Lecture 8 - antisocial behavior Flashcards

1
Q

antisocial behaviour

A
  • antisocial behaviour is a kind of atypical development. Involves
    > actions damaging to others (violence, theft, damage)
    > actions damaging indiv (poor achievement, criminal record)
  • high prevalence in teenagers (Eaton et al 2006) - 14-18y 19% carried weapon, 30% had their property stolen or damaged, 6% missed school
  • often one time thing
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2
Q

conduct problems

A
  • range of behaviours from mild to serious
  • change across development as indiv develops physical strength, cog abilities and sexual maturity
  • less severe earlier & more later on as inc opportunities
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3
Q

conduct disorder categories in DSM5

A
  • ODD(oppositional defiant disorder) - ongoing anger guided disobedience, hostility & defiant to authority beyond normal.
  • CD (conduct disorder) - prolonged antsocial behaviour & violations to laws & norms. seen as precursor to aspd (18)
  • ODD often precursor for CD
  • ODD onset at 6y average, CD onset 9y.
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4
Q

diagnosis of ODD DSM5

A
  • features: excessive persistent anger, frequent tantrums, disregard for authority
  • 6m+ symptoms
  • e.g. actions to annoy others, argue, often lose temper
  • high comorbidity with adhd & learning disorders
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5
Q

diagnosis of CD DSM5

A
  • repetitive and persistent pattern of behaviour where the basic rights of others or major age appropriate societal norms or rules are violated
  • 3 or more of these in past 12m:
    > aggression to people and animals
    > destruction of property
    > deceitfulness or theft
    > violations of rules
  • these cause significant social, academic or occupational impairments
    > child vs adolescent onset
    > mild moderate or severe
  • presence of callous-unemotional (CU) traits or not
  • high comorbidity with ADHD (65-90%)
  • more serious behaviours than ODD
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6
Q

CD associated problems

A
  • poor academic achievement
  • lower IQ
  • truancy
  • suspension
  • accidents
  • risky sexual behaviours
  • unplanned pregnancy
  • earlier onset of sexual behaviour
  • problems in work adjustment
  • legal difficulties
  • physical injury
  • risk of criminal victimisation
  • adulthood health problems, occ difficulties, family problems, offending
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7
Q

prevalence of ODD and CD

A
  • Loeber et al (2000)
    > CD: 1.8-16% boys, <1-9% girls
    > ODD: 2-15% boys, 1.5-15% girls
  • costello et al (2003) - both ODD and CD rates stable over ages 9-16
  • CD rate 3x for boys than girls, ODD 1.5x
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8
Q

antisocial behaviour over the lifespan

A
  • ODD and CD defined and diagnosed as disorders of childhood & adolescence (Aspd adult version)
  • one source of data is legal data but definitions may differ
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9
Q

age crime curve

A
  • peak in prevalence and incidence of offended in teenage and early adulthood followed by gradual decline
  • does not tell you if people continue to commit crimes or if one off
  • Eisner (2003) - huge historical dec in violent crime contrasts with stable age profile of offenders (peak offenders is 20-19). could be explained by adultification of teenagers now
  • possible factors: dev physical strength, independence, out of sync with cog and moral development
  • limitations: no info on antisocial behaviour, cross sectional
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10
Q

early vs late starter model

A
  • antisocial behaviour steadily inc at ages prior to crime stats
  • peak shows a large number of adolescent limited indivs whose antisocial behaviour does not continue
  • there is small sub-pop of life course persistent indivs
  • evidence:
    > moffitt 1993 - NZ longitudinal assessments. 5% boys had ASB rated above average and very antisocial = life course persistent antisocial behavioir
    > in contrast 2/3 remaining boys above average only at some time points = adolescent limited antisocial behavioir.
  • in teenage years antisocial behaviour inc making the two groups hard to distinguish.
  • at 15: BOTH groups had broken 5 laws, but most had broken fewer than 1
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11
Q

early vs late starters implications

A
  • implications: frameworks for assessment & treatment, predicts prognosis, recognises distinctions
    > early starters: usually male, family dysfunction, deficits in verbal ability, deficits in social cognition, adhd, deviant peer
    > late starters: lower m:f ratio, less aggression, fewer prior difficulties, more normative peer relationships, poor parental supervision, deviant peer group
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12
Q

early vs late starter model update - more than two trajectories?

A
  • assessed follow up at 38 & assessed by developmentally appropriate questions at each age
  • tested whether pop is made up of 2 or more discrete classes of individuals
  • new group: childhood limited - conduct problems in childhood drop away
  • Barker & Maughan (2009) - maternal report on if child is high risk based on norms. can get a probability of a high risk child and if they are on track for adolescent onset
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13
Q

child biological risk factors (CD, ODD, antisocial behaviour)

A
  • genetics
  • intergenerational transmission
  • neuroanatomy
  • neurotransmitters
  • other neurochemicals
  • ANS underarousal
  • prenatal and perinatal problems (alcohol in preg)
  • neurotoxins
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14
Q

psychosocial risk factors (CD, ODD, antisocial behaviour)

A
  • psychosocial factors
  • parenting
  • assortative mating
  • child abuse
  • peer effects
  • neighbourhood & socioeconomic factors
  • life stressors & coping skills
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15
Q

child functional risk factors

A
  • temperament
  • attachment
  • neuropsychological functioning
  • intelligence
  • reading problems
  • impulsivity and behavioural inhibition
  • social cognition
  • sociomoral reasoning
  • puberty and adolescent development
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16
Q

callous-unemotional traits and antisocial behaviour

A
  • dimension of psychopathy in children
  • traits: lack of empathy & guilt, unconcerned about school work, does not keep promises, does not show emotions, does not keep friends
  • small prop of antisocial children but at high risk of antisocial behaviour
  • a specifier for CD. high CU traits is bad, but can still have CD with no CU traits.
17
Q

temperament model of development of conduct problems

A
  • callous-unemotional pathway (genetic basis & heritability)
    > strong genetic basis
    > low emotional reactivity
    > punishment insensitivity & reward dominance
    > proactive aggression
    > violence
    > resistant to treatments
  • hostile-impulsive pathway
    > highly reactive to emotional & threatening stimuli
    > respond more strongly to provocations in social situations
    > hostile attribution bias
    > conduct problems associated more strongly with parenting practices
    > responsive to parental discipline.