Lec 7 Hearing Brain 2 Flashcards

1
Q

nature of sound

A

sound as outcome of physical movement, changes in air pressure

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2
Q

role of outer ear

A

amplifies frequencies, key in locating sounds

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3
Q

role of middle ear

A

converts airborne vibrations into liquid-borne vibrations

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4
Q

role of inner ear

A

converts liquid-borne sounds into neural impulses

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5
Q

what is tonotopic organisation

A

different regions in the brain area process different frequencies

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6
Q

Explain the ‘what’ pathway

A

Ventral route, found in temporal lobe, semantic content coded hear, also responsible for identifiyng ‘who’

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7
Q

Explain the ‘where’ pathway

A

Dorsal route, found in parietal lobe, codes where sound is coming from , also responsible for the ‘how’

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8
Q

define amplitude

A

loudness- high/low- the energy in a sound wave

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9
Q

define pitch

A

frequency in a sound wave

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10
Q

what is inter-aural differences

A

Difference in time taken for sound to reach one ear, dependent on position of sound source relative to head position.
Youmay hear a sound first/more intensely informing direction/location of sound source

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11
Q

head-related transfer function

A

Brain accounts for and corrects for the distortion of sound created due to head and outer ear (pinna) that are in the way of travelling sound

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12
Q

auditory stream segregation

A

complex auditory scenes (loud party/gig) can be divided into different processing streams for pitch, melody, location in space

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13
Q

Cocktail party problem (fMRI study)- method

A

p’s asked to attend to one of 3 people ina group i.e. low/middle pitched speakers

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14
Q

Cocktail party problem (fMRI study)- what 4 brain regions activated? what does each activatr?

A

IFG (inferior frontal gyrus) activation from pitch cues

Dorsal precentral sulcus (DPresCS) and superior parietal lobule (SPL) activation for location, IPS too

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15
Q

what can a spectogram show

A

physically depict speech as cmplex sound waves (darker areas are vowel- since they have a higher frequency

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16
Q

What features are used to produce the sound in our voices

A

Larynx and articulators

17
Q

In a voice processing study, what happens in silent/passive conditions in the brain

A

Compared with silent c, bilateral activation in voice-selective regions of human auditory cortex

18
Q

What does the adaptation paradigm show in a voice processing study

A

(listening to spoken syllables)
voice-selective area (right anterior superior temporal sulcus) sensitive to to change in speaker identity but NOT lexical-phonetic change
= voice processing NOT specific to humans- monkeys do it too

19
Q

TMS effect on voice processing

A

TMS over the voice-selective area of brain disrupts the ability for voice detection. NOT for loudness judgement of same stimuli

20
Q

What is the problem for Pure Word Deafness patients ?

Cause by what?

A

NOT actually dea, but cannot identify speech. Perceive speech as distorted, people sound too fast.
Causedby bilateral STG damage (Superior Temporal gyrus)

21
Q

what is speech perception

A

set of operations transforming auditory signal into mental representations

22
Q

Explain motor theory of speech perception

A

We perceive spoken words by reproducing a speaker’s movement in our vocal tract as opposed to identifying the sound patterns the speech generated by the speaker

23
Q

What does the strong version of motor theory suggest?

A

the motor system for speech production is NECESSARY for speech perception

24
Q

Name of key brain area associated with speech production

A

Broca’s area

25
Q

Support for the Motor Theory

A

1) McGurk Effect (integration of visual info into what we hear)
2) Categorical speech perception- means we cant perceive sounds we cant produce

26
Q

Neuroscientific evidence for motor theory

A

motor system is used in speech perception in a process of auditory-to-articulatory mapping

27
Q

What is the opposing caveat to the supporting evidence for motor theory

A

If motor system is critical for perception of speech sound, then damage to motor speech production system should have substantial impact on ability to perceive speech.
(this is not always the case)

28
Q

Evidence against the strong version of motor theory

A

From aphasic patients-
Hickok et al- Broca’s aphasia are non-fluent in speech production BUT when given speech perception tasks they perform well, suggesting motor speech system NOT necessary for speech perception

29
Q

D’Ausilio et al’s virtual lesion study as more evidence against strong version of motor theory

A

The virtual lesions disrupted recognition of phonemes (labial/dental) after TMS applied but ONLY when presented in background noise

30
Q

What does D’Ausilio’s virtual lesion study suggest about the PMC and speech perception

A

That the PMC (premotor cortex) only contributes to speech perception when the auditory signal is hard to disambiguate

31
Q

Opposing evidence to McGurk effect

A

When TMS applied over superior temporal sulcus (STS) McGurk effect is disrupted

32
Q

What has categorical speech perception study on phonemic discrimination taks suggested

A

Not just the motor cortex that processes speech categorically but whole network of brain areas
Conclude that there are multiple routes to speech perception, inc. WHAT (ventral) and HOW (dorsal) routes

33
Q

Which of the two routes are more agreed on in research

A
the WHAT (ventral) path for semantics is more agreed on in research.
The HOW (where, dorsal) route less agreed on- rather favoured by strong version of motor theory