Language Flashcards

1
Q

what 2 things does speech consist of

A

phonation and articulation

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2
Q

what controls speech

A

UMN control, modulated by extrapyramidal and cerebellar systems

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3
Q

what is phonation

A

sound produced by moving vocal cords

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4
Q

what controls phonation

A

laryngeal muscles innervated by CN X

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5
Q

what is dysphonia

A

hoarse, whispering, breathy

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6
Q

what is articulation

A

sound production by actions and varied posoitions of the lips, tongue, palate, pharynx

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7
Q

what muscles and nerves are responsible for articulation

A

facial, oral muscles innervated by CN VII, IX, X, XII

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8
Q

what is dysarthria

A

slurred, choppy, indistinct - (conversation will make sense)

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9
Q

T or F those unable to speak may still be able to use language

A

T - language is multimodal or symbolic communication. Those unable to speak may still communicate by reading and writing or using gestures

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10
Q

what is aphasia

A

a disorder of previously acquired language ability from a language center lesion in the dominant hemisphere

  • patients have impaired communication by means of any modality including gestures, signing, braille, and others
  • may speak but have trouble communicating

abnormal language

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11
Q

what artery feeds the cortical language centers

A

middle cerebral artery (MCA)

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12
Q

where are language centers located

A

around the lateral sulcus (sylvian fissure) of the dominant hemisphere

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13
Q

if a patient is right handed which hemisphere is the dominant one

A

the left

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14
Q

if a patient is left handed what hemisphere is dominant

A

50% left

50% right

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15
Q

what is Wernicke’s area responsible for

A

language comprehension, via listening, reading, and other modes

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16
Q

what is Broca’s area responsible for

A

language expression or execution, by speaking, writing and other modes

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17
Q

what can cause lesions in cortical areas that could cause aphasia

A
  • ischemic infarction (occlusion of dominant MCA or its branches)
  • hemorrhage, tumor, trauma or dementia
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18
Q

which modes of communication are affected by a lesion in the critical cortical areas of language

A

all modes (listening, speaking, reading and writing are similarly affected)

19
Q

what is fluency

A

ease, facility and quantity of speech, regardless of content or meaning

20
Q

what is paraphasia

A

word or syllable substituations (sully for silly or blue for green)

21
Q

what is neologism

A

nonsense words (type of paraphasia ) ie scatifang

22
Q

where is the lesion in Broca’s aphasia

A

posterior, inferior frontal lobe

  • frequently involves the adjacent motor cortex causes right hemiparesis and facial weakness
23
Q

How would a patient with Broca’s aphasia present

A
  • laborious, effortful, telegraphic nonfluency (I…up….early)
  • preserved comprehension (leads to frustration)
  • imperfect repetition
24
Q

where is the lesion in Wernicke’s aphasia

A

posterior, superior temporal lobe

25
how would a patient present with Wernicke's aphasia
- very fluent, but many filler words, paraphasic errors, few meaningful words: "if you saw it, redness would schloodder over the alls, anyway" - poor comprehension - imperfect repetition
26
what does nonsensical, spoken gibberish suggest in in a patient in absence of hemiparesis
patient is psychiatric or on drugs
27
where is the lesion in conductive aphasia
arcuate fasciculus (tract between Broca's and Wernicke's areas)
28
how does a patient with conductive aphasia present
- fairly fluent, with some paraphasic errors. - "intermediate" imperfect comprehension (like a milder version of Wernicke's aphasia) - imperfect repetition
29
how does a patient with global aphasia present
- severely nonfluent (mute), poor comprehension
30
where is the lesion in global aphasia
- extensive lesion of Broca's, Wernicke's and arcuate fasciculus
31
what is alexia
impaired reading when visual cortex is disconnected from language centers
32
what is agraphia
impaired writing when motor cortex for dominant hand is disconnected from the language centers
33
what clinical symptoms does a lesion in the posterior dominant hemisphere cause
alexia and agraphia
34
what does a lesion in the nondominant hemisphere cause
aprosodias (abnormal prosody)
35
what is prosody
semantic and emotional meaning conveyed by changes in vocal pitch, inflection, melody or tone of speech (rising pitch at the end of a sentence = a question)
36
what are the 2 types of Aprosodias
Sensory/receptive and motor/expressive
37
where is the lesion in a sensory/receptive aprosodia
nondominant "wernicke's area" lesion
38
how does a sensory/receptive aprosodia present
- poor prosodic comprehension when listening - speaks with normal prosody - unable to repeat prosody in other's speech can't tell the difference if I say "this is yours." vs "this is yours?" but can speak it themselves
39
where is a lesion causing motor/expressive aprosodia
nondominant Broca's area lesion
40
how does a patient with a motor/expressive aprosodia present
- good prosodic comprehension when listening - lack of prosody when speaking - unable to repeat prosody in other's speech
41
how is language defined neurologically
communication by means of symbols
42
what does perisylvian language center refer to
Braca's and Wernicke's areas
43
where is a lesion that causes imperfect repetition located
either the perisylvian language center (Broca's or Wernicke's areas) or the connecting arcuate fasciculus
44
paraphagia's are more common with lesions in what areas
Wernicke's ( lesions of the posterior perisylvian language center)