Lecture 10: Osteopathic Approach to GI Patient Flashcards

1
Q

What are the 2 goals of Mesenteric/Colonic Release?

A
  1. Enhance lymphatic and venous drainage
  2. Alleviate congestion secondary to visceral ptosis/dysfunction
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2
Q

OMT for GI dysfunctions is directed toward improving what 2 things?

A

Blood/lymphatic flow and balancing autonomics

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3
Q

Intense, dull discomfort in the RUQ w/ associated N/V and diaphoresis, which lasts 30 mins. to 1 hour and generally has a benign PE is characteristic of?

A

Biliary colic

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4
Q

When addressing the biomechanical component of GI pain, if there is failure of a SD to respond to OMT this point to what type of problem?

A

Viscerosomatic

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5
Q

The severity of palpated tissue texture abnormality = ?

A

Severity of visceral problem

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6
Q

An organ that is intra-peritoneal means that it has what?

A

Mesentery and covered by peritoneum

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7
Q

What are the abdominal peritoneal organs?

A
  • Stomach
  • Small intestine (jejunum, ileum, superior duodenum)
  • Spleen
  • Liver
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8
Q

What is the span of the abdominal cavity?

A

Diaphragm (excluding esophagus) —> Pelvic Diaphragm

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9
Q

In the GI tract what is found in the wall of the viscera in regards to viscerosensory/visceromotor?

A
  • Pacinian corpuscles
  • Free nerve endings
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10
Q

How does the localization of true visceral pain vs. true somatic pain differ?

A
  • True visceral pain = POORLY localized –> vague, cramping, achy
  • True somatic pain = WELL localized –> Sharp, asymmetric
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11
Q

How does visceral pathology lead to viscerosomatic reflexes?

A
  • Increased stretch/irritation to GI nerves –> increased afferent signals to CNS –> afferent fibers synapse in the dorsal horn of spinal cord
  • Prolonged afferent activity leads to FACILITATION of the neurons and the corresponding spinal segments
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12
Q

Visceral disturbances can cause activation of what leading to SD?

A
  • Activation of somatic muscle activity
  • Visceral pathology –> somatic changes paraspinally (TTC’s and increased tenderness)
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13
Q

What is the Percutaneous reflex of Morley?

Association with Appendicitis?

A
  • Direct transfer of inflammatory irritation from viscera –> peritoneum
  • Not reflexing thru visceral afferent reflex
  • Appendicitis –> Peritonitis = direct organ to peritoneum inflammation = abd. wall rigidity, pain, and rebound tenderness
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14
Q

In the GI the sympathetic component involving the thoracic splanchnic n. and lumbar splanchnic n. synapse with what ganglion?

A
  • Thoracic splanchnic n. –> Celiac and Superior Mesenteric Ganglion
  • Lumbar splanchnic n. –> Inferior Mesenteric Ganglion
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15
Q

Which spinal levels is the Celiac Ganglion involved with?

Organs?

A
  • T5-T9
  • Distal esophagus, stomach, prox. duodenum, liver, GB, spleen and portions of pancreas
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16
Q

Which spinal levels is the Superior Mesenteric Ganglion involved with?

Organs?

A
  • T10-T11
  • Distal duodenum, portions of pancreas, jejunum, ascending colon, proximal 2/3 of transverse colon
17
Q

Which spinal levels is the Inferior Mesenteric Ganglion involved with?

Organs?

A
  • T12-L2
  • Distal 1/3 of transverse colon, descending colon, sigmoid, and rectum
18
Q

The Upper GI + Lower GI (1/2) tract suppled by what nerve for parasympathetics?

A

Vagus n. (CN X)

19
Q

What does the Right Vagus N. vs. Left Vagus N. supply?

A
  • Right = lesser curve stomach, liver/GB, small bowel, right colon to mid-transverse colon
  • Left = greater curve stomach, ends at duodenum
20
Q

What is the parasympathetic supply for the descending colon, sigmoid, and rectum?

A

Pelvic splanchnic N. (S2-S4)

21
Q

What are the 3 large collecting interstitial lymph nodes for the GI?

A
  1. Celiac = stomach, duodenum, spleen and liver
  2. Superior mesenteric = jejunum, ileum, ascending/transverse colon
  3. Inferior mesenteric = descending/sigmoid colon, rectum
22
Q

Which metabolic disturbances (hormones, electrolytes, and acid base) lead to/associated w/ diarrhea?

A
  • Hyperthyroidism
  • Hypocalcemia and Hyperkalemia
  • Metabolic acidosis
23
Q

Which OMM treatment modalities can be used to normalize sympathetic activity in the GI?

A
  • ME
  • Soft tissue/MFR
  • Still’s
  • Chapman’s points
  • HVLA
  • Rib raising
  • Paraspinal inhibition
24
Q

How do anterior vs. posterior Chapman’s points differ in their utility?

A
  • Anterior are primarily diagnostic
  • Posterior are focus of treatment
25
Q

Increased sympathetic tone may cause what 3 GI problems?

A
  1. Ileus
  2. Constipation/flatulence –> Increased H2O absorption
  3. Abdominal distention
26
Q

Increased parasympathetic tone may cause what GI disturbances?

A
  • Increased secretion rate of all GI glands
  • Diarrhea/fecal incontinence –> decreased H2O absorption
27
Q

Proximal 2/3 of colon vs. distal 1/3 of colon spinal cord levels and SNS/PNS innervation?

A

Prox. 2/3 = T10-T11

- SNS = lesser splanchnic n. + superior mesenteric ganglion

- PNS = Vagus n.

Distal 1/3 = T12-L2

- SNS = least splanchnic n. + inferior mesenteric ganglion

  • PNS = sacral (pelvic) splanchnic (S2-S4)
28
Q

Any form of SD to the pelvic diaphragm can lead to fluid stasis within the pelvis, which then can cause what?

A

Pelvic congestion, viscerosomatic pain, and inability to clear infections

29
Q

How do the pelvic and thoracic diaphragm work together?

A
  • Both contract and expand during respiratory cycle; in unison
  • Act as a pump, thereby stimulating movement of the vasculature and lymphatic fluids
30
Q

In relation to the Posterior Chapman point for the colon there is a direct fascial relationship between the descending/ascending colon and which muscle?

A

Quadratus Lumborum