Management of breathlessness Flashcards

1
Q

definitions- tachypnoea, hyperpnoea, hyperventilation

A

T- rapid breathingq, Hyper- increased ventilation in response to metabolic requirements (exercise), ventilation- ventilation in excess of metabolic requirements

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2
Q

definitions- dyspnoea, breathlessness,

A

dyspnea- a subjective term generally applied to the unpleasant sensation of an awareness of breathing discomfort- breathing laboured or distressing
breathlessness- one of many descriptions used by patients to convey their experience of dyspnea- is an awareness of the intensity of breathing or suggests unrewarding respiration/chest tightness or inability to get air in

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3
Q

what is work of breathing

A

work done to overcome the resistive forces of the airways, lungs and chest wall.

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4
Q

during quiet respiration

A

the WOB is performed entirely by the inspiratory muscles, expiration is passive, powered by elastic recoil of the lungs, as breathing becomes more difficult the muscles work harder and the WOB increases

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5
Q

conditions that affect the efficiency of respiratory muscles

A

increased WOB- emphysema,a post op patients (GA), rib fractures, kyphoscoliosis, obesity and pregnancy, any cardiac disease

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6
Q

dyspnea/breathlessness- influenced by

A

perceived threats to respiratory homeostasis are unpleasant and accompanied by emotional responses, breathlessness and be perceived as life threatening, dyspnea is affected by- psychological state/ experiences/memory/fear/anxiety/depression/ anger/effort/discomfort

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7
Q

measures of dyspnoea/breathlessness

A

modified borg scale of perceived breathlessness, dyspnea intensity can be easily quantified

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8
Q

other causes of breathlessness

A

increased metabolic rate- increased ventilation e.g. fever, exercise
cardio-vascular issues- inadequate cardiac output, anaemia, deconditioning- lactate accumulates at low exercise levels causing increased ventilation, perfusion limitations- large V/Q mismatch due to wasted ventilation

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9
Q

what is anaemia

A

affects carrying capacity of haemoglobin, present with breathlessness on minimal activity, can’t be treated by physios

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10
Q

other causes of breathlessness

A

metabolic, neurogenic, neuromuscular

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11
Q

mechanical causes of increased WOB- increased resistive load

A

pathology- obstructive airway disease, asthma, chest infection, lung tumour
problem- increased secretions, inflammation in the airway, bronchospasm, obstruction in the airway

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12
Q

mechanical causes of increased WOB- increased elastic load

A

pathology- fibrotic lung, surfactant depletion, hyperinflation, pregnancy, distended abdomen, obesity, abdominal surgery, kyphoscoliosis, ankylosing spondylitis
problem- reduction in lung compliance increases the inspiratory muscle work required to overcome the elastic recoil of lungs, increase insp muscle work, increased alveolar surface tension, reduction in chest wall compliance

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13
Q

mechanical causes of increased WOB- decreased energy supply

A

pathology- eating difficulties, hypovolemic shock

problem- malnutrition, lack of perfusion to the respiratory muscles

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14
Q

mechanical causes of increased WOB- increased drive to breath

A

pathology- parenchymal disorders- pneumonia or fibrosis, acidosis and anemia
problem- stimulates nerve impulses from interstitial receptors increasing drive to breathe

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15
Q

mechanical causes of increased WOB- respiratory muscle dysfunction

A

pathology- neuromuscular disorders (MND, MS, GBS), chronic lung disease, chest wall disorders (kyphoscoliosis and malnourished)
problem- reduced ability to cope with normal WOB, neuromuscular deficiency, disadvantaged diaphragm due to HI lung, fatigue, weakness

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16
Q

mechanical causes of increased WOB- increased alveolar surface tension

A

pathology- pulmonary oedema, acute respiratory distress syndrome, surfactant depletion
problem- increased resistance to expansion

17
Q

mechanical causes of increased WOB- rib fracture

A

disrupted mechanics of thoracic cage, a segment of the chest wall which is flail unable to contribute to lung expansion

18
Q

dyspnea- emphysema

A

increase in expiratory airflow resistance/ increases expiratory muscle work, severe hypoxaemia may contribute to WOB by stimulating drive to breathe, muscles and joints are at a mechanical disadvantage due to passive and dynamic hyperinflation, increased inspiratory muscle work occurs to hold open floppy airways, even during exhalation

19
Q

increased WOB signs

A

use of accessory muscles, disturbed speech inability to complete sentences, pursed lip breathing, prolonged expiratory time, paradoxical breathing (Hoovers sign), in-drawing/recession/retraction of soft tissue of chest wall on inspiration caused by excessive negative pressure in the chest, this destabilizes chest wall increasing the WOB further

20
Q

increased WOB in acutely unwell

A

increase RR/ HR, decreased SpO2, mouth breathing, altered depth and pattern of breathing, use of accessory muscles, deranged ABG’s, CO2 retention, peripheral vasodilation can= warm hands/ bounding pulse/ flapping tremor of the hands

21
Q

increased WOB in the acutely unwell- later signs

A

restless, irritable, confused, and coma, increase or decreased HR/ BP, cardiac arrest, fatigue

22
Q

what do do with a breathless patient

A

treat by addressing the cause where possible, medication if appropriate, physiotherapy, pulmonary rehabilitation, breathlessness clinic if severe/end stage disease

23
Q

treatments for breathlessness patient

A

positions of ease- allows respiratory muscles to work on respiration, purse lip breathing, breathing re-educate, relaxation- mindfulness apps, balance between supply and demand of energy, fan therapy, sleep, pacing and work sequencing

24
Q

why do we position patients for breathlessness

A

many breathless patients automatically adopt a posture which eases breathing, allows accessory muscles to work on expiration

25
Q

breathing re-educating for breathlessness

A

to reduce WOB, increase confidence, desensitisation to breathlessness, pulmonary rehabilitation, breath enhancing positions, increase awareness of the pattern that is being used, encourage relaxation general or specific, nose breathing slow and low, facilitation of lower chest breathing through relaxation

26
Q

balance between supply and demand

A

increase supply- sleep, relaxation, rest, education, nutritional management, oxygen therapy, exercise training, purse lip breathing
decrease demand- treatment of cause, pacing, work sequencing, positioning, breathing control, stress reduction, exercise training

27
Q

how does fan therapy work

A

works with patients who are breathless, reduced perceived breathlessness, blows air across the Trigeminal nerve- alters feedback to brain which reduces feeling of breathless

28
Q

purse lip breathing

A

breathing out through actively pursed lips keeping facial muscles relaxed, patients can learn technique themselves, the positive pressure on exhalation prolonged expiratory time and reduces airway collapse in floppy airways of emphysema, particularly good during activity as reduces dynamic hyperinflation during exercise

29
Q

pacing, work sequencing

A

energy conservation, coordination of activity with breathing, sequencing, pacing- ensure breaks

29
Q

pacing, work sequencing

A

energy conservation, coordination of activity with breathing, sequencing, pacing- ensure breaks

30
Q

NIV

A

generated by volume or pressure cycled machines, patient triggered or automatic, synchronised assisted TV
acute effects- reduced PaCO2, increased pH, increased alveolar ventilation, rest respiratory muscles, decrease load in the respiratory muscles

31
Q

NIV- indications for use

A

hypercapnia, respiratory acidosis, respiratory muscle fatigue, ventilatory failure, weaning from ventilator, physiotherapy, palliative care- symptom control

32
Q

NIV- set up

A

blood gases, mask fit (nose, face or full)- check skin, granuflex to nose, no leaks
aim to capture patients RR and pattern, set trigger level, airway pressure should be 10-35cm H20, no set recipe for set up

33
Q

selection of patients for NIV

A

must be able to control airway, cough reflex, spontaneous respiration, functioning GI tract, some degree of understanding and cooperation

34
Q

advantages and problems for NIV

A

advantages- non-invasive, no sedation, can be ward managed, low cost, easy to withdraw, easy to manage at home, allows communication, mobility, sleep
problems- facial surgery, problems with nasal breathing, eating and drinking, gastric surgery, paralytic ileus, head injury fractured BOS, undrained pneumothorax, hypotension

35
Q

other treatment ideas- respiratory muscle training

A

offers a resistance to inspiration and/or expiration and aims to strengthen the respiratory muscles, from a physiological and pathological point of view

36
Q

other treatment ideas- mannual therapy

A

stretches to the vertebral and shoulder girdle joints to reduce tension, thoracic mobility exercises,