Lecture 2: Osteopathic Reflexes Flashcards

1
Q

What do dorsal horn neurons respond to?

A

Both visceral and somatic stimuli

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2
Q

What are the specific layers of the upper layers of the spinal cord?

A
  • Layer 3,4 - mechanoreceptors
  • Layers 1, 5 - A delta fast pain fibers
  • Layer 2 - small c fibers of slow pain
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3
Q

What types of neurons are included in the lower layers?

A
  • Interneurons

- Motoneuron cell bodies

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4
Q

What layers are afferent fibers located?

A

End mostly in layers 1 & 5 but lots of overlap

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5
Q

What % of interneurons receive input from both visceral and somatic afferents?

A

70-80%

May account for visceral pain being so diffuse and poorly located

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6
Q

What is responsible for the activation of somatic muscle activity seen with visceral disturbances?

A

The overlap of afferents received by interneurons

  • Visceral afferents activate sympathetic outflows and skeletal muscle motoneurons (increase tone)
  • Reverse is also possible
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7
Q

What affects Visceral/Somatic reflexes?

A
  • Descending influences
  • Affect the long-lasting excitability of the outflows by maintaining the reflex
  • Sensitization of interneurons acts as an amplifier so outputs are more than expected
  • May inhibit somatic and autonomic outflow
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8
Q

What is the result of visceral disturbance?

A

Reflexively cause activation in the somatic musculature creating somatic dysfunction in the facilitated segment

*contributes to decompensation of homeostasis

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9
Q

What is the result of somatic disturbance?

A

Reflexively alter visceral function

*contributes to decompensation of homeostasis

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10
Q

What is the somatic component of disease?

A

MSK palpatory findings may correlate with visceral disturbances

Normalizing MSK component may allow normalization of autonomic outflows resulting in restoration of homeostasis

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11
Q

What is sensitization?

A

When a stimulus is repeated at a rate of every second or two, response to the stimulus may continue to grow then levels out despite increase of the stimulus

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12
Q

What is habituation?

A
  • Opposite of sensitization
  • Process of decreasing response of a neural pathway with a continuous stimulation
  • Ubiquitous phenomenon
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13
Q

What is facilitation (facilitated segment concept)?

A
  • Habituation and Sensitization work together to keep a pool of neurons in a state of subthreshold excitation
  • Less afferent stimulation is required to trigger discharge of impulses
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14
Q

What causes Facilitation?

A

May be due to sustained increase in afferent input, aberrant patterns of afferent input, or charges within the affected neurons or their environment

Once established, facilitation can be sustained by normal CNS activity

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15
Q

What is the nociception theory?

A
  • Once a stimulus is strong enough to depolarize nociceptive pathways, impulses travel to cord then branch to multiple sites
  • Results in peptide release at motoneuron level in peripheral tissues
  • Peptides are important in inflammatory cascade and initiate release of prostaglandins and bradykinins
  • This results in lowering nociceptor threshold thus increasing input to cord (balance between habituation and sensitization is disrupted)
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16
Q

What is short term excitability?

A
  • 1-2 seconds of afferent input

- Excitability lasts 90-120 seconds

17
Q

What is long term sensitization?

A
  • Inputs of several minutes

- Excitability lasts hours

18
Q

What is fixation?

A
  • 15-40 minutes of afferent input

- Excitability lasts days or weeks

19
Q

What is permanent excitability?

A
  • Lasts forever

- Death of inhibitory interneurons

20
Q

What is allostasis?

A

Our balance of health

  1. stimulus applied to tissue
  2. dev’t inflammation
  3. causes primary afferant sensitization
  4. results in exaggerated response to noxious stimulus (hyperalgesia)
  5. secondary hyperalgesia develops (central sensitization)
  6. change of fxn of viscera and muscle spasm causing asymmetry and altered ROM
  7. decrease in endogenous descending pathway causing loss of control of protective mechanisms aka allostasis overload
21
Q

What are the effects of somatovisceral reflexes?

A

CV - HTN, increased risk of MI
Neuro - Depression, anxiety, memory loss, decreased cognition
Immune - immunosupression, autoimmune

22
Q

somatosomatic osteopathic reflexes

A

localized somatic stimuli produce patterns of reflex response in segmentally related somatic structures

23
Q

somatovisceral osteopathic reflexes

A

localized somatic stimulation producing patterns of reflex in segmentally related visceral structures

24
Q

viscerosomatic osteopathic reflexes

A

localized visceral stimuli producing patterns of reflex response in segmentally related somatic structures

somatic pain referral due to visceral nociceptive stimuli

25
Q

viscerovisceral osteopathic reflexes

A

localized visceral stimuli producing patterns of reflex response in segmentally related visceral structures

26
Q

withdrawl reflex

A

type of somatosomatic reflex

noxious stimulus applied to somatic structure

27
Q

myotatic response

A

type of somatosomatic reflex

stretch receptor stimulated and stretched muscle fires while antagonist is inhibited

28
Q

What type of reflex is abdominal distention after eating?

A

viscerovisceral - distention of gut results in contraction of stomach muscle

why spinal cord injured patients are able to digest food

29
Q

Somatocardiac reflex

A

nociceptive somatic stimuli result in elevation of HR and BP

30
Q

Somatogastric reflex

A

nociceptive somatic stimuli results in inhibition of peristalsis in stomach

31
Q

Somatoadrenal reflex

A

nociceptive somatic stimuli results in release of catecholamines from adrenal medulla

32
Q

What is important about the vagus nerve?

A

it’s 80-90% sensory!

All visceral nerves contain sensory fibers

33
Q

Where are the cell bodies of sensory neurons located?

A

Dorsal root ganglion - create complex communication network connecting visceral and somatic systems

34
Q

What are chapman’s reflexes?

A

Group of palpable points occurring in predictable locations on the anterior and posterior surfaces of the body that are considered to be reflections of visceral dysfunction or disease

35
Q

What are the components of Chapman’s reflexes?

A
  1. Viscerosomatic reflex of both diagnostic and treatment value
  2. Gangliform contraction that blocks lymphatic drainage and causes SNS dysfunction (neurolymphatic)
  3. A consistent reproducible series of points both anterior and posterior related to specific organs or conditions
36
Q

What might you palpate at one of Chapman’s points?

A

Small, smooth, firm nodule approximately 2-3 mm in diameter on deep fascia or periosteum

37
Q

How to you treat an abnormality at a Chapman’s point

A

Once found and isolated, apply gentle but firm pressure, move tip of finger in circular fashion in attempt to flatten the mass (10-30 seconds)

Stop when the mass disappears or the patient can no longer tolerate the procedure

38
Q

What might the patient experience during treatment?

A

A deep, disagreeable pain that they usually don’t know is there otherwise

Pinpoint, sharp, and non-radiating pain located directly under the physician’s fingertip

39
Q

Contraindications to Chapman’s treatment

A
  • have critical airway, breathing, or circulation problems
  • no consent
  • relative contraindications: fracture, cancer, and other patient instability