Maternal Flashcards
definition and etiology of maternal mortality
death while pregnant or within 42 days of TOP from a cause related to or aggravated by the pregnancy
CVD, suicide, ectopic, HTN disorders, haemorrhage, sepsis, DVT, PE, AFE
Mx of GORD
non pharm - don’t over eat, avoid acidic and spicy food, avoid caffeine, smoking and alcohol
pharm - antacids, PPI, histamine receptor antagonists
Mx of constipation
metamucil
movicol
coloxyl and senna
Mx of back pain
non pharm - light exercise, physiotherapy, hot and cold packs
pharm - paracetamol (avoid NSAIDs and aspirin)
Clinical features of obstetric cholestasis
pruritus of hands and feet without rash
rarely other biliary symptoms like jaundice, dark urine, pale stool
clinical features of acute fatty liver of pregnancy
malaise N&V abdominal pain jaundice acute liver failure
anaemia defintion in pregnancy
1st trimester <110
2nd and 3rd <105
postpartum <100
maternal anaemia - complications for foetus and mother
foetus - IUGR, prematurity, B12 deficiency causes neuro deficits
maternal - fatigue, SOB, dizziness, lack of reserve for PPH
Mx of anaemia in pregnancy
- how to correct ie meds and diet
- SE of medication
- F/U
iron supplementation 100mg/day
diet - leafy greens, red meat, vitamin C, avoid coffee and tea
SE PO iron - abdo discomfort, constipation, black stool, N&V
F/U: Hb 2/52 until corrected, continue supplements until 6 weeks postpartum
complications and management of varicella in pregnancy
complications
- maternal varicella pneumonia, neurological spread
- foetal congenital varicella syndrome
Mx in non immune mother
<96 hours from exposure, VZV Ig
>96 hours acyclovir PO
mother has varicella - acyclovir PO, USS monitoring, C/S if foetal or maternal compromise
Parvovirus B19 complications and management
foetus - miscarriage, anaemia, heart failure and death (hydrops fetalis)
no treatment available - supportive, USS monitoring
Hep B and C management in pregnancy
hep B - 3rd trimester medication to reduce viral load
Hep C - medication NOT recommended in pregnancy, wait until finished breast feeding
both - clean skin of neonate prior to injections, no scalp electrode, no foetal scalp sampling
pathophysiology of GDM
placenta secretes anti-insulin hormones ie glucagon, cortisol, lactose
maternal anti-insulin hormones increase ie thyroid hormone, cortisol
insulin resistance –> increased pancreatic production of insulin
not all women have enough reserve to increase insulin production –> GDM
pathophysiology of hypoglycaemia in the neonate born to mother with GDM
maternal glucose crosses placenta but insulin does not –> foetus increases insulin production
post partum - no maternal glucose, remaining increased insulins –> hypoglycaemia
risk factors for GDM
age >40 personal hx of GDM family hx of diabetes PCOS obesity multiple pregnancy ATSI steroids antipsychotics