Lecture 2 Gastric and Duodenal Disorders Flashcards
what is gastritis?
inflammation of gastric mucosa
what is the typical duration of gastritis?
can last hours or days
risk factors for acute gastritis?
food/alcohol
viruses
stress
ASA and NSAIDs
risk factors for chronic gastritis?
H pylori
repeated exposure to agents that cause acute gastritis
complications of chronic gastritis
atrophy
ulcers
cancer
bleeding if inflammation is severe
clinical manifestations of acute gastritis
anorexia
rapid onset epigastric pain
clinical manifestations of chronic gastritis
belching, nausea, vomiting, pyrosis
vague epigastric pain relieved by eating
nursing management of gastritis
GI rest
slow reintroduction starting with clear liquids
bland small meals
eliminate irritating foods, alcohol, smoking
IV fluids and electrolytes
pharmacologic management of gastritis
PPI, H2 antagonist, antacids
sucralfate
B12 - impaired absorption
antibiotics for H pylori
risk factors for peptic ulcer disease
H pylori
NSAID use
Alcohol
Smoking
acid hypersecretory disorders
stress
types of peptic ulcers (location)
duodenal
gastric
esophageal
pathophysiology of peptic ulcer disease
erosion from gastric juice can occur if the epithelium has been damaged
can penetrate mucosa and extend into smooth muscle
how do clinical manifestations of duodenal ulcer and gastric ulcer differ?
duodenal ulcer causes epigastric pain 2-3 hours after eating and improves with food and antacids
gastric ulcer causes epigastric pain immediately after eating and is not relieved by antacids
how are clinical manifestations of duodenal and gastric ulcers the same?
epigastric pain
pyrosis
constipation or diarrhea
vomiting
bleeding
sour burps
diagnostic tests for PUD
EGD
CBC
fecal occult blood test
H pylori test