M13: Multiples in Preg & Doppler Flashcards

1
Q

SEE NORMAL TWINNING FROM OB/GYN 1

A

/

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2
Q

why does chronicity refer to

when is it best determines

A

of placentas

b/w 10-14 wks

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3
Q

most important predictor of preg outcomes in twins

what signs do we look for

A

of placentas

lambda - means dichorionic, some of the placental tissue growing up b/w the membranes

T sign - monochorionic…. would have to be Mono/Di to see this

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4
Q

what does a thicker membrane b/w the fetuses suggest

A

dizygotic twins

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5
Q

most common type of monozygotic twins

A

monochorionic, diamniotic

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6
Q

whats a structure we can look for when to determine if there are 2 membranes

A

yolk sac

1 yolk sacs mean 1 membrane

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7
Q

least common type of monozygotic twins

A

monochorionic, monoamniotic

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8
Q

describe a papyraceous fetus

A

mummified skeletal remains of a demised twin… would occur later in gestation (if the demise is earlier body would reabsorb it)

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9
Q

when would mono/mono twins be conjoined

where are they most commonly conjoined

prognosis

A

if the egg splits after day 13

chest (thoracopagus) or abdomen (omphalopagus)

more are still born or die after 1 day of life

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10
Q

the majority of conjoined twins are what gender

A

female (70%)

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11
Q

what is diprosopus

A

duplication of anatomy or a single body part

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12
Q

what does TRAP syndrome stand for

other names

A

twin reversed arterial perfusion

acardiac parabolic twin
acardiac monster

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13
Q

w/ what type of twining does TRAP occur

A

mono/mono only

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14
Q

w/ TRAP, what type of connections are formed in the placenta

A

can be artery - artery

vein - vein

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15
Q

describe how TRAP syndrome works

A

the pump/donor twin pumps deoxygenated blood from the umbilical artery to the placenta… that blood is shunted along the surface of the placenta to the second acardiac twin… the deoxygenated blood enters the acardiac twin through the umbilical artery and leaves deoxygenated through the umbilical vein.

there is reversed flow in the umbilical artery of the acardiac twin

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16
Q

how does TRAP affect the development of the acardiac twin

A

-only the lower limbs and trunk will develop due to lack of nutrients and 02
-no heart of very abnormal, often no head
… acardiac twin is completely supplied by the donor twin

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17
Q

US appearance of TRAP

A

lack of separating membrane
inability to separate fetal parts
more than 3 vessel cord
complex anomalies

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18
Q

donor twin is at high risk for developing which condition

A

hydrous due to volume overload and high cardiac output… also leads to cardiac failure

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19
Q

what is a fetus in fetu

US appearance

A

a parasitic twin w/in the abdomen of its sibling

teratoma

20
Q

what is a heterotypic preg

A

one normal preg

one ectopic

21
Q

describe superfetation

A

fertilization of 2 separate ova months apart

22
Q

on other type of abnormal twinning variation

A

hydatidiform mole w/ coexisting twin

23
Q

complications w/ twins

A
twin to twin transfusion syndrome (TTTS)
twin embolization
premature delivery
congenital anomalies
cord accidents
24
Q

w/ what type of twinning does TTTS occur

what is TTTS

A

monochorionic only

arteriovenous fistula in the placenta (A-A, V-V, A-V), high mortality rate

25
which type of fistula in the placenta is the worst
artery to vein due to difference is press
26
describe what happens to the donor w/ a TTTS
small and hypotensive oligohydramnios stuck twin appearance Usually anemic
27
describe what happens to the recipient w/ a TTTS
large and hypertensive due to fluid overload polyhydramnios oedematous/hydropic appearance
28
general US appearance of TTTS
``` size discrepancy of >20% disparity of amniotic fluid single placenta thin or non membrane seen hydrous of one twin ```
29
what is a stuck twin
when the donor twin has little to no amniotic fluid and the amniotic membrane hold the baby tight against the uterine wall
30
one other cause of stuck twin other than TTTS how can you tell the two apart
a dichorionic preg w/ placental insufficiency causing IUGR... one baby will be small, the other will be normal
31
describe twin embolization syndrome
occurs when theres a demise of a twin in monochorionic twins.... clots can pass from the demised twin to the surviving twin and cause an infarct in the live fetal brain or liver... ... or it can cause sever hypotension in the surviving twin due to the demised twins placenta becoming low pressure and allowing blood to pool in the placenta. would lead to asphyxia
32
in what type of twining is twin embolization syndrome
monochorionic only
33
US appearance of twin embolization syndrome
ventriculomegaly, porencephalic cysts (from brain hemorrhage), cerebral atrophy or micoencephaly
34
normal length for cervix
~2.5 cm
35
genetic and developmental abnormalities are more common in what type of twins
monozygotic
36
do dizygotic twins have the same risk of developing anomalies as a singleton
yes
37
why would a fetal reduction be performed
improve morbidity and mortality for a successful preg
38
which vessels to we do doppler on w/ a TTTS
for each fetus: umbilical artery middle cerebral artery ductus venosus
39
what are the normal waveforms for all the vessels sampled w/ doppler w/ TTTS
umbilical artery: low resistance w/ lots of diastolic flow middle cerebral artery: high resistance ductus venosus: triphasic venous flow (like the HV's)
40
w/ fetal doppler, what measurement do we use for each vessel
pulsatility index to look for changes in diastolic flow
41
describe an abnormal waveform for the umbilical artery
no diastolic flow is bad reversed diastolic flow is worse
42
how to located the ductus venosus whats its appearance w/ colour doppler
from the AC, find the ductus by following the umbilical vein posterior to the IVC aliasing due to high velocity
43
describe abnormal flow for the ductus venosus why does it occur
reversal of the A wave due to myocardial impairment and increased ventricular EDP from increased RV afterload
44
which middle cerebral artery should you sample at what angle
the one closest to the transducer 0 degrees
45
what can cause an abnormal waveform of the MCA how will it change
vasodilation in the fetal brain w/ brain sparing IUGR reduced PI and increased diastolic flow