Mallory–Weiss tear Flashcards

1
Q

What is a MWT?

A

MWT = tear/laceration of mucosa at/near the gastro-oesophageal junction

Arterial bleeding
Tear - longitudinal

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2
Q

What is the aetiology of MWT?

A

Factors causing increased thoracic/intragastric pressure
Vomiting

Coughing - Whooping cough, bronchitis, bronchiectasis,
emphysema, COPD, LC

Retching/vomiting - Alcohol excess, GI: Food poisoning, PUD, volvulus,Hepatobiliary: Hepatitis, gallstones, Hyperemesis gravidum, Renal: UTI, RF, Neurological, Psychiatric: Anorexia nervosa, bulimia, Toxins: Chemotherapy, Post-anaesthesia

Straining

Hiccups

CPR

Acute abdominal blunt trauma

Hiatus hernia

Transoesophageal echocardiogram

Medications

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3
Q

What are the risk factors for MWT?

A
  • Hiatus hernia (present in 40-100%)
  • Condition predisposing to retching/vomiting
  • Chronic cough
  • Retching during endoscopy
  • Male
  • Middle age
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4
Q

What is the epidemiology of MWT?

A

3-15% of upper GI bleeding

Less common in children
More common in men

In women of child bearing age, the most common cause is hyperemesis gravidarum

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5
Q

What are the symptoms of MWT?

A
  • Haematemesis (vomit normal, then blood streaked)
  • Light-headedness
  • Dysphagia
  • Odynophagia
  • Melena
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6
Q

What are the signs of a MWT?

A

• Postural hypotension

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7
Q

What are appropriate investigations for MWT?

A
Bloods
FBC
Usually unremarkable
May show low HB
Urea - High (if ongoing bleeding)

Flexible OGD - DIAGNOSTIC
Red, longitudinal defect with normal surrounding mucosa
From few mm to several cm

CXR
Normal

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8
Q

What is management for MWT?

A

Urgent evaluation and monitoring: ABCDE + resuscitation

Commonly – self limits in 12-24 hours before OGD

OGD – if active bleeding (Cauterization or epinephrine injection may be used)

Anti-emetic
PPI (IV) - Anti-gastric acid therapy

If altered respiratory/mental status = endotracheal intubation

IF endoscopic haemostasis fails - Surgical intervention,
Senstaken-Blakemore tube

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9
Q

What is the prognosis of MWT?

A

Bleeding usually self-limited and stopped by the time of the OGD

Prognosis is excellent without associated disease

Re-bleeding occurs in 8-15% - Usually within first 24 hours

High risk patients = old age, haematemesis at presentation, haemodynamic instability, alcoholism, SID use, comorbidities

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