Language Flashcards

1
Q

what 2 things does speech consist of

A

phonation and articulation

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2
Q

what controls speech

A

UMN control, modulated by extrapyramidal and cerebellar systems

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3
Q

what is phonation

A

sound produced by moving vocal cords

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4
Q

what controls phonation

A

laryngeal muscles innervated by CN X

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5
Q

what is dysphonia

A

hoarse, whispering, breathy

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6
Q

what is articulation

A

sound production by actions and varied posoitions of the lips, tongue, palate, pharynx

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7
Q

what muscles and nerves are responsible for articulation

A

facial, oral muscles innervated by CN VII, IX, X, XII

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8
Q

what is dysarthria

A

slurred, choppy, indistinct - (conversation will make sense)

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9
Q

T or F those unable to speak may still be able to use language

A

T - language is multimodal or symbolic communication. Those unable to speak may still communicate by reading and writing or using gestures

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10
Q

what is aphasia

A

a disorder of previously acquired language ability from a language center lesion in the dominant hemisphere

  • patients have impaired communication by means of any modality including gestures, signing, braille, and others
  • may speak but have trouble communicating

abnormal language

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11
Q

what artery feeds the cortical language centers

A

middle cerebral artery (MCA)

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12
Q

where are language centers located

A

around the lateral sulcus (sylvian fissure) of the dominant hemisphere

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13
Q

if a patient is right handed which hemisphere is the dominant one

A

the left

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14
Q

if a patient is left handed what hemisphere is dominant

A

50% left

50% right

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15
Q

what is Wernicke’s area responsible for

A

language comprehension, via listening, reading, and other modes

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16
Q

what is Broca’s area responsible for

A

language expression or execution, by speaking, writing and other modes

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17
Q

what can cause lesions in cortical areas that could cause aphasia

A
  • ischemic infarction (occlusion of dominant MCA or its branches)
  • hemorrhage, tumor, trauma or dementia
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18
Q

which modes of communication are affected by a lesion in the critical cortical areas of language

A

all modes (listening, speaking, reading and writing are similarly affected)

19
Q

what is fluency

A

ease, facility and quantity of speech, regardless of content or meaning

20
Q

what is paraphasia

A

word or syllable substituations (sully for silly or blue for green)

21
Q

what is neologism

A

nonsense words (type of paraphasia ) ie scatifang

22
Q

where is the lesion in Broca’s aphasia

A

posterior, inferior frontal lobe

  • frequently involves the adjacent motor cortex causes right hemiparesis and facial weakness
23
Q

How would a patient with Broca’s aphasia present

A
  • laborious, effortful, telegraphic nonfluency (I…up….early)
  • preserved comprehension (leads to frustration)
  • imperfect repetition
24
Q

where is the lesion in Wernicke’s aphasia

A

posterior, superior temporal lobe

25
Q

how would a patient present with Wernicke’s aphasia

A
  • very fluent, but many filler words, paraphasic errors, few meaningful words: “if you saw it, redness would schloodder over the alls, anyway”
  • poor comprehension
  • imperfect repetition
26
Q

what does nonsensical, spoken gibberish suggest in in a patient in absence of hemiparesis

A

patient is psychiatric or on drugs

27
Q

where is the lesion in conductive aphasia

A

arcuate fasciculus (tract between Broca’s and Wernicke’s areas)

28
Q

how does a patient with conductive aphasia present

A
  • fairly fluent, with some paraphasic errors.
  • “intermediate” imperfect comprehension (like a milder version of Wernicke’s aphasia)
  • imperfect repetition
29
Q

how does a patient with global aphasia present

A
  • severely nonfluent (mute), poor comprehension
30
Q

where is the lesion in global aphasia

A
  • extensive lesion of Broca’s, Wernicke’s and arcuate fasciculus
31
Q

what is alexia

A

impaired reading when visual cortex is disconnected from language centers

32
Q

what is agraphia

A

impaired writing when motor cortex for dominant hand is disconnected from the language centers

33
Q

what clinical symptoms does a lesion in the posterior dominant hemisphere cause

A

alexia and agraphia

34
Q

what does a lesion in the nondominant hemisphere cause

A

aprosodias (abnormal prosody)

35
Q

what is prosody

A

semantic and emotional meaning conveyed by changes in vocal pitch, inflection, melody or tone of speech (rising pitch at the end of a sentence = a question)

36
Q

what are the 2 types of Aprosodias

A

Sensory/receptive and motor/expressive

37
Q

where is the lesion in a sensory/receptive aprosodia

A

nondominant “wernicke’s area” lesion

38
Q

how does a sensory/receptive aprosodia present

A
  • poor prosodic comprehension when listening
  • speaks with normal prosody
  • unable to repeat prosody in other’s speech

can’t tell the difference if I say “this is yours.” vs “this is yours?” but can speak it themselves

39
Q

where is a lesion causing motor/expressive aprosodia

A

nondominant Broca’s area lesion

40
Q

how does a patient with a motor/expressive aprosodia present

A
  • good prosodic comprehension when listening
  • lack of prosody when speaking
  • unable to repeat prosody in other’s speech
41
Q

how is language defined neurologically

A

communication by means of symbols

42
Q

what does perisylvian language center refer to

A

Braca’s and Wernicke’s areas

43
Q

where is a lesion that causes imperfect repetition located

A

either the perisylvian language center (Broca’s or Wernicke’s areas) or the connecting arcuate fasciculus

44
Q

paraphagia’s are more common with lesions in what areas

A

Wernicke’s ( lesions of the posterior perisylvian language center)