Lecture 9: Osteopathic Approach to Head Pain Flashcards

1
Q

What nerves should be considered for head pain of anterior 2/3 vs. posterior 1/3?

A
  • Anterior 2/3 –> Trigeminal n.
  • Posterior 1/3 –> Lesser Occipital (C1-3), Recurrent branches of IX and X
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2
Q

Relevant sympathetic spinal levels associated with head pain?

A

T1-T4

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3
Q

Mean age of onset for tension HA?

Peak prevalence?

A
  • MAO = 25-30 yo
  • Peak prevalence = 30-39 yo
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4
Q

Anxiety and depression often associated w/ what type of headache?

A

Chronic tension-type HA

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5
Q

Peripheral pain mechanisms are more likely important for what type of tension HA?

A

Episodic tension-type HA

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6
Q

Central pain mechanisms are more likely involved in what type of tension HA?

A

Chronic tension-type HA

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7
Q

What are the clinical features (signs/sx’s) of Tension-type HA’s?

A
  • Bilateral
  • Mild to moderate intensity
  • Pressure or tightening quality (nonpulsating)
  • Absence of N/V
  • Not aggravated by routine physical activity
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8
Q

How long can Episodic Tension HA’s last?

A

30 minutes to 7 days

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9
Q

Chronic Tension HA’s are defined as having episodes for how long (days/months)?

A

≥ 15 days/month on average for ≥ 3 months

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10
Q

What is the most common abnormal PE finding for Tension HA’s?

A

Pericranial muscle tenderness - tends to be mostly scalp

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11
Q

How to treat the neurological component (5-models) of a tension-type HA?

A
  • Analgesics and NSAIDs = 1st line
  • Combination analgesics w/ caffeine = 2nd line
  • Metaclopramide
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12
Q

3 focuses for the Metabolic component (5-models) in tx of Tension-HA?

A
  • Sleep hygiene
  • Hormonal influences - menstrual
  • Hydration
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13
Q

Using the 5-models what component should be addressed first in the treatment of Migraine, Cervicogenic, and Tension HA’s?

A
  • Respiratory/circulatory
  • Address lymphatics first to reduce irritants from inflammatory milieu
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14
Q

Using the 5-models approach what should be addressed via the neurological component for Migraine, Cervicogenic, and Tension HA’s?

A
  • Address counterstrain points anteriorly and posteriorly in the cervicals, upper thoracics, upper ribs, and UE’s
  • Use cranial to address other contributing SD’s
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15
Q

Which treatment technique for SD’s has been shown to be an effective method for treating tension HA?

A

MFR

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16
Q

How does a menstrual migraine differ than a normal one?

A

Typically w/o aura and more severe

17
Q

Using the 5-models approach what should be addressed via the Biomechanical component for Tension HA’s?

A
  • Address myofascial SD’s
  • Address joint SD’s w/ cranial, MET, Still’s, HVLA, or FPR
18
Q

What are 2 likely risk factors for Migraines?

A
  1. Medication overuse
  2. MS
19
Q

What is the mnemonic used for Migraine Diagnosis?

A
  • POUND
  • Pulsating
  • Duration 4-72 hOurs
  • Unilateral
  • Nausea or vomiting
  • Disabling

*4/5 criteria met has + likelihood ration 24 for definitive possible migraine

20
Q

Differential Diagnoses for Migraines?

A
  • Tension HA or Cluster HA
  • Cervical spine dz –> greater occipital neuralgia
  • Acute cervical strain –> Whiplash
  • Intracranial mass or Meningitis or Subarachnoid hemorrhage or TIA
  • Sinusitis
  • Cavernous sinus thrombosis
  • Optic neuritis or Acute Glaucoma
  • SLE
  • TMD
  • Epilepsy
21
Q

What is pathognomonic for migraine w/ aura?

A

Spreading oligemia (reduced blood volume) in brain

22
Q

Migraine w/o aura seems to be associated with what messenger molecules?

A
  • NO
  • 5-HT
  • CGRP
23
Q

Using the 5-models how do you treat the Biomechanical components of Migraines?

A

Address joint SD with Cranial, MET, Still’s, HVLA or FPR

24
Q

Cervicogenic HA is caused by disorder of what?

A

Cervical spine and its component bony, joint, and/or soft tissue elements

*Usually NOT associated w/ neck pain

25
Q

Involvement of which cervical facet is the most frequent source of Cervivogenic HA?

A
  • C2-C3 = most frequent
  • AA is probably 2nd
26
Q

Facet pain from C5-C6 and C6-C7 may contribute to what type of relfex resulting in TrP pain referral to the head?

A

Somato-somatic reflex

27
Q

For diagnosis of Cervicogenic HA, evidence of causation by at least two of the following 4 items must be met?

A
  1. Developed in temporal relation to the onset of cervical disorder/lesion
  2. Significantly improved or resolved w/ improvement in or resolution of cervical disorder/lesion
  3. Cervical ROM is reduced AND HA is made significantly worse by provocative maneuvers
  4. Abolished following diagnostic blockade of a cervical structure or its nerve supply
28
Q

What are 5 differential diagnoses for Cervicogenic HA?

A
  1. Migraine - POUND
  2. Tension HA
  3. C2 neuralgia
  4. Neck-tongue syndrome
  5. Occipital Neuralgia
29
Q

If the pain associated with a HA is myofascial what category does it best fit under?

A

Tension HA

30
Q

What are the common signs/sx’s of C2 neuralgia?

A
  • Paroxysmal sharp or shock-like pain centered in occipital region
  • Ipsilateral eye lacrimation and conjunctival injection are common
31
Q

What is Neck-tongue syndrome?

Onset typicall when?

A
  • Rapid head turning causes subluxation of the posterior AA joint and C2 spinal root compression
  • Sx = neck pain, may be asscociated w/ ipsilateral tongue sensory sx’s
  • Onset typically during childhood or adolescence
32
Q

Using the 5-model approach how should the Biomechanical component of Cervicogenic HA’s be treated?

A
  • Address joint SD w/ MET, Stills, or FPR
  • HVLA may irritate facilitated segments!