Lecture 3 and 4 Flashcards

1
Q

Why do people use substances

A
To enjoy the experience and short term effects
To feel confident
To “break the rules”
To be part of the subculture
To relieve boredom
Peer influence
Family factors
Community factors
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2
Q

Why do people continue to use addictive substances

A

To counter the unpleasant effects of prescribed medications
To continue the habit
To avoid unpleasant feelings
To satisfy cravings
To avoid withdrawal symptoms
To counter the effects of other drugs (e.g., benzos after speed)
Gateway theory

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3
Q

Theories of addiction

A

Moral: sign of moral weakness, deviance
Disease: “sick role”, not reversible, genetic
Genetic/neuro-pharmacological: based on adoption and twin studies, limbic system, reward pathways/neurotransmitters
Psychological: classical/operant conditioning, positive reinforcement, associations
Socio-cultural: gender, age, SES, subculture
Bio-psycho-social: multiple contributing factors operate in combination

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4
Q

Psychological Theories

A
  1. Disease theories
  2. Behavioural theories (reinforcement)
  3. Cognitive/Cognitive behavioural theories
  4. Motivation and change theories
  5. Family and systems theories
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5
Q

The case of Mike

A

22 year-old male
Started binge drinking and cannabis when 15 yo
Currently drinking 10 standard drinks 5+ days/week
Cannabis use increased since failing uni and relationship break-up with his first serious boyfriend. Smoking cannabis nightly.
Reports feeling hopeless, depressed and anxious
States he was never close with his family. Sees them regularly, but things have become more difficult.
His father is a heavy drinker and is concerned to see his son making the same mistakes. High expectations to succeed from father.
Mike says drug use helps him to “feel better and forget”. He knows that his use is high and causing him harm, but feels there is nothing he can do about it.
He feels terrible after a heavy drinking session.
He believes he gets on better with people after he has had a few drinks, and can cope better with conflict with his family.

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6
Q

Disease Theories overview

A

Dependence is a symptom of physical or mental illness or disease
Dependence is caused by the presence of a specific gene or biochemical abnormality that precedes substance use
Inability to discriminate blood alcohol level
Increased sensitivity to alcohol
Abnormal brainwaves
Evidence comes from family, twin and adoption studies

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7
Q

12 Steps models

A

AA, NA, Al-Anon
Dependence as a “spiritual disease” and “lack of control”
Main premises (see Heather and Robertson 1997; Kaskutas, 2009)
Alcoholism is “all or nothing”. Problem or no problem.
Alcoholics are powerless over alcohol and experiences.
Alcoholism cannot be “cured”, only managed.
Disease is progressive and deterioration in condition is inevitable if drinking continues.
Support through attending groups, peer support, submitting to a higher power
Mechanisms of theory not well explained
Limited research evidence base
Heavy drinkers can control use, many dependant users do return to controlled use, no evidence that the disease is ‘progressive’.
This is an example of abstinence vs. harm reduction approaches.
Treatment outcomes from 12 step programs-associated with greater friends, spiritual connection, finding meaning in life (Kaskutas, 2009)

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8
Q

Psychoanalytic View

A
Nature and nurture 
Unconscious processes that govern the id, ego, & superego
Importance of childhood experiences
Variety of theories:
Fixated at the “oral” stage
Id –drive reduction theory.
Ego – “self medication”
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9
Q

Psychoanalytic Shared Assumptions

A

Psychoanalytic shared assumptions:
Drug use is a symptom of an underlying psychological disorder
Indicates severe psychopathology
Psychological problems are assumed to cause substance abuse, but not usually recognised as a consequence of use
Addiction is considered a uniform disorder
Treatment via the therapeutic relationship and specific psychoanalytic techniques
Consumption provides relief from conflict generated by oral fixation; or repressed psychological conflict (see Rassool, 2011)

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10
Q

Jellinek 5 Types of Alcoholics ABGDE

A

alpha - pure psychological addiction
beta - continuous heavy drinking leading to medical complications, but psychological or physical dependence not evident
gamma - increased tolerance, withdrawals, cravings and loss of control
delta - as above with inability to abstain
epsilon - intermittent binge drinking interspersed with abstinence/controlled use

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11
Q

Cloninger, Wennberg 1987 Two Alcoholic Types

A
Type 1 (late onset): high reward dependency, high harm avoidance, and low novelty seeking
Type 2 (early onset): lower reward dependence, low harm avoidance*, high novelty seeking
Some research is inconsistent*
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12
Q

Personality Theory

A

Group of distinct personality characteristics that distinguish alcoholics from other individuals
Many ideas:
Predisposing factor (‘addictive personality’)
Consequence of dependence
Independent (personality trait and dependence)
Limited evidence of ‘addictive personality’ per se
Related to specific personality characteristics (e.g., sensation seeking)

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13
Q

Personality Predictors of Drug Use

A

Personality predictors of drug use (e.g., McGue 1995; Rassool, 2011)
Behavioural disinhibition (impulsivity)
Emotional negativity (negative mood, depressive PD)
Other personality features (e.g., narcissism)
Used to enhance sensation and avoid pain

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14
Q

Personality characteristics are also related to drug of choice

A
  • Heroin patients show greater novelty seeking than alcohol patients (Le Bon et al, 2004)
  • Some research considers internal vs. external stressors and uses MMPI personality profiles to categorise drug of choice (Lawson & Winstead, 2008)
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15
Q

Parts of the brain involved in addiction

A
  • Cortex
  • Limbic System
  • Brain stem
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16
Q

Endorphin-Deficiency Theory

A

Drugs of abuse target the brain’s pleasure centre

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17
Q

Behavioural Theories

A
Only considers observable/measurable behaviour
Behaviour is a consequence of learning
Four main types of conditioning
Classical conditioning
Operant conditioning
Modelling 
Tension reduction
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18
Q

Classical Conditioning Models

A
Classical conditioning models
Sights, smells and sounds consistently associated with drug use elicit physiological and psychological responses that lead to drug seeking behaviour
Conditioned stimuli (CS) – cues and triggers
Conditioned response (CR) – physiological and psychological responses
CS more important than CR
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19
Q

Operant conditioning models

A

Focuses on reinforcing properties of drugs, and the likelihood of people repeating immediately pleasurable experiences (and avoiding unpleasurable experiences)
Three main reinforcement types:
Positive reinforcement (i.e., drugs can cause pleasurable sensations)
Negative reinforcement
Punishment

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20
Q

Modelling behavioural theories

A

People learn favourable attitudes and expectation about drinking based on how the behaviour is modelled.
Lowers the risk of harm if one does not have to experience negative consequences personally
Increases the likelihood of pleasant experiences learned from others
Maintenance associated with past associations with drug-taking environments/situations (see Rassool, 2011)

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21
Q

Cognitive Model

A

Focuses on the thoughts/beliefs, and impact on behaviours and feelings
The way people interpret specific situations influences feelings, motivations and actions.
Layers of beliefs

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22
Q

Cognitive Dissonance

A

Conflicting beliefs (cognitive dissonance)
Conflict between the desire to use drugs and the desire to be free of drugs e.g. “I should not use alcohol” vs “It’s OK to have a drink just this one time”
Leads to a cycle of behavioural, emotional and thinking patterns
Cognitive-behavioural: thoughts and behaviours are learnt and therefore can be ‘unlearnt’ (CBT)

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23
Q

Cognitive Model

A
Circle 
-Activating stimulus: 
   Internal cues 
   External cues 
-Beliefs activated 
-Automatic thoughts 
-Cravings/urges 
-Facilitating beliefs (permission)
-Focus on instrumental strategies (Action) 
-Continue to use or relapse
24
Q

Social Learning theory

A

Triangle
Personal factors environment behaviour
Example: Adolescents who view substance use in a positive light, whose peers use drugs, and whose parents and peers have attitudes that condone substance use are more likely to use substances

25
Q

Expetancy Theory Bandura

A

Two specific cognitions:
Outcome expectancy – beliefs about effects and outcomes of using
Self efficacy – belief in ones own ability to effect change

26
Q

Implications for treatment: Relapse prevention

A

Interaction of high risk situations (cues/triggers) with coping response and expectancy (outcome and self-efficacy)
Aim is to reduce high risk situations and increase alternative coping strategies and self-efficacy

27
Q

Motivation and change theories

A

‘Transtheoretical’
Stages of change (Prochaska & DiClemente, 1986)
Clients do not necessarily enter treatment committed to action and making changes
Popular model – growing evidence base
Motivational interviewing - intrinsic motivation is a necessary and sufficient factor to initiate behaviour change

28
Q

Motivation and Change theories circle 6 things

A

Circle thing that was in lecture

1) Precontemplation: No intention of changing the behaviour
2) Contemplation: Aware a problem exists. No commitment to action
3) Preparation: Intent upon taking action
4) Action: Active modification of behaviour
5) Maintenance: Sustained change-new behaviour replaces old
6) Relapse: Fell back into old patterns of behaviour

29
Q

Family and systems theory

A

Determinants of behaviour are based on an individuals role within a system
Family and other systems (e.g., peers) have role in starting/maintaining substance use
Many differing theories
Shared common elements:
Boundaries
Reciprocal causality
Homeostasis
Importance of placing emphasis on interventions for the system.

30
Q

Deteminants

A

The range of risk and protective factors that make someone more or less likely to develop a drug use disorder

31
Q

Major risk and protective factors (Loxley et al., 2004) Prior to birth

A
Prior to birth
Risk factors: 
-Social Disadvantage 
-Family breakdown 
-Genetic influences 
-Maternal smoking and alcohol use 
Protective factors: 
Birth outside of Australia
32
Q

Risk factor: Socio economic status

A

Low Social Status and the increasing disparity between the rich and the poor have been noted to impact upon morbidity, morality, and other health-risk behaviours such as drug use. This most likely arises through economic discrepancies producing personal and interpersonal insecurities, tensions, and conflict, which negatively influence physical and mental health, well-being, coping, and competence of individuals in the community

33
Q

Risk factors: Trauma

A

Children affected by abuse or neglect have a higher risk of problems later in life

34
Q

Risk factors: Genetics

A

Genetic / biological

Examples:  
Variations in the metabolism of substances
Neurotransmitters
Genetic temperament
Personality factors
35
Q

Protective factors- Ethnicity?

A

Depends on a number of factors
Some indication being born outside Australia is protective
family cohesion, rules and cultural norms, and parental supervision
Also can be a risk factor
Low SES communities, family trauma, social isolation
Indigenous risk factor
Not biological/genetic
http://blogs.abc.net.au/queensland/2008/09/indigenous-drin.html
Related to poverty, disadvantage, cultural dispossession & exclusion, & some other cultural factors (sharing culture, no traditional rules for alc.)

36
Q

Aboriginal people who drink do so at harmful levels

A

While Aboriginal people generally drink less than non-Aboriginal people, those who do are more likely to drink at hazardous levels. Unfortunately, many reports focus on these results rather than the fact that generally they drink less.”

37
Q

Major risk and protective factors: Infancy/pre school

A

Risk factors
Parental neglect & abuse
Protective factors
Easy temperament

38
Q

Major risk and protective factors: Primary school 5-11

A
Risk factors: 
- Early school failure 
-Conduct disorder 
-Aggression 
Protective factors: 
-Social and emotional competence 
-Shy and cautious temperament
39
Q

Risk factors: Family factors

A

Many family risk factors to consider
Harmony vs. conflict
Family relationships/support

Parent and family drug use
Parental role modeling risks
https://youtu.be/jJNNtsJmZb4
Modeling less influential than quality of relationships & parental family management techniques

40
Q

Risk factors: Education

A

Academic failure / Learning or behavioural difficulties at school
May be both a contributor to and a result of drug use
Can result in detachment from school community
Impact on self-perception
Timing important
Grade 1 academic failure does not predict later delinquency
Grade 5 academic failure does predict later delinquency
Ongoing implications
Poor academic performance can reduce and lead to lack of training or employment opportunities

41
Q

Protective factors: Education

A

Pro-social contact
Including positive relationships with adults
Minimises boredom
Enhances intelligence
Development and identification of talents
Explore future aspirations
Environment supporting positive health behaviours

42
Q

Risk factors: Personality

A

Personality traits

Rebelliousness, non-conformity, low sense of responsibility, resistance to authority…

Sensation-seeking traits / adventurous personality

43
Q

Risk factors: Age of initiation

A

Earlier initiation increases risk of later abuse

Developmental impact of drug use

Reasons for earlier onset?

Peer associations
Familial and societal norms
Trauma/mental health issues

44
Q

Risk factors: Peer factors

A

Strong predictor of later drug abuse

Not “just” peer pressure
Peers have a strong immediate influence on choices
Peer modelling (social learning theory)
Shared sense of:
Alienation/Rebelliousness/Sensation seeking

45
Q

News reports

A

Youth already tried drugs more likely to be influenced by media coverage

Other factors involved though (e.g. social norms and behaviour of friends)

46
Q

Major risk and protective factors: Adulthood 18-64

A
Risk factors
- Frequent drug use in adolescence 
-Unemployment in early adulthood 
-Mental health problems 
Protective Factors: 
-Well-managed environment  for alcohol use 
-Marriage in early adulthood
47
Q

Major risk and protective factors: Retirement/old age 65+

A
Risk factors: 
-Losing a spouse 
-Retirement 
-Loneliness and reduced social support 
-Poor health 
Protective factors: 
-Good health 
- Social support
48
Q

General protective factors

A
Positive family relationships
Clear, consistent boundaries
Healthy attachments / social bonding
Stability and connectedness
Proactive problem solving - resilience
Development of a special talent
Can lead to pro-drug using peer groups though
Personality and temperament
Career goals / aspirations
Above average intelligence
49
Q

Prevention

A

“Prevention refers to measures that prevent or delay the onset of drug use as well as measures that protect against risk and reduce harm associated with drug supply and use”

50
Q

What exactly are we trying to prevent

A

Drug use
Or at least delay initiation
Harm associated with drug use
Route of administration risks
Overdose/excessive use risks
Avoidance of help seeking – e.g. due to legal concerns
Risks of associated problems – e.g. accidents
Harm to others – e.g. Laws prohibiting smoking in public places
Rather than preventing something:
Increasing resilience/other protective factors?

51
Q

Prevention levels: Levels of prevention strategies (3)

A

Levels of prevention strategies
Primary: Preventing uptake
Preventing non-users starting or delaying first use
E.g. Supply reduction methods, education on harms to developing body, school-based programs
Secondary: Preventing harm
Reducing risks to experimental/social users & avoiding transition to more regular use or possible harms
E.g. education on risks of addiction, advice on route of administration options
Tertiary: Reducing harm
Reducing use or potential harms among regular users
E.g. NSPs, controlled drinking, drink driving campaigns

52
Q

Prevention targets

A

Targets of prevention strategies
Universal: targeting whole populations
National campaigns, advertising programs

Selective: subgroups with above-average risk
School-based programs, Alcohol free communities

Indicated: individuals with detectable symptoms
Counselling, advice lines, educational pamphlets

53
Q

Clarifying Prevention Objectives

A

When to Target?
Early intervention
As early as possible in developmental pathway
What goals or purposes?
Attitudes
Knowledge
Expectancies
Behaviour associated with drug use (risks)
Targeting specific risk factors
Peer factors, educational involvement etc…
Best approach would be a combination of factors

54
Q

Research Support for Prevention

A

Adolescent Interventions
Support for classroom based education programs
More effective if interactive, time-intensive and led by students
Support for regulation of price and availability of tobacco to young people
Lack of support for effectiveness of recent alcohol price increases
More research required?

55
Q

APS Position

A

“While the ‘alcopop’ tax is an example of a targeted measure designed to offset the enticement of young people into an alcohol-soaked culture, such legislation—by itself—will not change Australia’s drinking culture.”

56
Q

Research support for prevention

A

Universal interventions for illicit drug use
Diversion programs
Prohibition
May reduce drug use among non-users (Primary)
Not much evidence it affects current users
International changes to Cannabis laws have not been associated with significant increase in use
Other Universal Interventions
Good support for effectiveness of drink driving laws in Australia
Support for public education campaigns
Reductions in cigarette smoking and risky drinking
Multi-faceted campaigns (advertising, laws, taxes, etc…`