

What is the nerve supply for the submandibular gland?

A

Parasympathetic innervation from the chorda tympani branch of facial nerve which unifies with lingual branch of mandibular nerve at the submandibular ganglion.

49
Q

What secretions do the submandibular gland produce?

A

Mixed - serous and mucous

50
Q

What is the innervation of the parotid gland?

A

Sensory innervation – auriculotemporal nerve and greater auricular nerve
o Parasympathetic – glossopharyngeal nerve and auriculotemporal nerve

51
Q

What is the nerve supply of the sublingual gland?

A

Parasympathetic – chorda tympani of facial nerve which unifies with lingual branch of mandibular nerve at submandibular ganglion.

52
Q

What secretions do the sublingual gland produce?

A

predominantly mucous but some serous

53
Q

What secretions does the parotid gland produce?

A

Serous

54
Q

Patient with extra oral large swelling
What information is required when taking a history and investigating a patent with swelling before looking in the mouth? (11)

A

How long swelling has been present?(continued swelling after 48hrs= infection)
cause - how did it start
Pain Hx- SOCRATES
fluctuation in size
Feeling generally unwell
Problems with breathing/swallowing (straight to A&E)
Palpation (firm/mobile)
Pus
Heat
Colour.

Fever-
Any suspicion of sepsis?-urgent referral to A &E.

MH- any allergies
DH- any previous treatment that could have caused this ?

Record:
Temperature
Respiratory rate
Heart rate
pulse
O2 saturation

55
Q

What things would you note about a facial swelling? (6)

A

size
location
Firm or soft
if mandible can be palpated
is it causing orbital swelling
encroaching on the soft tissues of the neck = airway compromised

56
Q

What is the SIRS criteria?

A

SIRS-systemic inflammatory response syndrome

  • Increased HR
    pulse >90/min
  • Increased respiratory rate
    > 20/min
  • temperature < 36 or >38 degrees
  • WCC
    < 4000 or > 12000/mm3
57
Q

Px has a large extra-oral swelling - You suspect sepsis. What do you do? (2)

What is done in secondary care?(6)

A
  • Spot and Diagnose (Take OBS e.g. O2 sat)
  • Urgent call for advice/referral to Max Fax

Hospital:
- Sepsis 6
1. Give high flow oxygen
2. Take blood cultures
3. Give IV AB’s
4. Give a fluid challenge
5. Measure lactate
6. Measure urine output

58
Q

What is Ludwig’s angina?

List the E/O & I/O features. (6)

A

Bilateral cellulitis (infection/swelling) of the sublingual and submandibular spaces (from dental infection)
- Swelling on both sides
= compromise airway

Features:
What are the extra-oral features?
- Diffuse redness
- Bilateral swelling in submandibular region

What are the intra-oral features?
- Raised tongue
- Difficulty breathing
- Difficulty swallowing
- Drooling

59
Q

Name 4 maxillary and 4 mandibular spaces

A

Maxillary Spaces:
buccal space.
the palate
(maxillary sinus)
infraorbital space
Infratemporal region

Mandibular Spaces:
* Mental space
* Submental space
* Buccal space
* Submasseteric space
* Sublingual space
* Submandibular space
* Lateral pharyngeal space

60
Q

What post op advice should be given to patients after Zygomatico-orbital fracture? (3)

A

Pain management, avoid nose blowing, finish course of antibiotics.

?observe for retrobulbar haemorrhage?

61
Q

Where does an odontogenic keratocyst develop from?

A

Rests of Serres from dental lamina.

62
Q

How do odontogenic keratocysts appear histologically? (4)

A
  • Wall - Daughter/satellite cysts found within the wall
  • epithelial lining: keratinised– parakeratinised
    Multicentric growth – parts of epithelial lining grows faster than other sites and have finger like projections (not a nice balloon shape)
  • No rete pegs within the epithelium (allows the epithelium to separate from the wall)
  • Palisading arrangement (basal cells at same height)
63
Q

How do odontogenic keratocysts appear radiographically? (5)

A
  • Well defined and well corticated radiolucent lesion
  • A/P growth along the body, angle and ramus of the mandible
  • Truly multilocular
  • Can cause displacement of adjacent teeth
  • Scalloped margins

**The cyst has no specific relationship to teeth **

64
Q

Why are odontogenic keratocysts problematic? (3)

A

High risk of recurrence
Multicentric growth – parts of epithelial lining grows faster than other sites and have finger like projections
If projections left behind = reoccurrence
Daughter/satellite cysts found within the wall – if left behind they form a new keratocyst

Risk To IDN - pressure

pathological fracture of jaw

65
Q

What syndrome is associated with the presence of many odontogenic keratocysts?

A

Basal cell naevus syndrome/Gorlin-Goltz syndrome

66
Q

What does a radicular cyst develop from?

A

Epithelial rests of Malassez from HERS (hertwig’s epithelial root sheath)

67
Q

What does a dentigerous cyst develop from?

A

Dental follicle at reduced enamel epithelium and crown.

68
Q

How do dentigerous cysts appear histologically? (4)

A
  • Thin non-keratinised stratified squamous epithelium
  • May resemble radicular cyst if inflamed - however no inflammation usually
  • Attached to the ACJ of the unerupted tooth
  • flat basement membrane
  • no rete pegs
69
Q

How do dentigerous cysts appear radiologically? (7)

A
  • Radiolucent with well-defined corticated margins which
  • associated with cemento-enamel junction of an unerupted/impacted tooth (neck of the tooth)
  • Larger cysts may begin to envelope root of tooth
  • May displace the involved tooth
  • Tend to be symmetrical initially - Larger cysts may begin to expand unilaterally
  • Variable displacement of cortical bone (i.e. bony expansion)
  • Unilocular
70
Q

where are dentigerous cysts most commonly found?

A

Unerupted M3M and U3s.

71
Q

Name 1 epithelial derived odontogenic tumour.

What is it like histologically?

A

Ameloblastoma
folicular ameloblastoma
- Islands present within a fibrous tissue background
- Islands Bordered by cells that resemble the ameloblast e.g. columnar cells with a dark staining nucleus
- Tissues in the middle of the follicle are loose (resemble stellate reticulum of tooth germ)
- Can be cystic changes within the follicle
- Other changes within the stellate reticulum like tissue involve squamous metaplasia or cells become granular etc
- No connective tissue capsule (in any of the types) = cells can grow and infiltrate into the jaw bone = reason for high recurrence rate

plexiform ameloblastoma:
- Amelobastoma cells arranged in strands
- Between the strands there is stellate reticulum like tissue
- Some Cells can form back to back with hardly any SR like tissue between
- No connective tissue capsule (in any of the types) = cells can grow and infiltrate inot the jaw bone = reason for high recurrence rate

adenomatoid odontogenic tumour:
- Epithelial cells arrange in duct like structures, sheets or rosette appearance
- There is a degree of calcification which are reflected in the radiological appearance
- Has a well developed fibrous tissue capsule surrounding therefore removal is easier and has a lower recurrence rate

calcifying epithelial odontogenic tumour
* Loose myxoid tissue (connective tissue) with stellate cells within
* May contain islands of inactive odontogenic epithelium (vital but inactive)
* No fibrous tissue capsule  locally invasive into adjacent bone and can cause problems with surgical removal and recurrence

72
Q

Name a mixed epithelium tumour.

A

Odontoma

73
Q

Name a mesenchymal tumour

A

odontogenic myxoma