Lecture 3 Flashcards

1
Q

describe the first stage of reading, visual word recognition

A

getting letters to the meaning of a word

a small set of symbols in combination makes up an infinite set of words

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2
Q

define mental lexicon

A

systematic organisation of words in your brain (+_ 60,000 - 70,000)

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3
Q

how does frequency affect word recognition speed?

A

high frequency words are recognised faster than low frequency words

general information-retrieval mechanism

less frequent words also tend to be less familiar, meaningful, and more nonword-like; if more like non words, more difficult to say “yes, it is a word” -> post-lexical factors

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4
Q

define the age of acquisition effect

A

words that you learned at a younger age are recognised faster

overlap with frequency, but not completely, e.g. kite

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5
Q

what did RAYNER say about transposed and substituted letters?

A

substituted letters more disruption than transposed letters

BUT transposed letters still costly compared to identical controls

t and s letter do slow down understanding, especially if the change is at the beginning or, to a lesser extent, the end of a word

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6
Q

what did REY say about transposed and substituted letters?

A

letter detection: is there an “A” in BOARD / BRASH?

A = grapheme, OA = grapheme

easier to indicate “yes there is an A” in BRASH than BOARD
-> Graphemes are processed as perceptual reading units. To get to the “A” in “BOARD”, you have to break apart the “OA”

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7
Q

define graphemes

A

letters and letter groups that correspond to one sound (phoneme)

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8
Q

what are morphemes?

A

the smallest meaningful unit of a language

can be a word itself (e.g., deck) or part of a word (de-brief)

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9
Q

explain roots and affixes in a morpheme

A

(prefixes and suffixes)

“unreal” has two morphemes “un-“ (prefix) and “real” (root)
“farmhouse” has two morphemes “farm” and “house”

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10
Q

what is compound decomposition?

A

tests whether compounds are decomposed into their constituents

immediate effects of lexeme (constituent) frequency
later effects of whole-compound frequency
-> immediate decomposition, but also whole word representation

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11
Q

What are pseudo-affixes?

A

CORNER (pseudo-suffix: CORN + ER)
BROTHEL (no pseudo-suffix, but contains existing word broth)

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12
Q

what occurs during word recognition with pseudo-affixes?

A

if pseudo-affixes are not distinguished as a unit, then priming comparable for both conditions

if pseudo-affixes are used to divide up words, then greater priming for CORN+ER -> CORN than for BROTHEL -> BROTH

suffixes (and possible suffixes - pseudo-suffixes) are extracted as units early during word recognition

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13
Q

are semantically rich words recognised faster or slower?

A

faster,
more semantic features; more semantic neighbours higher imageability, more concrete; easier to bodily interact with; degree of emotional valence (positive, negative, neutral: lucky - angry - plain); degree of arousal (high, low: snake -sleep)

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14
Q

how can semantics (supposed to come in only after accessed) affect recognition?

A

feedback from semantic to orthographic and phonological layer (interactive)?
marker or “flag” associated with orthography?

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15
Q

what is parafoveal processing?

A

lots of processing is done even before we look at a word

evidence that frequency of next word is extracted within ~100ms of looking at the word before

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16
Q

what is the first model of word recognition?

A

word entries are searched one at a time (in a series or serially)

word entries are searched all at once (in parallel)

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17
Q

what is the second model of word recognition?

A

information flows in strictly one way: letters -> words

interactive: letters <-> words

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18
Q

considering information flow, are there feedback connections between letters & words?

A

letters in words detected better than letters in nonwords

feedback from words to letters

letters in words detected better than letters on their own -> word superiority effect

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19
Q

describe Forster’s search model: a simple serial search

A
  1. recognise letters “COW”
  2. get some initial unit e.g. first letter
    this choses a bin (e.g. the letter “C” bin for the word “COW”)
  3. once in a bin, entries are searched one by one (bins are ordered by frequency)
  4. use master file to get to meaning
20
Q

describe the parallel model: the Logogen model

A

send info about letters to all word detectors (logogens) at once

info only feeds forward from letters to words

each logogen has a threshold level based on e.g. frequency

when activation passes threshold -> fire -> word recognised

21
Q

summarise the Interaction Activation Competition (IAC) model

A

parallel activation of letters and words

feedback from words to letters

high frequency words have higher resting levels of activation (not lower thresholds - note: difference from logogen mechanism)

all words have the same threshold

22
Q

describe ASIDE: Nettalk

A

connectionist neural network from the 1980s

1 input layer, 1 hidden layer, 1 output layer

about 300 “neurons”, interconnected -> 18,000 “synapses”

first random sound generation, with backwards propagation, starts to learn what is good and not (adjusting strength of connections)

does not contain any spelling-to-sound rules

after 1/2 day - can learn 1,000 words

23
Q

what is the dual-route model?

A

not just word recognition, but also from letters on a page/screen to speech

reading predictable words out loud
- pronunciation is predictable based on how the word is spelled
e.g. “SAT” “SPEAK” (called regular words)

reading unpredictable words out loud
- pronunciation is not predictable based on how the word is spelled
e.g. “YACHT” “STEAK” (called irregular words)

reading new words
- in experiments we use “nonwords” (nonsense words)
nonwords are letter strings which aren’t real words

two types:
pronounceable (e.g. chotel)
unpronounceable (e.g. rpteeof)

24
Q

what is neuropsychology?

A

neuropsychology is the study of patients who have suffered brain damage

25
Q

how will we expect a patient to respond after damage to the lexical system?

A

read of words will be difficult

reading of non-words/novel words will be normal

impairments of irregular words are expected

26
Q

what is the non-lexical route to speech?

A

GPC

grapheme / phoneme conversion system

converts letters into sounds

27
Q

what is surface dyslexia?

A

an “acquired” dyslexia (results from a stroke/brain injury)

28
Q

what is phonological dyslexia?

A

another acquired dyslexia resulted from stroke/brain injury

29
Q

what is deep dyslexia?

A

possibly a more severe form of phonological dyslexia

difficulties in reading
- nonwords
- function words
- visual errors (e.g. “think” instead of “thing”)
- abstract words (imageability effect)
- and others

30
Q

what is the defining feature of deep dyslexia?

A

semantic paralexia

produce words related in meaning:
DUEL > sword, rapier
BIRD > canary

31
Q

what were the findings of the lexical effects on reading nonwords?

A

TAVE vs. TAZE

neighbours of TAVE: SAVE, CAVE, DAVE, NAVE
but also HAVE (expectation word)
neighbours of TAZE: HAZE, MAZE, GAZE, DAZE
no expectations in pronunciation

dual route model predicts TAVE = TAZE because
1. both are nonwords -> use GPC
2. GPC is independent -> words should have no influence

HENCE, lexical neighbours influence both pronunciation times and errors for nonwords -> not all nonwords processed in the same way (dual route model)

32
Q

what were the findings of regularity effects on reading words?

A

regular consistent words:
“wade” rhyming neighbours: made, jade, fade, bade
regular inconsistent words:
“wade”. rhyming neighbours: pave, gave, cave, save, rave, but also: have

Dual route model predicts WADE = WAVE because: 1. both are words -> use direct route 2. consistency should not influence RTs or errors

BUT regular consistent words named faster & more accurately

hence, not all words processed in the same way

33
Q

summarise unimpaired data

A

nonwords seem to be affected by word neighbours (words that look similar) - this should not happen if all words are simply looked up

words seem to be affected by grapheme-phoneme correspondence - this should not happen if all words are simply looked up

the solution to this is the race mode where both routes “race” against each other and the fastest route wins

34
Q

define developmental dyslexia

A

dyslexia is a learning difficulty that primarily affects the skills involved in accurate and fluent word reading and spelling

35
Q

what are the hallmarks of dyslexia?

A
  • poor reading (slow, inaccurate)
  • poor spelling
  • poor comprehension
  • secondary consequence - by product of not being able to read words fluently
  • reduced reading experience (bc reading can be exhausting, time consuming)
  • continuum
  • not related to intelligence or classroom instruction
36
Q

what are the common co -occurring conditions?

A

ADHD, dysgraphia, dyscalculia, stuttering?

37
Q

can dyslexia be found across any language?

A

yes, in all languages
though can be more apparent in languages with inconsistent orthographies

38
Q

what is etiology?

A
  • neurobiological origin
  • moderate heritability (0.50)
  • twice as common in males
  • cause = multifactorial, associated with multiple genes and environmental risk factors
  • children at genetic risk, who later become dyslexic, look less at books and avoid being read to more even before learning to read
39
Q

what is the treatment for etiology?

A

intensive, explicit instructions in phonics, phoneme awareness, word analysis, reading fluency, and reading comprehension

intervention better than remediation

40
Q

what is the first of four major theories of DD?

A

phonological deficit theory

41
Q

what is the phonological deficit theory?

A

most widely accepted
1. poor phonological awareness (ability to manipulate sounds) e.g. phoneme deletion/substitution, syllable counting
2. poor verbal short-term memory, remembering sequences of sounds or letters
3. slow lexical retrieval, ability to name aloud letters or objects rapidly

42
Q

what is the second of four major theories of DD?

A

Double Deficit Theory
extension of first theory

2 deficits: phonological deficit (mainly related to accuracy) and naming - speed deficit (related to fluency)

naming speed measured by RAN (rapid automatised naming)
often used to diagnose reading difficulties in children

43
Q

what is the third of four major theories of DD?

A

Magnocellular deficit

44
Q

what is a magnocellular deficit?

A

dyslexia results from reduced sensitivity in the neural pathways of the visual system

magnocellular pathway: fast transmission from retina to occipital and parietal brain regions

magnocells 27% smaller in people with dyslexia

visual stress (can result in headaches, eye strain, poor concentration)

45
Q

what is the fourth and final major theory of DD?

A

Cerebellar deficit

46
Q

what is a cerebellar deficit?

A

cerebellum not only for motor skills but also cognitive skills, including language

people with dyslexia: reduced activation in the right cerebellum -> temporal processing deficit

poor naming speed, poor time estimation, poor balance

47
Q

summarise this lecture

A

recognising a simple word is very complex
- effects of frequency, orthography, morphology, semantics (also phonology)

different models of word recognition differ in search and information flow

dual route model of reading can handle data from certain types of dyslexia, but struggles with “unimpaired” data