Maternal Infant Care and Anomalies Flashcards

1
Q

Intimate Partner Violence

Level 1,2,3

A

Level 1 – Maltreatment is occasional; consists of slapping, punching, kicking, verbal maltreatment. Contusions occur.

Level II – Maltreatment is becoming more frequent; beatings are sustained and cause fractures, such as broken jaw or rib fractures.

Level III – Maltreatment is even more frequent, perhaps daily. A weapon, such as a gun, baseball bat, or broom handle may be used. Permanent disability or death from injuries, such as intracranial hemorrhage or concussion may occur.

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2
Q

Intimate Partner Violence

Cycle of Violence

A

Build up phase - increased tension
Stand over phase - control, fear
Explosion
Remorse phase - justification, minimilization, guilt
Pursuit phase – pursuit and promises, helplessness, threats
Honeymoon phase - enmeshment, denial of previous difficulties

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3
Q

Characteristics of the abuser and the abused

A

1 cause of death during pregnancy is homicide from intimate partner violence

Prevalence of IPV witnessed
Abused as a child
Alcohol may contribute -> poor impulse control
Learned helplessness

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4
Q

Partner Violence Screening tool

Advocacy – universal screening of all patients at every health care encounter

A

Have you been hit, kicked, punched, or otherwise hurt by someone in the past year?

Do you feel safe in your current relationship?

Is there a partner from a previous relationship who is making you feel unsafe now?

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5
Q

Transcultural Care

A

Health care values and beliefs
Health maintenance during pregnancy
Modesty, Fate
Childbirth, Childrearing

Health care practices
Rituals for preventing illness, restoring health
Care of anatomy & physiology of women

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6
Q

Assessing Families

Family centered care

A

Structure, parent style, communication, religion, culture, socioeconomic, morbidity, mortality

in delivery room – father, significant other, grandparents
cultural sensitivity
Cultural competence

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7
Q

Inspect for Obvious Anomalies

A
Eyes-Cataract, Retinablastoma
Nose-Choanal Atresia
Mouth-Cleft lip, cleft palate
Tracheoesophageal fistula
Abdominal wall defects
    Omphalocele, Gastroschesis
    Anal malformations
Biliary Atresia
Cardiac Anomalies
Neural tube defects
    Anencephaly
    Encephalocele
    Meningocele
Musculoskeletal defects
   Hip dysplasia
   Talipes varus
   Polydactyl
   Osteogenesis imperfecta
   Amniotic band injuries
GU
   Bladder extrophy
   Intersex
   Hypospadias, Epispadias
   Phimosis
Trisomy 21, 18, 13, Fragile X
Fetal Alcohol Syndrome
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8
Q

Retinoblastoma

A
“Red reflex”
Malignancy of retina
Unilateral or bilateral (25%)
Autosomal dominant gene
		in 40% of children

Chemotherapy, Radiation, Laser photo-coagulation
Surgery -> enucleation

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9
Q
Congenital Cataracts
Maternal infections (11)
A
rubella (the most common cause)
rubeola
chicken pox
cytomegalovirus
herpes simplex
herpes zoster
poliomyelitis
influenza
Epstein-Barr virus
syphilis
toxoplasmosis

tetracycline antibiotics – correlation

EASILY TX

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10
Q

Choanal Atresia

A

Nasal passages are blocked by bone, soft tissue
70% are unilateral
Most are not dx at delivery
If bilateral = respiratory distress

Tx Stents are placed in nares

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11
Q

Cleft lip

Integrate nursing interventions (5)

A

Happens at about 6-8 weeks gestation

Respiratory status
Feeding behaviors
Parent/infant interactions - bonding
Skin integrity, Oral hygiene
Prevention of Infection

Breastfeed? YES
Hard? Yes

Cocaine increases incidence

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12
Q

Cleft lip

Treatment

A

Surgery, based on severity

Modified feeding techniques or devices

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13
Q

Feeding Difficulties

ESSR

A
ESSR
E = Enlarge the nipple
S = Stimulate the suck reflex
S = Swallow fluid appropriately
R = Rest when infant signals with facial expression

Lamb’s nipple with X
Haberman

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14
Q
Esophageal Atresia (EA) and Tracheoesophageal Fistula (TEF)
Esophagus ends as...
A

Esophagus and trachea do not develop as parallel tracts

Esophagus ends as either…
Blind pouch (baby would need a G-tube since may not stretch down to reach stomach)
Connected to trachea by a fistula

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15
Q

TEF and EA Clinical Manifestations

5

A

“The Three C’s”: coughing, choking, cyanosis
Excessive salivation and drooling: frothy mucus
Apnea
Respiratory distress after eating
Abdominal distention

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16
Q

TEF and EA – Diagnostic testing and treatment plan

A

CXR -> radio-opaque or
Pediatric surgeon endoscopy or bronchoscopy

Surgery asap

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17
Q

TEF and EA – Nursing Interventions – Prioritize
Airway Management, Aspiration Precautions
Impaired Gas Exchange

A

Ensure NPO (nothing per os) preoperatively.
Assess Emma’s respiratory status for rate, depth, and ease, and breath sounds q 2-4 hrs and before and after suctioning
Assess Emma’s oxygen saturation level continuously
Elevate Emma’s head of bed (HOB) 30
Administer supplemental oxygen as indicated.
Perform oropharyngeal suctioning as appropriate.
Analyze Emma’s ABG, confer with provider

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18
Q

Abdominal Wall Defects
Two Variations of abdominal hernias
Omphalacele

A

Omphalacele
Intra-abdominal contents herniate through umbilical cord
Covered with peritoneal membrane

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19
Q

Abdominal Wall Defects
Two Variations of abdominal hernias
Gastroschisis

A

Abdominal organs herniate through abdominal wall
Not covered with peritoneal membrane

Stress on respiratory system and may have compromise

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20
Q

Anorectal Malformations

Types

A

Common congenital defects
Minor to complex
Occur in isolation or with other defects

Types
Anal stenosis - narrowing
Anal atresia - absent -> blind pouch or fistulas
Imperforate anus - blind pouch or membrane
Fistula – recto-urethra, recto-vaginal, recto…

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21
Q

Biliary Atresia

A

Absence or constriction of common bile duct
Bile cannot flow from liver into duodenum
Results in
Cholestasis
Fibrosis
Cirrhosis
Death

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22
Q

Clinical Manifestations of biliary atresia

A
Bile backup in liver
   Inflammation, edema, hepatic degeneration
   Malabsorption of fats and vitamins
Jaundice
Dark urine
Acholic stools = WHITE STOOLS
Weight loss
Irritability
Enlarged liver and abdomen
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23
Q

CF: Impermeable Epithelial Cells

A

Excessive mucous production in bronchioles and in pancreas, bile ducts, and small intestine

Cystic fibrosis is a hereditary disorder characterized by lung congestion and infection and malabsorption of nutrients

Instead of producing thin mucous, they produce thick mucous that leads to lung congestion

24
Q

CFTR gene: autosomal recessive

A

Important Facts:
Both parents contribute the altered gene
Each pregnancy has 25% chance of developing altered gene
Higher incidence in Caucasians and Ashkenazi Jews

Prenatal tests moms are getting blood drawn to test for CF gene 00

25
Q

CF Initial Symptom

A

meconium ileus – may be first sign of CF
No stool in 24 hours – obstruction because of thick mucous in intestine gets impacted

Steatorrhea – bulky, frothy, foul-smelling stool

26
Q

Fetal circulation

A

Ductus arteriosus
Foramen ovale
Ductus venosus

ALL CLOSE

27
Q

Factors associated with CHD
Prenatal
Genetic

A
Prenatal
Maternal insulin-dependent diabetes
Maternal rubella
Maternal alcoholism
Maternal age > 40 yrs
Genetic
Chromosomal – 50% risk Down Syndrome
Sibling with heart defect
Parent with CHD
Other non-cardiac congenital anomalies

Sx. Poor oxygenation, Murmur

28
Q

Diagnosing Heart Anomalies

A

Echocardiogram

Cardiac catheterization

29
Q

CHD
Acyanotic
Increased pulmonary blood flow
Obstructed blood flow

Cyanotic
Decreased pulmonary blood flow
Mixed blood flow

A

Acyanotic
Increased pulmonary blood flow –> VSD, ASD, PDA, AVC
Ventricular septal defect, atrial septal defect, patent ductus arteriosus, atrioventricular canal

Obstructed blood flow –> COA, AS
Coarctation of aorta, aortic stenosis

Cyanotic
Decreased pulmonary blood flow –> TOF, PS, T/PA
Mixed blood flow –> TGA, HLHS, TAPVR, TA
Transposition of great arteries, hypoplastic left heart syndrome,

30
Q

Ventricular Septal Defect

A

Small or large opening in septum between left and right ventricles
Left to right shunting

31
Q

VSD Assessment

A

Tachypnea, dyspnea
Poor growth, reduced fluid intake
Frequent respiratory infections
Onset of Heart Failure

32
Q

Atrial Septal Defect

A

Small or large opening in septum between the left and right atria
Foramen ovale doesn’t close

33
Q

Patent Ductus Arteriosus

A

Connection between the aorta and the pulmonary artery

Blood flows from the aorta (left side) to the pulmonary artery (right side) increasing blood flow to the lungs

34
Q

Coarctation of the Aorta

A

Aorta narrows, usually near the ductus arteriosus, obstructing blood flow
Ejection click
Systolic murmur

Persistent hypertension is common – restenosis can occur

35
Q

Transposition of the Great Arteries

A

Parallel circulation
Aorta originates from the right ventricle
Pulmonary artery originates from the left ventricle
Prostaglandin to maintain PDA

36
Q

Genetics and environment +

Folic acid deficiency can lead to…

A

Anencephaly
Encephalocele
Spina Bifida Occulta
Spina bifida cystica

37
Q

Head deformities

A

Microcephaly – small skull

Anencephaly – skull does not form over brain

38
Q

Neural tube defects

A

Encephalocele

Nasoencephalocele

39
Q

Pathophysiology of Neural Tube Defects

Prenatal Dx

A

In first few weeks:
Neural Tube fails to close, or
Neural Tube splits after closing

Prenatal Dx
Ultrasound
Elevated AFP

40
Q

Spina Bifida (3)

A

Usually cannot identify which lesion from viewing exterior

spinda biffida occulta
- Can be fistula or fissure

Meningocele
Sac contains meninges (membranes) and CSF
No spinal cord abnormalities

Myelomeningocele
Usually in lumbar area but can occur anywhere on spinal column
Impact depends on location
Infection, lots of compromise

Last two Spina Bifida Cystica

41
Q

Impact of neural tube defects
L2
S3

A

Below L2
Partial paralysis of lower extremities
Incontinence

Below S3
No motor impairment
May be incontinent or have some control

42
Q

Nursing Interventions

Pre-op (5) for neural tube defects

A
Pre-Op
Cover sac with warm NS sterile dsg
Monitor for CSF leakage
Prone with knees slightly flexed
Assess bowel, bladder function
Monitor for signs of infection

Family Centered Care
Involve parents in care
Hold the baby

43
Q

Developmental Dysplasia of Hip

Ortolani’s maneuver – physical exam test for hip dysplasia

A

Instability
Dislocation
Subluxation
Acetabular dysplasia

Pavlik harness –dynamic splint that allows movement

44
Q

Talipes Varus

A

aka club foot

45
Q

Polydactyly

A

extra digits

46
Q

Osteogenesis Imperfecta

A

Genetic disorder - “brittle bone disease”

Autosomal dominant - affecting production of collagen, the major protein of the body’s connective tissue
Less than normal or poor collagen leads to weak bones that fracture easy
4 types of OI, mildest may not be dx all until a routine xray discloses multiple fractures

47
Q

Hypospadias and Epispadias

A

Epispadias
“top” of the penis
(dorsal)
Ends in an opening at the upper aspect of the penis

Hypospadias
“under” the penis
(ventral)
A condition in which the opening of the penis is on the underside rather than the tip.

48
Q

Phimosis

Bladder Exstrophy

A
Foreskin doesn’t retract
   Anomaly vs Develop. Delay
 Tx with cortisone cream
 Surgical intervention:
		Circumcision for adults

Bladder exstrophy – protrusion of the urinary bladder through a defect in the abdominal wall

Intersex
ambiguous genitalia and/or gender chromosomes

49
Q

Down Syndrome

Phenotype - observable

A

Trisomy 21
Advanced maternal age
Advanced paternal age
Ultrasound, amniocentesis

Nuchal fold translucency
Epicanthal eye folds
Simian crease on palm
Flat nose
Wide, short neck
Hypotonia
50
Q

Edwards Syndrome

Ultrasound

A
Trisomy 18
90% stillbirth
Rare to live beyond first 1 yr
Advanced maternal age
Advanced paternal age

Ultrasound:
Microcephaly, small jaw
Low set ears
Rocker feet

Other anomalies: cleft lip/palate, cardiac, meningocele, kidney,

51
Q

Patau Syndrome

A

Trisomy 13
90% stillbirth
Rare to live beyond first 1 yr

Multiple anomalies

Ultrasound:
Microcephaly, sloping forehead
Low set ears
Rocker feet

52
Q

Fragile X

A

X chromosome disorder
Neural development impaired due to a malfunctioning protein in the gene
Female carriers

Phenotype - Facial Characteristics
Long face
Prominent jaw
Large ears
Strabismus
53
Q

Fetal Alcohol Syndrome

A

Most common cause
of intellectual disability

Phenotype
Small eye opening
Flat nasal bridge
Short upturned nose
Smooth philtrum
Thin vermilion

Infant Behavior
Dysphagia

54
Q

Prenatal Core Measures

A

Elective delivery
Induced deliveries less than 39 weeks gestation
Why? There is a higher rate of c/s and neonatal morbidity and mortality when patients are induced when they are less than 39 weeks.

Cesarean section
Deliveries by c/s
Why? C/S are a riskier delivery procedure with a higher rate of maternal and neonatal morbidity and mortality.

Antenatal steroids
Preterm laboring patients that received antenatal steroids
Why? Antenatal steroids reduce the risk and incidence of premature neonatal morbidity and mortality from respiratory distress syndrome.

Health-care associated bloodstream infections in newborns
Newborns with HCA bloodstream infections
Why? High neonatal morbidity and mortality from preventable infections (handwashing, sterile technique).

Exclusive breast milk feeding
Newborns fed breast milk only from delivery
Why? The benefits to newborns from breast milk feeding are significant

55
Q

Amniotic Band Syndrome

A

Amniotic Band Syndrome occurs when the unborn baby (fetus) becomes entangled in fibrous string-like amniotic bands in the womb, restricting blood flow and affecting the baby’s development.
Lose fingers, toes, cleft lip, club feet