Mallory–Weiss tear Flashcards
What is a MWT?
MWT = tear/laceration of mucosa at/near the gastro-oesophageal junction
Arterial bleeding
Tear - longitudinal
What is the aetiology of MWT?
Factors causing increased thoracic/intragastric pressure
Vomiting
Coughing - Whooping cough, bronchitis, bronchiectasis,
emphysema, COPD, LC
Retching/vomiting - Alcohol excess, GI: Food poisoning, PUD, volvulus,Hepatobiliary: Hepatitis, gallstones, Hyperemesis gravidum, Renal: UTI, RF, Neurological, Psychiatric: Anorexia nervosa, bulimia, Toxins: Chemotherapy, Post-anaesthesia
Straining
Hiccups
CPR
Acute abdominal blunt trauma
Hiatus hernia
Transoesophageal echocardiogram
Medications
What are the risk factors for MWT?
- Hiatus hernia (present in 40-100%)
- Condition predisposing to retching/vomiting
- Chronic cough
- Retching during endoscopy
- Male
- Middle age
What is the epidemiology of MWT?
3-15% of upper GI bleeding
Less common in children
More common in men
In women of child bearing age, the most common cause is hyperemesis gravidarum
What are the symptoms of MWT?
- Haematemesis (vomit normal, then blood streaked)
- Light-headedness
- Dysphagia
- Odynophagia
- Melena
What are the signs of a MWT?
• Postural hypotension
What are appropriate investigations for MWT?
Bloods FBC Usually unremarkable May show low HB Urea - High (if ongoing bleeding)
Flexible OGD - DIAGNOSTIC
Red, longitudinal defect with normal surrounding mucosa
From few mm to several cm
CXR
Normal
What is management for MWT?
Urgent evaluation and monitoring: ABCDE + resuscitation
Commonly – self limits in 12-24 hours before OGD
OGD – if active bleeding (Cauterization or epinephrine injection may be used)
Anti-emetic
PPI (IV) - Anti-gastric acid therapy
If altered respiratory/mental status = endotracheal intubation
IF endoscopic haemostasis fails - Surgical intervention,
Senstaken-Blakemore tube
What is the prognosis of MWT?
Bleeding usually self-limited and stopped by the time of the OGD
Prognosis is excellent without associated disease
Re-bleeding occurs in 8-15% - Usually within first 24 hours
High risk patients = old age, haematemesis at presentation, haemodynamic instability, alcoholism, SID use, comorbidities