Lecture 6 Flashcards

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1
Q

Externalizing Behavior Disorders

A

ADHD, conduct disorder, oppositional defiant disorder
Externalizing disorder- uncontrolled impulsive potentially aggressive behavior
Three main types

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2
Q

What is adhd

A

Persistent age-inappropriate symptoms of:
Inattention Hyperactivity Impulsivity

These symptoms cause impairment in major life activities
Umbrella term- categorize and diagnose persistent symptoms
Show all symptoms to some degree
Inattention- cant focus when expected- can result in mistakes and forgetful behaviour
Hyperactivity- what ppl think of when adhd- active, fidgeting, cant stay still, multitasking, restlessness- uncontrollable, difficulty engaging in relaxing and quiet tasks
Impulsivity- self control issues, act without thinking, more likely to interrupt conversations, ask irrelevant questions, make inappropriate comments, cant inhibit behaviours
Blanket term that refers to these behaviours- must cause impairment

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3
Q

Adhd history

A

Early 1900-Poor “inhibitory volition” and “defective moral control”
1917 to 1926- “Brain- injured child syndrome”
1940-1950-
“Minimal brain damage” and “minimal brain dysfunction”
Late 1950- Hyperkinesis “hyperactive child syndrome”
- hyperactivity

1970- Deficits in:
- attention
- impulse control
- hyperactivity

1980 Increased interest in ADHD and rise in stimulant use

Since 90s- inc interest in identifying symptoms and causes
Earliest descriptions-1902- George still said these behaviours caused my moral inhibition- couldn’t control behaviour but still intelligent
Throughout 1900s- more children in settings where these behaviours are more apparent- cat sit still, pay attention
More attention drawn to these behaviours
Following influenza epidemic- inc of children experiencing inphaliytis- swelling of brain causing adhd symptoms
1950- kids without brain trauma show symptoms, led to new disorders and terms
Beginning of biological cause of adhd
Not used anymore- doesn’t correlate with adhd
Hyperkinesi- focusing on hyper behaviours
In 1970- more research- more elements on attention and impulsivity and how it’s affects the disorder
1980- more interest in adhd and more treatment for adhd- medication
How we got to adhd today
Used to be called add

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4
Q

ADHD in the DSM-5

A

Neurodevelopmental disorder
Adhd- neuro developmental disorder
Early onset= start before age 12, may be noticeable at age 3, even earlier
Persistent course= persistent course for some individuals, persists through life, some out grow, some worsen symptoms
Differences in neural development = associated wth differences in neural development- delayed prefrontal maturation and activity differences= differences in thinking, behaviour, emotions
Associated with other neuro developmental disorders =adhd associated with delayed in language, motor development- linked with autism and learning disorders

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5
Q

CORE CHARACTERISTICS

A

Key symptoms fall under two well-documented categories:

I attention and hyperactivity- impulsivity
3- core groups of adhd fall into two categories
Highly correlated and interrelated- biut do predict differences
1- predicts school issues
2- peer rejection and impulsivity
Hyperactivity and impulsivity linked- because when display one often display the other
Using these dimensions can oversimplify the disorder- lots of variablity even with those who present the disorder textbook definition

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6
Q

Inattention

A

Inability to engage and sustain attention, and follow through on directions or rules while resisting distractions
Inattention can result from a failure in one or more cognitive processes:
Deficits in Attentional Capacity, selective attention and distracting Lott, sustained attention and alerting
Attentional capacity= numbers o items one can remember and attend to in short period- remember phone number- this is not a challenge associated with adhd- can hold same amount as those without adhd
Selective attention ability to focus on most important info while tuning out distractions
Distractibility= defecit in selective attention- adhd more likely to focus on salient info
Sustained attention/vigilance- ability to maintain concentration over long period of time- adhd struggle with this
Alerting- a deficit that happens before task has begun- ability to listen to instructions/prepare for task- have difficulty doing tasks cause miss instructions

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7
Q

Hyperactivity-Impulsivity

A

Undercontrol of motor behavior, challenge inhibiting behavior, inability to delay a response or defer gratification

Hyperactive behaviors
Activity is excessively energetic, intense, and not goal-directed

Impulsive behaviors
Inability to control immediate reactions or think before acting

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8
Q

DSM-V Diagnostic Criteria for ADHD

A

A. A persistent pattern of inattention and/or hyperactivity-impulsivity that interferes with functioning or development, as characterized by (1) and/or (2):
Inattention: Six (or more) of the following symptoms have persisted for at least 6 months to a degree that is
inconsistent with developmental level and that negatively impacts directly on social and academic/occupational activities:
Often fails to give close attention to details or makes careless mistakes in schoolwork, at work, or during other activities (e.g., overlooks of misses details,
work is inaccurate).
Often has difficulty sustaining attention in tasks or play activities (e.g., has difficulty remaining focused during lectures, conversations, or lengthy reading).
Often does not seem to listen when spoken to directly (e.g., mind seems elsewhere, even in the absence of any obvious distraction).
Often does not follow through on instructions and fails to finish schoolwork, chores, or duties in the workplace (e.g., starts tasks but quickly loses focus and is easily sidetracked).
Often has difficulty organizing tasks and activities (e.g., difficulty managing sequential tasks: difficulty keeping materials and belongings in order; messy, disorganized work; has poor time management; fails to meet deadlines).
Often avoids, dislikes, or is reluctant to engage in tasks that require sustained mental effort (e.g., schoolwork or home-work; for older adolescents and adults, preparing reports, completing forms, reviewing lengthy papers).
Often loses things necessary for tasks or activities (e.g., school materials, pencils, books, tools, wallets, keys, paperwork, eyeglasses, mobile telephones).
Is often easily distracted by extraneous stimuli (for older adolescents and adults, may include unrelated thoughts).
Is often forgetful in daily activities (e.g., doing chores, running errands; for older adolescents and adults, returning calls, paying bills, keeping
appointments).
Won’t expected to memorize
What are different dimensions and potential symptoms?
1st criteria- pattern of innattention or impulsive/hyperactivity that impairs functioning
Six or more symptoms- at least 6 months and inconsistent with development
Child fails to. Pay attention to details, make careless mistakes, difficult remaining focused
Hyperactivity and Impulsivity: Six (or more) of the following symptoms have persisted for at least 6 months to a degree that is inconsistent with developmental level and that negatively impacts directly on social and academic/occupational activities:
Often fidgets with or taps hands or feet or squirms in seat.
Often leaves seat in situations when remaining seated is expected (e.g., leaves his or her place in the classroom, in the office or other workplace, or in other situations that require remaining in place).
Often runs about or climbs in situations where it is inappropriate. Note: In adolescents or adults, may be limited to feeling restless.
Often unable to play or engage in leisure activities quietly.
Is often “on-the-go and acting as if “driven by a motor” (e.g., is unable to be or is uncomfortable being still for extended time, as in restaurants, meetings; may be seen by others as being restless or difficult to keep up with).
Often talks excessively.
Often blurts out answers before a question has been completed (e.g., completes people’s sentences; cannot wait for a turn in conversation).
Often has difficulty waiting his or her turn (e.g., while waiting in line).
Often interrupts or intrudes on others (e.g., butts into conversations, games or activities; may start using other people’s things without asking or receiving permission; for adolescents and adults, may intrude into or take over what others are doing).
Specify if:
In partial remission: When full criteria were previously met, fewer than the full criteria have been met for the past 6 months, and the symptoms still result in impairment in social, academic, or occupational functioning.
Specify current severity:
Mild: Few, if any, symptoms in excess of those required to make the diagnosis are present, and symptoms result in no more than minor impairments in social or occupational functioning.
Moderate: Symptoms or functional impairment between “mild” and “severe” are present.
Severe: Many symptoms in excess of those required to make the diagnosis, or several symptoms that are particularly severe, are present, or the symptoms result in marked impairment in social or occupational functioning.

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9
Q

Adhd presentation types

A

Presents- inattention only= inattentive type adhd- predominantly inattentive presentation Inattentive, easily distracted, slow-moving, spacey, easily confused, disorganized, daydreamy

Hyperactivity and impulsivity= hyperactive impulsive type Hyperactivity, fidgeting, excessive talking, impulsivity, argumentative

All =combined
Hyperactive, restlessness, disorganized, inattention, impulsivity
Most commonly referred for treatment
This is newly added to dsm- newer area needs more research on accuracy
Some suggests it may not be the best- 50% were reclassified to a type depending on methods, how info was combined and amount of symptoms needed

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10
Q

Additional DSM-5 Criteria For Diagnosis

A

Appears prior to age 12
Occurs across two or more settings (e.g., home, school, or work, with friends or relatives, in other activities)
Symptoms interfere with or reduce the quality of social or academic performance
Symptoms are not better explained by another disorder
In addition to the 6 symptoms present for 6+ months
Rely on retrospective reporting for those who come for treatment older

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11
Q

Limitations of DSM Criteria for ADHD

A

Limitations in developmental sensitivity
Categorical view of ADHD

DSM criteria shape our understanding of ADHD
DSM criteria are also shaped by, and in some instances lag behind, new research findings
1- the criteria are used across lifespan- for older adults= 5 symptoms, the symptoms decline over age but this is only adjustments ,age. Concerns about using this approach across lifespan- older ppl
2- these symptoms exist o dimension- see these behaviours in regular population- those who present 5 symptoms- may be the same as those with 6- why the cut off

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12
Q

Associated characteristics

A

Children with ADHD often have additional challenges:
Cognitive Deficits
Deficits in executive functions
Intellectual and academic deficits
Learning disorders
Distorted self-perceptions
Problems in executive functioning 50%- show deficits in one or more areas of EF
Refers to cognitive processes- integrate and manage other brain functions
Involves their ability to control, plan and organize
Organizing- have trouble starting planning, organizing work and likely to misunderstand instructions
Focus- loose focus, reread and forget what they read
Regulating alertness- procrastination, drowsiness
Manage frustration- easily frustrated, cant regulate emotions
WM- forgetting to do task, cant follo instructions
Regulate action- interrupting, blurting out
Intellectual and academic cognitive deficits Do not differ in levels of intelligence- difficulty applying intelligence- loss of potential to suceed
Can impact iq performance and intelligence task- cant focus, inattention
Poorer academic outcomes- lower grades imp
Difficulty in academic settings
Intellectual deficits and impaired academic functioning
Cognitive Deficits: Learning Disorders

Challenges with reading, spelling, and math
45% have specific learning disorder
Self perception- distorted self perception Some linking to distorted self perception
How diffferent factors present can predict this
Mood disorders make tis more likely
Hyperactive and conduct problem– show positive illusory bias- exaggerated self perception in relation to behaviour and abilities – show more agression- serve as self protective mechanisms- cope with symptoms- may not recognize what successful behavior/performance is

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13
Q

Speech and language impairments

A

Challenges with formal speech
Co-occurring language disorder
In some cases- these interrelated with other symptoms- impulsivity, inattention

~30–60% of children with ADHD
have a speech and/or language impairment

Difficulty understanding others speech
Formal speech and language disorders
Speech production errors
Excessive and loud talking
Inability to listen

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14
Q

Medical and physical conditions

A

Health-related problems

Accident-proneness and risk-taking behaviors
Over 50% of parents of a child with ADHD describe their child as being accident-prone
At higher risk for health related problems- asthma and bed wetting, poor fitness, eating disorder sleep disturbance- may be because of stimulant medication
More accident prone- more likely to show up in hospitals with broken bones, bruising- experience accidents due to impulsive behaviour
Inc risk or car accidents and substance abuse

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15
Q

Social problems

A

Family problems

Peer problems

Close friendships may buffer negative outcomes
Sibling conflict- may feel that one with adhd is victimizing them or getting more attention
Parents more likely to be stressed, maternal depression, father substance abuse
Family concern about outside perception and stigma of mental disorders
Peer difficulties- cant apply experiences to interactions, friends= protective factor

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16
Q

Commonly Co-Occurring Psychological Disorders

A

Up to 80% of children with ADHD have a co-occurring psychological disorder and up to 50% have 2 or more disorders
ODD CD anxiety disorder mood disorder motor coordination and tic disorder CD= more severe
As adhd symptoms inc- anxiety inc
Adhd diagnosed at 4-6= higher chance of suicidal behaviour and depression especially in females

17
Q

Prevalence and Course

A

Prevalence rates vary widely with sampling methods
5–9% of children and adolescents in North America
~5% worldwide have ADHD
Factors to consider:
Gender, socioeconomic status and ethnicity Vary depend on method and symptoms used
Adhd most common
Gender- adhd occurs more in boys- boys 2-3x more likely to be diagnosed
Adhd may be unreported in girls- may not report because not disruptive
SES- lower income families associated with higher risk- likely driven by other cooocuring conduct problems- also associated with low income and low SES
Difficult to say if link is caused by adhd or CD
Race-findings are inconsistent
Adhd is in all cultures
Methodology- not developed with all ethnicity in mind- adhd tools may not be as effective for different ethnicities
When receiving treatment t- don’t differ from treatment benefits
ADHD is slightly more prevalent among lower SES
ADHD is slightly more prevalent among lower SES

18
Q

Course and outcomes

A

Infancy- signs are likely present- hard to depict symptoms before age 3
Temperament- may indicate adhd- not great marker as large number of kids with temper ant issues- don’t ave adhd
Preschool- more easy to recognize and cause more problems- more likely to have problems later on – disruptions in parent child relationship= predicts worsening f symptoms
Elementary- starting school, recognizable, wen they are first referred and treated, ODD- behaviours inc and develop in this period
Adulthood- 50% of diagnosed in childhood- persist symptoms into adulthood- others cope/outgro- especially for those wth mild severity and no CD

19
Q

Interrelated Theories of ADHD

A

Cognitive function deficits, rewards and motivation deficits, arousal level deficits and self regulation deficits Number of proposed theories of cause of adhd
All work together to inc understanding if cause of adhd
Cognitve function- sustained attention, ef deficits- lead to cognitive language and motor difficulties of adhd
Only 50% of children present cog deficit – 50% this is not the factor leading to adhd
Reward –higher reward hreshold- need bigger reward- have difficulty motivating self with no rewards
Have difficulty motivating self when delay of rewards
Adhd associated with disruptions in dopamine paths
Arousal- have difficulties with arousal- under aroused- engage in behaviours to achieve optimal arousal level
Self regulation- higher order deficits- interrelated with reward and arousal deficits- integration of theories, self regulation link these factors

20
Q

Potential Causal/Risk Factors for the Emergence of ADHD

A

Genetic Influences
ADHD runs in families

Adoption studies

Twin studies

Gene studies
Strong =genetic influence of adhd
Adhd is inherited
60 % of child getting when parent has adhd
1/3- of biological relatives have adhd
Adoption- 3x higher in biological vs adoptive parents genetic component
Twin studies- identical twins= 65%
Gene studies- genomic risk- involved in neurotransmission- genes involved in dopamine regulation- dopamine= psychomotor and award seeking- brain regions with adhd- rich in dopamine
Stimulants- affect dopamine in synapse
Pregnancy, Birth, and Early Development
Pregnancy and birth complications
Young maternal and paternal age (< 20 years old)
Mother’s use of cigarettes, alcohol, or other drugs during and after pregnancy, low birth weight, malnutrition
These are contributing factors (rather than a causal association)
May create a “malleable” state
Difficult to decipher role these factors play- associated with other variables, low SES, family conflict
Exposure to these during development- may create development cascade – inc risk of adhd

21
Q

Neurological causes

A

Brain abnormalities and developmental delays:
Abnormalities in the frontostriatal circuitry (prefrontal cortex and basal ganglia)
Smaller right prefrontal cortex
Brain maturation delayed in prefrontal regions
Some neurotransmitters may be involved
Dopamine, norepinephrine, epinephrine, and serotonin
Selective deficiency in the availability of dopamine and norepinephrine
Using medication for effective treatment of ADHD symptoms does not prove that deficits are the cause of symptoms
Frontostriatal- connect frontal lobe to basal ganglia- motor and higher functioning
Evidence is variable- need more research
Dopamine, norephonephrine- adhd has deficits in these

22
Q

Diet and lead

A

Sugar is NOT the cause of hyperactivity

Diet
Focus on micronutrients with an interest in essential fatty acids
Possible moderating role of genetic factors may explain why food additives affect the behavior of some children

Lead exposure has been associated with ADHD symptoms
Early research on diet- has been debunked
May be moderator of adhd- certain foods may affect symptoms in some
Lead exposure inc- adhd inc- only for certain people

23
Q

Family influence

A

Family influences may lead to ADHD symptoms or to greater severity of symptoms

Family problems may result from interacting with a child who is difficult to manage

Family conflict may lead to the presence, persistence, or later emergence of co-occurring oppositional and conduct disorder symptoms
More strong of a genetic than psychological component
Parenting style- don’t cause adhd- exacerbate symptoms
Adhd- result of incentive parent practices
Parenting style and tempermant
Environment and child interact with each other. Difficult Holden elicit negative parent behaviour
High family conflict- inc risk of codevelopement especially ODD and CD

24
Q

Treatment

A

Many do not receive treatment for end treatment early
Combining= most effective

Stimulant medication
Stimulants have been used to treat ADHD since the 1930s
E.g., Dexedrine, Dextrostat, Adderall, Ritalin
Function by:
Altering activity in the frontostriatal region by affecting neurotransmitters (dopamine)
May help normalize frontostriatal structural abnormalities and functional connections
Stimulants have been used to treat ADHD since the 1930s
E.g., Dexedrine, Dextrostat, Adderall, Ritalin
Function by:
Altering activity in the frontostriatal region by affecting neurotransmitters (dopamine)
May help normalize frontostriatal structural abnormalities and functional connections
Other medications then stimulants may be used
Goal to manage adhd at home and school

Pros
Improve symptoms for ~80% of children
Young people are generally positive about their experiences using stimulants
When used appropriately and with proper supervision, usually quite safe
Most side effects are benign
Not addictive for most children
Not associated with increased risk for substance abuse
Cons
Effects are temporary and occur only while medication is taken
Long term impacts of stimulant use are uncertain
There is some evidence stimulants can be addictive if misused

25
Q

Parent Management Training (PMT)

A

Parents are taught about adhd and it’s biological basis. Given a set of guiding principles. Taught behaviour management they. Encourage ti share enjoyable activities with children. Taught how to reduce levels of arousal. Focuses on parents and improving parenting styles
2- immediate powerful consequences and immediate rewards
Consistency
3- rewards and sanctions

26
Q

Educational Intervention

A

Teacher and child set realistic goals and objectives
Mutually agreed upon reward system: carefully monitor performance and reward the child for meeting goals
Disruptive or off-task behaviors may be punished with response-cost procedures (loss of privileges, activities, points, or tokens)
Promote good teaching methods
Identifying target behaviours and potential rewards
What to do= general teaching methods

27
Q

Intensive intervention

A

Summer Treatment Programs (STP)
8-week program, 360 hours (equivalent of 7 years of weekly therapy)
Maximize opportunities to build effective peer relations in natural settings
Provide continuity with academic work

28
Q

Multimodal Treatment Study of Children with ADHD (MTA Study)

A

Large-scale, randomized clinical trial
Children ages 7 to 9 were assigned to 1 of 4 groups and followed for 14 months:
Medication management (stimulant medication 7 days a week)
Behavioral treatment (35 sessions of PMT, 10 teacher/school visits, 8-wk STP)
Combined behavioral treatment and medication
Routine community treatment (treatment delivered in community care)
Findings:
Combined treatment > medication > behavior therapy > routine community treatment
Results held at the 24-month follow-up
By 36 months there were no group differences in ADHD symptoms

29
Q

Additional Interventions

A

Family counseling and support groups
Help family members develop new skills, attitudes, and ability to relate more effectively
Individual counseling
Helps children with ADHD deal with their problems and feelings of isolation and abnormality
Helps build their sense of self-competence