malnutrition and nutritional assessment Flashcards

1
Q

what is the definition of malnutrition?

A

a state resulting from lack of uptake or intake of nutrition leading to altered body composition and body cell mass leading to diminished physical and mental function and impaired clinical outcome from disease”.

highest prevalence is in the youngest and oldest age groups (distribution like a halfpipe)

higher prevalence in women, especially onlder

also highest prevalence in oncology

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2
Q

what is a general person who would have malnutrition like?

A
someone how is admitted to hospital who is:
over 65
with a long term condition
people with progressive condition
people with drug or alcohol problems
people with and GI disorders

1/3 people admitted to hospital are malnourished

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3
Q

what is the impact of malnutrition?

A
  1. Surgeon Hiram Studley.

Surgery for perforated duodenal ulcer.

Postoperative mortality 10 x greater in those who had lost  20% bodyweight preoperatively, compared with those who had lost less.

  1. England and Wales (ONS). Malnutrition:

Direct cause 66 hospital deaths

Contributory factor 285 hospital deaths

the cost of malnutrition every year is £ 19.6 billion 15% of the total public expenditure on health and social care.

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4
Q

why does malnutrition have an impact, what does it do to the body?

A

Physical and functional decline and poorer clinical outcomes

↑ Mortality, septic and post surgical complications, length of hospital-stay, pressure sores, re-admissions, dependency

↓ Wound healing, response to treatment, rehabilitation potential, quality of life

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5
Q

how do we identify and diagnose malnutrition in the clinical setting?

A

Screen

assess - dietician

diagnose:
nutrition diagnosis

Plan

Implement

Monitor

Evaluate

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6
Q

what happens during the screen for malnutrition?

A

A simple tool to identify risk.
Carried out by any HCP.
This is not assessment or diagnosis.
“MUST” - malnutrition universal screening tool

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7
Q

what happens during the assessment for malnutrition?

A
by a dietician
A systematic process of collecting & interpreting information to determine the nature and cause of the nutrient imbalance. (BMI plays very little role)
anthropometry
biochemistry
clinical
dietary
social and physical
nutrition requirements |(estimations of resting metabolic rate)
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8
Q

what are the indications for nutritional support?

A

Nutrition support should be considered in people who are either:

  1. Malnourished =

BMI < 18.5 kg/m2 or

Unintentional weight loss >10 % past 3 - 6 / 12 or

BMI < 20 kg/m2 + unintentional weight loss > 5 % past 3 – 6 / 12.

  1. At risk of malnutrition =

Have eaten little or nothing for > 5 days and / or are likely to eat little or nothing for the next 5 days or longer or

Have a poor absorptive capacity, and / or have high nutrient losses and/or have increased nutritional needs from causes such as catabolism.

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9
Q

what is artificial nutrition support?

A

The provision of enteral or parenteral nutrients to treat or prevent malnutrition.

there is an algorithm for this (stratton and elia)

enteral (intestines) is superior to parenteral (veins)

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10
Q

what are the methods of enteral artificial nutrition support?

A

Route:

Enteral nutrition (EN) is superior to parenteral nutrition (PN).

Where parenteral nutrition is used, the aim is to return to enteral → oral feeding as soon as (where) clinically possible.

Access:
Is gastric feeding possible?

Yes	=  Naso-gastric tube (NGT) 
No  	=  Naso-duodenal (NDT) / naso-jejunal tube (NJT)

Long term (> 3 months) = Gastrostomy/jejunstomy

Nutritional feeds → renal, low sodium, respiratory, immune, elemental, peptide.

Gastric outlet obstruction = NGT feeding contraindicated → NJT

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11
Q

What are the complications associated with enteral feeding?

A

Mechanical: misplacement, blockage, buried bumper

Metabolic: hypergylcaemia, deranged electrolytes

GI: Aspiration, nasopharyngeal pain, laryngeal ulceration, vomiting, diarrhoea.

Misplaced NGTs
2005 – 2011 = 21 deaths + 79 cases of harm, National Patient Safety Agency (NPSA).
Aspirate pH  5.5
If pH > 5.5 → chest x-ray, interpreted by trained professional following NPSA guidelines.

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12
Q

what are the methods of parenteral artificial nutrition support?

A

Parenteral nutrition (PN): The delivery of nutrients, electrolytes and fluid directly into venous blood.

Indications:
An inadequate or unsafe oral and/or enteral nutritional intake
OR
A non-functioning, inaccessible or perforated gastrointestinal tract

Composition:

Ready made / bespoke “scratch” bags.

MDT → fluid and electrolyte targets

Access:

Central venous catheter (CVC): tip at superior vena cava and right atrium.

Different CVCs for short / long term use.

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13
Q

What are the complications associated with parenteral nutrition?

A

Metabolic:
Deranged electrolytes, hyperglycaemia, abnormal liver enzymes, oedema, hypertriglyceridaemia

Mechanical:
(when inserting catheter)
Pneumothorax, haemothorax, thrombosis, cardiac arrhythmias, thrombus, catheter occlusion, thrombophlebitis, extravasion

Catheter related infections

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14
Q

Does Nutrition Support Benefit the Malnourished Patient?

A

yes, reduced mortality

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15
Q

what is albumin?

A

Albumin synthesised in the liver.
most. abundant circulating protein in the plasma
Low plasma albumin = poor prognosis.
A negative acute phase protein = ↓ plasma albumin when ↑ inflammation. (Pretty sure this is wrong)

The acute phase response:
Inflammatory stimulus → activation of monocytes & macrophages → release cytokines.
Cytokines act on liver to stimulate production of some proteins whilst downregulating production of others e.g. albumin.
LESS ALBUMIN

A moderate inflammatory stimulus will induce plasma acute phase protein changes.
The negative acute phase protein, albumin, will↓.

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16
Q

is albumin a valid marker of malnutrition in the acute hospital setting?

A

No.
Albumin synthesis ↓es in response to inflammation ∴ it is not a valid marker of nutrition status nor an indication for nutrition intervention in the acute setting.
Best evidence = hypoalbuminaemia in obese trauma patients.
Dietitian focused on the aetiology/impact of the inflammatory state on nutrition status.

17
Q

what is refeeding syndrome?

A

A group of biochemical shifts & clinical symptoms that can occur in the malnourished or starved individual on the reintroduction of oral, enteral or parenteral nutrition.

during malnutrition there is a reduction in insulin secretion and an increase in glucagon secretion

cellular pumps stop working to conserve energy, so ions are free to diffuse across the plasma membrane

leads to: 
hypokalaemia
hypophospataemia
salt and water retention - oedema
thiamine deficiency
18
Q

what are the consequences of refeeding syndrome?

A

Arrhythmia, tachycardia, CHF → Cardiac arrest, sudden death

Respiratory depression

Encephalopathy, coma, seizures, rhabdomyolysis,

Wernicke’s encephalopy

19
Q

According to the National Institute for Health and Care Excellence (NICE), what are the criteria for defining the risk of RFS?

A

At risk:
Very little or no food intake for > 5 days

High risk: 
more than 1 of the following:
BMI < 16 kg/m2
Unintentional weight loss > 15 % 3 – 6 /12
Very little / no nutrition > 10 days
Low K+, Mg2+, PO4 prior to feeding

Or more than 2 of the following:
BMI < 18.5 kg/m2
Unintentional weight loss > 10 % 3 – 6 / 12
Very little / no nutrition > 5 days
PMHx alcohol abuse or drugs (insulin, chemotherapy, antacids, diuretics)

Extremely high risk:
BMI < 14 kg/m2
Negligible intake > 15 days

20
Q

how is refeeding syndrome managed?

A

start: 10-20 kcal/kg
CHO (carbohydrate) 40-50% energy
micronutrients from onset of feeding

Correct and monitor electrolytes daily following Trust policy

Administer thiamine from the onset of feeding following Trust policy

Monitor fluid shifts and minimise risk of fluid and Na+ overload