Lecture 8 - Brain & instinctual Behaviour Flashcards
What is meant by homeostasis?
Regulation of optimum levels of bodily functioning, for instance:
- Blinking - cleans the eyes
- Keeping body temp around 37’c
Doing this stuff voluntarily takes quite a lot of effort
We use two cues to help us do this
- Internal Cues
- External Cues
Why is it important to retain body temp of 37?
If you go slightly above 37, by 2 or so degress
- can cause cramps, sweats, nausea
If you go below 37, by 2 or so degrees
- causes shivering
If you go further
- heat exhaustion or further: heat stroke/ death
- Mild hypothermia or further: severe hypothermia - frost bites - moves blood supply to vital organs sacrifices extremities
Introduce/ define internal cues for homeostasis
Internal cues inform us about: Sleep, warmth, thirst, hunger etc
- PNS and CNS are both used
- often see it as a choice, e.g. to get a drink, but it’s actually our body telling us to do that
- Every animals do it
What are the 2 internal cue systems for hunger?
- Glucose/ Glycogen Model - (stomach and liver) - PNS
- much slower
- Signals from stomach & Liver indicate about eating/ fullness - vital organs when it comes to feeding - Hormonal model? Ghrelin/Leptin - CNS
- much faster
Evolutionarily beneficial to have both of these options available depending on how fast you need to do it
Outline the Glucose/ Glycogen system as the first half of the 2 internal cue systems
AUTONOMIC NERVOUS SYSTEM
- Food enters stomach
- Food is bathed in stomach acid to extract the nutrients - especially sugars
- specifically glucose which is pumped into the muscles for energy - Stomach produces glucose which is pumped into the bloodstream
If not immediately needed: we dont waste it, we store it, would be evolutionarily stupid to let it go to waste. But we can’t store it as glucose because that is a transactional chemical. So we convert it into glycogen for storage
When there is low levels of glucose in the blood, ANS sends signals to the liver to get it out of storage:
- Glycogen is converted back into glucose and released into the blood stream
What are the 2 regulation pathways in the Glucose/ Glycogen system that monitor how much glucose is in the body
- IF NOT ENOUGH GLUCOSE IN BLOODSTREAM
- either: a) Eat, or b) convert stored glycogen into glucose
- if nothing left to convert in stores, you get very hungry - IF NOT CONVERTING GLYCOGEN INTO GLUCOSE
- if the liver is not converting glycogen into glucose, the ANS see’s this and concludes that there must be enough glucose in the bloodstream already
- dont need to eat
LIVER tells ANS when and when you dont need to eat
Outline how diabetes is involved in this glucose/ glycogen system
In both types of diabetes, type 1 and type 2, there is too much sugar staying in the blood (hyperglycemia), therefore not enough getting to muscles
- This is because there is a lack of INSULIN
Not enough INSULIN being produced by the PANCREAS
Insulin is the gatekeeper that allows glucose to LEAVE the bloodstream and go into the muscles - a lack of insulin would mean a lack of glucose leaving the blood and energising the muscles
- this can cause fainting or worse
- sudden collapse in energy - need to use all the energy we have to keeping the vitals going
Eventually, with no energy, heart will stop
What is the Hormonal Model of eating/ fullness
2 Hormones in the CENTRAL NERVOUS SYSTEM important here:
- this system works through signals from both of these hormones:
- Leptin - indicates satiety (fullness) via complicated receptors from the gustatory system that indicate sateity
2 Ghrelin - indicates low glucose in blood and hunger
- uses blood stream glucose levels
Outline a rat with defective Leptin receptors
When a rat had defective/ deactivated Leptin receptors
- Leptin is still being produced and sending signals
- but the Leptin receptors on the post-synaptic cells have been deactivated
so it just couldnt stop eating
How is the hypothalamus involved in this hormonal model?
Hypothalamus is where all the information regarding insluion, glucose, eating and hunger goes
It’s in the midbrain, above the pituitary (secreates growth hormones), made up of lots of structures
Multi-functional too, bascially is responsible for all functions related to the survival of the self and the species
• Hunger, thirst, reproduction, sleep, body temp
THE LATERAL HYPOTHALAMUS IS IN CHARGE OF HUNGER
What is the debate here surrounding eating disorders and these systems?
If we all have these 2 systems, that are all the same, we should anticipate that we all have the same eating habits - but we do not
- we see eating disorders
And numbers of eating disorders are all increasing, and fluctuating each years
- so are eating disorders are leptin related problem - NO because eating disorders prevelance changes each year, but our Leptin systems havent change in millions of years
Outline the internal and external cues to Eating Disorders
It is unclear why people get Eating Disorders and why the numbers are increasing
Internal - not clear
- could be a glucose/ glycogen problem, hypothalamus problem or a leptin problem
External:
- Cognition - the way we think about food
- personality - Anorexic personality?
- social psychology - belonging to certain groups? identify with an ED identity?
- Sociology - feel this way due to society, SE status, gender, education, jobs
Either way: Physiology is not the whole story - as it hasnt changed, something else might be influencing these fluctuations
Outline Pain
Pain prompts regulation via action - need to do something about it
- pain signals indicates disequilibrium - that something isnt right here - so we need to do something about it
Sympathetic ANS involved here - fight or flight to get out of pain
- heart, lungs, blood flow
What are evolutionary differences in pain - what would be best
Evolutionarily there were genes that were better/ quicker at:
- recognising (externally/ sensorily) a dangerous/threatening/ painful stimuli
- Responding to that stimuli
Evolutionarily, it would be beneficial to be fast at both of these - it would be more adaptive
What is the James-Lange theory surrounding pain?
Argued that once our body has automatically responded - to either fight or take flight
- IT IS AFTER THIS that our brain kicks in to interpret the events, and what is happening to us
I’m, running so i must be scared
a) external cues become associated with pain via learning
b) Fight/ flight is triggered unconsciouslly and fear is inferred from the action