L4 Reading Difficulties Flashcards

1
Q

What is dyslexia?

A

Learning difficulty affecting skills involved in accurate and fluent word reading and spelling
- slow word processing
- spelling
- visual perception
- working memory
- attention
- planning

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2
Q

Who has dyslexia?

A

9-12% of the population
More frequent in males than females

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3
Q

What is the multi-deficit model by Pennington 2006 ?

A

Can be used for multiple behavioural disorders but made specifically for dyslexia
Look at behaviour when trying to diagnose
Behavioural disorders,
Cognitive processes,
Neural systems,
Aetiological risk and protective factors

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4
Q

What evidence is there to suggest that there is a genetic basis?

A

Reading ability seems to be heritable
Heritability estimates 40-60% - varies
Child is at greater risk if parents have dyslexia (Schulte-Korne 1996)
From twin studies Stevenson 2987 found heritability estimates of 29% for reading abilities and 73% for spelling abilities

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5
Q

What are candidate genes?

A

Many genes that may be used when spelling and writing
- KIAA0319
- DCDC2
- ROBO1

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6
Q

What is the evidence for candidate genes?

A

KIAA0319 - affects neural migration and radial glia adhesions, evidence from genetic pooling of 223 subjects with DD
DCDC2 - particularly associated with the severe phenotype of dyslexia, longitudinal study found that genetic risk from DCDC2 strengthens the link between pre-reading maze learning

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7
Q

What is the difference of pleiotropy and polygenicity?

A

Pleiotropy - one gene effects many things
Polygenicity - many genes contribute to single trait

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8
Q

What is the generalist genes hypothesis?

A

Genes for LD are same as those for normal variance
Genes for symptom A are same as those for symptom B
Genes for one LD are same as those for another LD

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9
Q

How do genes influence dyslexia?

A

Genetic correlates with other types of neurodiversity and psychiatric disorders
- associations between dyslexia risk and ADHD risk + bipolar, schizophrenia
Genes interact with the environment
- interacts change overtime
- higher influence when socio-economic status is high

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10
Q

What are some environmental factors?

A

Home learning environment
School conditions
Socio-economic status
Stress

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11
Q

What did Dilnot et al 2017 research?

A

Investigates risk factors for predicting attention, behaviour and reading readiness in those at risk of dyslexia
Home literacy environment
No main effect or family risk once environment controlled
Environment had a large effect for expression of dyslexia

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12
Q

What is the phonological deficit theory?

A

Deficit in phonological awareness - recognising spoken parts of words

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13
Q

What did Swan and Goswami 1997 find about the phonological deficit theory?

A

Measured picture naming, and phonological abilities
Found children with dyslexia had poorer performance for picture naming than neurotypical controls
Linguistic skills - children with dyslexia performed similarly to reading age controls, poorer performance for phenomics tasks
Both groups had better performance for high frequency words

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14
Q

What is the atomisation hypothesis?

A

Dyslexia is the result of a deficit in automatic processing
Difficulty atomising reading behaviours once learnt
Might be caused by reduced activation in the cerebellum

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15
Q

What was Moores 2003 research into the atomisation hypothesis?

A

People with dyslexia and controls took part in tasks that required focusing and shifting attention between two targets
Found both groups similar results for focusing
But dyslexia group performed worse than controls when the task relied on shifting attention
Recognition di not generalise when criteria were changed

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16
Q

What is the single deficit model?

A

One to one relationship between an explanation and the presentation of the disorder
Singular explanation

17
Q

What are the multiple deficit model?

A

Complex eitology
Shared factors contributing to a cognitive skill
Genes have a probabilistic influences

18
Q

What is comorbidity?

A

Twos simultaneous diagnosis
May be associated but are not necessarily related

19
Q

What is the first explanation for comorbidity?

A

One diagnosis increases the likelihood of receiving another diagnosis
Do not have to be related

20
Q

What is the second explanation for comorbidity?

A

Shared liabilities
Genetic, cognitive or cellular basis

21
Q

What is the third explanations of comorbidity?

A

Correlated liabilities
Associated but not risk factors

22
Q

What was Snowling et al 2019 research into comorbidity?

A

Longitudinal study 260 children
Observed before entering school and during
Measured a range of cognitive predictors
- poor language as preschool precursor
- dyslexia and DLD show different developmental trajectories
- very important to examine the influence of time on outcomes

23
Q

Why does comorbidity occur between dyslexia and other disorders?

A

Shared genetic eitology
Shared cognitive bases
Evidence points towards similar underlying mechanisms rather than causality

24
Q

What is an evidence based intervention?

A

Intervention should be based on:
- theory of how a skill develops
- theory of how a skill is promoted
- high quality evidence

25
Q

What is a theory based intervention?

A

Theories inform which skills are targeted by the intervention
e.g. phonological deficit theory identifies phonemic awareness as a key skill
- intervention based on isolating, identifying and blending phonemes

26
Q

What is the review of phonological awareness training?

A

Highly effective across literacy domains
Not effective outside domain
Improved in combination with letter knowledge

27
Q

What is the theory of how a skill is developed?

A

Phonics instruction can be used to improve letter sound knowledge
- synthetic phonics
- analytic phonics
Both techniques are useful but a combined approach with synthetic phonics is most successful

28
Q

What does it mean by high quality evidence?

A

Rigorous trials
Educationally realistic trials
Randomised controlled trials
Scaled up classroom research

29
Q

What are the important things to consider?

A

Cost
How are they implemented
How can we accommodate for those if intervention is unsuccessful