L9 Pulp Reaction to Restorative Procedures Flashcards
What features have been measured in research to histologically assess the composition of pulp beneath cavities?
- Reduction in odontoblast number
- Displacement of odontoblast nuclei
- Inflammatory cells in parts of pulp normally occupied by odontoblasts
- Inflammation elsewhere in the pulp
- Reparative dentine formation betneath the cavity
- Distance of inflammation from cavity floor (mm)
What are the effects of cutting a tooth?
Creates heat and pressure, and desiccates (dries) the tooth.
What factors influence the degree or likelihood of pulp injury during the cutting process?
- Residual dentine thickness
- State of pulp prior to treatment e.g. pre-existing inflammation
- Quality of dentine e.g. reactionary
- Mass of material
- Area of cavity floor (no. of tubules occluded)
When does reparative dentine production begin after cavity preparation?
Approx. 20 days after cavity prep.
After 100 days 0.1mm will be deposited.
Why are most materials most likely to be irritants when first placed?
Because when they are first placed they have not yet fully polymerised and most of the irritant constituents are only partially reacted, so are free to cause tissue damage.
How can toxic constituents be released from an already set material?
- By corrosion
- By abrasion
- By leaching (if soluble) e.g. fluoride leaching from GIC
Which materials were found to have the most and least effect on fibroblasts in vitro?
- Amalgam was the least toxic
- Zinc eugenol was the most toxic
What is a key factor that contributes to pulp damage in restorative procedures and how can it be overcome?
Bacterial microleakage
- A base must be used to prevent microleakage between material and tooth tissue
- E.g. Kalzinol (zinc oxide eugenol cement)
- Bacterial infection is more important than the restorative material of choice itself
- More likely to be penetration from the tooth surface than proliferation of
bacteria trapped in the cavity at the time of restoration
- Extent of bacterial microleakage seems to be the most important factor
determining the extent of the inflammatory changes in the pulp beneath cavities