Lecture 2- specific phobias Flashcards

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1
Q

what is anxiety

A

Anxiety is “a future-orientated mood state associated with preparation for possible, upcoming negative events” (Craske et al., 2009, p. 1067)
Anxiety can be helpful

o Study harder for an exam

o Stay focused on important tasks

  • Anxiety can be disruptive

o When targeted at ‘unfounded’ threat

o When out of proportion to the situation

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2
Q

what is fear

A

Not to be confused with fear

  • Fear is an alarm response to threat
  • Anxiety is apprehension towards anticipated problem
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3
Q

Difference between normal and abnormal anxiety

A

Fear can be adaptive

o Avoid or get out of dangerous situations

  • Anxiety can be helpful

o Study harder for an exam

o Stay focused on important tasks

  • Anxiety can be disruptive

o When targeted at ‘unfounded’ threat

o When out of proportion to the situation

  • When does normal anxiety become a disorder?

o When it interferes with normal functioning

o Quite normal and adaptive to be anxious about something

  • But what if it stops you ever leaving the house?
  • Or stops you interacting with others?
  • Or if it makes you late for work or other important commitments?
  • Then it is clearly a disabling disorde
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4
Q

prevelance of anxiety

A

Kessler, Meron Ruscio, Shear, and Wittchen (2008):

  • Most common psychiatric disorders
  • Age of onset: early compared to some other disorders – specific phobias and SAD often start in childhood

o Associated with onset of other disorders

o But first treatment for anxiety disorders usually not till adulthood

  • Wittchen et al. (2011)

o EU 12-month prevalence: 14% of population (14 years old +)

o 61.5 million

o F/M ratio: 2

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5
Q

describe the cognitive features of anx

A

Anxiety manifest in thoughts

o From mild worry to panic and terror

  • Perception of impending disaster

o End of the world or death

  • Morbid preoccupation with uncertainty
  • Negative thoughts
  • Biased information processing
  • Negative schema (mental frameworks that organise beliefs and information about the world, self, other people etc. – the mental filters through which we experience)
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6
Q

describe the somatic features of anx

A

Anxiety shows in any one or more of following

o Shallow breathing

o Dry mouth

o Diarrhoea

o Frequent and urgent urination

o Faintness

o High blood pressure

o Rapid heartbeat

o Sweating

o Muscle tension

o Shaking

  • These symptoms make sense in terms of an automatic physiological ‘fight or flight’ response to perceived threat
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7
Q

what is the dsm for a specific phobia

A

A. Marked fear and anxiety about a specific object or situation (e.g. flying, heights, animals, receiving an injection, seeing blood)

  • B. The phobic object or situation almost always provokes an immediate fear or anxiety
  • C. The phobic object or situation is actively avoided or endured with intense anxiety or distress
  • D. The fear or anxiety is out of proportion to the actual danger posed by the specific object or situation, and to the sociocultural context
  • E. The fear, anxiety, or avoidance is persistent, typically lasting for 6 months or more (NEW)
  • F. The fear, anxiety, or avoidance causes clinically distress or impairment in social, occupational or other important areas of functioning
  • G. The disturbance is not better explained by another mental disorder (particularly fear/anxiety related to other anxiety, and related, disorders)
  • GONE: The person recognises that the fear is excessive or unreasonable+ Some additional specifiers relate to specific objects or situations
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8
Q

what are the 4 types of SP and desrcribe them

A

Animal Type

o Spiders (very common)

o Snakes

o Dogs

o Cats

o Bees/wasps

o Rats/mice

  • Natural Environment Type

o e.g. heights, storms, water

  • Situational Type

o e.g. airplanes/flying, lifts, bridge

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9
Q

prevelance of SP

A

Wittchen et al. (2011)

o EU 12-month prevalence: 6.4% of population (14 years old +)

o 22.7 million

o F/M ratio: 2.4:1

  • Kessler et al. (2005)

o Lifetime prevalence in USA: 12.5%

  • Most common form of anxiety disorder
  • Phobias of animals, natural environment, & blood/injections/injury often start in childhood
  • Most situational phobias start in early adulthood
  • Typical childhood fears (the dark) often resolve without treatment
  • But phobias tend to be very persistent in adults
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10
Q

physcial syptoms of SP

A

Increased heart beat

  • Increased blood pressure
  • Shortness of breath
  • Except blood, injection, injury phobia

o Fear targeted towards anything ‘medical’

o Or sight of blood and gore

  • Physical symptoms can be the opposite of other phobias
  • Decreased blood pressure
  • May occasionally cause person to faint
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11
Q

DESCRIBE THE FEatures pf S[

A

Irrationality

o Sufferer may know the fear is irrational

  • But cannot do anything about it

o No matter how they try to face fear

  • Reinforced by avoidance behaviour
  • Confirms the feeling of safety
  • Clinically significant?

o Many specific phobias can be unpleasant or inconvenient for the person

  • Change route to avoid driving over bridges

o But only clinically noteworthy if significantly interfere with life

  • Most specific phobias probably do not
  • Disgust

o Often fear of object is associated with disgust

  • Especially with animal phobias- Spiders, snakes

o Fear and disgust in spiders (Olatunji & Deacon, 2008)

  • Those with specific phobia report elevated fear and disgust when viewing pictures of spiders- Compared to those without phobia
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12
Q

DESCRIBE 2 PSYCHODYNAMIC THEORIES

A

Freud: phobias: anxiety is displaced onto ‘neutral’ object

o People do not fear the object itself

o But have displaced anxiety over other issue onto it

o Original conflict resides with Oedipus complex

o Too unbearable, so transferred onto neutral object

o Avoiding neutral object ð avoiding original conflict

  • Arieti: interpersonal childhood problem is repressed

o Children originally trust those around them

o Protect from danger

o Then fear that adults/parents cannot be trusted

o To deal with mistrust they displace fear onto other objects

o Enable them to trust people again

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13
Q

2 behavioual models of SP

A

Eysenck’s incubation theory (1968)

  • Phobia started with development of conditioned stimulus (CS)
  • Neutral S (bee) + sting (unconditioned S) -> bee = CS (now triggers fear and avoidance)
  • Does repeated exposure to bee (CS) without sting ð extinction?

o Not necessarily

  • Eysenck argued that incubation may occur instead

o Phobia is maintained even with non-reinforced CS

  • May be due high trait anxiety overriding extinction

o Predisposition to attend to fear and aversion

o Overly ruminate on original conditioni
Specific phobias as learned behaviour

o Classical conditioning can account for fear of objects

  • Previously neutral stimuli paired with traumatic event
  • Becomes feared object

o Someone with claustrophobia

  • May have been shut in confined space as child
  • Famous study of Little Albert (Watson)

o Albert plays white rat with no fear

o Loud noise sounded above head scares Albert

o Repeated pairing of sound with presence of ratð

  • Albert associates rat with distress- Becomes scared of rat
  • Mowrer’s two-factor theory (1939)

o Phobias acquired through classical conditioning

o Maintained through operant conditioning

  • Avoidance relieves anxiety
  • Negatively reinforces phobia
  • But a person with a bee phobia is not stung every time they see a bee ð so fear should disappear?

o So why is fear maintained despite no repeat of sting? Eysenck’s incubation theory may offer an explanation …

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14
Q

DESCRIBE eyesnecks incubation theory

A

Phobia started with development of conditioned stimulus (CS)

  • Neutral S (bee) + sting (unconditioned S) -> bee = CS (now triggers fear and avoidance)
  • Does repeated exposure to bee (CS) without sting ð extinction?

o Not necessarily

  • Eysenck argued that incubation may occur instead

o Phobia is maintained even with non-reinforced CS

  • May be due high trait anxiety overriding extinction

o Predisposition to attend to fear and aversion

o Overly ruminate on original conditioning event

  • Also referred to as ‘paradoxical enhancement’
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15
Q

describe mowrers 2 factor theory

A

Phobias acquired through classical conditioning

o Maintained through operant conditioning

  • Avoidance relieves anxiety
  • Negatively reinforces phobia
  • But a person with a bee phobia is not stung every time they see a bee ð so fear should disappear?

o So why is fear maintained despite no repeat of sting? Eysenck’s incubation theory may offer an explanation …

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16
Q

describe 2 biological theories of SP

A

enetics (see Hettema, et al. 2001)

o Little evidence to support hypothesis that specific phobia is genetically determined

  • But propensity to anxiety is moderately heritable
  • direct explanation
  • Autonomic lability

o Autonomic nervous system (ANS) controls arousal

o Some people more labile (jumpy) than others

o ANS involved in phobic behaviour

  • This propensity may be genetic
  • Hyperactivation in the amygdala and insula – which is the case for Social Anxiety Disorder too& Wagner
17
Q

describe treatments for SP

A

Fewer than 1 in 5 seek any form of treatment (Wang, et al. 2002)

  • Pharmacological treatments

o People with phobias often self-medicate to relieve anxiety

o Some people take benzodiazepines before confronting feared stimulus

o But equally might drink alcohol

o Some evidence of short term relief

o But phobia remains

o Overall, pharmacological treatment is rare – regular/long-term use of benzodiazepines not advised - addiction

  • Psychological treatments are more popular

o Considerable evidence of swift and successful therapies

Choy, Fyer, and Lipsitz (2007) Review of treatment options/ evidence

  • Behavioural
  • Exposure Therapy

o In vivo vs. imaginal vs. virtual reality

o Gradual vs. flooding

o With vs. without relaxation (systematic desensitisation)

o External vs. internal (interoceptive exposure)

  • Often quick and effective
  • Can improve even well-established and severe phobias
  • We will focus on a form of in vivo exposure in the seminars this week: Ost’s One Session Treatment (OST; Ost, 1985; Zlomke, 2008)
18
Q

DESCRIBE 2 behaviourist treatments of SP

A

Modelling

o Therapist engages with fear object (e.g. handling spider)

  • Client watches this
  • Before being asked to directly confront fear

o Client observes the calmness of therapist

  • Helps reduce client’s own fears about fear object

o Works on principles of social learning theory

  • But usually part of exposure treatment
  • Flooding

o Client is exposed to intensive dose of their feared object – all at once (compare to prolonged but gradual exposure in OST)

o Therapist prepares client with relaxation techniques

  • Reduce fear during flooding

o Effective and quick therapy

  • But compliance difficult!

o Not well favoured by therapists due to client (and therefore therapist) discomfort

o Not available via NHS

19
Q

describe cognitive models of treatment for sp

A

ognitive therapy alone less likely than behavioural to benefit

o Client is already likely to be aware their phobia is illogical

o So little point in just focusing on the faulty cognition – though addressing catastrophic nature of thoughts might be helpful

  • Cognitive-behavioural (CBT)

o Adding cognitive component to behavioural methods contributes little extra benefit for some specific phobias

  • Purely or mostly behavioural interventions appear to be more effective for specific phobias
20
Q

Evaluate treatments for SP

A

Some evidence that benzodiazepines help short-term

o But precaution needed due to severe risk of dependence

o …and phobia remains anyway

  • Behavioural treatments

o By far and away the most successful

  • Cognitive treatments – not as effective as behavioural
  • Choy, Fyer, and Lipsitz (2007):
  • “Most phobias respond robustly to in vivo exposure, but high dropout rates and low treatment acceptance
  • Response to systematic desensitisation is moderate
  • Virtual reality exposure ? effective for flying/ height phobia
  • Blood/injury phobias respond uniquely to applied tension”