Lamb and Kid Disease Flashcards

1
Q

what is lamb mortality rate between pregnancy and weaning? when do most of losses occur?

A

10-30%
70% in lambs under 3 weeks; 80-90% of these losses in first week

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2
Q

70% of lamb mortality is related to _______ rather than _______

A

management; infectious disease

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3
Q

what are some general causes of lamb death

A

abortion
weak lambs at birth
parturition issues
hypothermia
starvation*
hypoglycemia
*
septicemia
penumonia
diarrhea
predation***

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4
Q

out of all the stillbirth, abortion, and weak newborn causes, how many are due to infectious disease? what are some common infectious diseases?

A

60-70%
Toxoplasma, Chlamydiophila abortus (enzootic abortion), Campylobacter fetus (vibriosis)

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5
Q

which are ZOONOTIC: Toxoplasma, Chlamydiophila abortus (enzootic abortion), Campylobacter fetus (vibriosis)

A

ALL of these

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6
Q

in late winter and early spring, producer finds these lesions on placenta of stillborn/aborted/weak newborns. you diagnose ______ based on the placental lesions, H&E stain, and serology? how do you treat the flock?

A

Toxoplasma
decoquinate

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7
Q

a producer reports fevers, abortions 2 weeks before lambing, stillbirths, and weak lambs. you diagnose Chlamydophila abortus (enzootic abortion). how do you treat? what vaccination do you recommend?

A

tetracycline
vaccine: Bacteria available from US (Colorado serum cpy)

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8
Q

you are investigating a rash of 3rd trimester spring abortions and weak limbs born dead. you note thickened placentas with grey cotyledons and fetal livers with necrotic foci 1-2 cm wide (image). what are you suspicious of?
how will you diagnose, treat, and vaccinate for this?

A

vibriosis from Campylobacter fetus
Giemsa stain or PCR
tetracycline to treat
Bacterin vaccine

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9
Q

what are 5 potential things to include in a protocol for abortion

A
  • separate animal from flock
  • wearing a DISPOSABLE GLOVE remove fetus and placenta, place in a strong garbage bag and REFRIGERATE, or put somewhere cool out of reach of animals
  • monitor rest of flock for abortions and manage as above
  • develop intervention level, eg. 2% of flock aborts –> submit to lab
  • do not be PREGNANT OR ILL and around flock
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10
Q

what % of mortality related to dystocia

A

18% mortality related to dystocia
(think he is saying dystocia has 18% mortality rate? or maybe that of all lamb/kid mortality 18% is related to dystocia?)

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11
Q

what factors may contribute or cause mismothering

A

lambing in group housing - more likely to have mismothering with many ewes who are close together
physical conditions preventing ewe to lick off lamb - primiparous or systemic illness of mom
stealing of lamb by another ewe - often once thief has own lamb will lose interest
iatrogenic

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12
Q

the top cause of death in lambs and kids in the first 48 hours

A

hypothermia - 19% of lamb death

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13
Q

why are lambs and kids terrible at avoiding hypothermia

A

born wet
outdoors is windy (strictly speaking this is not their fault)
lack of fleece (depending on breed)
large surface area, small BW
sometimes twins and triplets
lack of shelter
thin doe or ewe
ewe not licking enough/effectively bc primiparous or systemic illness of dam

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14
Q

how to treat hypothermic newborn

A

warm colostrum, give external source of heat (heat lamp, hairdryer), dextrose 20% IP or OV at 10 ml/kg!, milk or replacer 50 ml/kg 6-8h
and bonus, prevent: avoid drafts

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15
Q

what are some causes of hypoglycemia/starvation in lambs and kids

A

plugs teats, mastitis/hard udder (OPP), poor bonding (various causes/contributors to this), congenital defects, large litter/poor production

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16
Q

treatment for starvation/hypoglycemia in babies

A

recognize early, warming, glucose or dextrose 20% 10/mL/kg

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17
Q

what does colostrum provide a source of

A

ENERGY (heat and glucose)
IgG and other immune things
laxative
macro and microminerals
fat soluble vitamins

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18
Q

what is adequate transfer of immunity

A

delivery of SUFFICIENT QUANTITY of GOOD QUALITY colostrum in GIT and ADEQUATE ABSORPTION of Abs from colostrum to blood (a 2 actors event)
need IgG at least 12 g/L

19
Q

name 4 categories of problems with adequate transfer of passive immunity

A

problems with quality: young, sick, undernourished, poorly vaccinated dam
with availability: prepartum leakage, nursing from another lamb
with ingestion: weak neonate, competition with other lambs
with absorption: weakness, sickness, hypothermia, dehydration, delay in ingestion

20
Q

what are colostrum volume requirements

A

50-75 ml/kg TID first day, first by 2h
- housed kids need 200 ml/kid/day
kids outside in cold need 280 ml/kg/day

21
Q

how is diagnosis of FTPI (<12 g/L IgG) made

A

RID, Zn sulfate turbidity test, GGT, BRIX refractometer, or easiest method is TP (>5.5 g/dL)

22
Q

what are the best sources of colostrum

A

dam best
another ewe
cow colostrum
(can be frozen in 2-3 oz (ice cube tray) then use 8fr rubber feeding tube lubricated and 60cc catheter tip syringe)
commercial colostrum replacers or colostrum supplements also available

23
Q

name several diseases transmissible through colostrum

A

OPP, CAE, paratuberculosis if using cow colostrum, E. coli, Salmonella

24
Q

what are some methods of colostrum sterilization

A

pasteurization at high temp for short period of time (note may cause immune denaturation), or heat treatment

25
a lamb/kid that didn't ingest much colostrum becomes pyrexic, congested mucus membrane, collapses, then develops diarrhea and swollen joints. what is wrong
septicemia
26
what can omphalophlebitis cause? how is omphalophlebitis treated?
septicemia, peritonitis, UTI, arthritis antibiotics and supportive therapy
27
what are 3 ways to prevent omphalophlebitis
sanitation, navel dipping, colostrum
28
what are the 2 most common causes of predation of lambs and kids
large domestic dogs, then coyotes
29
describe spider lamb syndrome / hereditary chondrodysplasia, including susceptible breeds and genetic mutation
deformity of legs and vertebral column by 4-6 weeks of age. C/S include angular limb deformities (arthrogryposis), scoliosis, hypothesis, facial deformities. possible signs at birth include long legs and Roman nose caused by mutation in gene for fibroblast factor receptor 3, FGFR3, and is recessive black face sheep
30
what is wrong
spider lamb syndrome, hereditary chondrodysplasia
31
what is Cache valley virus? how is it transmitted? which species does it affect?
cause of ovine/caprine pregnancy failure and neonatal malformations it is an Orthomunyavirus transmitted by mosquitoes and endemic in NAm affects goats, cattle, horses, deer, HUMANS
32
what is wrong
Cache valley virus
33
a goat is normal at birth, but at age 3-10 days presents with sudden onset of classic paralysis. PE reveals decreased muscle tone, anorexia, apathy. BW shows low pH, low HCO3, increased D-lactate, increased anion gap. what is wrong?
floppy kid syndrome - this is a metabolic acidosis without signs of dehydration; it is a transient and reversible condition that can occur in any goat breed and there is no proven etiology. differentials would be vitamin E/Se deficiency, enterotoxemia, sepsis
34
how to treat floppy kid disease
oral sodium bicarb (1/2 teaspoon) maybe antacids IV fluids if severe
35
several lambs/kids aged 1-3 weeks present with fever and tachypnea. one dies and this is a portion of the lungs. what is wrong? how to treat?
pneumonia - can be due to virus, P. multocida, or likely in this case Mannheimia haemolytica (portion of lungs affected) tetracycline or of fails ceftiofur
36
what are some causes of nutritional diarrhea
heavy milker, singleton or death of twin, change from milk to milk replacer, error in preparation of milk replacer
37
what are some C/S of infectious diarrhea
wet fleece and tail, then if more severe: lethargy, staggering, sunken eyes, inability to stand, possibly hypothermia
38
how is infectious diarrhea diagnosed and treated
lab evaluation tx: calf oral electrolytes 100 ml QID and possibly IV electrolyte solutions
39
how is infectious diarrhea prevented
isolate affected lambs or kids, move to another area clean udder ensure adequate colostrum there is no scours vaccine labelled for small ruminants
40
this disease can affect adults and young SR. C/S include shock, diarrhea with mucus and blood, abdominal pain, opisthotonus, paddling, and glucosuria when acute. or sudden death if parachute. on necropsy you can find pericardial fluid accumulation, petechial hemorrhages on serosal surfaces, and a soft pulpy kidney. what is it? describe the disease a bit
enterotoxemia or pulpy kidney disease caused by C. perf type D, which is normally in intestines, but can proliferate when changes in diet occur. epsilon toxin increases gut permeability, moves into bloodstream, and causes edema and vascular damage of brain, kidney, heart and lungs
41
describe treatment for pulpy kidney disease / enterotoxemia
pain relief with flunixin 2 mg/kg IV or meloxicam 1 mg/kg SQ antitoxin 200 IU/kg IV tetracycline in feed this is for outbreaks.
42
describe prevention of pulpy kidney disease / enterotoxemia
multivalent vaccine 2 doses 4-6 weeks apart and a booster at 6 months avoid digestive disturbances (avoid bacterial proliferation in gut)
43
Schmallenberg virus is currently in Europe only, but could become an issue here as well. describe which species it affects, how it is transmitted, and what is banned from entry to Canada (from the EU)
cattle, small ruminants, probably deer transmitted by biting midges and mosquitoes, probably starting in 2012, semen and embryo had to test negative for the virus before entering Canada, but since 2020 they are banned (bovine, bison, water buffalo, sheep, goat semen; bovine, bison embryos)
44
what are the two forms of Schmallenberg virus
adults: fever, drop in milk production, sometimes diarrhea abortion, mummification, newborn calves and lambs with malformations