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Oral Path 1 > L4 > Flashcards

L4 Flashcards

1
Q

Localized Juvenile spongiotic gingival hyperplasia Clinical

A

Bright red, velvety or papillary plaque

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2
Q

Localized Juvenile spongiotic gingival hyperplasia location

A

Facial gingiva; maxillary predilection

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3
Q

Localized Juvenile spongiotic gingival hyperplasia Tx

A

No response to improved oral hygiene

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4
Q

Necrotizing Ulcerative gingivitis

A

Mixed bacterial infection

Stress, poor oral hygiene, poor diet, immune suppression, smoking

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5
Q

Necrotizing Ulcerative gingivitis Clinical

A

Punched out interdental papillae

Localized or diffuse gingival involvement

Severe pain, oral malodor, spontaneous hemorrhage

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6
Q

Necrotizing Ulcerative gingivitis occasionally the process spreads

A

To adjacent soft tissues

Necrotizing ulcerative mucositis, stomatitis

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7
Q

Necrotizing Ulcerative gingivitis if infection extends through mucosa to cutaneous surface of face termed

A

noma (cancrum oris)

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8
Q

NUG Treatment

A

Debridement
Mild salt water rinse or chlorhexidine
Improve oral hygiene and diet

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9
Q

Desquamative Gingivitis

A

Sloughing of the gingival epithelium

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10
Q

Desquamative Gingivitis associated with

A

Several different immune mediate vesiculobullous diseases

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11
Q

Desquamative Gingivitis is not

A

A diagnosis

Clinical description

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12
Q

Desquamative Gingivitis patient management

A

Incisional biopsy is necessary for definitive diagnosis

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13
Q

Drugs-Related Gingival Hypeplasia -Degree of clinical enlargement related to patients susceptibility and level or oral hygiene

A

Abnormal growth of gingival tissues secondary to use of systematic medication

-diffuse involvement

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14
Q

Drugs-Related Gingival Hypeplasia -Degree of clinical enlargement related to

A

patients susceptibility and level or oral hygiene

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15
Q

First drug associated with drug related gingival hyperplasia

A

Dilantin (phenytoin)

Then came

Nifedipine
Cyclosporin

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16
Q

Drug-related gingival hyperplasia Tx

A

Removal of the offending medication may result in cessation and some regession of the gingival enlargement

home plaque control

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17
Q

Gingival Fibromatosis.

A

Slowly progressive collagenous overgrowth of the gingiva

Isolated or familial; localize or generalized

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18
Q

Gingival Fibromatosis gingiva is

A

Firm and normal color

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19
Q

Gingival Fibromatosis other findings sometimes observed

A

Hypertichosis
Epilepsy
Intellectual disability

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20
Q

Gingival Fibromatosis Tex

A

Oral hygiene instruction

Gingivectomy

Selective tooth extraction sometimes necessary in severe cases

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21
Q

Gingival Fibromatosis gingivectomy

A

Ideally delayed until after complete eruption of permanent dentition;
Reduced tendency for recurrence

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22
Q

Impetigo

A

Superficial infection of the skin causes but Staph aureus or Strep pyogenes

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23
Q

Impetigo is easily

A

Spread in crowded unsanitary living conditions

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24
Q

Impetigo peak occurrence

A

During summer or early fall in hot moist climates

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25
Q

Impetigo is most common in

A

School aged children

26
Q

Impetigo clinical presentation

A

Facial lesions often around nose and mouth

Erythema with superficial vesicles that quickly rupture and become covered in a thick amber crusts

27
Q

Impetigo cases may arise in areas

A

Of damaged skin; predicting dermatitis, cuts, scratches, insect bites

28
Q

Impetigo diagnosis

A

Presumptive diagnosis based on clinical presentation

Definitive diagnosis requires isolation of causative organisms in culture of skin

29
Q

Impetigo treatment

A

Topical or systemic antibiotics

30
Q

Tonsillolithiasis

A

Calcified structures that develops in enlarged Tonsillar crypts

31
Q

Tonsilar confections

A

Convoluted crypts of the tonsils are commonly filled with desaquamted cells, foreign debris and bacteria

32
Q

Tonsilloliths

A

Aggregates of desquamated cells that undergo calcification

33
Q

Tonsillolithiasis clinical presentation

A

Enlarged crypts filled with yellowish debris varies from soft to fully calcified

Variable size

Foul smelling

Solitary or multiple

34
Q

Tonsillolithiasis Tx

A

No treatment necessary unless associated with clinical symptoms

35
Q

Syphilis

A

Chronic infection caused by the spirochete treponema pallidum

36
Q

Syphilis spread by

A

Intimate sexual contact
Transplacental transmission
Contaminated blood exposure

37
Q

Syphilis is highly infectious during ________

A

First two stages

38
Q

Primary Syphilis

A

Chancre

Resolves spontaneously in 3+ weeks

39
Q

Secondary Syphilis

A

Develops 4-10 weeks after initial infection

  • Lymphadenopathy
  • Erythematous maculopapular cutaneous eruption
  • Mucous patches and condylomata lata or ora mucosa
  • Split papules at angles of mouth
40
Q

Untreated secondary patients

A

Will enter latent period

41
Q

Tertiary Syphilis

A

Develops 1-30 years after latency period

May affect any tissue

Gumma Formation

Oral involvement may produce palatal perforation

42
Q

Congenital Syphilis

A

Saddle nose deformity
Saber Shins
Hutchinson’s triad

43
Q

Hutchinson’s Triad

A

Malformed incisors and molars
Ocular interstitial keratitis
Eight nerve deafness

44
Q

Primary and secondary lesions show

A

Intense plasmacytic infiltrate

45
Q

Tertiary histopath

A

Granulomatous inflammation

46
Q

Spirochete can be identified using

A

Warthin-Starry Stain

47
Q

Syphilis serology

A

Screening Tests
Specific antibody tests

Dark Filed Microscopy for non-oral lesions

48
Q

Syphilis Tx

A

Penicillin remains the drug of choice

49
Q

Tuberculosis caused by

A

Mycobacterium tuberculosis

50
Q

TB transmission

A

Droplet

51
Q

TB Clinical features

A

Low grade fever, night sweats, fatigue

Weight loss

Chronic bloody cough

52
Q

TB oral lesions

A

Rather uncommon
Solitary chronic painless ulcer or granular lesion

Most common on gingiva and tongue

May be due to hematogenous or Direct implantation of organims

53
Q

TB diagnosis

A

Positive skin test with PPD

Chest radiograph

Identification of organims in biopsy material or sputum

Culture

Molecular testing

54
Q

TB histopath

A

Usually necrotizing granulomatous inflammation

Multinucleated giant cells

Organisms stain suing the acid fast method

55
Q

TB Tx

A

Combination of antibiotics

Isoniazid (INH) rifampin pyrazimide and ehtybutol

Then INH and rifampin for 4motnsh

56
Q

Actinomycosis

A

Caused by several actinomyces species that normally inhabit the mouth

Often associated with local trauma

57
Q

Cervicofacial Actinomycosis may follow

A

May follow dental extraction or untreated dental disease

58
Q

Cervicofacial Actinomycosis

A

Diffuse swelling erythema
Diffuse swelling and erythema

Draining sinus tracts

59
Q

Sulfur granules

A

Colonies of origins in purulent exudate

60
Q

Actinomycosis histo

A

Filamenotus bacteria that form colonies

bacterial colonies surrounded by neutrophils

Adjacent tissue may show granulomatous inflammation or granulation tissue

61
Q

Actinomycosis

A

Removal of offending tooth

High dose antibiotics usually IV PCN for 2 weeks then oral PCN for 2 weeks

Periapical actino usually responds to less aggressive treatment

Good prognosis with appropriate therapy

Oral Path 1 (4 decks)

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