Gait Flashcards

1
Q

gait apraxia

A

Memory or concept of walking “put into action” (praxis: concept is executed or performed “on command”; patients with gait apraxia are immobile when asked to walk, despite having all the other essentials

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2
Q

what 5 things are needed for normal walking

A
  1. Strength (UMNs, LMNs, NMJs, muscle)
  2. Coordination (cerebellar system)
  3. Postural control (extrapyramidal system)
  4. Sensation (particularly proprioception)
  5. Memory or concept of walking
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3
Q

what kind of lesion does a Romberg sign indicate and what function is lacking as a result

A
posterior column (or sensory nerve) lesion
lack of proprioception
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4
Q

where is the problem if a patient cannot stand with feet together with eyes open

A

cerebellar disease

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5
Q

describe a broad based gate

A
  • feet spread wide apart for stability;

* gait much more unsteady walking a straight line (tandem or heel-to-toe);

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6
Q

where is a lesion in a patient with a broad based gait

A

lesion of the posterior columns or sensory (proprioceptive) nerves (worse with eyes closed) or cerebellum

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7
Q

describe a hemiplegic gait

A
  • affected lower limb is stiffly extended and
    swung or circumducted;
    • affected ipsilateral upper limb is flexed at
      elbow and wrist with decreased armswing;
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8
Q

when is a hemiplegic gait normally seen

A

stroke patients

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9
Q

describe a tabetic gait

A

“foot slapping” gait, where patient compensates for impaired sensation by forcibly planting the feet down to “feel” the floor;

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10
Q

what is a tabetic gait associated with

A

neurosyphilis (tabes dorsalis) - would also see Argyle-Robinsin pupils (constrict to near reflex but not light)

or severe neuropathy

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11
Q

describe a steppage gait

A
  • caused by foot drop (weak dorsiflexion);
    • to prevent tripping over the toes, the hip
      is flexed even higher to elevate the
      drooping foot, which is lowered to the
      floor toe first;
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12
Q

what is a steppage gait associated with

A

peroneal nerve or L5 root lesions, or severe peripheral neuropathy

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13
Q

describe a waddling gait

A
  • when walking, weak pelvic or hip muscles cannot support the body “on one leg” while the opposite foot is lifted off the ground;
  • patient compensates by swaying or leaning to the left when the right foot is raised and vice versa, alternately tilting the pelvis from side to side, reminiscent of a waddling duck;
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14
Q

what is a waddling gait associated with

A

myopathy

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15
Q

describe a scissors gait

A
  • although the legs are weak, marked spasms and tightness in the adductor muscles of the thighs force the knees stiffly together when walking;
  • legs tend to cross over each other, like the closing blades of a scissors;
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16
Q

what is a scissors gait associated with

A

due to corticospinal tract lesions affecting the legs (spastic paraparesis) as in cerebral palsy or MS

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17
Q

describe a Parkinsonian gait

A
  • slow, shuffling gait, with “stooped forward” posture and lack of a normal arm swing;
  • festination—leaning further and further forward to walk, the patient then runs to “catch up” with the center of gravity;
  • turning around is laboriously slow, requiring multiple, small steps, often with a tendency to fall over;
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18
Q

what are some ways to test cerebellar function

A

finger to nose
heel to knee to shin
hand flip flop
rebound

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19
Q

describe the characteristics of cerebellar dysarthria and what part of the cerebellum is affected

A

slurred, thick, scanning (erratic, jerky, explosive or hyphenated quality) speech

left cerebellum

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20
Q

what types of eye movements may be seen with cerebellar problems

A

nystagmus with erratic, jerky, eye movements

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21
Q

what kinds of abnormal limb control may be seen in cerebellar disease

A

kinetic tremor
dysmetria
decomposition of movement
loss of check response

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22
Q

what is a kinetic tremor

A

rhythmic oscillations during limb movement toward a target

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23
Q

what is dysmetria

A

overshooting or undershooting a target

24
Q

what is decomposition of movement

A

a normally smooth movement becomes jerky, “broken down”

25
Q

what is loss of check response

A

sudden release of contracted biceps leads to striking the face; triceps does not “check” this action due to imbalance of agonist/antagonists muscle

26
Q

what does a midline cerebellar lesion mostly affect

A

the trunk

27
Q

what does a cerebellar lesion of the hemispheres mostly affect

A

the limbs

28
Q

what movement disorders are seen in hemispheral sydrome

A
  • predominantly affects ipsilateral limbs (kinetic tremor, limb dysmetria, dysdiadochokinesia, rebound phenomenon);
29
Q

what causes hemispheral syndrome

A

ipsilateral infarction, hemorrhage, tumor or multiple sclerosis lesion (bilateral lesions in degenerative diseases)

30
Q

what movement disorders does vermal (vermian) syndrome cause

A

predominantly affects the trunk - truncal unsteadiness with standing or walking, tremor, postural imapairment, gait ataxia

31
Q

what can cause vermal syndrome

A

hemorrhage, tumor, MS, degenerative disorders, alcoholic cerebellar degeneration

32
Q

what is alcoholic cerebellar degeneration

A

atrophy of anteriorsuperior vermis, with gait ataxia and lower limb dysmetria

33
Q

how is spinocerebellar degeneration aquired

A

hereditary degenerative disorders of unknown cause and no curative treatment

34
Q

what is predominantly affected in spinocerebellar degeneration (ataxias)

A

the nuclei and tracts of the cerebellum and spinal cord in a progressive fashion (older patients become wheelchair dependent )

35
Q

what is the most common type of Spinocerebellar degeneratiion

A

Friedreich’s ataxia

36
Q

what disease is associated with a predominantly resting tremor

A

parkinsons

37
Q

what is a familial essential tremor

A

a postural tremor and a kinetic tremor

38
Q

what is Choreoathetosis

A

slow, writhing, continual limb movements (athetosis) plus brief, irregular, flowing “dancelike” movements (chorea) affecting limbs, trunk and face

39
Q

what can cause choreoathetosis

A

lesions in the caudate nucleus (Huntington’s disease) or its connecting pathways

or high levels of dopaminergic medications may produce choreoathetosis or dystonia

40
Q

what is hemiballismus

A

rapid, violet (“ballistic”), flinging movements of proximal upper and lower limbs on one side

41
Q

what causes hemiballismus

A

lesion (usually infarction) of the contralateral subthalamic nucleus

42
Q

what is dystonia

A

continual or sustained painful contraction of muscles, causing turning and spasms of the limbs or neck, with fixed, unnatural postures

no specific lesion or pathology has been correlated with this disorder

43
Q

what type of dystonia is focal

A

torticollis

44
Q

what type of dystonia is generalized

A

dystonia musculorum deformans

45
Q

what is a tic

A

brief, semipurposeful, stereotyped, repetitive contractions of groups of muscles (e.g., eye blink, facial twitch, sniff)

46
Q

what is thought to cause tics

A

decreased motor inhibition in the basal ganglia possibly

47
Q

What is seen in Tourette’s syndrome

A
  • –motor and vocal (e.g., grunts, growls) tics;
  • –inherited, with variable penetrance; more often in boys;
  • –attention deficit disorder;
  • –behavioral problems;
48
Q

what is myoclonus

A

rapid, shocklike movements of the limbs or body, usually bilateral, but often asynchronous

49
Q

what causes myoclunus

A

due to diffuse encephalopathies from neurological (e.g., Creutzfeldt-Jakob disease) or medical diseases (e.g., renal or hepatic failure, anoxia);

50
Q

what is asterixis

A

a flapping tremor of the extended hand or foot due to loss of postural tone

51
Q

when do you see bilateral asterixis

A

diffuse encephalopathies from medical diseases (renal or hepatic failure)

52
Q

when is unilateral asterixis seen

A

in structural brain lesions

53
Q

what medications can be given to help alleviate a parkinsonian tremor

A

anticholinergics, L-dopa, dopamine agonists

54
Q

what medications can alleviate an essential tremor

A

beta-adrenergic blockers, barbiturates

55
Q

what medications can alleviate choreoathertosis, hemiballismus, and tics

A

dopamine antagonists

56
Q

what medications can be given to alleviate dystonia

A

anticholinergics, benzodiazepines, botulinum toxin injections