Lecture 11 Flashcards
eating behaviour regulated by:
-signals and mechanisms that stimulate eating
-signals and mechanisms that to stop eating or lead to refrain from eating
hunger
-unpleasant sensation that signals for food
-4-6hrs after eating
-food has left stomach & nurtients absorbed
what triggers hunger
-triggered by contracting empty stomach and empty small intestine
-triggered by stomach hormone ghrelin
what drives eating
appetite
appetite
-phychological desire to eat
-experienced without hunger
-stimulated by sight & smell of food
-illness or stress may result in loss of appetite
factors affecting appetite
-hormones
-inborne appetites
-learned preferences
-habits
-social interactions
-disease states
-stimulates & drugs
-environement conditions
satiation
perception of fullness, determines how much food is eaten in a sitting, stretch receptors in stomach send signals to brain
satiety
suppresses hunger, determines length of time between meals, perception of fullness lingering after a meal
sensory specific satiety
-concept that we tend to get bored of a food as we eat it
-more variety=more likely we will increase overall consumption
buffet
people generally overeat
are hunger and satiety equal
no - hunger is stronger
Leptin
- a satiety hormone
–appetite-suppressing
-produced by adipose tissue in stomach
-travels to brain
-directly linked to appetite control & amount of body fat
gain of body fat stimulates
leptin production> reduces consumption resulting in fat loss>reduces leptin secretion increasing appetite
studies on glycemic index
some show low-glycemic index diet reduced or delayed hunger, other found opposite or no effect
of the energy yielding nutrients, which is the most satiating
protein
fat is known for its effect on
satiety
protein & fat trigger release of
intestinal hormone (CCK) that slows stomach emptying and prolongs feelings of fullness
fat
-weak effect on satiation
-provides a lot of kcals in small volume
Theories of metabolic causes of obesity
- Set point theory
- Fat cell number theory
- Thermogenesis 1: brown fat theory
set point theory
body somehow attempts to maintain a stable body weight
fat cell number theory
fat cells may increase faster in children who are obease contributing to obesity as adults
Thermogenesis 1: brown fat theory
brown fat (brown adipose tissue) has abundant energy wasting protiens. lean people have more brown fat, infants have abundant brown fat
genetics & obesity
-in rare cases, primary cause of obesity is genetic
-infleunce how body stores & uses energy
-genes not solely responsible
-adopted people have similar weight to birth parents
likelihood of genetic obesity
if a child has 1 parent obease, they are 40-70% likely to be obease