Lecture 4: Surgery of the Hip 1 (Exam 1) Flashcards

1
Q

What is canine hip dysplasia (CHD)

A

A Hereditary dev condition of the coxofemoreal (hip) joint that leads to degenerative joint disease (DJD)

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2
Q

What is the most prevalent genetic based ortho disease of dogs

A

Canine Hip Dysplasia (CHD)

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3
Q

Define hip dysplasia

A

Abnorm dev of hip joint characterized by subluxation or complete luxation of the femoral head in younger px & mild to severe DJD in older px (Hip laxity!)

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4
Q

What can be found w/ DJD

A
  • Cartilage damage
  • Osteophyte formation
  • Subchondral sclerosis
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5
Q

Define luxation of the hip joint

A

Complete separation btw/ the femoral head & acetabulum

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6
Q

Define subluxation

A

Partial or incomplete separation btw/ femoral head & acetabulum

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7
Q

What cause hip dysplasia pain in juvenile dogs

A
  • Articular cartilage wear exposes pain fibers in the subchondral bone
  • Laxity causes stretching of soft tissue
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8
Q

What causes hip dysplasia pain in older dogs

A

Osteoarthritis

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9
Q

Describe the clinical signs in canine hip dysplasia

A
  • Exercise intolerance is the most common sign
  • Clinical signs often don’t correlate w/ radiographic findings
  • Some dogs w/ mod or severe dysplasia are asymptomatic
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10
Q

Describe the etiopathogenesis of hip displasia

A
  • Hereditary: polygenetic multifactoral
  • Envi influenced
  • Hips are norm at birth
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11
Q

What will reduce onset, severity, & incidences of CHD

A

Restricting growth rate

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12
Q

T/F: CHD can be reduced & eliminated by breeding only dogs w/ norm hips

A

False; it is only reduced & not eleiminated

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13
Q

Describe the signalment of CHD

A
  • Sometimes seen in toy breeds & cats (bony changes rare)
  • Highest incidence in large breed dogs
  • Rapid weight gain & growth causes probs w/ dev of supporting soft tissue which contributes to hip laxity
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14
Q

Describe hip laxity

A
  • Decreases SA of articulation (concentrating stress over a smaller area)
  • Favors the dev of CHD
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15
Q

What are the physiologic responses to laxity

A
  • Increased joint fluid vol
  • Proliferative fibroplasia of joint capsules
  • Increased trabecular bone thickness
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16
Q

What is the mechanical response to laxity

A
  • Joint capsule stretching
  • Acetabular bone deformation
  • Periosteal nerve tearing
  • Sharpey’s fibers rupture, bleed, & form osteophytes
  • Microfractures of acetabular trabecular cancellous bone
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17
Q

What structures act together to support the hip

A
  • Round ligament
  • Joint capsule
  • Periarticular musculature
  • Capsular hydrostatic constraints
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18
Q

What are the clinical sx in young dogs (4 - 12 M)

A
  • Most often: sudden onset of unilateral lamenes
  • Abnorm gait (swaying, short stride, or bunny hopping)
  • Pain
  • Poor muscle dev in the hind limbs
  • Joint laxity
  • Positive ortolani sign
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19
Q

what is the angle of reduction

A

Point where femoral head slips back into the acetabulum when abducted

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20
Q

What is the angle of subluxation

A

Point where the femoral head slips out of the acetabulum when adducted

21
Q

What are the clinical signs in adult dogs ( > 15 M)

A
  • Chronic lameness that is worse after exercise
  • Often bilateral lameness
  • Decreased muscle mass in pelvic limbs
  • Waddling gait/bunny hopping
  • Crepitus & pain on palpation
  • Rises slowly w/ difficulty
  • Shoulder muscle hypertrophy
  • Difficulty climbing stairs
22
Q

What can be found in both a young dog & older dog differential dx

A

Cranial cruciate injury

23
Q

What is a major sign of laxity in the PE of young dogs

A
  • Barlow test (first part of ortolani test subluxation)
  • Positive ortolani sign (reduction of the femoral head)
24
Q

Which organizations say radiography is req for definitive dx

A
  • Ortho foundation of animals (OFA)
  • Univ of Pennsylvania Hip Improvement Program (PennHIP)
25
Q

What does radiography eval

A
  • Sublux/lux
  • Acetabular margin
  • Size, shape, & architecture of femoral head & neck
  • Presence of exostosis or osteophytes
  • Subchondral bone eburnation
26
Q

What does the OFA do

A
  • Collate & disseminate info on ortho dx of animals
  • Advise, encourage, & establish control programs to lower disease incidence
  • Encourage & finance research
  • Receive fund & make grants
27
Q

Describe the OFA: dysplasia control registry

A
  • 24 M or older to register
  • Positioning specific VD radiograph (Hip extended view)
  • Film ID req
  • Evaluated indep by 3 radiologist based on breed, sex, & age
  • Consensus repor produced
28
Q

What are the 7 grades of the OFA consensus report

A
  • Excellent hip conformation
  • Good h8ip conformation
  • Fair hip conformation
  • Near normal
  • Mild hip dysplasia
  • Mod hip dysplasia
  • Severe hip dysplasia
29
Q

How is the px positioned for a VD radiograph

A
  • Extend the hips & internally rotate the tibias (patellas are directly over the trochlear grooves)
  • Be sure the pelvis is straight (the obturator foramina are symmetric)
30
Q

When is a px a candidate for total hip replacement (THR) or femoral head ostectomy (FHO)

A

If clinical signs cannot be managed medically

31
Q

List the criticisms of the OFA

A
  • test non physiologic hip position
  • Joint laxity is dynamic
  • Subjective/intra & inter observer variation
  • Influence of age on reliability
  • Variation in ax
  • Hormonal effects on hip laxity
  • Variation w/ health status of dog
  • Lack of uniform reporiting
32
Q

What is PennHIP

A
  • Stress radiographic dx method
  • Database/registry
  • International network of hip eval centers
33
Q

Describe the PennHIP measures max passive hip laxity

A
  • Passive vs. functional laxity
  • Shows ~ 2.5x > laxity than seen on the hip extended view
  • Statistically predictive @ 16 W of age
  • Distraction view very reliable to show laxity (highly repeatable & objective; has a distraction index
34
Q

Describe the PennHIP radiographic procedure

A
  1. Hip extended radiograph - the hind legs are placed in “extension.” Used to ID radiographic signs of hip osteoarthritis (OA)
  2. Compression radiograph - hip placed in neutral stance position & the femoral heads are seated in the acetabula
  3. Distraction radiograph - hips are places in the same neutral position as compression radiograph; a special device called a distractor is used to reveal the joint laxity
35
Q

Label these steps of a PennHIP readiographic procedure

36
Q

What is the PennHIP distraction index (DI)

A
  • DI = measure of hip laxity (distance the ball is distracted from the hip socket
  • Expressed as a # btw/ 0 & 1
37
Q

What does a DI near 0 =

A

little joint laxity (very tight hips)

38
Q

What does a DI closer to 1 =

A

High degree of laxity (very loose hips)

39
Q

Dogs w/ (tighter/looser) hips are less likely to develop hip dysplasia than dogs w/ (tighter/looser) hips

A

tighter;looser

40
Q

What does a level below of 0.30 mean

A

Below .30 hip dysplasia is very unlikely to occur

41
Q

What are the vet req in PennHIP

A
  • Training
  • Certification
  • Mandatory submission of ALL films
  • Encourage positive ID (microchip or tattoo)
42
Q

What are the criticisms of PennHIP

A
  • Training req time & cost
  • Special equipment
  • Potential for injury (unfounded)
43
Q

What factors influence the treatment of CHD

A
  • Px age
  • Degree of discomfort
  • Physical & radiographic findings
  • Client expectation
  • Finances
44
Q

What percent of young px return to acceptable function w/ medical or conservative management

45
Q

When is sx treatment done for CHD

A
  • When conservative tx is not effective
  • When athletic performance is desired
  • Slow progression of DJD & enhance probability of good long term limb fxn
46
Q

Describe short term medical/conservative management

A
  • Complete rest 10 - 14 days
  • Moist heat
  • PT
  • NSAIDs
  • Chondroprotective agents
47
Q

Describe long term medical/conservative management

A
  • Weight control
  • Exercise like walking & swimming
  • NSAIDs
  • Chondroprotective agents
48
Q

What NSAIDs are used to treat CHD

A
  • Carprofen
  • Derocoxib
  • Meloxicam
  • Previcox
49
Q

List chondroprotective agents w/ the potential to treat CHD

A
  • Parental polysulfated glycosaminoglycans (adequan)
  • Oral glucosamine & chondroitin sulfate (glycoflex & cosequin)