LA Acute Abdomen Diagnosis and Plan Flashcards

1
Q

describe what to do for a nonstrangulating obstruction in the field versus in sx referral hospital

A

small intestine:
-field: refer! fluid can’t get to LI to get absorbed, so needs IV fluid support
–fluid could also back up to need to check for reflux regularly

-surgical referral hospital: medical to start; often best response from horse, or surgery as indicated

large intestine:
-field: treat or refer
–often can respond to medical management (CARE WITH CECAL IMPACTIONS though; high incidence of rupture); refer as indicated

-referral hospital: often respond best to medical therapy, best response with horses; surgery as indicated

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2
Q

describe next steps for a strangulating obstruction in field versus surgical referral hospital

A

small intestine:
-field: refer or euthanize
–need surgery to survive
–can refer for further diagnostics if need confirmation
–if sx not an option, euthanasia

-sx referral hospital: need surgery to survive, euth if sx not an option

large intestine:
-field: refer or euthanize, same as small
-sx referral: same as small

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3
Q

describe next steps for an inflammatory lesiol

A

small intestine:
-field: REFER
–lots of reflux: frequent check
–shock, SIRS: intensive management

-sx referral:
–medical: supportive care is mainstay of treatment
–surgery if indicated: decompression

large intestine:
-field: REFER
–shock, SIRS: intensive management
–zoonotic, contagious risk

-referral hospital:
–medical: supportive care is mainstay of treatment
–surgery if indicated decompression

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4
Q

what goes in to making the decision to treat colic in field?

A
  1. diagnosis: confidence in the diagnosis
  2. PAIN:
    -severity of pain
    -duration/recurrence
  3. response to treatment
  4. expected treatment requirements
    -frequency and skill required to perform
  5. owner:
    -comfort with monitoring and treatment
    -cost, convenience
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5
Q

what goes in to the decision to recommend surgery for colic?

A
  1. diagnosis: confidence in diagnosis that requires surgery
  2. PAIN:
    -severity
    -duration/recurrence
  3. response to treatment
  4. systemic/gastrointestinal deterioration
  5. owner: risks of surgery vs risks without surgery
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6
Q

describe the types of medical treatment for colic

A
  1. non-specific, symptomatic:
    -analgesics (alpha-2 agonists)
    -anti-spasmodics (Buscopan)
    -trocharization (relieve gas inside a GI structure; can be part of stabilization and also pain relief)
  2. non-specific, supportive care:
    -fluids, electrolytes, acid base
    -nutrition
  3. specific therapies:
    -impactions: laxatives
    -endotoxemia
    -LDDLC
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7
Q

describe the types of surgical treatment

A
  1. remove contents:
    -alleviate distension, remove obstruction, remove source of endotoxin
    -needle decompression, move SI contents into cecum, enterotomy, high enema
  2. correct positioning:
    -alleviate obstruction of GI +/- blood supply
    -for displacements and strangulating lesions
  3. resection and anastomosis
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8
Q

give specifics for ileal impactions (NSO SI)

A
  1. history of coastal/Bermuda hay
  2. tapeworms often involved
  3. sometimes palpable on rectal exam
  4. treatment: most resolve with supportive care and analgesia
  5. prognosis: for survival is good for medical and surgical treatment
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9
Q

give specifics for ascarid impaction (NSO SI)

A
  1. 5-8 month old foals most common; weanlings
  2. often occur after 1st antiparasitic administration
  3. can sometimes visualize in SI lumen with ultrasound
    -look like traintracks/double lines
  4. less severe cases can be medically managed then give slow kill dewormer
  5. if unresponsive, surgery is indicated
    -prognosis considered poor
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10
Q

describe treatment and prognosis for SI bezoars in camelids

A
  1. treatment: surgery!
  2. prognosis better with earlier intervention and duodenal (vs jejunal which is poor prognosis)
  3. rupture/leakage reported before, during, and after surgery, so DONT WAIT, refer!
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11
Q

describe specifics for impactions (NSO LI)

A
  1. history clues: cecal bute admin, stall rest, small colon diarrhea, sandy area
  2. pelvic flexure, small colon, and cecum often palpable on rectal exam
  3. treatment:
    -medical management often includes laxatives: water, electrolyte water, MgSO4, oil, psyllium for sand
    -water is better than oil! will dissolve poop, oil will not

-cecal: rupture more common than others so consider surgery more rapidly

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12
Q

describe specifics for enteroliths (NSO LI)

A
  1. history: california, alfalfa hay
  2. radiographs can be diagnostics
    -except for RDC
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13
Q

describe specifics for displacements (NSO LI)

A
  1. LDDLC: approx 20% reflux!
    -rectal findings (tightly stuck squished colon with a tight band) are diagnostic!
    -can rule out with ultrasound if see spleen under kidney
  2. RDDLC: colonic mesenteric vessel on right side and rectal transverse band suggestive on ultrasound
  3. treatment and prognosis:
    -recurrence reported at a wide range
    -RDDLC: increased incidence of recurrent colic post-op
    -LDDLC: specific medical management phenlyephrine, jogging, rolling
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14
Q

describe general treatment and prognosis for horse NSO LI

A
  1. often resolve with medical management
  2. surgical prognosis good to very good
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15
Q

describe diagnostics, treatment, and prognosis for spiral colon bezoars in camelids (LA NSO LI)

A
  1. treat with surgery
  2. prognosis better with earlier intevrention and better for LI and SI
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16
Q

describe diagnostics, treatment, and prognosis atresia coli (LA NSO LI)

A
  1. signalment: within days of birth
  2. dx: contrast radiography
    -enema = no fecal staining! (normal - meconium)
  3. prognosis for productivity poor
17
Q

describe diagnostics, treatment, and prognosis for cecal dilation/dislocation (LA NSO LI)

A
  1. signalment/history:
    -dairy
    -within 60 days of freshening
    -concurrent peripartum disease
  2. dx: rectal, ping, distension
  3. tx: may be able to treat with prokinetics and supportive care
  4. prognosis: high rate of recurrence

above also applies to torsion and volvulus (signalment, treatment)

18
Q

describe working diagnosis specifics for horse strangulating obstruction in small intestine

A

signalment/history:
1. strangulating lipoma: older horses (10 to >15)
2. epiploic foramen entrapment: cribbers
3. inguinal hernia: stallions, recent breeding
4. intussusceptions:
-jejuno-jejunal: foals
-ileocecal: young adult or tapeworm infection

exam:
1. inguinal hernia: testicular and rectal palpation, ultrasound scrotum

  1. intussusceptions: ultrasound bulls-eye, abdominocentesis changes may lag (sequestered)
19
Q

describe working diagnosis specifics for other LA SO SI

A
  1. intussusception:
    -hx/signalment:
    –calves <2 months
    –Brown swiss
    –other GI dz: enteritis, parasites: change motility
    -dx: ultrasound bullseye
  2. volvulus:
    -hx/signalment: calves, 1wk-6mo
  3. HBS:
    -hx/signalment: >4 y/o, brown swiss in peak milk production (3-4 months)
    -tarry (raspberry jam), scant feces
    -C. perfringens A +/- aspergillus fumigatus
20
Q

describe treatment and prognosis for SO SI

A
  1. horses:
    -variable prognosis to discharge
    -better without resection and anastomosis
    -prognosis better for foals and older horses
    -prognosis for J-J may be better than J=I and/or J-C
  2. other LA:
    -intussception: often need resection and anastomosis, reported prognosis variable
    -volvulus: prognosis best if catch early and less intestine involved
    -HBS: high mortality, particularly poor if require resection and anastomosis
21
Q

describe working diagnosis specifics for SO LI

A

horse:
-LCV: hx/signalment is post-partum mare

-intussusception:
–hx/signalment: more ccommon <3 years old parasitism might play a role
–dx: can present acute or chronic, bulls-eye on US, abdominocentesis may not change as expected if sequestered

other LA:

-intussusception, mesenteric volvulus like small intestine
-cecal torsion/volvulus for dilation/doslocation

22
Q

describe treatment and prognosis for SO LI

A

horse:

  1. LCV: variable prognosis, duration likely plays a role
  2. intussusception: prognosis 50-80%
    -Ce-ce better than ce-colon
    -ranked best to worse prognosis: reduce only, typhlectomy, colotomy and typhlectomy

other LA:
-like with SO SI, prognosis likely better with faster diagnosis and treatment

23
Q

describe anterior enteritis/duodenal proximal jejunitis (AE/DPJ)

A

etiology: C. difficile, salmonella, mycotoxins, C. perfringens

signalment/hx:
-avg 5-10 yr
-mares may be more predisposed
-stress: foaling, other changes
-high concentrate diet

treatment:
-aggressive medical supportive care
-surgery: does not appear to decrease reflux volume post op

prognosis:
-good in most

24
Q

describe idiopathic peritonitis

A

etiology: unknown; transmural parasite migration proposed

clinical signs: lethargy, inappetance, colic, depression

PE: fever

ultrasound: +/- increase echogenic fluid, often see thickened SI/LI

abdominocentesis:
-grossly discolored, cloudy
-VERY increased TP, lactate, and WBCC (avg >150,000 cells/ul)

bloodwork:
-dehydration
-WBC abnormalities common

culture: not always positive (blood culture media may be better)
-actinobacillus spp seems most common

treatment: broad spectum abx and supportive care
-prognosis to discharge and 1 year very good
-recurrent colic does not appear increased relative to other causes of colic